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1  ATP-sensitive K(+) (K(ATP)) channels is the sulfonylurea receptor.
2 inward rectifier, plus SUR2A, a low-affinity sulfonylurea receptor.
3 or family, in keeping with the presence of a sulfonylurea receptor.
4  enzymes, beta-cell-specific glucokinase and sulfonylurea receptor.
5 inwardly rectifying Kir channel (Kir6.x) and sulfonylurea receptors.
6 nnels, with SUR 1 and SUR 2, probably SUR2B, sulfonylurea receptors.
7             Recent studies demonstrated that sulfonylurea receptor 1 (SUR 1) regulated nonselective c
8 eric complexes, (SUR1/Kir6.2)(4), comprising sulfonylurea receptor 1 (SUR1 or ABCC8) and a K(+)-selec
9 sisting of octamer complexes containing four sulfonylurea receptor 1 (SUR1) and four Kir6.2 subunits.
10 otassium (K(ATP)) channel, a complex of four sulfonylurea receptor 1 (SUR1) and four potassium channe
11  ABCC8 and KCNJ11, which encode the subunits sulfonylurea receptor 1 (SUR1) and inwardly rectifying p
12 tive potassium (K(ATP)) channels composed of sulfonylurea receptor 1 (SUR1) and Kir6.2 regulate insul
13 h defects in ABCC8 and KCNJ11 genes encoding sulfonylurea receptor 1 (SUR1) and Kir6.2 subunits, whic
14 ATP-sensitive K(+) (K(ATP)) channel proteins sulfonylurea receptor 1 (SUR1) and Kir6.2, encoded by AB
15 caused by mutations in the channel proteins: sulfonylurea receptor 1 (SUR1) and Kir6.2, results in lo
16 sitive potassium (KATP) channels composed of sulfonylurea receptor 1 (SUR1) and Kir6.2.
17                       Recessive mutations of sulfonylurea receptor 1 (SUR1) and potassium inward rect
18 ensitive potassium (K(ATP)) channel subunits sulfonylurea receptor 1 (SUR1) and the inwardly rectifyi
19 tive potassium (KATP) channels consisting of sulfonylurea receptor 1 (SUR1) and the potassium channel
20 ed glucose sensitivity after deletion of the sulfonylurea receptor 1 (SUR1) both in man and mouse.
21  outward current that was antagonized by the sulfonylurea receptor 1 (SUR1) channel blocker tolbutami
22 ium (K(ATP)) channels composed of Kir6.2 and sulfonylurea receptor 1 (SUR1) couple glucose metabolism
23                                              Sulfonylurea receptor 1 (SUR1) is a molecule with more d
24 tions in ABCC8 or KCNJ11, genes encoding the sulfonylurea receptor 1 (SUR1) or the inwardly rectifyin
25 tudies using transfected COSm6 cells, mutant sulfonylurea receptor 1 (SUR1) protein was expressed on
26 ium channel and a regulatory ABC transporter sulfonylurea receptor 1 (SUR1) regulate insulin secretio
27 s to nucleotide binding fold-1 (NBF1) of the sulfonylurea receptor 1 (SUR1) subunit of the KATP chann
28 e-lining Kir6.2 subunits and four regulatory sulfonylurea receptor 1 (SUR1) subunits, control insulin
29 ctifying potassium channel (Kir6.2) and four sulfonylurea receptor 1 (SUR1) subunits.
30                             Mutations in the sulfonylurea receptor 1 (SUR1), a subunit of ATP-sensiti
31  in ischemic astrocytes that is regulated by sulfonylurea receptor 1 (SUR1), is opened by depletion o
32                                          The sulfonylurea receptor 1 (Sur1)-NC(Ca-ATP) channel plays
33 scovery of a disease-causing mutation in the sulfonylurea receptor 1 (SUR1)/ABCC8 from a patient with
34                                 Diazoxide, a sulfonylurea receptor 1 (SUR1)selective KATP-channel ope
35 racellular calcium in islets from normal and sulfonylurea receptor 1 knockout (SUR1-/-) mice.
36 ns in ABCC8 or KCNJ11, genes that encode the sulfonylurea receptor 1 or the inward rectifier Kir6.2 s
37  ATP-sensitive K(+) channel (K(ATP) channel) sulfonylurea receptor 1 subunit, and decreased inhibitor
38 ying potassium channel Kir6.2 assembles with sulfonylurea receptor 1 to form the ATP-sensitive potass
39 cases are associated with mutations in SUR1 (Sulfonylurea receptor 1) or KIR6.2 (Inward rectifier K(+
40 Kir6.2 or its associated regulatory subunit, sulfonylurea receptor 1, causes congenital hyperinsulini
41  K(ATP) channel, a functional complex of the sulfonylurea receptor 1, SUR1, and an inward rectifier p
42  cervical SCI, we tested the hypothesis that sulfonylurea receptor 1-regulated (SUR1-regulated) Ca(2+
43                                          The sulfonylurea receptor 1-transient receptor potential mel
44                                              Sulfonylurea receptor- 1 was quantified by enzyme-linked
45 inemic hypoglycemia, similar channel loss in sulfonylurea receptor-1 (SUR1) and Kir6.2 null mice yiel
46 he absence of nucleotide stimulation through sulfonylurea receptor-1 (SUR1).
47   There was no association between mean/peak sulfonylurea receptor-1 and mean/peak intracranial press
48      There was a temporal delay between peak sulfonylurea receptor-1 and peak intracranial pressure i
49  is available, assessing cerebrospinal fluid sulfonylurea receptor-1 in larger studies is warranted t
50                 Because a therapy inhibiting sulfonylurea receptor-1 is available, assessing cerebros
51                         We hypothesized that sulfonylurea receptor-1 is measurable in human cerebrosp
52                          However, decreasing sulfonylurea receptor-1 trajectories between 48 and 72 h
53                                              Sulfonylurea receptor-1 trajectories between 48 and 72 h
54                                              Sulfonylurea receptor-1 was detected in severe traumatic
55                                Mean and peak sulfonylurea receptor-1 was higher in patients with CT e
56                                         MAIN Sulfonylurea receptor-1 was present in all severe trauma
57                                              Sulfonylurea receptor-1-transient receptor potential cat
58 report quantifying human cerebrospinal fluid sulfonylurea receptor-1.
59 nes were generated and independently bred to sulfonylurea receptor 2 (SUR2) null mice to generate mic
60  ABCC9 (c.1320 + 1 G > A), which encodes the sulfonylurea receptor 2 (SUR2) subunit of K(ATP) channel
61 tations in the genes encoding the regulatory sulfonylurea receptor 2 (SUR2) subunits of the ATP-sensi
62                    The distribution of human sulfonylurea receptor-2 (SUR2)-containing K(ATP) channel
63 T-PCR) using primers specific for Kir6.2 and sulfonylurea receptor 2A (SUR2A) subunits was performed
64                                          The sulfonylurea receptor 2B (SUR2B) forms the regulatory su
65                             Sulfhydration of sulfonylurea receptor 2B (SUR2B) was induced by NaHS and
66  kidney 293 cell-attached patches expressing sulfonylurea receptor 2B and Kir6.2, we found K(ATP) sti
67 ed equivalent of this channel comprising the sulfonylurea receptor 2B and the inward rectifier 6.1 su
68                            The high-affinity sulfonylurea receptor, a novel member of the ATP-binding
69 in the second nucleotide-binding fold of the sulfonylurea receptor, a subunit of the pancreatic-islet
70 iant in the gene encoding a component of the sulfonylurea receptor (ABCC8 p.A1369S) promotes closure
71 se, two insulin-dependent diabetes loci, the sulfonylurea receptor, and ataxia telangiectasia.
72 l beta-cells, including rat insulin, amylin, sulfonylurea receptor, and glucokinase, are stably expre
73  both nucleotide-binding-fold regions of the sulfonylurea receptor are required for normal regulation
74 Superfusion of tolbutamide, a K(ATP) channel sulfonylurea receptor blocker, elicited identical glucos
75  a transgenic strategy where the full-length sulfonylurea receptor containing exon 40 was expressed u
76 osinophils do express mRNA homologous to the sulfonylurea receptor family, in keeping with the presen
77 effective after procedures that decouple the sulfonylurea receptor from the K(ATP) channel.
78  in the first nucleotide-binding fold of the sulfonylurea-receptor gene.
79 lurea 1 and 2 failed to show expression of a sulfonylurea receptor in the parietal cell, thus further
80                 Transgenic expression of the sulfonylurea receptor in vascular smooth muscle cells wa
81                        Administration of the sulfonylurea-receptor inhibitor glibenclamide promptly r
82                                          The sulfonylurea receptor is present in membranes from a num
83 o disrupt the normal splicing pathway of the sulfonylurea-receptor mRNA precursor.
84 ce probe BODIPY-FL-glyburide labeling of the sulfonylurea receptor of mitoK(ATP) from brain and liver
85 ct with ABC proteins beyond the subfamily of sulfonylurea receptors provides an intriguing explanatio
86  inwardly rectifying potassium channel and a sulfonylurea receptor regulatory subunit (SUR1 or SUR2).
87 yeast cadmium resistance transporter and the sulfonylurea receptor share a conserved topology disting
88 aining a pore-forming subunit (Kir6.1) and a sulfonylurea receptor subunit (SUR2B).
89 he potentiatory effects are conferred by the sulfonylurea receptor subunit, SUR.
90  pore-forming subunit (Kir6.2) but different sulfonylurea receptor subunits (SUR1 and SUR2A, respecti
91 r6.2 subunits complexed with four regulatory sulfonylurea receptor subunits (SUR1 in pancreatic beta-
92 ore-forming subunits (Kir6.2) and modulatory sulfonylurea receptor subunits (SUR1).
93 ed from pore-forming (Kir6.x) and regulatory sulfonylurea receptor subunits, are critical electrical
94 , an inwardly rectifying K+ channel, and the sulfonylurea receptor SUR, an ATP binding cassette prote
95 e components of KATP channels in beta-cells, sulfonylurea receptor (SUR) 1 and Kir6.2, have operation
96 he ATP-gated K(+) (K(ATP)) metabolic sensor [sulfonylurea receptor (SUR) 1 and potassium inwardly rec
97 rd rectifier potassium channel (Kir) 6.2 and sulfonylurea receptor (SUR) 1 critically regulate pancre
98            Mutations in either the Kir6.2 or sulfonylurea receptor (SUR) 1 subunit of the channel hav
99                           The ability of the sulfonylurea receptor (SUR) 1 to suppress seizures and e
100 eter regulation of wild-type [SUR2(+/+)] and sulfonylurea receptor (SUR) 2-deficient [SUR2(-/-)] mous
101        These channels are heteromultimers of sulfonylurea receptor (SUR) and KIR6.X subunits associat
102                                          The sulfonylurea receptor (Sur) and somatostatin receptor ty
103 ultimers assembled by an ion conductor and a sulfonylurea receptor (SUR) ATPase.
104                               Using putative sulfonylurea receptor (SUR) coiled-coil domains as baits
105 timeric structures formed by a member of the sulfonylurea receptor (SUR) family and a member of the i
106                                          The sulfonylurea receptor (SUR) gene, now known to encode an
107  (HBB) and markers D11S899/D11S1324 near the sulfonylurea receptor (SUR) gene.
108                                          The sulfonylurea receptor (SUR) is a member of the ATP-bindi
109                                          The sulfonylurea receptor (SUR) is another atypical ABC prot
110 second nucleotide-binding fold (NBF2) of the sulfonylurea receptor (SUR) of an individual diagnosed w
111 a member of the ATP binding cassette family (sulfonylurea receptor (SUR) or cystic fibrosis transmemb
112 e KATP channel is formed from four each of a sulfonylurea receptor (SUR) regulatory subunit and an in
113 el subunit (Kir6.1, Kir6.2) and a regulatory sulfonylurea receptor (SUR) subunit, an ATP-binding cass
114 ATP), channels are comprised of K(IR)6.x and sulfonylurea receptor (SUR) subunits that assemble as oc
115 -forming Kir6.2 subunits and four modulatory sulfonylurea receptor (SUR) subunits.
116           Compounds acting on the pancreatic sulfonylurea receptor (SUR) to increase (e.g., glibencla
117 ssion of the inward rectifier Kir6.2 and the sulfonylurea receptor (SUR), a member of the ATP-binding
118                                          The sulfonylurea receptor (SUR), an ATP-binding cassette (AB
119  transmembrane conductance regulator (CFTR), sulfonylurea receptor (SUR), and heavy metal tolerance f
120 rectifying K(+) channel 6.2 (Kir6.2) and the sulfonylurea receptor (SUR), now renamed SUR1, subunits
121 -dependent stimulatory action of the ATPase, sulfonylurea receptor (SUR), on K(IR) sufficient to elic
122                           It is comprised of sulfonylurea receptor (SUR)-1 and Kir6.2 proteins.
123               Mutations in the high-affinity sulfonylurea receptor (SUR)-1 cause one of the severe re
124 provides a template for the MDR-like core of sulfonylurea receptor (SUR)-1.
125 nsitive K+ (KATP) channel and its associated sulfonylurea receptor (SUR).
126 um channel (Kir) and an ABC transporter-like sulfonylurea receptor (SUR).
127  glybenclamide from its binding sites on the sulfonylurea receptor (SUR).
128                             ATP/ADP-sensing (sulfonylurea receptor (SUR)/K(IR)6)(4) K(ATP) channels r
129  novo L225P mutation in the L0 region of the sulfonylurea receptor (SUR)1, the regulatory subunit of
130 sized that the mitoK(ATP) channel contains a sulfonylurea receptor (SUR)2 regulatory subunit and aime
131                           They comprise four sulfonylurea receptors (SUR) and four potassium channel
132 els (K(ATP) channels) are heteromultimers of sulfonylurea receptors (SUR) and inwardly rectifying pot
133 mponent gene expression including regulatory sulfonylurea receptors (SUR) SUR1 and SUR2B but not SUR2
134 channels are heteromultimers of KIR6.2 and a sulfonylurea receptor, SUR, an ATP binding cassette (ABC
135 s tissues contain subtypes of the regulatory sulfonylurea receptor, SUR, and pore-forming, K(+) inwar
136 itive potassium (K(ATP)) channel composed of sulfonylurea receptor SUR1 and potassium channel Kir6.2
137                                          The sulfonylurea receptor SUR1 associates with Kir6.2 or Kir
138 ective opener for K(ATP) channels containing sulfonylurea receptor SUR1 subunits, but not with cromak
139                                              Sulfonylurea receptors SUR1 and SUR2 are the regulatory
140 , is composed of two types of subunits: 1) a sulfonylurea receptor (SUR1) and 2) an inwardly rectifyi
141 are an octameric assembly of two proteins, a sulfonylurea receptor (SUR1) and an ion conducting subun
142  channel, composed of the beta-cell proteins sulfonylurea receptor (SUR1) and inward-rectifying potas
143                  These channels comprise the sulfonylurea receptor (SUR1) and Kir6.2, a member of the
144  probands were screened for mutations in the sulfonylurea receptor (SUR1) gene by single-strand confo
145             Mutations in both the Kir6.2 and sulfonylurea receptor (SUR1) genes have been associated
146 l membranes indicated that the high affinity sulfonylurea receptor (SUR1) is not present on eosinophi
147 tween them and a nearby pancreatic beta-cell sulfonylurea receptor (SUR1) missense variant (S1370A),
148  complex of pore-forming Kir6.2 subunits and sulfonylurea receptor (SUR1) subunits with two nucleotid
149 m (KATP) channels, which comprise Kir6.2 and sulfonylurea receptor (SUR1) subunits.
150 r6.2) and four regulatory beta-subunits, the sulfonylurea receptor (SUR1, SUR2A or SUR2B).
151 ore-forming subunit (Kir6.1 or Kir6.2) and a sulfonylurea receptor (SUR1, SUR2A, or SUR2B).
152                            The genes for the sulfonylurea receptor (SUR1; encoded by ABCC8) and its a
153 no evidence for the presence of either known sulfonylurea receptors (SUR1 or SUR2) in the mitochondri
154 mily (Kir6.1, KCNJ8, and Kir6.2 KCNJ11) with sulfonylurea receptors (SUR1, ABCC8, and SUR2, ABCC9) of
155 l KATP (surfaceKATP) channels encoded by the sulfonylurea receptor SUR2A and the pore-forming subunit
156 rmed from pore-forming Kir6.2 and regulatory sulfonylurea receptors (SUR2A in heart and skeletal musc
157 ssium inward rectifier (Kir6.x) subunits and sulfonylurea receptors (SURs).
158 rvations and the unexpected partnership with sulfonylurea-receptors (SURs) makes the TRPM4 channel a
159 ir6.x pore-forming subunit proteins and four sulfonylurea receptor (SURx) subunit proteins and has si
160 ifier potassium (K(ir)6.x) subunits and four sulfonylurea receptor (SURx) subunits.
161 K(ATP) channels are formed as a complex of a sulfonylurea receptor (SURx), a member of the ATP-bindin
162                                              Sulfonylurea receptors (SURx) are multi-spanning transme
163                                              Sulfonylurea receptors (SURx) are required subunits of t
164                                              Sulfonylurea receptors (SURx), members of the ATP bindin
165 r pore forming (Kir6.x) and four regulatory (sulfonylurea receptor, SURx) subunits that each contain
166 d of a pore-forming protein, Kir6.x, and the sulfonylurea receptor, SURx.
167 osynthesis and were thought to interact with sulfonylurea receptors that mediate chitin vesicle trans
168 rough association of the Kir6.2 pore and the sulfonylurea receptor, the stress-responsive ATP-sensiti
169 ck by adenine nucleotides similar to that of sulfonylurea receptor type 1 (SUR1).
170 ; however, the evidence is strong that SUR1 (sulfonylurea receptor type 1) subunits are also expresse
171           K(ATP) channels, (SUR1/Kir6.2)(4) (sulfonylurea receptor type 1/potassium inward rectifier
172 -rectifier potassium channel 6.2) and SUR2A (sulfonylurea receptor type 2A) subunits; however, the ev
173                                     The gene sulfonylurea receptor was eliminated as a candidate gene
174 ic ATP-binding cassette regulatory subunits (sulfonylurea receptors), which counterbalance the nearly
175      The gene responsible (SUR1) encodes the sulfonylurea receptor, which maps to chromosome 11p15.1.

 
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