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2 of 50 and 8 of 50 otic, 14 of 47 and 4 of 47 submandibular, 18 of 58 and 10 of 58 superior cervical,
6 e activity appears to be directly related to submandibular acinar cell loss in NOD-scid mice involvin
7 were also observed in freshly dispersed rat submandibular acinar cells but not in three immortalized
10 cusing on the membrane potential in isolated submandibular acinar cells revealed mechanistic details
11 ed that NOD-scid mice develop severe loss of submandibular acinar cells with concomitant appearance o
13 mphatic transport and drainage of Gd-DOTA to submandibular and deep cervical lymph nodes was demonstr
14 p progressive histopathologic lesions of the submandibular and lachrymal glands similar to NOD mice,
15 vere chronic periductal inflammation in both submandibular and lacrimal glands in contrast to the muc
16 nto several stages: 1) initial injury to the submandibular and lacrimal glands via an environmental i
23 alyses of 14,441 cells from embryonic day 12 submandibular and parotid salivary glands to characteriz
25 higher uptake of (18)F-PSMA-1007 in muscle, submandibular and sublingual gland, spleen, pancreas, li
30 e cranial ciliary, pterygopalatine, lingual, submandibular, and otic ganglia--arise from glial cells
33 progressively expanding lingual, sublingual, submandibular, and submental hematomas have the tendency
34 into the rat glioma cell line C6B4, the rat submandibular cell line RSMT-A5, and the rat pancreatic
35 operties of the conductances in lacrimal and submandibular cells were similar, e.g. halide selectivit
36 was significantly underexpressed in AdCC of submandibular compared to parotid and sublingual glands
38 rotid artery (eICA) stenoses, detectable via submandibular Doppler sonography and cervical magnetic r
39 s are co-expressed in the apical membrane of submandibular duct cells, consistent with the significan
42 ed the expression of muscle-related genes in submandibular fibroblasts that began to mimic parotid fi
43 hout any signs of distress and had bilateral submandibular firm nonpulsatile tender masses, each esti
44 termine variations in stimulated parotid and submandibular flow rates over 6 hours and to define sali
45 of neuronal cells within the parasympathetic submandibular ganglion (PSG) express the catecholaminerg
46 e mouse superior cervical ganglion (SCG) and submandibular ganglion (SMG) to examine the assembly of
47 pre- and postganglionic neurons in the mouse submandibular ganglion in vivo, focusing on the mechanis
52 H+ exchanger (NHE) isoforms expressed in the submandibular gland (SMG) acinar and duct cells and thei
53 l-]i and the Cl- current in the rat salivary submandibular gland (SMG) acinar and duct cells was used
54 ned miRNAs expressed in the mouse developing submandibular gland (SMG) and found that miR-200c accumu
55 y studies have been done to understand mouse submandibular gland (SMG) branching morphogenesis, littl
57 Parasympathetic innervation is critical for submandibular gland (SMG) development and regeneration.
58 -inducible Cre recombinase Ela-CreERT in the submandibular gland (SMG) ductal cells, was established
59 Here, our results show that in primary mouse submandibular gland (SMG) epithelial cells, P2X7R activa
61 f the vector to the parotid gland (PTG), the submandibular gland (SMG) or to the liver via the tail v
62 epth analysis of male and female adult human submandibular gland (SMG) samples by bulk RNA sequencing
63 y the latter two develop inflammation in the submandibular gland (SMG), a critical target of Sjogren'
64 ery (BA), middle cerebral artery (MCA)], the submandibular gland (SMG), and pineal gland was quantifi
65 king the regenerative response of the murine submandibular gland (SMG), following innate immune-media
66 infiltration and caspase-3 activation in the submandibular gland (SMG), production of antinuclear and
69 nd the Cl- current were measured in isolated submandibular gland acinar and duct cells to characteriz
70 int of the Ca(2+)-activated Cl(-) current in submandibular gland acinar cells from Best2-deficient mi
72 ected HEK293 cells and from the pancreas and submandibular gland also coimmunoprecipitated EBP50.
73 tive, non-vasomotor sympathetic axons in the submandibular gland and at the base of piloerector hairs
74 1 IFNR (IL14alphaTG.IFNR(-/-)) had the same submandibular gland and lacrimal gland injury as did the
75 lized in the plasma membrane region of human submandibular gland and Madin-Darby canine kidney cells.
76 sly and exogenously expressed TRPC1 in human submandibular gland and Madin-Darby canine kidney cells.
77 glands, specifically in acinar cells of the submandibular gland and palatine minor glands, as well a
78 termined by recording salivary flow from the submandibular gland and temperature changes on the tongu
80 cover that decreasing MT-MMP activity during submandibular gland branching morphogenesis decreases pr
85 tro stimulation with a muscarinic agonist of submandibular gland cells isolated from mice treated wit
92 renergic receptors) in pancreatic acinar and submandibular gland duct cells, respectively, evoke a Ca
93 and membrane current were measured in human submandibular gland ductal (HSG) cells to determine the
94 PC3 was detected in the apical region of rat submandibular gland ducts, whereas TRPC6 was present in
97 deficient adenoviruses, lymphocytes from the submandibular gland express T-bet, GATA3, and RAR-relate
98 show that calcineurin is required for normal submandibular gland function and secretion of digestive
99 the question of a role for 5-HT in mammalian submandibular gland function was re-addressed, using iso
102 ations of this stem cell population into the submandibular gland of irradiated mice successfully rest
106 measurements from time-lapse images of mouse submandibular gland organ explants to construct a tempor
107 chain reaction studies in cells of mammalian submandibular gland origin using consensus sequence prim
108 noculation with tcMCMV, lymphocytes from the submandibular gland preferentially express the transcrip
110 ped large ductal-type adenocarcinomas in the submandibular gland region, where islets were transplant
111 tion of Hedgehog signaling within the murine submandibular gland rescued radiation-induced salivary g
112 ike the induced pancreatic tumors, all three submandibular gland tumors that were examined had the mu
113 bulated solid mass was detected in the right submandibular gland with similar sonographic findings.
114 previously demonstrated, immunization of the submandibular gland with tissue culture-derived murine c
117 ively influences gene expression in the male submandibular gland, and that many of the sex difference
118 f androgen control of gene expression in the submandibular gland, and to explore the degree to which
119 s observed in cultured cells in vitro and in submandibular gland, cortex, and caudate nucleus for as
120 embryonic parotid gland as compared with the submandibular gland, focusing on the mesenchymal cell po
121 ion of adenylyl cyclase and, at least in rat submandibular gland, involved in modifying the volume an
122 ncreased by LPS-induced periodontitis in the submandibular gland, returned to control values after HU
123 ce lacked comparable defects in the lung and submandibular gland, suggesting that MT1-MMP acts via me
124 Of the different cells isolated from the submandibular gland, this specific population, Lin-CD24+
125 odel of severe glandular injury in the mouse submandibular gland, we show that de novo formation of a
126 ean dose, 0.26 Gy/GBq, was seen in the right submandibular gland, whereas the lowest mean dose, 0.029
136 mSv/MBq), the kidneys (0.1722 mSv/MBq), the submandibular glands (0.1479 mSv), and the parotid gland
137 on in the parotids (24% +/- 14%, P = 0.001), submandibular glands (35% +/- 11%, P < 0.001), and kidne
138 ration in the parotids (24% 14%, P = 0.001), submandibular glands (35% 11%, P < 0.001), and kidneys (
139 15 and 5-lipoxygenase) is expressed in mouse submandibular glands (mSMG), using qPCR and Western blot
141 93), spinal cord (PRP, 64%; mean SUV, 2.12), submandibular glands (PRP, 53%; mean SUV, 2.11), parotid
142 ain/neck muscles, and sialorrhea/parotid and submandibular glands (reporting odds ratios 0.79-0.27).
143 ctive of this study was to determine whether submandibular glands (SMG) from ALX/FPR2(-/-) mice displ
144 involved in SPM biosynthesis were altered in submandibular glands (SMG) from NOD/ShiLtJ female mice a
145 sequently, this DC subset became resident in submandibular glands (SMGs) and nasal passages (NPs) in
147 cumulation of PSMA-targeting radioligands in submandibular glands (SMGs) can be explained with PSMA e
148 issues such as the nasal passages (NPs), the submandibular glands (SMGs), and nasopharyngeal-associat
152 e acinar cells in developing rat parotid and submandibular glands and are also products of the sublin
153 s re-addressed, using isolated, perfused rat submandibular glands and dispersed-cell aggregates from
154 ved in cells isolated from mouse and opossum submandibular glands and rat sublingual and parotid glan
155 ein occurred in the nasal lamina propria and submandibular glands and the frequencies of CD11c+CD8+ d
157 , we report that ex vivo branching of intact submandibular glands decreases when either FGFR2 express
161 cells as well as lymphocytic infiltrates in submandibular glands from patients with pSS demonstrated
163 t adenoviral-mediated gene transfer to mouse submandibular glands is possible by intraductal cannulat
165 respectively), AdhAQP1 administration to rat submandibular glands led to a two- to threefold increase
166 an hamsters were transplanted into the right submandibular glands of 50 female hamsters that were or
167 of immunoglobulin A (IgA) was studied in the submandibular glands of anaesthetized rats by stimulatin
169 y of a replication-deficient adenovirus-5 in submandibular glands of C57BL/6 mice through retrograde
170 Temporal expression of IL-17 and IL-23 in submandibular glands of C57BL/6.NOD-Aec1Aec2 mice correl
171 d enriched the number of functional acini in submandibular glands of irradiated animals and enhanced
173 rt a RCC case with metastasis to parotid and submandibular glands that has the same sonographic and s
174 pine-stimulated in vivo fluid secretion from submandibular glands was essentially normal in double-nu
175 diffuse lymphocytic infiltration was seen in submandibular glands, a major target of pSS, by age 6 wk
176 of primary salivary tissue, both parotid and submandibular glands, and differentiating hS/PCs, we con
177 ences in ABPs secreted by mouse lacrimal and submandibular glands, and in ABPs secreted by male and f
178 ed phosphorylation of SMAD1/5/8 in the mouse submandibular glands, and led to a recovery of SG functi
179 ed ipsilateral/contralateral parotid glands, submandibular glands, and oral cavity surrogates for eac
180 for each organ at risk (kidney, parotid and submandibular glands, bone marrow, liver, and lacrimal g
182 caspase-3, lack of leukocyte infiltration of submandibular glands, reduced synthesis of disease-assoc
183 DNA levels were lower in the spleen than in submandibular glands, the number of individual viral gen
184 al saliva obtained from both the parotid and submandibular glands, with highest levels of activity pr
191 egions of interest on lacrimal, parotid, and submandibular glands; left ventricle; liver; spleen; kid
192 receptors were found in cell lines from the submandibular (HSG) and parotid (HSY) salivary glands.
195 e day 3 accumulation of KJ1-26+ cells in the submandibular LN was inhibited if the eye was removed wi
198 ng OVA peptide in vivo was only found in the submandibular lymph node and not in other lymph nodes, s
199 rP(Sc), was first detected in the tongue and submandibular lymph node at 1 to 2 weeks following inocu
202 ll clonal expansion was only observed in the submandibular lymph node following conjunctival applicat
204 0(+) macrophage infiltration of gingival and submandibular lymph node tissues and significantly (p <
206 he a.c. OT-I T cells increased 9-fold in the submandibular lymph nodes and 3-fold in the spleen follo
207 phenotype (M2) of macrophages isolated from submandibular lymph nodes as observed by flow cytometry.
208 Nasal mucosa-associated lymphoid tissue and submandibular lymph nodes initially accumulated PrP(d) a
209 ad bronchial tree-associated lymph nodes and submandibular lymph nodes that were 5-10 times larger th
210 including teeth and surrounding tissues, and submandibular lymph nodes were obtained for histomorphom
212 ity in the spleens, superficial cervical and submandibular lymph nodes, and inoculated eyes peaked at
213 in the spleen, the superficial cervical and submandibular lymph nodes, and the inoculated eye by lys
214 ans, including hypertrophy of the spleen and submandibular lymph nodes, glomerulonephritis, and periv
217 8(+) T cells first appeared in the draining (submandibular) lymph node on day 5 and were detectable i
221 22-kilodalton protein was isolated from the submandibular (mental) gland of the male terrestrial sal
222 s indicated that the direct contact of mouse submandibular (mSMG) cell clusters and hHF-MSCs was not
223 These results indicate that a decrease in submandibular NO-BH4 protein expression may provide insi
224 andibular PP, 4%-31%; parotid NUR, 2.2-16.0; submandibular NUR, 1.4-16.2; parotid TNUR, 8-45 min; sub
227 xpression of SLPI transcripts was evident in submandibular, parotid, and minor salivary glands from b
228 g ranges were observed: parotid PP, 22%-49%; submandibular PP, 4%-31%; parotid NUR, 2.2-16.0; submand
229 mbryonic cells are tagged by AID(cre) in the submandibular region, where conditional deletion of the
230 In addition, the introduction of the mouse submandibular renin (mRen-2d) transgene into Sprague-Daw
233 dentify the salivary protein(s) responsible, submandibular saliva was fractionated by anion- exchange
235 and stimulated glandular (i.e., parotid and submandibular) saliva flow rate and composition alterati
236 orted to affect electrolyte transport by the submandibular salivary gland (adrenaline, carbachol, iso
238 lished the epigenomic landscape of the mouse submandibular salivary gland (SMG) by performing chromat
239 d H(+) and HCO3- transport mechanisms in the submandibular salivary gland (SMG) ducts of wild type, N
241 artial sialoadenectomy, in the female murine submandibular salivary gland (SMG) to establish a model
243 ductal (mSG-DUC1) cell line, from the murine submandibular salivary gland (SMG), which recapitulate d
244 ly, SERCA2b was found in the luminal pole of submandibular salivary gland acinar and duct cells.
245 pared the properties of native CaCC in mouse submandibular salivary gland acinar cells to the Ca(2+)-
249 e in mobilizing Ca2+ from internal stores of submandibular salivary gland but not pancreatic acinar c
250 ession of ryanodine receptor was detected in submandibular salivary gland cells but not pancreatic ac
251 ClC-3 was examined in rat lacrimal gland and submandibular salivary gland cells using RT-PCR and West
252 n profiles at five different stages of mouse submandibular salivary gland development provide insight
256 asympathetic innervation of the lacrimal and submandibular salivary gland is dramatically reduced in
259 (GS-AgNPs) can selectively accumulate in the submandibular salivary gland, followed by being excreted
263 urce of immature T cells in parotid (PG) and submandibular salivary glands (SMG) were studied in Fisc
264 In the present study we investigated whether submandibular salivary glands (SMGs) in F508 mice (Cftr(
266 he role of LIMK signaling in mouse embryonic submandibular salivary glands using ex vivo organ cultur
267 re inoculated by retrograde perfusion of the submandibular salivary glands via Wharton's duct with tc
268 ithelial basal periphery in developing mouse submandibular salivary glands, and that ROCK inhibition
269 than GS-AuNPs and can rapidly penetrate into submandibular salivary glands, be efficiently taken up b
273 ntly and focally in forming cleft regions of submandibular salivary-gland epithelia, accompanied by a
277 major salivary glands: the parotid (PG), the submandibular (SMG), and the sublingual glands (SLG).
281 fferent anatomic subsites including parotid, submandibular, sublingual, and minor salivary glands dif
282 y(A)+ RNA isolated from harderian, lacrimal, submandibular, sublingual, and parotid glands and the li
284 ory activity was present in whole saliva and submandibular-sublingual saliva, but it was essentially
285 35%), stimulated parotid (47%), unstimulated submandibular/sublingual (23%), and stimulated submandib
287 ates for unstimulated whole and unstimulated submandibular/sublingual (SMSL) saliva as well as citrat
289 sucrose diets, removal of the parotid or the submandibular/sublingual glands, and diets in powder or
290 bserved for protein secretion in parotid and submandibular/sublingual glands, and that the secretion
294 encoded by MUC7), a major component of human submandibular/sublingual saliva, is a bacterial receptor
295 cin MG1 and other salivary proteins in human submandibular/sublingual secretion (HSMSL) could have a
296 Protein samples from parotid secretion, submandibular/sublingual secretion, whole saliva, and pe
299 bular NUR, 1.4-16.2; parotid TNUR, 8-45 min; submandibular TNUR, 2-45 min; parotid DF, 20%-99%; and s