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1                                    ABSTRACT: Sudomotor activity is modified by both warm and cold flu
2                                              Sudomotor activity is modified by both warm and cold flu
3 had laboratory evidence of severe adrenergic sudomotor and cardiovagal failure.
4 sed autonomic testing, including adrenergic, sudomotor and cardiovagal functions and Thermoregulatory
5 ardiovascular, gastrointestinal, urogenital, sudomotor, and pupillomotor systems, occur in varying co
6 ic tests (Valsalva maneuver, deep breathing, sudomotor, and tilt test).
7 ts using the methodology of the quantitative sudomotor axon reflex test (QSART).
8 tor function as measured by the Quantitative Sudomotor Axon Reflex Test, most significantly in the fo
9     Participants also completed quantitative sudomotor axon reflex testing, quantitative sensory test
10       Nerve conduction studies, quantitative sudomotor axon testing, and intraepidermal nerve fiber d
11 he presynaptic portion of the postganglionic sudomotor axon.
12 ve power despite matched WBGT; and (iii) the sudomotor but not behavioural thermoregulatory responses
13 tudy documented the pathological evidence of sudomotor denervation in FAP.
14 riers, both skin denervation and peptidergic sudomotor denervation were evident: (1) IENF density was
15 e the pathology and clinical significance of sudomotor denervation.
16 s designed to test for the early presence of sudomotor dysfunction and to characterize its relation t
17 llary impairments, 7 of 8 had postganglionic sudomotor dysfunction, 9 of 11 had urinary retention and
18  thermoregulatory sweat test (67%) confirmed sudomotor dysfunction.
19 thy (FAP) due to mutated transthyretin, with sudomotor failure as a common manifestation.
20 autonomic failure with severe adrenergic and sudomotor failure combined with the clinical phenotype i
21  primarily due to a significant reduction in sudomotor function as measured by the Quantitative Sudom
22                          Previous studies of sudomotor function in diabetes have included patients wi
23 cally, autonomic evaluation often focuses on sudomotor function yet this is rarely assessed in animal
24 tology, amino acid profiles, metabolism, and sudomotor function.
25                    Biomarkers of sensory and sudomotor innervation exhibited a stage-dependent progre
26                                              Sudomotor innervation was stained with 2 markers: protei
27 sed with cardiovascular, pupillary, urinary, sudomotor, lacrimal and salivary testing, and Composite
28 , and vasoactive intestinal peptide (VIP), a sudomotor nerve functional marker, followed by quantitat
29          We interpret this redistribution of sudomotor responses to be indicative of sympathetic nerv
30                            The MCASS and its sudomotor subscore (reflecting peripheral sympathetic fu
31 (MCASS) and its adrenergic, cardiovagal, and sudomotor subscores, breath testing for small intestinal
32 scular, gastrointestinal, genitourinary, and sudomotor symptoms in all subjects.
33 her this supersensitivity also occurs in the sudomotor system.
34 cale for grading of cardiovascular reflexes, sudomotor tests and skin biopsies (QASAT), and Composite
35 ilomotor nerves accompanied by impairment of sudomotor, vasomotor, and pilomotor function.
36   Topical capsaicin leads to degeneration of sudomotor, vasomotor, and pilomotor nerves accompanied b
37                     There were reductions in sudomotor, vasomotor, pilomotor, and sensory function in
38 owed for 6 months with serial assessments of sudomotor, vasomotor, pilomotor, and sensory function wi
39                     There were reductions in sudomotor, vasomotor, pilomotor, and sensory nerve fiber