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1 ontal pockets with PD of >/=5.4 mm, a single subgingival administration of a 0.4% moxifloxacin gel as
2 is study was to compare trehalose powder for subgingival air-polishing with sonic debridement in resi
3                                              Subgingival air-polishing with trehalose powder showed c
4                                              Subgingival and peri-implant biofilm samples were collec
5 MP Jumpstart data on 24 subjects for saliva, subgingival, and supragingival samples.
6 s were randomly assigned to receive a single subgingival application of a 0.125%, 0.4%, or 1.25% moxi
7 tive periodontal pathogens and resistance of subgingival bacteria against moxifloxacin were assessed.
8 ined potential correlations between detached subgingival bacteria collected in gingival crevicular fl
9 obial mouthrinse on levels of representative subgingival bacteria in subjects with mild to moderate p
10 ion on the prevalence and levels of selected subgingival bacteria using molecular approaches for bact
11                 Levels and proportions of 40 subgingival bacteria were determined using checkerboard
12  of local inflammation on the composition of subgingival bacteria, and 3) to understand how inflammat
13  determine the effect on clinical variables, subgingival bacteria, and local immune response brought
14 mographics, PD, clinical loss of attachment, subgingival bacteria, serum hsCRP, interleukin (IL)-1bet
15 cols on salivary nitrite and its relation to subgingival bacteria.
16 , a chronic inflammation driven by dysbiotic subgingival bacterial flora, is linked on clinical level
17            We investigated the prevalence of subgingival bacterial infection with eight species, test
18                                 The observed subgingival bacterial patterns in these elderly individu
19 ts did not sterilize or substantially reduce subgingival bacterial populations compared to negative c
20  study characterizes the association between subgingival bacterial profile and periodontal parameters
21               We analyzed the association of subgingival bacterial profiles and clinical periodontal
22                                       Pooled subgingival bacterial samples were collected from 477 de
23 ngival crevicular fluid (GCF) biomarkers and subgingival bacterial species in periodontally healthy s
24                                   Samples of subgingival biofilm and gingival crevicular fluid were c
25 s identified by polymerase chain reaction in subgingival biofilm and serum.
26 l role for smoking cessation in altering the subgingival biofilm and suggest a mechanism for improved
27  effects on major periodontopathogens in the subgingival biofilm as well as on biomarkers in the ging
28 that SubGPAP is more efficacious in removing subgingival biofilm in moderate-to-deep periodontal pock
29            We tested the hypothesis that the subgingival biofilm is compositionally different in curr
30                        A 10-species in vitro subgingival biofilm model was exposed to metronidazole (
31 Aggregatibacter actinomycetemcomitans in the subgingival biofilm of individuals with and without seve
32 hing appears to be a promising procedure for subgingival biofilm removal in periodontal treatment.
33                                              Subgingival biofilm samples from each tooth were analyze
34  studies comparing microbiologic outcomes of subgingival biofilm samples from healthy implants and im
35                                              Subgingival biofilm samples were obtained, and the level
36                                        Seven subgingival biofilm samples were taken successively, usi
37 ed to full-mouth periodontal examination and subgingival biofilm sampling (14 sites/patient).
38                                              Subgingival biofilm specimens from inflamed deep periodo
39 is is related to the presence of a dysbiotic subgingival biofilm that elicits the immune response.
40                                              Subgingival biofilm was analyzed regarding 11 periodonto
41                                              Subgingival biofilm was collected at the deepest site of
42 sythia (previously T. forsythensis) in their subgingival biofilm was determined by polymerase chain r
43  forsythia, and Fusobacterium nucleatum from subgingival biofilm were determined by quantitative poly
44 VL and the levels of 35 microbial species in subgingival biofilm, adjusted for confounders.
45 olymerase chain reaction on the basis of the subgingival biofilm, and IL-1beta and TNF-alpha were qua
46 of periodontitis; however, its effect on the subgingival biofilm, the primary etiological agent of pe
47 ts the composition of the disease-associated subgingival biofilm, yet little is known about its effec
48 s, C. gingivalis, and D. pneumosintes in the subgingival biofilm.
49                              Oral supra- and subgingival biofilms are complex communities in which hu
50                            Both marginal and subgingival biofilms in smokers are characterized by ear
51 d by significant changes in the marginal and subgingival biofilms, with a decrease in the abundance o
52 le initial colonization of both marginal and subgingival biofilms, with lower niche saturation than t
53 ine the reproducibility of curet sampling of subgingival biofilms.
54 nfounding factors and effect modification by subgingival calculus and age.
55  levels, bleeding upon probing, or extent of subgingival calculus comparing subjects assigned to prot
56  the 655-nm InGaAsP diode laser in detecting subgingival calculus in patients with periodontal diseas
57  associations between bone density, CAL, and subgingival calculus require further research, particula
58 ulus deposition over tooth surfaces, and the subgingival calculus that enables the enlargement of the
59 ratively a mucoperiostal flap was performed, subgingival calculus was visualized, and photographic im
60  The overall probability to correctly detect subgingival calculus with the laser (accuracy) was 0.82
61 ans with type 2 diabetes had more supra- and subgingival calculus, an increased extent and severity o
62                             Among women with subgingival calculus, there were no associations between
63    The 655-nm diode laser was able to detect subgingival calculus.
64 aser radiation to be a useful tool to detect subgingival calculus.
65           In multivariate regression models, subgingival colonization of A. actinomycetemcomitans and
66                   Periodontitis is caused by subgingival colonizing bacteria in the oral cavity.
67 ures on restoration of microbial eubiosis in subgingival communities, confirming the important role f
68 ed to perform a directed survey of the human subgingival crevice and to isolate bacteria having rod-l
69 reating host-bacterial disequilibrium in the subgingival crevice is poorly understood.
70 ts were identified who underwent surgery for subgingival curettage and/or periodontal flap and are co
71 riodontal disease who undergo procedures for subgingival curettage and/or periodontal flap have a rem
72 h were stained with 0.5% toluidine blue, and subgingival debridement efficacy was assessed.
73  group (one session of full-mouth ultrasonic subgingival debridement followed 1 week later by Er:YAG
74 ip (CHX chips) as an adjunctive treatment to subgingival debridement in patients afflicted with peri-
75  (Er:YAG) laser application as an adjunct to subgingival debridement in the treatment of chronic peri
76                                              Subgingival debridement was carried out using either tre
77 ssesses the efficacy of combining full-mouth subgingival debridement with Er:YAG laser application in
78 ed (SLA) titanium disks were inoculated with subgingival dental plaque and cultured anaerobically for
79 onstellatus and Streptococcus intermedius in subgingival dental plaque biofilms may contribute to for
80 acterial DNA in both prostatic secretion and subgingival dental plaque from the same individual.
81 g the restorative phase of treatment, and in subgingival dental plaque of periodontitis patients, ind
82 cter rectus, and Fusobacterium nucleatum, in subgingival dental plaque of pregnant women in the OPT S
83  levels of P. gingivalis and S. cristatus in subgingival dental plaque.
84 y of oral bacterial surfaces and to colonize subgingival dental plaque.
85 al flap surgery is frequently used to remove subgingival deposits, yielding consequential reductions
86 gh-throughput in vitro model that replicates subgingival dysbiosis and normobiosis, with a tool to me
87 crobiomes in parallel, and we describe a new subgingival dysbiosis index.
88                          Pure cultures of 47 subgingival E. faecalis clinical isolates were each inoc
89 evealed poor in vitro activity against human subgingival E. faecalis clinical isolates, and would lik
90 developed to facilitate visualization of the subgingival environment as an aid in diagnosis and non-s
91 simulate nutritional aspects of the inflamed subgingival environment.
92  development of antibiotic resistance in the subgingival flora in either group.
93  influence the composition of the contiguous subgingival flora.
94 lant pocket depths (IPD) of 5-8 mm underwent subgingival implant surface debridement followed by repe
95 ine digluconate, or 7.5% povidone-iodine for subgingival irrigation during SRP.
96 eatment outcome after 12 months of different subgingival irrigation solutions during scaling and root
97 is known of the antibiotic susceptibility of subgingival isolates of these two bacterial species, thi
98                    In response to persistent subgingival ligature-mediated challenge, Rac-null mice h
99 ontium in response to periodontitis-inducing subgingival ligatures compared with wild-types.
100                    Inflammatory responses to subgingival ligatures, assessed by changes in peripheral
101 entification microarray (HOMIM) was used for subgingival microbial assessment.
102 -mouth clinical measures of extent/severity, subgingival microbial burden by several species, and sel
103 eractions between the host immune system and subgingival microbial communities during the resolution
104                           The ability of the subgingival microbial community to induce an inappropria
105 ing cessation altered the composition of the subgingival microbial community, by means of a quantitat
106 e next-generation sequencing to evaluate the subgingival microbial composition of young patients with
107 ompiled the results of all studies comparing subgingival microbial data between these clinical condit
108 reports, case series, and reviews) comparing subgingival microbial data from patients with CP and AgP
109 monstrated that smoking cessation alters the subgingival microbial profile; however, the response of
110 ailable data on clinical periodontal status, subgingival microbial profiles, and serum IgG antibodies
111 ng (BOP), plaque index (PI), and count of 40 subgingival microbial species (checkerboard DNA-DNA hybr
112  Among older women, taxonomic differences in subgingival microbiome composition and diversity were ob
113 stic effects of smoking and pregnancy on the subgingival microbiome have never been studied.
114                                          The subgingival microbiome is largely uncultivated, and ther
115 riodontal therapy and smoking cessation, the subgingival microbiome is recolonized by a greater numbe
116 sively assessed the changes occurring in the subgingival microbiome of young patients with periodonti
117                                          The subgingival microbiome was evaluated via 16S rRNA gene s
118 EMD treatment predictably alters a dysbiotic subgingival microbiome, decreasing pathogen richness and
119 or smokers in order to effectively alter the subgingival microbiome.
120 d to investigate the effect of HA gel on the subgingival microbiome.
121 associated with shifts in the balance of the subgingival microbiome.
122 h 5% human serum was optimal for replicating subgingival microbiomes from health and disease.
123 to grow health- and periodontitis-associated subgingival microbiomes in parallel, and we describe a n
124                        Assessment of the rat subgingival microbiota after RvE1 treatment revealed mar
125 ncing was used to compare the composition of subgingival microbiota and establish correlations betwee
126 tudy was to quantify the association between subgingival microbiota and periodontal disease progressi
127 hese prospective results affirm clearly that subgingival microbiota are measurably elevated several y
128                                          The subgingival microbiota as well as determination of marke
129 gates HGF expression and its relationship to subgingival microbiota in medically healthy individuals
130           This comprehensive analysis of the subgingival microbiota in patients with PLS used 16S rib
131                                          The subgingival microbiota in PLS is diverse.
132                                          The subgingival microbiota observed in patients with new-ons
133 ective of this study was to characterize the subgingival microbiota of African-American children with
134                                          The subgingival microbiota of cats diagnosed with chronic pe
135       This study compares the changes to the subgingival microbiota of individuals with "refractory"
136 on-periodontal-related microorganisms in the subgingival microbiota of individuals with HIV.
137 e and number of periodontal pathogens in the subgingival microbiota of smokers versus never-smokers w
138 t literature regarding the complexity of the subgingival microbiota of the domestic cat and reveal bo
139 t in patients with a metronidazole-sensitive subgingival microbiota on the clinical parameters of CAL
140                                              Subgingival microbiota predict 2-y glucose change among
141 We investigated whether baseline measures of subgingival microbiota predicted fasting plasma glucose
142                                          The subgingival microbiota profile in patients with new-onse
143  translocation of periodontal pathogens from subgingival microbiota to the bloodstream and then to at
144                       The composition of the subgingival microbiota was characterized by 16S rDNA seq
145  major differences in the composition of the subgingival microbiota were observed between shallow and
146                               Changes in the subgingival microbiota were similar between the groups (
147 AgP periodontitis present differences in the subgingival microbiota when compared with patients with
148 he relationships among these biomarkers, the subgingival microbiota, and the clinical parameters of p
149 major role in shaping the composition of the subgingival microbiota.
150         The composition and diversity of the subgingival microflora and their oligotypes in health an
151  in health at low levels, but changes to the subgingival nutritional environment increase their compe
152  C. albicans) were more often present in the subgingival OB of patients with and without type 2 diabe
153  was to assess the presence of yeasts in the subgingival OB of patients with type 2 diabetes and peri
154                                              Subgingival OB samples were collected and oral yeasts sp
155 e investigated the presence of yeasts in the subgingival oral biofilm (OB) of type-2 diabetic and non
156       Treponema denticola is a predominantly subgingival oral spirochete closely associated with peri
157  antimicrobial sensitivity of enterococci of subgingival origin, this study evaluates the in vitro an
158                                              Subgingival P. endodontalis levels and serum immunoglobu
159                                              Subgingival P. endodontalis was defined by checkerboard
160                                              Subgingival P. endodontalis, its immune response, and se
161 ere found between the presence of individual subgingival pathogens and cancer risk.
162 fect of alcohol consumption on the levels of subgingival periodontal pathogens and proinflammatory cy
163 n vitro antibiotic resistance among selected subgingival periodontal pathogens in patients with CP.
164 h CP in the United States frequently yielded subgingival periodontal pathogens resistant in vitro to
165 rall, 74.2% of the patients with CP revealed subgingival periodontal pathogens resistant to at least
166         Fifteen percent of patients harbored subgingival periodontal pathogens resistant to both amox
167 e significant antimicrobial activity against subgingival periodontopathogens.
168 y failed to observe any adjunctive effect of subgingival placement of chlorhexidine chips after scali
169 clinical examination during which samples of subgingival plaque and buccal epithelial cells were obta
170 n of a mucoperiosteal flap in each quadrant, subgingival plaque and calculus were removed.
171                                              Subgingival plaque and crevicular fluid samples were col
172                                 Marginal and subgingival plaque and gingival crevicular fluid samples
173                                 Marginal and subgingival plaque and gingival crevicular fluid samples
174 mean +/- SD, 34 +/- 10 y) from whom baseline subgingival plaque and longitudinal FPG were measured.
175 ed at baseline and after 3 and 6 months, and subgingival plaque and sulcus fluid samples were taken f
176 g of the bacterial species present in canine subgingival plaque and their associations with health an
177 nse to specific bacterial species within the subgingival plaque biofilm.
178 hod to detect three periodontal pathogens in subgingival plaque collected before treatment and at 2 a
179 ipid extracts derived from diseased teeth or subgingival plaque do not contain free lipid A constitue
180      Microbiologic differences were found in subgingival plaque for patients with DG and pGI.
181                                              Subgingival plaque from deep sites of current and never-
182                                              Subgingival plaque from diseased sites strongly activate
183 laser on titanium surfaces contaminated with subgingival plaque from patients with peri-implantitis a
184                                              Subgingival plaque from three healthy and three disease
185 [MMP]-8, elastase, and sialidase) in GCF and subgingival plaque levels of Porphyromonas gingivalis, T
186                                              Subgingival plaque microbial profiles and gingival crevi
187      Changes in individual species levels in subgingival plaque microbiota were not detectable; howev
188  strain D11S-1, which was recovered from the subgingival plaque of a patient diagnosed with generaliz
189 ntal examination was performed, and a pooled subgingival plaque sample was collected from the deepest
190             EBV DNA was detected in 18.5% of subgingival plaque samples (72/390) and in at least one
191                                              Subgingival plaque samples and gingival biopsies were co
192                                              Subgingival plaque samples and serum samples were subjec
193 cant differences in bacteria in salivary and subgingival plaque samples between AgP and CP.
194                                              Subgingival plaque samples collected at baseline and day
195 ase chain reaction analysis was performed on subgingival plaque samples for the detection of A. actin
196                                              Subgingival plaque samples from 35 patients (healthy and
197                               In this study, subgingival plaque samples from both healthy and disease
198 of different bacterial species in saliva and subgingival plaque samples from individuals with aggress
199 ecies biofilms were derived using supra- and subgingival plaque samples from mesio-buccal aspects of
200              Here, saliva, supragingival and subgingival plaque samples from periodontitis patients a
201                             Whole saliva and subgingival plaque samples from the deepest pocket of ea
202                                  We analyzed subgingival plaque samples from women who experienced fe
203                            The collection of subgingival plaque samples is the common way for the det
204                                         Nine subgingival plaque samples per patient were analyzed usi
205                                   Up to four subgingival plaque samples per person, each obtained fro
206                                              Subgingival plaque samples pooled from 5 healthy subject
207 nt positive correlation between salivary and subgingival plaque samples was detected in patients with
208                          The microbiology of subgingival plaque samples was evaluated using DNA check
209       Clinical parameters were recorded, and subgingival plaque samples were analyzed at baseline, pe
210                                              Subgingival plaque samples were analyzed for Aggregatiba
211                                              Subgingival plaque samples were analyzed using semiquant
212                                              Subgingival plaque samples were assessed for counts of 4
213                                     Baseline subgingival plaque samples were assessed for the presenc
214                                              Subgingival plaque samples were assessed using immunoflu
215                                              Subgingival plaque samples were collected at baseline; 0
216                          Gingival tissue and subgingival plaque samples were collected from 19 patien
217                               In this survey subgingival plaque samples were collected from 223 dogs
218                                         Nine subgingival plaque samples were collected from each subj
219 n to clinical measurements and GCF sampling, subgingival plaque samples were collected from four post
220                                              Subgingival plaque samples were collected to determine t
221                     Microbial species within subgingival plaque samples were identified by human micr
222                     Pooled saliva and pooled subgingival plaque samples were injected into pregnant m
223                                      GCF and subgingival plaque samples were obtained from healthy si
224                                              Subgingival plaque samples were obtained from periodonta
225                                      GCF and subgingival plaque samples were obtained from the mesio-
226                                              Subgingival plaque samples were obtained pre- and postop
227                                              Subgingival plaque samples were taken at baseline and 15
228                                              Subgingival plaque samples were taken from diseased (DD)
229                                              Subgingival plaque samples were taken from the mesio-buc
230                                              Subgingival plaque samples were taken, and the presence
231 eeding on probing (BOP), were performed, and subgingival plaque samples were taken.
232                            The collection of subgingival plaque samples with paper points is time-con
233                                     In 1,188 subgingival plaque samples, 11 bacterial species were as
234 terial species were detected in salivary and subgingival plaque samples.
235 d A in lipid extracts from diseased teeth or subgingival plaque samples.
236 in the placenta than in the pooled saliva or subgingival plaque samples.
237             Oral infection was assessed from subgingival plaque samples.
238                                     Multiple subgingival plaque samples/patient (1,057 in total) were
239 t be discussed as a potential alternative to subgingival plaque sampling for microbiologic analysis i
240                                              Subgingival plaque specimens were detected by PCR with s
241         Baseline microbiota were measured in subgingival plaque using 16S rRNA sequencing.
242                                              Subgingival plaque was collected at baseline, and 3, 6,
243                                              Subgingival plaque was collected from 22 smokers at the
244                                              Subgingival plaque was collected from 44 systemically an
245 probing, and plaque index were measured, and subgingival plaque was collected from LAgP diseased and
246                                              Subgingival plaque was collected with sterile paper poin
247                  A heavily diluted sample of subgingival plaque was inoculated onto culture plates su
248                                              Subgingival plaque was sampled at baseline (13 to 16 wee
249           In 50 patients with periodontitis, subgingival plaque was sampled from the deepest pocket o
250 regatibacter actinomycetemcomitans levels in subgingival plaque were analyzed by quantitative real-ti
251 aseline, gingival crevicular fluid (GCF) and subgingival plaque were collected and clinical periodont
252                 Clinical data and samples of subgingival plaque were collected at baseline, 45 days a
253          Gingival crevicular fluid (GCF) and subgingival plaque were collected from 77 patients at th
254  all the samples (saliva, supragingival, and subgingival plaque) and was correlated with AAA diameter
255           Saliva, gingival crevicular fluid, subgingival plaque, and blood samples were obtained at t
256 s included a periodontal examination; blood, subgingival plaque, and crevicular fluid specimen collec
257                       DNA was extracted from subgingival plaque, and V3 to V4 regions of the 16S rRNA
258 dontitis, is abundant at the leading edge of subgingival plaque, where it interacts with gingival epi
259 ion to the highly proteolytic environment of subgingival plaque, which is exposed continually to an a
260                                              Subgingival plaque-coated titanium disks with a moderate
261 sythia, but not A. actinomycetemcomitans, in subgingival plaque.
262 h the levels of key periodontal pathogens in subgingival plaque.
263 re performed on saliva and supragingival and subgingival plaque.
264 rrelated with the number of P. gingivalis in subgingival plaque.
265 es in the frequency of bacteria in serum and subgingival plaque.
266 46 in the tongue, saliva, supragingival, and subgingival plaque.
267                                              Subgingival plaques were analyzed for the presence of 18
268 ence of all three species in the reservoirs (subgingival pockets and blood DCs) of PD patients before
269  in reducing clinical parameters of LAgP and subgingival presence of JP2 in diseased and healthy site
270  the levels of plaque bacteria suggests that subgingival recolonization was occurring.
271 orus HD100 was topically administered in the subgingival region of MFMs at days 0, 3, and 7.
272 tis) HN019 was topically administered in the subgingival region of MFMs on days 0, 3, and 7.
273                                              Subgingival S. constellatus and S. intermedius exhibited
274                                              Subgingival samples from healthy individuals and shallow
275 gnosed with severe chronic periodontitis had subgingival samples harvested from four sites (the deepe
276 ans was demonstrated in vascular, blood, and subgingival samples in one of 36 patients.
277                                              Subgingival samples were collected from two sites (probi
278                     Before treatment, pooled subgingival samples were obtained from the five deepest
279 tal, 44 domestic cats were enrolled, and 139 subgingival samples were subjected to 16S rRNA gene sequ
280 T. forsythia, or C. rectus) were detected in subgingival samples, with a prevalence rate of 72.2%, 47
281 ed a vascular lesion, a blood sample, and 36 subgingival samples.
282 annerella forsythia) in vascular, blood, and subgingival samples.
283 a reliable and reproducible method to obtain subgingival samples.
284               Intervention trials, including subgingival scaling and/or root planing, were systematic
285 temic antibiotics or with plaque control and subgingival scaling significantly reduces CRP levels aft
286 ent and CMV was rarely present in individual subgingival sites affected by chronic periodontitis.
287  surfaces-is also a significant colonizer of subgingival sites in patients with chronic periodontitis
288 rease in microbial diversity was observed in subgingival sites of ailing implants, compared with heal
289                                              Subgingival sites were sampled every 6 months to assess
290 to periodontal breakdown in heavily infected subgingival sites, particularly in patients responding p
291  explain the persistence of this organism at subgingival sites.
292 ade the host defence system and colonize the subgingival space.
293 aim of the present study is to analyze which subgingival species are associated with SUP in patients
294  of 33 S. constellatus and 17 S. intermedius subgingival strains, each recovered from separate patien
295 was developed to aid in the visualization of subgingival structures and to improve the diagnosis and
296 al crevicular fluid (GCF) and a selection of subgingival/submucosal plaque bacteria from clinically h
297 CAL, and significantly reduced the amount of subgingival Tf and Td.
298                               The CFU/mL for subgingival yeasts were higher in group A than groups B
299                               The CFU/mL for subgingival yeasts were higher in group B than group C (
300                                              Subgingival yeasts were more often isolated from the OB

 
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