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1 , pendrin has been proposed to function as a sulfate transporter.
2 tion of sulfate uptake and expression of the sulfate transporters.
3 ane glycoprotein and a member of a family of sulfate transporters.
4 ough the interaction between its cytoplasmic sulfate transporter and anti-sigma (STAS) domain and the
5 tructures also show interactions between the Sulfate Transporter and Anti-Sigma factor antagonist (ST
6 ansmembrane (TM) domains and two cytoplasmic sulfate transporter and anti-sigma factor antagonist (ST
8 panning alpha-helices and a C-terminal STAS (sulfate transporters and anti-sigma-factor) domain homol
10 cal for the activity of Arabidopsis thaliana sulfate transporters, and specific lesions in this domai
11 impaired, but deletion of the adjacent STAS (sulfate transporter anti-sigma factor antagonist) domain
12 gulator (CFTR), (R)CFTR, via the Slc26-STAS (sulfate transporter anti-sigma) domain, resulting in mut
13 s of SULTR1;2, which encodes a high-affinity sulfate transporter, are defective in sulfate transport
14 conserved miRNA that targets a low-affinity sulfate transporter (AST68) and three ATP sulfurylases (
15 th treatments also caused up-regulation of a sulfate transporter, but only in the case of sulfur defi
17 e exchange of STAS domains between different sulfate transporters, can severely impair transport acti
18 ed sulfate/anion transporters that contain a sulfate transporter domain and are found in a widely dis
19 ral residue substitutions near the conserved sulfate transporter domain of prestin either greatly red
20 nsporter (encoded by DTD; 32%) and the human sulfate transporter 'downregulated in adenoma' (encoded
24 e identity), the human diastrophic dysplasia sulfate transporter (encoded by DTD; 32%) and the human
25 5'phosphosulfate reductase genes, along with sulfate transporters, especially those involved in sulfa
26 e; however, root proliferation and increased sulfate transporter expression occurred as in S-deficien
28 d functionally characterized a high-affinity sulfate transporter from the endophytic fungus Serendipi
33 he sequence analysis classified the Brassica sulfate transporter genes into four different groups.
38 SLC26A2 is the primary sodium-independent sulfate transporter in cartilage and bone and is importa
39 ion exome analysis, we identify SLC26A1 as a sulfate transporter in humans and experimentally validat
43 reased expression of genes encoding specific sulfate transporters in roots and other plant parts.
44 ing or modifying the STAS domain of dominant sulfate transporters in roots of Arabidopsis thaliana.
45 ons 260 and 288, are conserved in all of the sulfate transporters in the family whereas the NaDC cont
47 teractions between residues surrounding the "sulfate transporter motif" is essential for normal prest
48 ing invasive pulmonary aspergillosis since a sulfate transporter mutant strain and a sulfite reductas
50 pecifically, uptake of selenate (probably by sulfate transporters) occurred at a much higher rate tha
52 ls all the criteria expected of a functional sulfate transporter responding to sulfur limitation: SiS
54 logy and the presence of a slightly modified sulfate-transporter signature sequence comprising its pu
55 y analyzing the expression of genes encoding sulfate transporters (STs) of groups 1, 2, and 4 (SlST1.
59 , such as late embryogenesis abundant (LEA), sulfate transporters (SULTR), NAM-ATAF-CUC (NAC), and ni
60 d constitutive upregulation in S. pinnata of sulfate transporters SULTR1;2 (root influx) and SULTR2;1
61 icroarray studies identified a high-affinity sulfate transporter (SULTR1;2) among the most robust and
62 ion to its known function as a high-affinity sulfate transporter, SULTR1;2 may have a regulatory role
63 fate assimilation, as well as a low-affinity sulfate transporter, SULTR2;1, is strongly induced by su
64 show that SLC26a2/7 is a ventrally expressed sulfate transporter that promotes a ventral accumulation
65 in has a role in controlling the activity of sulfate transporters, their stability, or their localiza
67 ux pumps employed to remove cadmium, while a sulfate transporter was down-regulated to reduce nonspec
68 rivation, although the expression of Group 3 sulfate transporters was not affected by the sulfate sta
69 pecies, abundant gene expression of putative sulfate transporters was observed for both Se-hyperaccum
70 d the sphX gene, and transcripts of multiple sulfate transporter were also significantly more abundan
71 quisition genes encoding for a high-affinity sulfate transporter were significantly induced, while de