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1 .10+/-0.05 versus -0.04+/-0.05; P<0.05); (2) subepicardial (0.16+/-0.15 versus 0.09+/-0.08; P<0.05) a
4 e most likely related to the same structural subepicardial abnormalities, but the mechanism is differ
5 epicardial attachment to the myocardium and subepicardial accumulation of epicardial-derived cells.
7 al portion (subendocardial, mid-portion, and subepicardial activity: 90 +/- 3, 96 +/- 2 and *80 +/- 5
8 local amplitude dependent upon the immediate subepicardial activity; the combination of these effects
11 I(Ca) was recorded in acutely dissociated subepicardial and subendocardial murine left ventricular
12 ank tests were used for paired comparison of subepicardial and subendocardial MVD and SI within group
14 t frequent location of LGE in all groups was subepicardial at the basal inferolateral wall, although
15 ach facilitated the precise visualization of subepicardial autonomic nerves in the ventricles using w
18 ted with ECG and vectorcardiogram (VCG), and subepicardial biopsies were taken at 5 to 120 minutes an
19 GRK activity was measured in arrhythmogenic subepicardial border zone (EBZ) tissue overlying the inf
20 terns tended to be dense and linear, usually subepicardial but also midmyocardial and transmural with
24 tion of FGFR-1 and VEGFR-2 in epicardial and subepicardial cells adjacent to FGF virus-infected myoca
27 ocardial longitudinal shortening at base and subepicardial circumferential shortening at apex continu
28 nd prevalence of resident macrophages in the subepicardial compartment of the developing heart coinci
30 letion results in defective formation of the subepicardial coronary veins, but had no significant eff
32 h a left ventricular pressure catheter and 2 subepicardial cylindrical ultrasonic dimension transduce
33 inium enhancement was present in a primarily subepicardial distribution in 40% of patients with DSP (
35 cardiac injury response by conditioning the subepicardial environment, potentially offering a new th
36 endo), basal midmyocardial (mid), and apical subepicardial (epi) regions of the left ventricular free
37 al cells labeled in ovo with DiI invaded the subepicardial extracellular matrix, demonstrating that m
39 rating characteristic curve [AUC], 0.82) and subepicardial GLS (AUC, 0.77) had the highest diagnostic
40 cute myocarditis, with midmyocardial GCS and subepicardial GLS providing the highest diagnostic perfo
42 ing and a loss in the natural subendocardial/subepicardial gradient, which roughly correlated inverse
44 of the hearts, transmural in 23.3%, midwall-subepicardial in 23.3%, and midwall-subendocardial in 20
45 is in POH-DCM was severe, subendocardial and subepicardial, in contrast with subendocardial fibrosis
46 was performed in normal rabbit hearts during subepicardial injections (50 muL) of norepinephrine (NE)
48 iants are associated with high prevalence of subepicardial late gadolinium enhancement and left ventr
49 ients with suitable images, LGE involved the subepicardial layer inferior and lateral wall in 154 pat
50 for distal sympathetic axon extension in the subepicardial layer of the dorsal ventricular wall of th
51 RBP-null cardiac myocytes, especially in the subepicardial layer, display increased cell proliferatio
53 with massive hyperinnervation of the intact subepicardial layers and heterogeneous distribution of n
58 l and posterolateral apical right ventricle, subepicardial left ventricular fibrofatty replacements (
59 t, formed a chimeric epicardium, invaded the subepicardial matrix and myocardial wall, and became cor
60 recursors from the proepicardium through the subepicardial matrix where the coronary arteries develop
61 most severe AS (n=15), the subendocardial to subepicardial MBF ratio decreased from 1.14+/-7 at rest
64 r and hypercellular epicardium with abundant subepicardial mesenchyme and a thin compact zone myocard
65 ion of the embryonic epicardium produces the subepicardial mesenchyme that is essential for normal co
66 ex communication between the epicardium, the subepicardial mesenchyme, and the myocardium mediated in
67 emonstrate differences in subendocardial and subepicardial microcirculation and to investigate the re
68 bserved significant transmural APD gradient: subepicardial, midmyocardial, and subendocardial APD80 w
71 with ventricular arrhythmias and isolated LV subepicardial/midmyocardial late gadolinium enhancement
73 d accuracy of quantifying subendocardial and subepicardial myocardial blood flow (MBF) and the relati
74 correlating activation maps of the surviving subepicardial myocardial layer with immunolocalization o
75 Twenty-seven dogs underwent placement of LV subepicardial myocardial markers to measure regional LV
77 1 and Scn5a expression remained lower in the subepicardial myocardium of the RVOT than in RV myocardi
79 fractionated electrograms in the ventricular subepicardial myocardium, which can be treated with abla
81 subendocardial myocytes but is prolonged in subepicardial myocytes (control: endo, 126+/-7 ms; epi,
82 eferential conduction from subendocardial to subepicardial myocytes is lost, and failing myocytes man
84 ural gradient, with faster repolarization in subepicardial myocytes than in subendocardial myocytes.
85 I(Ca) density was significantly larger in subepicardial myocytes than in subendocardial/myocytes.
86 ter in subendocardial myocytes compared with subepicardial myocytes, indicating stress-induced amplif
92 bal myocardial blood flow and subendocardial:subepicardial perfusion ratio were quantified using 3-Te
94 esent in the myocardium, particularly in the subepicardial regions of the right ventricular anterolat
95 ort-lived apex-to-base and subendocardial-to-subepicardial relaxation gradients at the onset of diast
97 A); and 2) scars restricted to the anterior subepicardial right ventricular outflow tract in 11 pati
98 ribes a novel clinical entity of an isolated subepicardial right ventricular outflow tract scar servi
100 The comparison of mean subendocardial and subepicardial SI within groups revealed significantly mo
101 s of induced VT arose from subendocardial or subepicardial sites distant from areas of marked conduct
102 ion of WT1 and RALDH2 initially populate the subepicardial space and subsequently invade the ventricu
104 tant mice, including failed expansion of the subepicardial space, blunted invasion of the myocardium,
108 e mean difference between subendocardial and subepicardial TOTv values versus that in the control reg
110 we found that cardiomyocytes throughout the subepicardial ventricular layer trigger expression of th
111 ndent action potential characteristics of LV subepicardial versus subendocardial myocytes in differen
114 ys occurred at the border between M-cell and subepicardial zones, where repolarization gradients were