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1 icit disorder), CNTF (psychosis), and OPRM1 (substance dependence).
2 ed in many brain regions in individuals with substance dependence.
3 redict suicidal behavior among patients with substance dependence.
4 e factors in the development of each type of substance dependence.
5 e, and 5) lifetime psychiatric disorders and substance dependence.
6 y of alcoholism predicted the development of substance dependence.
7 evelopment of mood and anxiety disorders and substance dependence.
8 is of hospital policies on the management of substance dependence.
9 childhood self-control, or family history of substance dependence.
10 ociations between mood/anxiety disorders and substance dependence.
11 focused on cannabis use or individuals with substance dependence.
12 associated with lower odds of many forms of substance dependence.
13 isorder, post-traumatic stress disorder, and substance dependence.
14 r rates of concurrent psychotic symptoms and substance dependence.
15 al ideation and clear intent, and those with substance dependence.
16 nosis of schizophrenia, bipolar disorder, or substance dependence.
17 y provide a biomarker for the development of substance dependence.
18 what has been found in schizophrenia without substance dependence.
19 in patients with comorbid schizophrenia and substance dependence.
20 sk of relapse for patients with co-occurring substance dependence.
21 severity without an increase in severity of substance dependence.
22 bens, holds promise as a pharmacotherapy for substance dependence.
23 sorder is associated with very high rates of substance dependence.
24 who met DSM-IV-TR criteria for both PTSD and substance dependence.
25 development of individualized treatments for substance dependence.
26 t GSK598809 may remediate reward deficits in substance dependence.
27 on of cognitive-behavioral therapy (CBT) for substance dependence.
28 findings regarding association of DRD2 with substance dependence.
29 disorders, including Parkinson's disease and substance dependence.
30 common clinical problem in individuals with substance dependence.
31 nd in relation to improvement in severity of substance dependence (0.43 vs 0.52; incidence rate ratio
33 Among individuals at higher genetic risk for substance dependence, ACEs were less associated with tha
34 nder balanced) and was associated with adult substance dependence, adult life impairment, and treatme
35 ted for major depression, anxiety disorders, substance dependence (alcohol and drug dependence), and
36 g 382 European Americans (EAs) affected with substance dependence [alcohol dependence (AD) and/or dru
37 A history of MDD prior to lifetime onset of substance dependence also reduced the likelihood of remi
38 d symptoms can be effective in patients with substance dependence, although results have not been con
45 cebo-controlled trials of patients with both substance dependence and depression, applied the inclusi
46 adjunct to standard outpatient treatment for substance dependence and may provide an important means
47 ining from smoking, overlap with genetics of substance dependence and memory, and nominate gene varia
48 ubsample, we tested polygenic scores for the substance dependence and mood/anxiety disorder factors a
49 related to maladaptive behaviors, including substance dependence and nonadherence to medical treatme
50 uals could improve effectiveness of treating substance dependence and preventing drug abuse relapse.
51 ses revealed that genes influencing risk for substance dependence and related phenotypes, such as bod
53 with diagnoses of a personality disorder or substance dependence and some normal comparison subjects
56 lifetime psychiatric disorders, and lifetime substance dependence, and 3) explore the stress-bufferin
57 SM-III-R diagnosis of nonalcohol nonnicotine substance dependence, and 8.1% and 5.2% for antisocial p
58 tent mental health problems (eg, depression, substance dependence, and additional suicide attempts) c
60 views to assess prevalence of mental health, substance dependence, and comorbid psychiatric disorders
61 ention-deficit/hyperactivity disorder, prior substance dependence, and posttraumatic stress disorder
62 ing the indexing terms naloxone, naltrexone, substance dependence, and substance withdrawal syndrome.
63 nment (SAGE); the Yale-Penn genetic study of substance dependence; and the National Health and Resili
65 etween the child's psychopathology, paternal substance dependence/antisocial personality disorder sta
66 cally for patients with bipolar disorder and substance dependence, appears to be a promising approach
67 r, becoming apparent that sex differences in substance dependence are also influenced by genetic fact
69 disorder, obsessive-compulsive disorder, and substance dependence are not likely to share a common ca
70 n which 77 individuals seeking treatment for substance dependence at an outpatient community setting
71 nmental influences increase in importance as substance dependence becomes more specialized in adultho
72 nternalizing psychopathology than those with substance dependence but not antisocial personality diso
73 ning systems on behavior in individuals with substance dependence, but it is unknown whether these di
75 iterion count was performed in 3 independent substance dependence cohorts (the Yale-Penn Study, Study
76 been routinely observed in individuals with substance dependence compared with nondependent control
77 r, psychosis, posttraumatic stress disorder, substance dependence, current abuse, intellectual disabi
78 participants had 1 or more mental health or substance dependence diagnoses; 1 in 5 (20.1%) had 2 or
79 lence and comorbidities of mental health and substance dependence disorders assessed via the Mini-Int
85 ) and externalizing (antisocial personality, substance dependence) disorders in statistically predict
88 icated in addictive-like eating behavior and substance dependence: elevated activation in reward circ
92 al population studies have demonstrated that substance dependence has a substantially heritable compo
93 eceptor genes that have been associated with substance dependence have been determined to be function
94 ls in neural functioning between obesity and substance dependence have been found, to our knowledge,
96 rder (HR, 1.70; 95% CI, 1.05-2.75; P = .03), substance dependence (HR, 2.96; 95% CI, 1.24-7.08; P = .
97 tions among ACEs, mood/anxiety disorders and substance dependence in 12,668 individuals (44.9% female
98 a weaker direct association between ACEs and substance dependence in both ancestry groups (reflecting
99 rogram GWAS of PTSD (N = 5200) with PTSD and substance dependence in COGA, and moderating effects of
100 the occurrence of depression in relation to substance dependence in evaluating suicidal risk among s
101 e for a genetic component in the etiology of substance dependence in Native Americans, including stud
104 ion for chromosome 11 gene cluster SNPs with substance dependence, including extension of liability t
105 entification of neurocognitive predictors of substance dependence is an important step in developing
106 with regard to the use of alcohol and drugs, substance dependence is one of the primary sources of he
107 tal circuits is an area of great interest in substance dependence literature, particularly as the fie
108 etime diagnoses of alcohol dependence, other substance dependence (marijuana, cocaine, other stimulan
109 r depressive disorder (MDD) on the course of substance dependence may differ depending on the tempora
110 irect and indirect associations of ACEs with substance dependence, mediated by mood/anxiety disorders
111 mes successfully classified individuals with substance dependence on alcohol or nicotine relative to
112 cluding 3,240 individuals, 2,140 of whom had substance dependence on one of five substances: alcohol,
113 uniformly stable against confounding: axis I substance dependence or abuse disorders and axis II pass
114 hat an individual with a lifetime history of substance dependence or habitual smoking at the first in
118 ther specific psychiatric disorders, notably substance dependence, panic and generalized anxiety diso
119 dhood self-control predicts physical health, substance dependence, personal finances, and criminal of
121 ied genes with variants associated with four substance dependence phenotypes or five psychiatric diso
122 d might biologically bridge OD and other non-substance dependence psychiatric traits where similar pa
123 ing childhood maltreatment, tobacco smoking, substance dependence, psychiatric medication use, poor p
124 h groups improved significantly over time on substance dependence, psychotic symptoms, homelessness,
126 ther a common set of regions are involved in substance dependence regardless of the substance used or
130 Subjects ascertained for genetic studies of substance dependence (SD) and diagnosed with ASPD and co
133 he timing of depressive episodes relative to substance dependence served as an important factor in th
135 on, patients with comorbid schizophrenia and substance dependence showed significant blunting of stri
136 further support the importance of examining substance dependence, specifically alcohol dependence, a
137 y subjects (control group), 26 subjects with substance dependence (substance-dependent group), and 21
139 CEs with mood/anxiety disorders, mediated by substance dependence (the reverse or 'substance-induced'
141 and NCAM1 are functional candidate genes for substance dependence; the TTC12 and ANKK1 loci are not w
142 reconcile the conflicting associations with substance dependence traits, we performed a meta-analysi
143 o patients with bipolar disorder and current substance dependence, treated with mood stabilizers for
144 tigated the effects of MDD on the outcome of substance dependence under 3 circumstances: (1) lifetime
146 9q34) likely to contain genes that influence substance dependence vulnerability (DV) in adolescence.
147 Major depression among 602 patients with substance dependence was classified as occurring before
149 environmental risk factors for psychoactive substance dependence was similar in males and females.
151 for anxiety disorders, major depression, and substance dependence were approximately three times as h
155 ies with the presence or absence of paternal substance dependence were subdivided into those with and
156 d the association between OPRM1 variants and substance dependence, when sex and age of subjects and a
157 with chronic psychotic illness and comorbid substance dependence who entered a specialized day hospi
158 ctional status of families with fathers with substance dependence with or without comorbid antisocial
159 eceptor system is increasingly recognized in substance dependence, with higher mu-opioid receptor (MO
160 filiation with deviant peers than those with substance dependence without antisocial personality diso
161 =34) did not differ markedly from those with substance dependence without antisocial personality diso