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1 8-4.03]) were associated with higher risk of subdural hematoma.
2             A CT scan of his head revealed a subdural hematoma.
3 lled adult patients with symptomatic chronic subdural hematoma.
4 eal artery embolization in the management of subdural hematoma.
5 in >/=2% of patients were hematuria (2%) and subdural hematoma (2%).
6 s contusion, subarachnoid hemorrhage, and/or subdural hematoma, 3.23 [95% CI 1.59-6.58]).
7 ere 128 subarachnoid hemorrhage (33.4%), 134 subdural hematoma (35.0%), and 121 intraparenchymal hemo
8          In that series, one patient died of subdural hematoma 380 days after implant.
9  (43%, 26/60); central pontine myelinolysis, subdural hematoma, acute infarcts, and Aspergillus brain
10      Among patients with symptomatic chronic subdural hematoma, adjunctive middle meningeal artery em
11 s old, 66% were male patients, and 62.6% had subdural hematoma; admission Glasgow Coma Scale score wa
12 tient experienced worsening of a preexisting subdural hematoma after USCDT and therapeutic anticoagul
13 TS: The SECA (Surgical Evacuation of Chronic Subdural Hematoma and Aspirin) trial was an investigator
14 ceiving apixaban who developed a spontaneous subdural hematoma and declining mental status that impro
15                        There were 6 cases of subdural hematoma and intracranial injury reported in fo
16  a trial that involved patients with chronic subdural hematoma and that was stopped early, dexamethas
17  drug use was associated with higher risk of subdural hematoma; and the highest odds of subdural hema
18                         Subacute and chronic subdural hematomas are common and frequently recur after
19 ming surgery for most patients with an acute subdural hematoma (ASDH) and traumatic brain injury (TBI
20 cus (SE) are frequent complications of acute subdural hematoma (aSDH) associated with increased morbi
21  agonist, BAY X3702, in a rat model of acute subdural hematoma (ASDH).
22 ognostic factors following surgery for acute subdural hematomas (ASDHs) in England and Wales over a 2
23 , status epilepticus in the acute phase, and subdural hematoma at presentation.
24 in injury, primary intracerebral hemorrhage, subdural hematoma, brain tumor, central nervous system i
25 ticularly among those >80 yrs of age (36% of subdural hematoma cohort), in lower income patients, in
26                           Background Chronic subdural hematoma (cSDH) is a common neurosurgical condi
27 A) embolization (MMAE) treatment for chronic subdural hematoma (CSDH) is limited.
28 perioperative phase of treatment for chronic subdural hematoma (cSDH) may reduce recurrence rates but
29 recurrence rate in the evacuation of chronic subdural hematoma (cSDH) needs further study.
30 roposed as a potential treatment for chronic subdural hematoma (CSDH).
31 anisms behind the pathophysiology of chronic subdural hematoma (CSDH).
32               Neurosurgical intervention for subdural hematoma decreased from 41% in 1998 to 31% in 2
33                                              Subdural hematoma evacuation was associated with decreas
34 djacent to cerebral contusions or underlying subdural hematomas, even brief periods of hyperventilati
35   Contusion, subarachnoid hemorrhage, and/or subdural hematoma features were associated with incomple
36                              Traumatic acute subdural hematomas frequently warrant surgical evacuatio
37                   The increased incidence of subdural hematoma from 2000 to 2015 appears to be associ
38 bably or definitely related to treatment): 1 subdural hematoma grade 4, 1 anemia grade 3, 1 thrombocy
39                                 Incidence of subdural hematoma has been reported to be increasing.
40            The prevalence and total cost for subdural hematoma has increased significantly in the las
41 thasone on outcomes in patients with chronic subdural hematoma has not been well studied.
42 nts receiving standard treatment for chronic subdural hematoma have a high risk of treatment failure.
43 y assigned symptomatic patients with chronic subdural hematoma in a 1:1 ratio to a 19-day tapering co
44 bdural hematoma risk and determine trends in subdural hematoma incidence and antithrombotic drug use
45                                              Subdural hematoma incidence and antithrombotic drug use
46                                  The overall subdural hematoma incidence rate increased from 10.9 per
47 dural hematoma with antithrombotic drug use, subdural hematoma incidence rate, and annual prevalence
48  may shorten detection time for epidural and subdural hematomas, increase sensitivity (especially for
49                         Hospitalizations for subdural hematoma increased from 59,373 (30 per 100,000
50                            The prevalence of subdural hematoma increased with age (p < .001), particu
51 : contusion, subarachnoid hemorrhage, and/or subdural hematoma; intraventricular and/or petechial hem
52                                      Chronic subdural hematoma is a common neurologic disorder that i
53                                              Subdural hematoma is a common type of intracranial hemor
54              Health resource consumption for subdural hematoma is increasing without clear evidence t
55 gical evacuation in the treatment of chronic subdural hematoma is unclear.
56                    Among 10010 patients with subdural hematoma (mean age, 69.2 years; 3462 women [34.
57 lowing events: recurrent or residual chronic subdural hematoma (measuring >10 mm) at 180 days; reoper
58        Among adults with symptomatic chronic subdural hematoma, most of whom had undergone surgery to
59                                              Subdural hematoma occurred in 18% of HI (5% of TP), with
60                                              Subdural hematoma occurred in 8 patients (2 in the core
61 of various ages, particularly rib fractures, subdural hematoma of the brain, and retinal hemorrhages.
62                Major discrepancies were four subdural hematomas, one pneumocephalus, one hemorrhagic
63 ciousness or amnesia for more than 24 hours, subdural hematoma, or brain contusion).
64 rge disposition, length of stay, and cost of subdural hematoma over time.
65 y (positive LR, 3.4 [95% CI, 1.8-6.4]), or a subdural hematoma (positive LR, 3.2 [95% CI, 2.6-3.8]) i
66 tients aged 20 to 89 years with a first-ever subdural hematoma principal discharge diagnosis from 200
67 tion between use of antithrombotic drugs and subdural hematoma risk and determine trends in subdural
68                                      The rat subdural hematoma (SDH) model produces a zone of ischemi
69                                              Subdural hematomas (SDH) can induce ischemia and neurona
70 for later seizures were brain contusion with subdural hematoma, skull fracture, loss of consciousness
71 ritical care unit with an acute nontraumatic subdural hematoma that required emergent surgical evacua
72 y assigned patients with symptomatic chronic subdural hematoma to undergo middle meningeal artery emb
73 f subdural hematoma; and the highest odds of subdural hematoma was associated with combined use of a
74                                  The risk of subdural hematoma was highest when a VKA was used concur
75 morrhagic contusions or underlying evacuated subdural hematomas was studied.
76 ients undergoing surgery for traumatic acute subdural hematoma were randomly assigned to undergo cran
77 ors of failure of MMAE treatment for chronic subdural hematomas were identified, with small diameter
78          Among patients with traumatic acute subdural hematoma who underwent craniotomy or decompress
79 atients with symptomatic subacute or chronic subdural hematoma with an indication for surgical evacua
80 atients with symptomatic subacute or chronic subdural hematoma with an indication for surgical evacua
81                               Association of subdural hematoma with antithrombotic drug use, subdural
82 rmatory cranial CT scan revealed a worsening subdural hematoma with midline shift, a single dose of f
83 echanical fall with head trauma resulting in subdural hematoma with no associated neurological defici