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1 e criteria of previous authors, although the sulcal anatomy of the human temporal pole has great vari
2           However, the relationships between sulcal anatomy, brain activity, and behavior are still p
3 tism and 14 healthy comparison subjects, and sulcal and gyral thickness were measured for the total b
4                  Increases in total cerebral sulcal and gyral thickness were observed in children wit
5  folding into a set of nested subcomponents: sulcal and hull area, and sulcal depth and length.
6 al cortical gray matter decline and cortical sulcal and lateral ventricular expansion.
7             Regionally specific increases in sulcal and subarachnoid CSF occur during adulthood and a
8 MH) grade, brain and hippocampal volume, and sulcal and ventricle atrophy using nested multivariate r
9 d white matter as well as greater volumes of sulcal and ventricular CSF than nonalcoholic women.
10 iations, except for a lack of association of sulcal and ventricular size with race.
11 sulted in a significant reduction in buccal, sulcal, and proximal dental caries compared to untreated
12                                       Total, sulcal, and smooth-surface molar caries in the GBP59- an
13 on could be further subdivided into an outer sulcal anterior auditory field (sAAF) and an inner field
14 tudinal allometric scaling points to loss of sulcal area as a principal contributor to loss of brain
15 ectories demonstrated steeper loss of SI and sulcal area through adulthood in SZ.
16 portionate contribution of changes in SI and sulcal area to TBV changes in SZ.
17 s revealed that both annual change in SI and sulcal area was significantly stronger related to change
18                               Scaling of SI, sulcal area, and sulcal length with TBV in SZ and FEP wa
19 ral region: the lateral rostral suprasylvian sulcal area.
20 lcal/cortical hull area) and determinants of sulcal area: sulcal length and sulcal depth.
21 an posterior cingulate gyrus without marking sulcal areas.
22 rtical plate (CP), subplate (gyral crest and sulcal bottom), and IZ.
23 espread as well as spatially coherent within sulcal boundaries and decreases in amplitude across the
24 y had statistically significant decreases in sulcal caries severity.
25 of smooth-surface caries and the severity of sulcal caries, relative to controls.
26 We found accelerated enlargement in cortical sulcal cerebrospinal fluid spaces early in the course of
27 n clusters were associated with formation of sulcal clots (P < 0.01), a high likelihood of adjacent c
28 al morphology of the cingulate/paracingulate sulcal complex and feedback-related activity.
29 rontal gyrus (MFG) shows that both gyral and sulcal components of the MFG have greater myelin content
30 yers of the gyral components is greater than sulcal components.
31 ical patches equated for size and shape; (2) sulcal connectivity did not consistently correspond with
32                    Tracer injection into the sulcal cortex demonstrated that at least some axonal fib
33 he anteroventral field and the pseudosylvian sulcal cortex.
34 ed subcomponents: sulcation index (SI, total sulcal/cortical hull area) and determinants of sulcal ar
35 d with larger left (9.66%) and right (9.61%) sulcal CSF relative volumes were found in women with SPD
36 y matter volume and significant increases in sulcal CSF volume compared with controls.
37 ng groups, gray matter volume reductions and sulcal CSF volume increases were significantly more pron
38 er neocortical gray matter volume and larger sulcal CSF volume provide evidence of the brain basis of
39 as segmented into gray matter, white matter, sulcal CSF, and ventricular CSF from a T(1)-weighted mag
40 al lobe gray matter, lateral ventricles, and sulcal CSF.
41 omical-behavioral studies, we share tertiary sulcal definitions with the field.
42                                      Maps of sulcal depth (distance from the cerebral hull) were comb
43 easures included cortical thickness (CT) and sulcal depth (SD) across the cerebrum.
44                         Regional analysis of sulcal depth and cortical shape showed the greatest impa
45 ted subcomponents: sulcal and hull area, and sulcal depth and length.
46          We performed a detailed analysis of sulcal depth based on geometric cortical surface represe
47 riability was also related to variability in sulcal depth but not cortical thickness, positively corr
48 ch significant sulcal widening and decreased sulcal depth co-occurred.
49 ior frontal gyrus that was associated with a sulcal depth difference in the anterior insula and front
50                                              Sulcal depth findings in the PS corresponded closely to
51 ross individuals to generate maps of average sulcal depth for WS and control subjects, along with dep
52 al thickness and surface area indicated that sulcal depth has higher locus yield, heritability, and e
53             We found bilateral reductions in sulcal depth in the intraparietal/occipitoparietal sulcu
54  negatively correlated with lobar and global sulcal depth in VPT infants.
55                     Our findings demonstrate sulcal depth is a promising neuroimaging phenotype that
56                                              Sulcal depth is a robust yet understudied measure of loc
57                                 We generated sulcal depth maps from structural magnetic resonance ima
58  effect of FCD size, network, histology, and sulcal depth on age of epilepsy onset.
59 ted with dominant network (p = 0.04) but not sulcal depth or histology.
60  fetal cortical local gyrification index and sulcal depth were associated with infant 18-month social
61  fetal cortical local gyrification index and sulcal depth were negatively associated with infant soci
62 , lobe volume, local gyrification index, and sulcal depth) in the frontal, parietal, temporal, and oc
63  on two measures (verbal memory and cortical sulcal depth).
64 lated with cortical thickness, gyrification, sulcal depth, and complexity metrics, particularly in th
65 thickness, surface area, gyrification index, sulcal depth, and curvature) and diffusion parameters (f
66  (measured by gray- and white-matter volume, sulcal depth, and gyrification index) that localize to t
67 al cortical volume, thickness, surface area, sulcal depth, and subcortical volume).
68 ersubject variability in cortical curvature, sulcal depth, cortical thickness, and cortical surface a
69 ogy measures (gyrification index, thickness, sulcal depth, curvature, surface area) at term-equivalen
70 increased cerebral cortical gyrification and sulcal depth, decreased brain metabolites (e.g., choline
71                   Population average maps of sulcal depth, depth variability, three-dimensional posit
72  B, -0.8; 95% CI, -1.6 to -0.1; P = .03) and sulcal depth, except for the frontal lobe (for example,
73 variants exhibit altered local gyrification, sulcal depth, surface area and thickness in their cortex
74 yzed regional changes in cortical thickness, sulcal depth, surface area, and volume between two timep
75         Here, we showed that WBV (thickness, sulcal depth, volume) and total cortical surface area we
76 t the first genome-wide association study of sulcal depth.
77 terminants of sulcal area: sulcal length and sulcal depth.
78  -54.62; 95% CI, -85.05 to -24.19; P < .001; sulcal depth: beta = -14.22; 95% CI, -23.59 to -4.85; P
79 = -24.01; 95% CI, -40.34 to -7.69; P = .003; sulcal depth: beta = -7.53; 95% CI, -11.73 to -3.32; P <
80  although subpial thorn-shaped astrocytes at sulcal depths were occasionally observed in aged control
81 including the subpial region of the cortical sulcal depths.
82 erations that included parenchymal edema and sulcal effusion leading to transient hyperintensities on
83 unassisted detection of ARIA-E (edema and/or sulcal effusion) and ARIA-H (microhemorrhage and/or supe
84 rmalities suggestive of vasogenic oedema and sulcal effusions (ARIA-E) and microhaemorrhages and haem
85 lume increases in the lateral ventricles and sulcal (especially frontal and temporal) CSF than Val ho
86           Prefrontal gray matter decline and sulcal expansion were associated with greater BPRS negat
87                      Faster rates of frontal sulcal expansion were related to greater BPRS total and
88 cortical gray matter volume loss, as well as sulcal expansion.
89 ution atlas (including labeling of gyral and sulcal features).
90 olding of the mammalian cerebral cortex into sulcal fissures and gyral peaks is the result of complex
91 ere observed in gyral crowns and deep within sulcal folds.
92 al human brains covering a period of primary sulcal formation (20-28 gestational weeks).
93 al curvature (gyral crowns in face areas and sulcal fundi in place areas).
94 ds to be higher in gyral crests and lower in sulcal fundi.
95 tion is mechanistically underpinned by trans-sulcal gene expression gradients enriched for developmen
96 generated flat maps in relation to gyral and sulcal geography, thereby facilitating the analysis of c
97 hat the gray matter volume loss and abnormal sulcal geometry may be related.
98                      Statistical analysis of sulcal geometry will facilitate multisubject atlasing, n
99 sion were used to characterize variations in sulcal geometry.
100 h increasing age, and higher ventricular and sulcal grades in men and in nonblack individuals.
101                             Inferior frontal sulcal hyperintensities (IFSH) observed on fluid-attenua
102 -ri (parenchymal hyperintensities 89% vs 3%, sulcal hyperintensities 78% vs 9%, and gyral swelling 86
103 ory changes (cortical or parenchymal oedema, sulcal hyperintensities).
104  (parenchymal white matter hyperintensities, sulcal hyperintensities, and gyral swelling) are more co
105 g parenchymal white matter hyperintensities, sulcal hyperintensities, and gyral swelling; and evidenc
106 ther, these results reveal that the MFS is a sulcal landmark that is shared between humans and other
107  By using models of the cortical surface and sulcal landmarks and a statistical model for gray matter
108  cortical pattern-matching technique used 72 sulcal landmarks traced on each brain as anchors to alig
109 olded cortical surface relative to gyral and sulcal landmarks.
110 ess cortical gray and white matter, and less sulcal, lateral, and third ventricular CSF.
111  hull area) and determinants of sulcal area: sulcal length and sulcal depth.
112 lation-based interhemispheric asymmetries of sulcal length existed that could be considered markers o
113  prefrontal cortex, with a 1 cm reduction in sulcal length increasing the likelihood of hallucination
114 s, photographs, morphological recordings and sulcal length metrics, our data illustrate the interindi
115              Scaling of SI, sulcal area, and sulcal length with TBV in SZ and FEP was allometric and
116  outer cortical surface (hull surface area), sulcal length, depth, and width.
117 nduced by head impact loading is greatest in sulcal locations, where pathology in cases of chronic tr
118 roughout cortex, we share pmfs probabilistic sulcal maps with the field.SIGNIFICANCE STATEMENT Latera
119 exome-wide associations of regional cortical sulcal measures derived from magnetic resonance imaging
120                     Also, we show systematic sulcal morphological organization of the medial frontal
121 retation of the evolution of primate brains, sulcal morphological variability of the medial frontal c
122 fining thousands of sulci revealed that LPFC sulcal morphology (depth, surface area, and gray matter
123 stion: What is the linking mechanism between sulcal morphology and cognition?
124 es did not consider the relationship between sulcal morphology and cognitive development in individua
125 ionship between variability in LPFC tertiary sulcal morphology and reasoning scores in a cohort of ch
126                 These results illustrate how sulcal morphology can be functionally relevant and provi
127 the interindividual variability in the vmPFC sulcal morphology in 57 humans (37 females) and demonstr
128 and provide a resource for future studies of sulcal morphology in health and disease.
129 folding, and there is mounting evidence that sulcal morphology is relevant to functional brain archit
130          Longitudinal allometric analysis of sulcal morphology provides deeper insight into lifespan
131              A model-based approach relating sulcal morphology to cognition identified that a subset
132 hese data provide a new framework to compare sulcal morphology, cytoarchitectonic areal distribution,
133                             Furthermore, the sulcal organization influences the location of the vmPFC
134 ralimbic sulcus influences significantly the sulcal organization of this region.
135                                              Sulcal outlines were reparameterized for surface compari
136                                   We combine sulcal pattern analysis with resting-state functional ma
137 nches, and connections) and for a particular sulcal pattern, namely, the interposition of a precentra
138 y and white matter, macrogyria, and abnormal sulcal pattern.
139 ese results require a reconsideration of the sulcal patterning in ventral temporal cortex across homi
140                           Although gyral and sulcal patterns are highly heritable, and emerge in a ti
141 Most of Einstein's sulci are identified, and sulcal patterns in various parts of the brain are compar
142  that taking into account the variability in sulcal patterns might be essential to guide the interpre
143                Two of the photographs reveal sulcal patterns of the medial surfaces of the hemisphere
144                       Heritability for gyral-sulcal patterns, as reflected in the cross-correlation d
145 functional activity can be affected by local sulcal patterns.
146 ral poles, and parietal cortex; and lower in sulcal, perirolandic, and medial occipital cortex.
147 etween cortical surface sulci to construct a sulcal phenotype network (SPN) from each magnetic resona
148 iderations the contact between the posterior sulcal plate and the first postcingular plate, as well a
149 allel to the cortical surface, most often in sulcal regions but also in many gyral crowns.
150                       The volume fraction of sulcal regions exceeding brain injury thresholds were si
151                                              Sulcal regions exhibited higher contrast compared to gyr
152 ickness, with positive relationships seen in sulcal regions in prefrontal and temporal cortices, and
153          Positive relationships were seen in sulcal regions in prefrontal and temporal cortices, whil
154 owed converging imaging abnormalities within sulcal regions with a significant decrease in fractional
155 ntal and orbitofrontal and superior temporal sulcal regions with areas of relative preservation in vi
156  measured in 20 tissue and 5 ventricular and sulcal regions.
157 n, namely, the interposition of a precentral sulcal segment between the central sulcus and one of its
158 anifested in global alterations of gyral and sulcal shape.
159                            Ventricular size, sulcal size, and white matter signal intensity changes w
160                                   Using peri-sulcal stimulation to evoke recurrent activity in deep l
161              The results show that gyral and sulcal structures are aligned with high but variable acc
162  the basilar cisterns (P <.001) and cerebral sulcal subarachnoid space (P <.001) after Fio(2) was red
163 tans and a cariogenic diet, total smooth and sulcal surface lesions are more than 2- and 1.6-fold hig
164 ts by using elastic transformations to align sulcal topography.
165 the relationship between thickness and local sulcal topology arises in development is still not well
166  vmPFC and show that taking into account the sulcal variability might be essential to guide the inter
167 roup had smaller thalamic and larger frontal sulcal volumes than control subjects.
168     Slopes relating cortical gray matter and sulcal volumes to age were steeper in alcoholic than in
169 x interactions for cortical white matter and sulcal volumes were due to abnormalities in alcoholic me
170 and occipital cortices, in which significant sulcal widening and decreased sulcal depth co-occurred.
171 ences were consistent with cortical thinning/sulcal widening and ventricular enlargement.
172                                    Globally, sulcal widening was associated with cortical thinning an
173 een cognitive function and brain morphology (sulcal width, SW) using three cross-sectional observatio
174                                              Sulcal width, ventricular size, and white matter signal
175           It invokes tangential tension and "sulcal zipping" forces along the outer cortical margin a

 
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