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1 ive interactions of bacteria at the membrane-substrate level.
2 tracking is integral to the structure at the substrate level.
3 ense oligonucleotides in the presence of low substrate level.
4 reased enzyme activity and not to changes in substrate level.
5 the enhancement of the metabolic rate at the substrate level.
6 of gut microbial carbohydrate metabolism at substrate levels.
7 ells were correlated with >50% reductions in substrate levels.
8 nd contribution to the control of conjugated-substrate levels.
9 ovide a complete model for the enzyme at all substrate levels.
10 des expressions for response time and active substrate levels.
11 autophagy upregulation and lowers autophagy substrate levels.
12 relative to the enzyme turnover at ug L(-1) substrate levels.
13 t to alter both dynamic and steady-state ADO substrate levels.
14 c reactions, transporters, cofactors, enzyme substrate-level activation and inhibition relationships,
16 doxin, which increases the efficiency of the substrate level and electron transport phosphorylations.
17 reference, adapting to the prevailing plasma substrate levels and hormonal milieu, but in type 1 diab
18 sm that allows SM to accommodate fluctuating substrate levels and may contribute to its widely report
19 mote interorgan signaling that alters muscle substrate levels and metabolism, thereby contributing to
20 n the KO background reverted both NUDT3 mRNA substrate levels and P-body counts to those of wild-type
21 rs to run with a constant speed at different substrate levels and, therefore, is a substantial criter
22 A (alpha-GalA) activities, glycosphingolipid substrate levels, and in vitro mutation expression were
23 y by acetylation and OAA accumulation at the substrate level are two strategies for the host to respo
25 tate production and a significant portion of substrate-level ATP produced anaerobically, were tested
27 rwise inaccessible parameters such as active substrate levels, b) accurate response-time predictions
29 t mammalian antioxidant, is regulated at the substrate level by cysteine, which is synthesized from h
32 unded plants, but that the AOS hydroperoxide substrate levels, controlled by upstream enzymes (lipoxy
33 rometry-based enzyme functional analysis and substrate level-controlled enzyme kinetics consistently
34 tegy that allows acetic acid removal without substrate-level (de)phosphorylation may instead be emplo
35 the effects of metabolite concentrations and substrate-level enzyme regulation while identifying meta
38 ced oxidants, O(2) evolution, and metabolite substrates levels for glycogen synthesis in normal mediu
39 rial oxidative phosphorylation, increases in substrate level generation of ATP and reducing equivalen
41 relates with lowered pERK but unchanged pPKA substrate levels in D1 medium spiny neurons as well as i
43 associated negative PE occurred even at low substrate levels in this study could be attributed to li
45 tive interventions aiming at alleviating the substrate-level inhibition of key enzymes in order to en
46 idic operations, instrument portability, and substrate-level integration with other pre- and post-PCR
47 proteasome inhibition, and this increase in substrate level is consistent with the observed loss of
48 zation of genome-annotated, respiratory, and substrate-level lactate dehydrogenases (LDHs) from the o
49 erability of DA neurons and that enhancing G-substrate levels may be a neuroprotective strategy for t
50 e the complete inhibition of MOC, indicating substrate level phosphorylation and explicit anaerobic s
51 ondrial enzyme capable of ATP production via substrate level phosphorylation in the absence of oxygen
52 PHOS) with BDQ and simultaneously inhibiting substrate level phosphorylation via genetic disruption o
53 cations of oxygen consumption rate (OCR) and substrate level phosphorylation via glycolysis (i.e., vi
56 elding succinyl-CoA supporting mitochondrial substrate-level phosphorylation (mtSLP), releasing succi
57 robically generate ATP by intramitochondrial substrate-level phosphorylation and maintain DeltaPsi(m)
60 ctions and is not fermentative, we find that substrate-level phosphorylation is its primary anaerobic
64 ed cells with Embden-Meyerhof glycolysis and substrate-level phosphorylation that lack the alpha-prot
66 tron acceptors, generates ATP primarily from substrate-level phosphorylation under anaerobic conditio
68 ive phosphorylation via the pmf, but also by substrate-level phosphorylation via the enzyme AckA.
69 te as the electron acceptor, consistent with substrate-level phosphorylation yielding a significant a
70 asses (e.g., fermentation pathways bypassing substrate-level phosphorylation), substrate channeling (
71 NAD(+)/NADH ratio, generate more ATP through substrate-level phosphorylation, and accumulate biomass
72 -derived polysaccharides that rely mostly on substrate-level phosphorylation, as they seem to lack mo
73 the central pathway does not gain net ATP by substrate-level phosphorylation, chemolithoautotrophic g
74 ding carbon fixation, the shikimate pathway, substrate-level phosphorylation, gluconeogenesis and gly
83 is significantly higher than of disulfides, substrate level regulation favors the synthesis of H2S o
85 ranscriptional and translational regulation, substrate-level regulation of enzyme activity, post-tran
86 se results reveal the critical importance of substrate-level regulation of spectrin cleavage for the
87 this balance between calpain activation and substrate-level regulation of spectrin cleavage is unkno
88 li74) core kinetic model for E. coli with 55 substrate-level regulations using the newly developed K-
90 differential effects of the two compounds on substrate levels that feed into starch synthesis and on
91 conditions of reduced mitogen or nutritional substrate levels, the serine/threonine kinase target of
93 suggest that the early kinetic events at the substrate level ultimately govern successful chaperonin-
94 ne was adaptively regulated by extracellular substrate level via transcriptionally mediated mechanism
96 enzyme); conversely, when the intracellular substrate level was reduced by methionine deprivation, t
98 ining other ESX systems, we found that ESX-1 substrate levels were increased in both the CF and OMM f