ライフサイエンスコーパス: subthalamic

1 re also important as they suggest that local subthalamic activity may potentially be decoded to enabl

2 y ON dopamine, the total duration over which subthalamic and pallidal populations were aligned to pha

3 equency and duration of periods during which subthalamic and pallidal populations were phase-locked t

4 mifying axons in the LH [11, 12], and nearby subthalamic and thalamic areas lack local synaptic conne

5 ients with Parkinson's disease to identify a subthalamic area with an analogous electrophysiological

6 More ventral subthalamic areas showed predominant connectivity to med

7 icycling and walking led to a suppression of subthalamic beta power (13-35Hz), and this suppression w

8 The designated subthalamic 'beta' area projected predominantly to motor

9 uld be modulated by the zona incerta (ZI), a subthalamic brain region that influences sensory discrim

10 While thalamic and subthalamic brain regions play important roles in sensor

11 iological abnormalities, including excessive subthalamic bursts, cortico-subthalamic synchronization,

12 g machine learning techniques we showed that subthalamic connectivity differentiates binge drinkers a

13 nts with PD played a virtual casino prior to subthalamic DBS (whilst 'on' medication) and again, 3-mo

14 However, it is not well known whether subthalamic DBS affects more complex aspects of decision

15 to what has been shown for patients with PD, subthalamic DBS reversibly decreased PAC in a subset of

16 hose persons who become more impulsive after subthalamic DBS, an intervention in which non-motor outc

17 and reduction of cortical synchronization by subthalamic DBS, providing an explanation for their simi

18 Subthalamic deep brain stimulation (DBS) for Parkinson's

19 atients with Parkinson's disease, on and off subthalamic deep brain stimulation (DBS), focussing on a

20 Subthalamic deep brain stimulation (STN-DBS) for Parkins

21 We simulated the effects of subthalamic deep brain stimulation both proximally to th

22 Subthalamic deep brain stimulation may alleviate bradyki

23 haracterize anatomical circuits modulated by subthalamic deep brain stimulation, and infer about the

24 tients with Parkinson disease with bilateral subthalamic deep brain stimulation.

25 Parkinson's disease with either thalamic or subthalamic electrodes (13 male and two female patients,

26 k performance has its origins in the pallido-subthalamic feedback loop.

27 Gamma oscillations arise in the subthalamic-globus pallidus feedback loop, and occur dur

28 Thus, we show that a prefrontal-subthalamic hyperdirect pathway is present in humans and

29 causes reorganization likely takes place at subthalamic levels.

30 t in mid-brain structures, in particular the subthalamic locomotor region (subthalamic nucleus, STN)

31 ponding and emphasized the relevance of this subthalamic network across alcohol misuse.

32 potential therapeutic targets for modulating subthalamic neuron activity in neurological disorders su

33 The activity of subthalamic neurons is tightly controlled by upstream in

34 Here, we study firing properties of human subthalamic neurons, using microelectrode recordings and

35 input and downstream structures reveals that subthalamic, not striatal, activity fluctuations correla

36 se patients with electrodes implanted in the subthalamic nuclei for deep brain stimulation.

37 35Hz) beta power changes were analyzed in 22 subthalamic nuclei from 13 Parkinson disease patients (5

38 ing from dorsal raphe, pedunculopontine, and subthalamic nuclei were tested for synaptic modification

39 le/ventral striatal (VC/VS) and anteromedial subthalamic nucleus (amSTN) DBS in the same patients and

40 ic) profiles in the dorsolateral part of the subthalamic nucleus (i.e. its sensorimotor territory) wa

41 tential of high-frequency stimulation of the subthalamic nucleus (STN HFS) for heroin addiction.

42 such speed-accuracy adjustments by recording subthalamic nucleus (STN) activity and electroencephalog

43 yperdirect pathway from the isocortex to the subthalamic nucleus (STN) adjacent to the PSTN.

44 Neural synchrony between the subthalamic nucleus (STN) and cortex is critical for pro

45 Deep brain stimulation (DBS), targeting the subthalamic nucleus (STN) and globus pallidus interna, i

46 Recent evidence implicates the subthalamic nucleus (STN) and globus pallidus internus (

47 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) and globus pallidus internus (

48 but varies as a function of activity in the subthalamic nucleus (STN) and is further modulated by tr

49 We recorded local field potentials in the subthalamic nucleus (STN) and scalp EEG (modified 10/20

50 connectivity profile of effective DBS to the subthalamic nucleus (STN) and test its ability to predic

51 otor cortex and by a network composed of the subthalamic nucleus (STN) and the external segment of gl

52 The subthalamic nucleus (STN) and the zona incerta (ZI) are

53 SMA) and inferior frontal gyrus (IFG) to the subthalamic nucleus (STN) are thought to support this fu

54 DBS.SIGNIFICANCE STATEMENT It is known that subthalamic nucleus (STN) beta activity is linked to sym

55 The striatum and the subthalamic nucleus (STN) constitute the input stage of

56 ses in parkinsonian nonhuman primates during subthalamic nucleus (STN) DBS and globus pallidus intern

57 Here, we developed a mouse model of subthalamic nucleus (STN) DBS for PD, to permit investig

58 changes in Parkinson disease (PD) induced by subthalamic nucleus (STN) DBS to determine whether these

59 Subthalamic nucleus (STN) deep brain stimulation (DBS) c

60 lls contribute to the therapeutic effects of subthalamic nucleus (STN) deep brain stimulation (DBS) i

61 Subthalamic nucleus (STN) deep brain stimulation (DBS) r

62 The glutamatergic subthalamic nucleus (STN) exerts control over motor outp

63 local field potential (LFP) activity in the subthalamic nucleus (STN) from electrodes implanted in p

64 ders and deep brain stimulation (DBS) of the subthalamic nucleus (STN) has been reported to improve s

65 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) has no effect on the AER, but

66 The neuronal population of the subthalamic nucleus (STN) has the ability to prolong inc

67 We recorded local field potentials from the subthalamic nucleus (STN) in 15 PD patients of both gend

68 regulated by the burst-firing pattern of the subthalamic nucleus (STN) in a feed-forward, or efferent

69 Recent studies have implicated the subthalamic nucleus (STN) in decisions that involve inhi

70 SIGNIFICANCE STATEMENT We tested whether the subthalamic nucleus (STN) in humans is causally involved

71 ans have demonstrated the involvement of the subthalamic nucleus (STN) in motivational and emotional

72 potential (LFP) activities recorded from the subthalamic nucleus (STN) in patients with deep brain st

73 ingers while recording LFP activity from the subthalamic nucleus (STN) in patients with Parkinson's d

74 The subthalamic nucleus (STN) is a critical excitatory signa

75 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is a highly effective symptoma

76 The subthalamic nucleus (STN) is a key area of the basal gan

77 ze DBS algorithms.SIGNIFICANCE STATEMENT The subthalamic nucleus (STN) is a pivotal element of the ba

78 Evidence across species has shown that the subthalamic nucleus (STN) is activated by scenarios invo

79 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is an effective therapy for th

80 The subthalamic nucleus (STN) is an element of cortico-basal

81 The subthalamic nucleus (STN) is hypothesized to play a cent

82 The subthalamic nucleus (STN) is the main target for neurosu

83 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is the most common neurosurgic

84 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is the most commonly used surg

85 Gamma activity in the subthalamic nucleus (STN) is widely viewed as a pro-kine

86 nt models of decision making assume that the subthalamic nucleus (STN) mediates this function by elev

87 ied by a reduction in the rhythmic output of subthalamic nucleus (STN) neurons and synchronization wi

88 ese models suggest that a network of GPe and subthalamic nucleus (STN) neurons computes the normaliza

89 rnal globus pallidus (GPe) and glutamatergic subthalamic nucleus (STN) neurons form a key network wit

90 rnal globus pallidus (GPe) and glutamatergic subthalamic nucleus (STN) neurons form a key, centrally

91 ng methods to predict firing patterns of rat subthalamic nucleus (STN) neurons when their rhythmic fi

92 ta and gamma ranges has been recorded in the subthalamic nucleus (STN) of Parkinson's disease (PD) pa

93 e applied glutamate receptor blockers to the subthalamic nucleus (STN) of parkinsonian rats and evalu

94 Although beta characterization in the subthalamic nucleus (STN) of PD patients undergoing deep

95 hether structural changes are present in the subthalamic nucleus (STN) of people with mild-to-moderat

96 The subthalamic nucleus (STN) of the basal ganglia appears t

97 er 5 pyramidal neurons, which project to the subthalamic nucleus (STN) of the basal ganglia, play a k

98 uppresses motor activity, ostensibly via the subthalamic nucleus (STN) of the basal ganglia.

99 Parkinson's disease (PD) patients to either subthalamic nucleus (STN) or globus pallidus internus (G

100 rget for deep brain stimulation (DBS) of the subthalamic nucleus (STN) remains controversial.

101 ion and blockade of neuronal activity in the subthalamic nucleus (STN) results in a hyperkinetic move

102 ocking of background spiking activity in the subthalamic nucleus (STN) to frontal electroencephalogra

103 during movement, we analysed human ECoG and subthalamic nucleus (STN) unit activity during hand grip

104 gyrus, presupplementary motor area (preSMA), subthalamic nucleus (STN), and primary motor cortex duri

105 d associated brainstem nuclei, including the subthalamic nucleus (STN), globus pallidus, striatum, an

106 projections to ventral tegmental area (VTA), subthalamic nucleus (STN), lateral hypothalamus, among o

107 pivotal element of the basal ganglia is the subthalamic nucleus (STN), which serves as a therapeutic

108 We hypothesize that repeated pairing of subthalamic nucleus (STN)-DBS and M1-TMS at specific tim

109 eated by deep brain stimulation (DBS) of the subthalamic nucleus (STN).

110 us (DR), pedunculopontine nucleus (PPN), and subthalamic nucleus (STN).

111 capsule (ALIC), the nucleus accumbens or the subthalamic nucleus (STN).

112 e-SMA), inferior frontal gyrus, caudate, and subthalamic nucleus (STN).

113 t and indirect pathways, interact within the subthalamic nucleus (STN).

114 were the major GPe cell type innervating the subthalamic nucleus (STN).

115 under different synaptic influences from the subthalamic nucleus (STN).

116 ency oscillations in the cerebral cortex and subthalamic nucleus (STN).

117 cognition is specifically attributed to the subthalamic nucleus (STN).

118 ated and coherent activity in the cortex and subthalamic nucleus (STN).

119 brain stimulation (DBS, n=164) at either the subthalamic nucleus (STN, n=84) or globus pallidus inter

120 Deep brain stimulation (DBS) of the subthalamic nucleus (STN-DBS) has largely replaced ablat

121 lation (HFS) via implanted electrodes at the subthalamic nucleus (STN-HFS).

122 When recording the subthalamic nucleus activity, we found a clear neural re

123 l and direct pathway and reduced coupling of subthalamic nucleus afferent and efferent connections.

124 The strength of effective subthalamic nucleus afferents and efferents were reduced

125 We show that VP output pathways to the subthalamic nucleus and also to the ventral tegmental ar

126 chronization and oscillatory activity in the subthalamic nucleus and basal ganglia (BG) output nuclei

127 a, premotor cortex) and subcortical network (subthalamic nucleus and cerebellum).

128 or performing simultaneous recordings of the subthalamic nucleus and cortex using magnetoencephalogra

129 BAergic transmission within the normal human subthalamic nucleus and evidence of GABA innervation thr

130 Using a computational model of subthalamic nucleus and external globus pallidus, we ext

131 served that certain phase alignments between subthalamic nucleus and globus pallidus amplified local

132 ronize a network of model neurons comprising subthalamic nucleus and globus pallidus external and sug

133 ed local field potential recordings from the subthalamic nucleus and globus pallidus of five patients

134 observed in the ventral border region of the subthalamic nucleus and in its sensorimotor subregion an

135 Fourth, we show that activity in the subthalamic nucleus and its effective connectivity with

136 Beta band coupling between the subthalamic nucleus and lateral motor areas was not infl

137 atter structures (caudate nucleus, thalamus, subthalamic nucleus and lentiform nucleus) was estimated

138 ssed synchronization of activity between the subthalamic nucleus and mesial premotor regions, includi

139 nvolvement of fibres connecting ventromedial subthalamic nucleus and orbitofrontal cortex.

140 ations, and make stronger projections to the subthalamic nucleus and parafascicular nucleus of the th

141 associated with anterior associative-limbic subthalamic nucleus and right dorsolateral prefrontal fu

142 o, with right-hemispheric tracts between the subthalamic nucleus and the pre-supplementary motor area

143 tudy, 40 received a bilateral implant in the subthalamic nucleus and their data contributed to the pr

144 fied cell classes projected primarily to the subthalamic nucleus and to the striatum, respectively.

145 gambles, weighted by connections between the subthalamic nucleus and ventromedial prefrontal cortex.

146 The striatum and the subthalamic nucleus are the main entry points for cortic

147 ived bilateral deep brain stimulation of the subthalamic nucleus at the National Institutes of Health

148 Accordingly, the subthalamic nucleus can be considered a key component of

149 tral striatum to right anterior ventromedial subthalamic nucleus consistent with previous observation

150 st to follow-up, the evidence indicates that subthalamic nucleus DBS improves motor function for up t

151 dence of the safety and clinical efficacy of subthalamic nucleus DBS with a novel MICC device for the

152 olled, randomised controlled trial to assess subthalamic nucleus DBS, with a novel multiple independe

153 c efficacy of STN DBS.SIGNIFICANCE STATEMENT Subthalamic nucleus deep brain stimulation (STN DBS) is

154 Subthalamic nucleus deep brain stimulation (STN DBS) pro

155 Bilateral subthalamic nucleus deep brain stimulation (STN-DBS) has

156 Subthalamic nucleus deep brain stimulation (STN-DBS) in

157 Parkinson's Disease patients both on and off subthalamic nucleus deep brain stimulation (STN-DBS), wh

158 onism with severe dyskinesias, and underwent subthalamic nucleus deep brain stimulation 8 years after

159 , attention/memory, and sleep outcomes after subthalamic nucleus deep brain stimulation depends on th

160 Subthalamic nucleus deep brain stimulation is an effecti

161 Specifically, we show that subthalamic nucleus deep brain stimulation modulates all

162 vivo evidence for the modulatory effects of subthalamic nucleus deep brain stimulation on effective

163 investigate the influence of the location of subthalamic nucleus deep brain stimulation on non-motor

164 subthalamic nucleus, respectively, and that subthalamic nucleus deep brain stimulation predominantly

165 results highlight that clinically effective subthalamic nucleus deep brain stimulation suppresses sy

166 Eleven patients with Parkinson's disease and subthalamic nucleus deep brain stimulation underwent fun

167 ng magnetoencephalography (during concurrent subthalamic nucleus deep brain stimulation).

168 ct rare functional MRI data in patients with subthalamic nucleus deep brain stimulation.

169 paradigm to test the hypothesis that pairing subthalamic nucleus deep-brain stimulation (STN-DBS) wit

170 ally effective deep brain stimulation of the subthalamic nucleus differentially modifies different os

171 oxygen-level-dependent response in the right subthalamic nucleus during an impulsiveness task.

172 such as the ventral intermediate nucleus and subthalamic nucleus fell outside our Holmes tremor circu

173 ency band oscillatory synchronization in the subthalamic nucleus have been associated with motor impa

174 levations in beta activity (13-35 Hz) in the subthalamic nucleus have been demonstrated to correlate

175 white matter structures in proximity to the subthalamic nucleus have been implicated in the clinical

176 effective high-frequency stimulation of the subthalamic nucleus imposes cross-frequency interactions

177 als from both the inferior frontal gyrus and subthalamic nucleus in 21 subjects.

178 In conclusion, our results implicate the subthalamic nucleus in a modulation of outcome value in

179 sfully to detect stimulated glutamate in the subthalamic nucleus in brain slices and in vivo.

180 ecificity of the anterior associative-limbic subthalamic nucleus in decisional impulsivity.

181 riatal, and hyperdirect connections with the subthalamic nucleus in modulating waiting and stopping a

182 a better understanding about the role of the subthalamic nucleus in non-motor functions is needed.

183 Deep brain stimulation (DBS) of the subthalamic nucleus in Parkinson's disease is known to c

184 ase, suggesting a more pervasive role of the subthalamic nucleus in the control of human decision-mak

185 sibility that cortical connectivity with the subthalamic nucleus in the high and low beta bands may r

186 or regions were predominantly coupled to the subthalamic nucleus in the high beta frequency range, bu

187 tion suppresses synchrony locally within the subthalamic nucleus in the low beta oscillatory range an

188 tal volatility) and implicate a role for the subthalamic nucleus in the modulation of outcome certain

189 e that the amplitude of beta activity in the subthalamic nucleus increases in proportion to burst dur

190 imulation of estimated predominant non-motor subthalamic nucleus induced hypomanic behaviour.

191 Deep brain stimulation (DBS) of the subthalamic nucleus is a symptomatic treatment of Parkin

192 Deep brain stimulation of the subthalamic nucleus is an effective and established ther

193 Deep brain stimulation of the subthalamic nucleus is an effective treatment for Parkin

194 Deep brain stimulation (DBS) of the subthalamic nucleus is an established therapeutic option

195 Deep brain stimulation of the subthalamic nucleus is an established treatment for the

196 We show that stimulation of the subthalamic nucleus is causally implicated in increasing

197 Moreover, the subthalamic nucleus is critical to balance inhibitory an

198 variability, functional organization of the subthalamic nucleus is difficult to investigate in vivo

199 thway between the inferior frontal gyrus and subthalamic nucleus is hypothesized to mediate movement

200 The subthalamic nucleus is implicated in inhibitory function

201 pears at odds with the current view that the subthalamic nucleus is important for adjusting behaviour

202 ese results provide strong evidence that the subthalamic nucleus is involved in response inhibition,

203 The subthalamic nucleus is the preferred neurosurgical targe

204 that low-frequency neuronal activity in the subthalamic nucleus may encode the information required

205 lationship using extracellular recordings of subthalamic nucleus neurons from 19 PD patients undergoi

206 FoxP2+ arkypallidal GPe neurons and subthalamic nucleus neurons were lost by 18 months but n

207 ocal field potentials were recorded from the subthalamic nucleus of 12 patients with advanced Parkins

208 corded local field potential activity in the subthalamic nucleus of 18 patients with Parkinson's dise

209 ants underwent bilateral implantation in the subthalamic nucleus of a multiple-source, constant-curre

210 ocal field potentials were recorded from the subthalamic nucleus of eight Parkinson's disease patient

211 z) oscillations recorded from the cortex and subthalamic nucleus of Parkinson's disease patients.

212 es by recording the neuronal activity of the subthalamic nucleus of patients with Parkinson's disease

213 ression, likely reflecting the impact of the subthalamic nucleus on basal ganglia output; then, at ~1

214 ests that the modulatory relationship of the subthalamic nucleus on intracerebellar connectivity is l

215 onse-conflict increases the influence of the subthalamic nucleus on M1-representations of incorrect r

216 Yet only the change in strength of local subthalamic nucleus oscillations correlates with the deg

217 First that the subthalamic nucleus plays a role in adjusting response t

218 Motor cortical coupling with subthalamic nucleus predominantly involved driving of th

219 basal ganglia pathways, and the hyperdirect subthalamic nucleus projections.

220 tterns in the striatum, globus pallidus, and subthalamic nucleus related to sensory and motor events

221 ectivity with the ventrolateral thalamus and subthalamic nucleus showed inverse correlation with keta

222 nts with idiopathic Parkinson's disease with subthalamic nucleus stimulation were analysed on externa

223 in the anterior frontal cortex, striatum and subthalamic nucleus suggests the striatal afferent conne

224 uency band having much shorter net delays to subthalamic nucleus than those in the lower beta band.

225 ociated suppression in their coupling to the subthalamic nucleus was not found to correlate with moto

226 ming asymmetric synapses in the dorsolateral subthalamic nucleus was reduced by 55.1% and 27.9%, resp

227 ral cortices, striatum, substantia nigra and subthalamic nucleus were assessed.

228 oing bilateral deep brain stimulation of the subthalamic nucleus were included, and we investigated N

229 as associated with lower connectivity of the subthalamic nucleus with ventral striatum and subgenual

230 ence of different functional circuits within subthalamic nucleus' portions deemed to be appropriate a

231 lysis exploring the effect of atrophy in the subthalamic nucleus, a cerebellar input source, confirme

232 internal capsule, the ventral striatum, the subthalamic nucleus, and a midbrain target.

233 r in all regions except the dentate nucleus, subthalamic nucleus, and corpus callosum of multiple sys

234 subthalamic nucleus, ramping activity of the subthalamic nucleus, and movement-related activity of th

235 Hz, a similar, though weaker, oscillation in subthalamic nucleus, and strong phase coherence between

236 ex preceded stopping-related activity in the subthalamic nucleus, and synchronization between these t

237 eiving more inputs from the globus pallidus, subthalamic nucleus, and zona incerta.

238 y to the thalamus and posteromedially to the subthalamic nucleus, in close proximity, mainly anterola

239 taucipir uptake in globus pallidus, putamen, subthalamic nucleus, midbrain, and dentate nucleus relat

240 emporally regulated sensory responses of the subthalamic nucleus, ramping activity of the subthalamic

241 by the hyperdirect and indirect pathways to subthalamic nucleus, respectively, and that subthalamic

242 particular the subthalamic locomotor region (subthalamic nucleus, STN) and the periaqueductal grey (P

243 tical nodes and is posited to project to the subthalamic nucleus, with a putative global suppressive

244 ucleus predominantly involved driving of the subthalamic nucleus, with those drives in the higher bet

245 ally and spectrally segregated resting state subthalamic nucleus-cortical networks.

246 The subthalamic nucleus-globus pallidus network is a potenti

247 ct evidence points to the involvement of the subthalamic nucleus-the most common target for deep brai

248 ith anterior and posterior subregions of the subthalamic nucleus.

249 ts localized within the anterior and ventral subthalamic nucleus.

250 emotionally evoked activity to right ventral subthalamic nucleus.

251 in stimulation of the limbic and associative subthalamic nucleus.

252 onnections of the pre-supplementary area and subthalamic nucleus.

253 atory input from the 'limbic' regions of the subthalamic nucleus.

254 des mapped onto a mesial-lateral axis of the subthalamic nucleus.

255 atients, after deep brain stimulation of the subthalamic nucleus.

256 in 4 patients with PD undergoing DBS of the subthalamic nucleus.

257 tual conditions in the cerebellum, pons, and subthalamic nucleus.

258 connects the inferior frontal gyrus and the subthalamic nucleus.

259 overlap the labeled neurons observed in the subthalamic nucleus.

260 ter-related hypotheses about the role of the subthalamic nucleus.

261 ions in deep-brain structures, including the subthalamic nucleus.

262 tween the inferior frontal gyrus and ventral subthalamic nucleus.

263 omass, reversed by deep brain stimulation of subthalamic nucleus.

264 e average improvement located ventral to the subthalamic nucleus.

265 mprovement were mainly located dorsal to the subthalamic nucleus.

266 anted with the DBS device bilaterally in the subthalamic nucleus.

267 s for the function of circuits involving the subthalamic nucleus.

268 l circuits accompanied by hyperactivation of subthalamic nucleus/putaminal regions.

269 that stimulation of three DBS targets (STN, subthalamic nucleus; GPi, globus pallidus internus; NAc,

270 Subthalamic, pallidal, and striatal neurons engaged and

271 Subthalamic principal neurons were found to express GABA

272 on through both GABAA and GABAB receptors on subthalamic principal neurons.

273 utamate transporter 1-positive (i.e. cortico-subthalamic) profiles in the dorsolateral part of the su

274 or a partial loss of the hyperdirect cortico-subthalamic projection in MPTP-treated parkinsonian monk

275 he following two questions about the cortico-subthalamic projections using the lentivirus anterograde

276 Zona incerta (ZI) is a largely inhibitory subthalamic region connecting with many brain areas.

277 n, at the single neuron level, the important subthalamic role in motor control and coordination and i

278 The well-known subthalamic somatotopy showed a large overlap of feet an

279 f the cortical motor network, and highlights subthalamic stimulation as a network-modulating therapy.

280 timulation in Parkinson's disease, P=0.0078, subthalamic stimulation in Parkinson's disease, P=0.0312

281 ven increasing interest in the potential for subthalamic stimulation in psychiatric disorders and the

282 bjects with obsessive compulsive disorder on subthalamic stimulation may be less likely to check for

283 Accordingly, subthalamic stimulation may release bilateral cortical c

284 sks and mathematical modeling, we found that subthalamic stimulation normalizes pathological hyperact

285 Cortical potentials evoked by subthalamic stimulation revealed short latency events in

286 Subthalamic stimulation shifts evidence accumulation in

287 ive effects as a function of localization of subthalamic stimulation.

288 constitute a less invasive alternative than subthalamic stimulation.

289 Parkinson's disease and its modulation with subthalamic stimulation.

290 clinical and reaction time improvement from subthalamic stimulation.

291 son's disease (PD) and tend to improve after subthalamic (STN) stimulation after a marked reduction o

292 , whereas the Dbx1 microdomain gives rise to subthalamic (STN), premammillary (PM) and posterior hypo

293 luding excessive subthalamic bursts, cortico-subthalamic synchronization, and in situ beta synchroniz

294 ded to electrical stimulation of the cortico-subthalamic system in parkinsonian monkeys.

295 To find out whether the primate cortico-subthalamic system is also subject to functionally relev

296 veractivation and synchronization of cortico-subthalamic transmission alone sufficiently and instanta

297 e found that inhibition of NMDAergic cortico-subthalamic transmission ameliorates parkinsonian motor

298 ways, our data suggest that deranged cortico-subthalamic transmission via the NMDA receptor also play

299 r and lower extremity movement kinematics in subthalamic units and observed evidence for re-routing t

300 tractography using the participant-specific subthalamic volume of activated tissue as a seed.