ライフサイエンスコーパス: superficial

1 our understanding of CSF leukocytes remains superficial.

2 ss lead to neurological disease remains very superficial.

3 oral tongue tumors received the traditional superficial 3 or 4 deposits of ICG-(99m)Tc-nanocolloid (

4 l injury with MW, RF, or cautery to create a superficial 3-mm lesion immediately after intrasplenic i

5 ulturing them around Matrigel beads allowing superficial access to the apical membrane and making the

6 show that each skin lesion of disseminated superficial actinic porokeratosis originates from a post

7 groups, receiving EA in the treatment group, superficial acupuncture at sham points in the control gr

8 increased the interplay between the cmA and superficial amygdala.

9 alence of microvascular abnormalities in the superficial and deep capillary bed with worse retinopath

10 to the surrounding retina, were selected in superficial and deep capillary plexus (SCP and DCP).

11 m(2) and 6 x 6-mm(2) angioscans for both the superficial and deep capillary plexuses (SCP and DCP, re

12 of the overlying skin has similar effects on superficial and deep cells: lamellipodia are lost, blebs

13 overt motor actions led to activity in both superficial and deep cortical layers, motor imagery enga

14 nduced significantly greater activity in the superficial and deep laminae than the innocuous control

15 on and of behavioral state by neurons within superficial and deep layers of area V4 in monkeys while

16 ilar to ocular dominance (OD) columns within superficial and deep layers of the primary visual cortex

17 Circuits in superficial and deep layers play distinct roles in corti

18 asciculation in ALS patients, involving both superficial and deep muscles, and at a frequency higher

19 nt decrease in mean signal index in both the superficial and deep plexus and binarized flow index in

20 ees, particularly for discriminating between superficial and deep second degree burns, which is chall

21 surgery were followed for the occurrence of superficial and deep SSI as defined by the Center of Dis

22 age, macular OCT-A shows less involvement of superficial and deep vascular plexus in NAION in contras

23 knesses were measured in addition to macular superficial and deep vasculature after projection remova

24 zed for foveal avascular zone (FAZ) area and superficial and deep vessel density.

25 epsis, acute kidney injury, wound infection (superficial and deep), rate of intraoperative need for v

26 Gram-positive bacterium that can cause both superficial and deep-seated infections.

27 At 1 year after surgery, patients with superficial and deep/organ space SSIs incurred higher he

28 ted that at 1-year following the index date, superficial and deep/organ space SSIs were significantly

29 ndogenous expression of Tacr1 throughout the superficial and deeper dorsal horn (DDH), as well as the

30 olarization of lamellipodia extended by both superficial and deeper protoneuromast-forming cells depe

31 The majority of subclones (29/46, 60%) share superficial and invasive phenotypes.

32 ne improved albuminuria, podocyte density in superficial and juxtamedullary nephrons, and podocyte fi

33 x, as indicated by stronger BOLD decrease in superficial and middle than deeper layers for gratings t

34 Fungal diseases range from relatively-minor superficial and mucosal infections to severe, life-threa

35 gistic regressions examined risk factors for superficial and severe hospital-acquired pressure injuri

36 therapy (EBT) is commonly used for treating superficial and subdermal tumors.

37 Prevalence of all, superficial, and severe hospital-acquired pressure injur

38 activation across cell types and layers; (b) superficial anodal stimulation is more effective than ca

39 novel data on how fiber architecture of the superficial anterior temporalis (SAT) varies dynamically

40 s to detect and count cells flowing in large superficial arteries and veins without drawing blood sam

41 Superficial biopsies of the right periorbital region wer

42 recharged by 400-nm photoexcitation light in superficial blood vessels during circulation, and turned

43 A decline in superficial, but not severe, hospital-acquired pressure

44 f the results, validated the measurements in superficial capillaries with known perfusion pathways, a

45 between sleep scores and vessel densities in superficial capillary plexus (P = .04).

46 one (FAZ) area and the vessel density of the superficial capillary plexus (SCP) and deep capillary pl

47 Retinal thickness, superficial capillary plexus (SCP) and deep capillary pl

48 gth density (VD) were examined from both the superficial capillary plexus (SCP) and deep capillary pl

49 The superficial capillary plexus (SCP) and the deep capillar

50 compare the retinal microvasculature in the superficial capillary plexus (SCP) in Alzheimer's diseas

51 En face OCTA images corresponding to the superficial capillary plexus (SCP), deep capillary plexu

52 Macular perfusion density in 3 plexuses [superficial capillary plexus (SCP), deep capillary plexu

53 measure retinal vessel density (VD) from the superficial capillary plexus in the foveal, parafoveal,

54 Superficial capillary plexus perfusion density was reduc

55 P = .011) and vessel densities (P = .002) in superficial capillary plexus was noted.

56 ffered saline)-pretreated mice, nonapoptotic superficial cell death from 2 to 6 hours and apoptosis i

57 Despite superficial cell loss, KGF suppressed intermediate and b

58 The proportion of anuclear and superficial cells increased and parabasal decreased (P <

59 cortical pyramidal cells and show that more superficial cells receive stronger synaptic inhibition o

60 Superficial cells, which are critical for maintaining th

61 ontrols, KGF-pretreated mice had regenerated superficial cells.

62 ransport as well as drainage to the deep and superficial cervical lymph nodes.

63 of this study was to investigate if a Flex-M superficial coil images would provide more information t

64 Compared to the superficial collicular layers, neuronal responses in the

65 tinal peptide-immunoreactive fibers was more superficial compared to the basal assessment and control

66 ities ("competence"), but instead because of superficial constraints on demonstrating that knowledge

67 he Lower Triassic of Hubei, China, revealing superficial convergence with the modern duckbilled platy

68 and open wounds of ocular adnexa, diplopia, superficial corneal and/or conjunctival injuries, and or

69 more strongly with glomerular volume in the superficial cortex.

70 of late components substantially reduces the superficial cortical depth bias of fMRI responses and he

71 d if this part of the volley is sensitive to superficial cortical excitability.

72 ment, imagined movements evoked responses in superficial cortical layers only.

73 cDCS reliably induced DCS-LTD in superficial cortical layers, and a long-term potentiatio

74 lvinar projections to area 17 was largely in superficial cortical layers, especially layer 1.

75 derin deposits in the subarachnoid space and superficial cortical layers, indicative of chronic bleed

76 corresponds to iron-positive deposits in the superficial cortical layers, representing the chronic ma

77 rges within modules, which in ASD implicates superficial cortical neurons.

78 months, first in frontal leptomeningeal and superficial cortical vessels followed by vessels penetra

79 we report that in macaque monkeys, deep and superficial cortical white matter neurons (WMNs), peri-c

80 arkers within the parasubiculum, across both superficial-deep and DV axes.

81 the donor site (whether anterior-, lateral-, superficial-, deep-palate or the maxillary tuberosity) c

82 l intelligence, so that we can separate more superficial differences from those that may be deep and

83 uantify transverse muscle oscillations (deep-superficial displacement of the muscle boundary relative

84 cs of dendritic spines over time on the same superficial dorsal horn (lamina II) neurons before and a

85 pressed within a subpopulation of excitatory superficial dorsal horn (SDH) neurons.

86 tory synaptic transmission within the spinal superficial dorsal horn (SDH) that include a reduction i

87 presumed glutamatergic neurons in the mature superficial dorsal horn (SDH), and modifies activity-dep

88 nges in synaptic signaling across the mature superficial dorsal horn (SDH), remains unknown.

89 idence that spinoparabrachial neurons in the superficial dorsal horn contribute to persistent pain st

90 pressing neurons in primary afferents and in superficial dorsal horn neurons, there is little to no i

91 distributed among excitatory interneurons in superficial dorsal horn.

92 y but not inhibitory interneurons in the rat superficial dorsal horn.

93 citatory neurons located in lamina II of the superficial dorsal horn.

94 ferent fibres regulate afferent input to the superficial dorsal horn.

95 tal study of dendritic spine dynamics in the superficial dorsal horn; (2) that nerve injury-induced p

96 al layer (K14-Dsg2/Ptc1(+/lacZ) mice) or the superficial epidermis (Inv-Dsg2/Ptc1(+/lacZ) mice) resul

97 dings with extensive axonal branching in the superficial epidermis and large receptive fields.

98 plaque rupture probably does not pertain to superficial erosion, a process heretofore little underst

99 earch should probe further the mechanisms of superficial erosion, and develop point-of-care tests to

100 tiple processes likely predispose plaques to superficial erosion, including experiencing disturbed fl

101 tes likely promote endothelial damage during superficial erosion.

102 dendrites via their lateral dendrites in the superficial external plexiform layer (EPL); (2) axodendr

103 ere as follows: inferior gluteal artery (2), superficial external pudendal artery (2), deep femoral a

104 e are "ms-type" secondaries, which innervate superficial fast-twitch and slow fibers via medial and s

105 ed consecutive participants with symptomatic superficial femoral and/or popliteal artery disease at 1

106 ard Balloon Angioplasty for the Treatment of Superficial Femoral Artery [SFA] and Proximal Popliteal

107 Treatment of Atherosclerotic Lesions in the Superficial Femoral Artery [SFA] and/or Proximal Poplite

108 Treatment of Atherosclerotic Lesions in the Superficial Femoral Artery and/or Proximal Popliteal Art

109 Treatment of Atherosclerotic Lesions in the Superficial Femoral Artery and/or Proximal Popliteal Art

110 the use of paclitaxel-coated devices in the superficial femoral artery.

111 N=8376) undergoing endovascular treatment of superficial femoral-popliteal artery disease in the Soci

112 To compare mortality after treatment of superficial femoral-popliteal artery disease with paclit

113 o better understand how experience with even superficial forms of democratic institutions across a di

114 Superficial GEM-PNST contained regions of nerve-associat

115 lutamatergic neurons that integrate into the superficial glomerular layer.

116 Our previous works have shown that superficial gross anatomical analyses of these specimens

117 ith a p53 mutation was sufficient to promote superficial growth melanomas, whereas BRAF(V600E) accele

118 icular cells induced or reduced responses in superficial gRSC, respectively.

119 ces could be detected in large quantities in superficial hair structures.

120 analyses showed a trend toward lower risk of superficial incisional SSI in the postimplementation per

121 d, indicating their impact is not limited to superficial infections.

122 injected cases were significantly lower when superficial injection was chosen first (P < 0.001).

123 though the best results were achieved when a superficial injection was chosen.

124 genation status of ganglion cells within the superficial inner retina, whether this is normoxic (OHM1

125 The superficial interneurons, SINs, of the zebrafish tectum,

126 Superficial (intradermal or periareolar) or deep (peritu

127 By genetically converting superficial IT pyramidal cells into PT-like deep-layer p

128 g enzymes, removed cellulose microfibrils in superficial lamellae sequentially, layer-by-layer, and s

129 embryos are embedded in Matrigel, basal and superficial lamellipodia are recovered; however, only th

130 that only a minority of neurons in the most superficial lamina of the SC display significant changes

131 wake, running vs stationary), SC depth (most superficial lamina vs deeper in the SC), research techni

132 ergic and IB4-binding C fiber populations in superficial laminae of the thoracic dorsal horn.

133 ereas noxious vFHs (26 g) also activated the superficial laminae.

134 target deep layers, late born ones innervate superficial laminae.

135 ized movements and interdigitate into a more superficial layer [6, 7].

136 c interlaminar astrocytes are located in the superficial layer and project long processes traversing

137 A), and 3x3-mm(2) en face OCTA images of the superficial layer and the deep layer were obtained at ba

138 sual improvement, outer parafoveal VD in the superficial layer at the baseline showed the largest are

139 calize with pericellular type VI collagen in superficial layer cells in the MCC perichondrium but is

140 or was altered, leading to abnormal deep and superficial layer neuron generation.

141 mammalian cerebral cortex generates deep and superficial layer neurons progressively.

142 ynaptic inputs on dendrites of individual V1 superficial layer neurons with high spatial and temporal

143 linking and stabilization of the lubricating superficial layer of cartilage by mediating interaction

144 esigned to treat RSV which replicates in the superficial layer of epithelial cells lining the airways

145 l association cortex, parasubiculum, and the superficial layer of medial entorhinal cortex.

146 bundant cell type in the epidermis, the most superficial layer of skin.

147 at fed forward up the cortical hierarchy via superficial-layer cortex.

148 ally generate deep-layer neurons followed by superficial-layer neurons directly or via the generation

149 of genetically defined neuron populations in superficial layers (L) of mouse primary visual cortex (V

150 on deep-layer neurons providing feedback to superficial layers (not to deep layers), suggesting that

151 d movements showed a reduced response in the superficial layers (repetition suppression) coupled with

152 ulation of layer 5; after 0.6 ms, stimuli to superficial layers 2/3 could also contribute.

153 the relative microbial abundance between the superficial layers and the deep layers.

154 at follow the hypothesized patterns: namely, superficial layers are preferentially active during the

155 ticipate in different inhibitory networks in superficial layers by targeting either parvalbumin (PV+)

156 ngle neurons and neuronal populations in the superficial layers conveyed more information about the o

157 deed, the oSVZ initially adds neurons to the superficial layers II and III, increasing their thicknes

158 ask-related variables are represented in the superficial layers of GC of alert, licking mice.

159 l and inhibitory neurons located in deep and superficial layers of GC.

160 and inhibitory neurons belonging to deep and superficial layers of GC.

161 tapping) evoked neural responses in both the superficial layers of M1 that receive cortical input and

162 We recorded ensembles of grid cells in superficial layers of medial entorhinal cortex during ac

163 we expressed GCaMP6f in Lm128C cells in the superficial layers of the motor cortex and performed in

164 eduction in perfusion was more pronounced in superficial layers of the peripapillary retina (NFLP and

165 sual information from the retina through the superficial layers of the superior colliculus and the pu

166 o-photon Ca(2+) imaging of dLGN afferents in superficial layers of V1 in female and male mice, we dem

167 lasticity of excitatory synapses observed in superficial layers of visual cortex is dependent on NMDA

168 isting modalities has been limited to either superficial layers or early developmental stages due to

169 rs of V1, acetylcholine is delivered to more superficial layers through volume transmission.

170 ntage of taste-coding neurons in deep versus superficial layers with chemosensitive neurons across al

171 middle cortical layer and weaker in deep and superficial layers, while top-down modulations were stro

172 the disinhibitory calretinin neurons in the superficial layers.

173 g specific inhibitory neurons in layer 1 and superficial layers.

174 e top-down modulations were strongest in the superficial layers.

175 om the deeper, proliferative zone toward the superficial layers.

176 cortical layers, motor imagery engaged only superficial layers.

177 es but is confined to the treatment of small superficial lesions.

178 Given the superficial localization of itch neuron terminals, cells

179 er proportion of CD4+CCR5+ cells with a more superficial location.

180 ompacta and ventral tegmental area homologs, superficial mamillary area, laterodorsal tegmental nucle

181 habenula pathway derives from neurons in the superficial mamillary area, which in terms of its connec

182 rsity across the mouse cortex, we identified superficial, mid and deep astrocyte identities in gradie

183 The superficial, middle, and deep regions showed significant

184 ey function, and structural pathology in the superficial, middle, and deep regions.

185 p molecular layer and overelaboration in the superficial molecular layer.

186 primarily driven by increased production of superficial neurons in the dramatically enlarged outer s

187 the direction of planned eye movements than superficial neurons.

188 we discovered FCSCs localized within the TMJ superficial niche exhibit Notch activity during TMJ morp

189 ary by lesion position (interaction P = .02; superficial, odds ratio [OR] = 17.7 [95% CI: 1.50, 207],

190 The primary outcome was superficial or deep surgical-site infection according to

191 Superficial or deep surgical-site infection was diagnose

192 capacities, vs. when such failures are only superficial or peripheral?

193 ates an increase in processing complexity in superficial PAC, which remains present throughout cortic

194 the thalamo-recipient middle PAC layers and superficial PAC.

195 as not associated with incompleteness of the superficial palmar arch ( P=0.13).

196 The more superficial part of the canal forms from an open primary

197 ice participated in enlarged ensembles, with superficial PCs (sPCs) having a higher probability of sp

198 ateral malleolus to identify and protect the superficial peroneal nerve (SPN).

199 Two nerve injuries of the superficial peroneal nerve were reported.

200 ant increase in FAZ major axis length in the superficial plexus and a significant decrease in binariz

201 e flow density in the inferior sector of the superficial plexus in the macular region was also signif

202 deep plexus and binarized flow index in the superficial plexus were found with increasing duration o

203 Retinal plexus flow deficit (superficial plexus: 0.10+/-0.12 mm(2); deep plexus: 0.29

204 is the first case reporting the findings of superficial "polka dots" pattern iridian atrophy in 360

205 We describe a superficial population of flat primordium cells that wra

206 with excellent recurrence-free survival for superficial premalignant, minimally invasive, and small

207 se observations support a key role played by superficial primordium cells and the skin in directed mi

208 ters orientation-specific representations in superficial rather than middle or deeper V1 layers, cons

209 crease in glomerulosclerosis with age in the superficial region, but larger glomerular volume was not

210 osis and ischemic-appearing glomeruli in the superficial region.

211 ns about immune memory responses in an acute superficial respiratory mucosal infection and their impl

212 En face images of the superficial retinal layer (SRL) and deep retinal layer w

213 ed with controls in the nonsegmented retina, superficial retinal layer (SRL), and deep retinal layer

214 also demonstrated delayed advancement of the superficial retinal vascular layer and aberrant vascular

215 Peripapillary superficial retinal vessel densities were significantly

216 The area of the foveal avascular zone in the superficial retinal vessel layer increased significantly

217 the size of the foveal avascular zone in the superficial retinal vessel layer, more pronounced in gro

218 Mechanistically, superficial revascularization is guided by epicardial Cx

219 % CH was suggested for delaying ripening and superficial scald of 'Bartlett' pears during the long-te

220 The superficial scald on pear peels was only observed on fru

221 nside a linear scleral tunnel underneath the superficial scleral flap.

222 erms of visualization at the level either of superficial (SCP) or deep (DCP) capillary plexus and the

223 zone (FAZ) diameter, and vessel densities of superficial (SCP-VD) and deep capillary plexuses (DCP-VD

224 Cortical superficial siderosis appears to be the result of predom

225 Histopathologically, cortical superficial siderosis corresponded to iron-positive haem

226 Cortical superficial siderosis is an established haemorrhagic neu

227 In fact, cortical superficial siderosis is emerging as a strong independen

228 llectively, these data suggest that cortical superficial siderosis on MRI corresponds to iron-positiv

229 nd pathophysiological mechanisms of cortical superficial siderosis remain elusive.

230 Next, cortical superficial siderosis was assessed on a total of 65 Perl

231 Cortical superficial siderosis was assessed on ex vivo gradient e

232 Moderate-to-severe cortical superficial siderosis was associated with concentric spl

233 y tissue injury, moderate-to-severe cortical superficial siderosis was associated with the presence o

234 Increased severity of cortical superficial siderosis was associated with upregulation o

235 Cortical superficial siderosis was present on ex vivo MRI in 8/14

236 s and vascular pathology underlying cortical superficial siderosis.

237 logical verification of MRI-defined cortical superficial siderosis.

238 Given the striking superficial similarities of these scattering features, t

239 t for the circular visceral muscles, despite superficial similarities, a significantly different spec

240 Despite superficial similarities, the signalling cascade underly

241 These Staphylococcus infections range from superficial skin infections to deep-seated invasive infe

242 "s-type" units, which exclusively innervate superficial slow muscle fibers via septal nerves.

243 The RNFL thickness maps were separated into superficial (SNFL) and deep (DNFL) slabs through a parti

244 ctive non-invasive tool for the diagnosis of superficial soft tissue lesions and may negate the need

245 Multiple superficial soft tissue lesions were studied, the majori

246 lecularly characterized neurons in the mouse superficial spinal cord dorsal horn that express estroge

247 In conventional melanoma, superficial spreading and nodular melanoma account for m

248 adipose tissue compartment volumes-abdominal superficial (sSAT), deep subcutaneous (dSAT), and intern

249 creased, there was a significant decrease in superficial SSI rates (lowest adherence quintile, 4.6% v

250 Lower superficial SSI rates are associated with the number of

251 Superficial SSIs were 20.7% (30/145 patients) in the pla

252 inatorial system may have been preceded by a superficial stage where signalers neither needed to be c

253 dritic outgrowth of pyramidal neurons to the superficial strata of the hippocampus.

254 located in periventricular, intermediate and superficial strata.

255 The light-driven superficial structuration observed on the surface of fil

256 flammatory perivascular injury and injury to superficial structures, including the subpial region of

257 pus from L2 of LEC arise from fan cells in a superficial sub-layer (L2a) that are immunoreactive for

258 , separately characterizing glomeruli in the superficial (subcapsular), middle, and deep (juxtamedull

259 We found that CA1 place cells located in the superficial sublayer were more active in cue-poor enviro

260 o determine the specific circuits within the superficial superior colliculus (sSC) that drive orienti

261 mellar vesicles, Cyt1Aa's insertion was more superficial, supporting the notion that a detergent effe

262 While superficial surgical site infection following SCS implan

263 rein deep synapses are potentiated more than superficial synapses.

264 ial nucleus (PBN) show that less than 20% of superficial Tacr1(CreER) dorsal horn neurons are spinal

265 xed through decoding accuracy-proceeded from superficial task cues, to stimulus locations, to feature

266 Burn injury therefore exemplifies a superficial temporally dynamic pathology for which exper

267 level in the range of complex light-induced superficial textures accessible onto azopolymer film sur

268 ic detection or excised SLNs was higher with superficial than deep injections.

269 anoparticle-doped fibers allowed an accurate superficial thermal map detected in real-time.

270 well recognized clinically in the context of superficial thrombophlebitis (thrombosis and inflammatio

271 aseline prevalence of recurrent DVT was 38%; superficial thrombophlebitis was diagnosed in 4.6%.

272 protocols resulted in a lower percentage of superficial Ti when compared with S/R-nC (P <0.002).

273 aoperative fluorescence for visualization of superficial tissue layers for image-guided surgery.

274 RSA) is a major human pathogen, which causes superficial to lethal clinical infections.

275 GAS) causes a wide range of infections, from superficial to life-threatening diseases, upon dissemina

276 cies cause infections in humans ranging from superficial to life-threatening.

277 rs allowed us to access mitral cell (MC) and superficial tufted cell (sTC) subpopulations separately.

278 s, are classified as non-muscle-invasive or 'superficial' tumours.

279 ing and normalization after ~4 months) and a superficial ulcerative lesion in a control group patient

280 ation: type 1: erythema; type 2: ulcers (2a: superficial ulcers; 2b: deep ulcers); type 3: perforatio

281 ciated with metastasis , but we still have a superficial understanding of how it affects cellular pro

282 s (NFLP), ganglion cell layer plexus (GCLP), superficial vascular complex (SVC [NFLP + GCLP]), deep v

283 OCT angiography images of the superficial vascular complex (SVC) and deep vascular com

284 ities were calculated on projection-resolved superficial vascular complex (SVC), intermediate capilla

285 e central 1-mm circle in projection-resolved superficial vascular complex (SVC), intermediate capilla

286 P) were primarily damaged by RP, compared to superficial vascular complex (SVC).

287 plexus capillary density (NFLP_CD), macular superficial vascular complex vessel density (mSVC_VD), a

288 ious injection number, microaneurysms in the superficial vascular complex, and microaneurysms in the

289 To describe patterns of reperfusion in the superficial vascular plexus (SVP), deep capillary plexus

290 to the resolved PAMM lesions, vessels in the superficial vascular plexus were traced to identify smal

291 ic nerve region in addition to peripapillary superficial vasculature.

292 onary embolism (I26) or any code for deep or superficial vein thrombosis was listed as the primary ca

293 mbophlebitis (thrombosis and inflammation of superficial veins); however, it is more dangerous when i

294 ematous and showed the presence of prominent superficial veins.

295 ably occupy tissues and cannot be sampled in superficial venous blood.

296 erences between risk factors associated with superficial versus severe hospital-acquired pressure inj

297 Whole image macular superficial vessel density was significantly lower in NA

298 ibution of retrogradely labeled cells in the superficial, visual layers of SC is consistent with the

299 He presented with multiple areas of superficial white circular spots of iridian atrophy in 3

300 lls (FCSCs) localized within the TMJ condyle superficial zone niche that regenerate cartilage and rep