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1         BAEPs are recorded at 6 or 24 h post-sulfa.
2  (50 mg/kg) administered 30 or 120 min after sulfa (200 mg/kg) in 16 days old jjs.
3                            Sulfadimethoxine (sulfa) administration to jjs, which displaces bilirubin
4                                    The term "sulfa allergy", originally coined for the sulfonamide an
5  precatalysts, a number of efficient carba-, sulfa-, and phospha-Michael additions were achieved very
6 ssociated uveitis, with special attention to sulfa class antibiotics, historically have been used to
7 designer drug consisting of sulfapyridine, a sulfa-containing antibacterial agent, and 5-amino-salicy
8                                Whereas these sulfa drug-like molecules might have acted pharmacologic
9 f such mutations in the United States, where sulfa-drug prophylaxis is widespread, to that in China,
10 of patients had DHPS mutations; 38% received sulfa-drug prophylaxis.
11 f patients had DHPS mutations; none received sulfa-drug prophylaxis.
12                                              Sulfa drugs are widely used in the treatment and prophyl
13 t nonlipidic small molecules, which resemble sulfa drugs implicated in systemic hypersensitivity and
14 s have identified mutations in the target of sulfa drugs that appear to represent emerging resistance
15 ynthase gene (dhps), the enzymatic target of sulfa drugs used for PcP prevention and treatment.
16 hich is notable for its similarity to common sulfa drugs, but its mechanism of NKT cell activation re
17 tibacterial substances, organoarsenicals and sulfa drugs, but these were soon outshone by a host of m
18 mycin, cetylpyridinium chloride, and several sulfa drugs.
19 lving under positive selective pressure from sulfa drugs.
20            Minocycline administered prior to sulfa in jjs protects against BAEP abnormalities.
21                  The highly enantioselective sulfa-Michael addition of alkyl thiols to unactivated al
22                              Stereoselective sulfa-Michael addition of appropriately protected thioca
23        The mechanism of the enantioselective sulfa-Michael addition reaction catalyzed by a cinchona
24   In this manuscript, a simple and efficient sulfa-Michael addition reaction of aryl thiols to trisub
25 honidine or cinchona alkaloid-urea catalyzed sulfa-Michael addition reactions, also applies to the ca
26          The products of the organocatalytic sulfa-Michael addition to alpha,beta-unsaturated alpha-a
27 denitrative C-S bond formation with xanthate/sulfa-Michael addition to chalcones.
28 ough C(aryl)-S coupling, thioester cleavage, sulfa-Michael addition, aldol reaction, and elimination
29          A novel triphenylphosphine-mediated sulfa-Michael addition-triggered cascade reaction has be
30  is a late-stage, highly diastereoselective, sulfa-Michael addition.
31                             Enantioselective sulfa-Michael additions to alpha,beta unsaturated diazoc
32                                              Sulfa-Michael additions to alpha,beta-unsaturated N-acyl
33 of the alpha,beta-unsaturated ester, affords sulfa-Michael adducts in excellent yields (up to >99%) a
34 fones in a simple and reliable way through a sulfa-Michael reaction that proceeds with high yield and
35 sequential domino reactions, namely a domino sulfa-Michael/aldol condensation of alpha,beta-unsaturat
36 e scope of the procedure, some NHC-catalyzed sulfa-Michael/aldol organocascades were also investigate
37            A highly enantioselective cascade sulfa-Michael/Julia-Kocienski olefination reaction betwe
38          The minocycline 120 min post-sulfa (sulfa/mino+120) group had a significantly decreased ampl
39           The minocycline 30 min post-sulfa (sulfa/mino+30) group was not significantly different fro
40 -2-yl)-1,3-thiazol-2-yl]amino}pyrimidin-2-yl)sulfa nyl]octanoic acid) with IC50 = 0.3 and 0.4 muM, re
41  pneumonia (PCP) are affected by duration of sulfa or sulfone prophylaxis and influence response to s
42                    In multivariate analysis, sulfa or sulfone prophylaxis and study site were indepen
43  in 76% of isolates from patients exposed to sulfa or sulfone prophylaxis compared with 23% of isolat
44               Thus, an increased duration of sulfa or sulfone prophylaxis increases the chance of dev
45 jority of patients with mutations respond to sulfa or sulfone therapy.
46 ulfone prophylaxis and influence response to sulfa or sulfone therapy.
47 e treatment; mutations increased the risk of sulfa or sulfone treatment failure (RR, 2.1; P=0.01).
48 ht percent of patients with mutations failed sulfa or sulfone treatment; mutations increased the risk
49 ed with 4% (3/76) of patients not exposed to sulfa prophylaxis (P=.017).
50 sing frequency and have been linked to prior sulfa prophylaxis and possible emergence of sulfa resist
51 association between DHPS mutations and prior sulfa prophylaxis and shows that the prevalence of DHPS
52 bserved in 19% (6/31) of patients exposed to sulfa prophylaxis, compared with 4% (3/76) of patients n
53  regions and are similar to those that cause sulfa resistance in other organisms.
54 ne are likely involved in the development of sulfa resistance in P. carinii.
55 tent with positive directional selection for sulfa resistance mutations.
56  sulfa prophylaxis and possible emergence of sulfa resistance.
57                                          The sulfa/saline treated jjs exhibited a significantly incre
58                 The minocycline 120 min post-sulfa (sulfa/mino+120) group had a significantly decreas
59                  The minocycline 30 min post-sulfa (sulfa/mino+30) group was not significantly differ
60 ed mutations in 16 of the 37 isolates; prior sulfa/sulfone prophylaxis was associated with the presen
61              The nucleotide sequences of the sulfa target enzyme, dihydropteroate synthase (DHPS), di
62                    We present herein several sulfa-Tn antigens incorporated in MUC1 sequences that po