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1 tial diameter, respectively, after 15 min of suffusion.
2 mination (94% for rash, 100% for hemorrhagic suffusion, and 95% for signs of secondary infection), ch
7 EDR impairment was reversed by adventitial suffusion of superoxide dismutase (SOD) of aortas from w
8 intravital microscopy, we found that 20-min suffusion of the extract elicited significant concentrat
10 diameter, n = 41) microvessels following the suffusion of vasoactive drugs was measured with video ca
12 nitroindazole (7-NI), followed by successive suffusions of the cGMP analogue, 8-bromo-cGMP (8Br-cGMP)
13 esis, addition of 10(-4) M furosemide to the suffusion still caused significant dilation in arteriole
14 pound 48/80, we examined permeability during suffusion with compound 48/80 (25 microg/ml) in the pres
16 disruption of the blood-brain barrier during suffusion with compound 48/80, we examined permeability
17 o 76.6% of initial diameter) after 20 min of suffusion with L-NAME, an inhibitor of NO synthesis.
20 uced slowing of flow was reversed rapidly by suffusion with UFH, provided flow had not already stoppe
22 ran (M(w)=10000 Da; FITC-dextran-10K) during suffusion with vehicle, S-nitroso-N-acetylpenicillamine