ライフサイエンスコーパス: superfusion

1 (P < 0.01 vs. glucose alone after 90 min of superfusion).

2 dominant regulator of surface pH under pH 5 superfusion.

3 ulation and elicited inward currents by NMDA superfusion.

4 developed after prolonged (more than 45 min) superfusion.

5 d neurotoxicity were examined using in vitro superfusion.

6 nule; P < 0.01 vs. control) after 120 min of superfusion.

7 e out of the vasculature in response to fMLF superfusion.

8 t occurred 2 min after onset of arachidonate superfusion.

9 ase when they were both included in the same superfusion.

10 ce imaging of [H(+)] dynamics in cells under superfusion.

11 qually during acute and chronic moxifloxacin superfusion.

12 additional prolongation occurred on chronic superfusion.

13 d intact neural mouse retinae, maintained by superfusion.

14 ed a marked fade during the 10-min period of superfusion.

15 ma xenograft in cranial window, whereas VEGF superfusion (10-1000 ng/ml) failed to increase permeabil

16 VEGF superfusion (100 ng/ml) significantly increased vascular

17 Furthermore, VEGF superfusion (1000 ng/ml) increased vascular permeability

18 y both in vivo electrochemistry and in vitro superfusion: (2) similar, albeit attenuated effects are

19 K(ATP) channel inhibition via glibenclamide superfusion (5 mg kg(-1) GLI; sulphonylurea diabetes med

20 With either pH 5 or pH 3 superfusion (a) gastric pit lumens had the most divergen

21 Ethanol superfusion also decreased paired-pulse facilitation of

22 Focal superfusion altered both the extent and the direction of

23 isation-induced release of [125I]BDNF during superfusion and batch protocol experiments.

24 Superfusion and electrical field stimulation of rat hipp

25 tal differences between in vitro brain slice superfusion and in vivo brain perfusion.

26 In vitro superfusion and in vivo electrochemistry were used to in

27 that the gastric mill rhythms elicited by PK superfusion and MCN1 stimulation in the isolated STG are

28 0, 3, 30 or 300 nM) estradiol throughout the superfusion and subsequently given a dopamine (1 microM)

29 stigated using both the Ussing chamber and a superfusion apparatus.

30 A small-volume superfusion assay and high-pressure liquid chromatograph

31 Using a small-volume superfusion assay and HPLC with fluorescence detection a

32 In a superfusion assay of DA efflux, cells overexpressing SYN

33 ed, rabbit neural retinas were maintained by superfusion at different times of the regular light/dark

34 +) channel blocker BaCl(2) is present in the superfusion bath.

35 elaxing factor and a neurotransmitter with a superfusion bioassay cascade technique.

36 These results show that (1) in vitro superfusion can be used to dynamically evaluate dopamine

37 Dopamine applied by superfusion caused a slow monophasic hyperpolarisation i

38 at specified times transferred to an acrylic superfusion chamber designed to allow controlled flow of

39 g video imaging with Fura-2 in a 37 degreesC superfusion chamber.

40 rescent Ca2+ indicator fura-2 and mounted in superfusion chambers for fluorometric measurement of [Ca

41 Dopamine output from superfusion chambers which received infusion of MPP(+) w

42 tion to shortening, depending on duration of superfusion, concentration, and CL.

43 red during a 30-s stimulation under constant superfusion conditions.

44 e circadian phenomenon is independent of the superfusion conditions.

45 Bioassay superfusion demonstrated a twofold augmentation of the b

46 iatal spiny projection neurons and histamine superfusion demonstrates expression of functional histam

47 Moreover, kisspeptin superfusion did not affect the frequency or amplitude of

48 Solution superfusion did not produce any obvious changes in cell

49 f-actin staining revealed that TNF alpha superfusion disrupted f-actin filaments when compared to

50 Short time-fraction (30 s) superfusion experiments showed that the [125I]BDNF relea

51 For in vitro superfusion experiments, dopamine and dihydroxyphenylace

52 milar results were observed with and without superfusion flow; (c) local mucus gel thickness was a po

53 Adding exogenous BH4 directly to the superfusion fluid leads to a massive increase in [3H] do

54 ent-clamp recordings, SP (100 nM) applied by superfusion hyperpolarized the membrane potential (7 +/-

55 larizations and caffeine applications during superfusion in Ca(2+)-free, Sr(2+)-containing solutions

56 pplying voltage clamp depolarizations during superfusion in Na(+)-free, Sr(2+)-containing solutions.

57 on of individual capillaries in EHP fixed by superfusion in situ revealed thickening of capillary end

58 Extended superfusion led to a local, apparent increase in light t

59 pattern of SP release was also observed when superfusion medium containing CO2-HCO3 buffer, pH 7.4, w

60 [Me-14C]Ch placed in the superfusion medium for 30 min during a 1-h stimulation p

61 Omission of calcium in the superfusion medium markedly attenuated hypoxia-induced S

62 ntal pulp was prepared and stimulated by the superfusion method with BK alone and in combination with

63 receptor in liver was analyzed using a novel superfusion method.

64 t up for studies of [3H]NE release using the superfusion method.

65 own on Cytodex-3 beads and placed in special superfusion microchambers.

66 Brief superfusion of 11-66 mM ethanol significantly increased

67 Superfusion of 2-methylthio-adenosine-5'-triphosphate (2

68 ression to produce axonal injury, unilateral superfusion of 3 microM TTX into the rat supraoptic nucl

69 Superfusion of 4-AP (0.8 mM) reversibly depolarized a lo

70 Superfusion of 4-AP (50 microM) induced two types of act

71 Acute superfusion of 44 mM ethanol increased the amplitude of

72 Cs(+)-based intracellular solution or during superfusion of 5 mm TEA, suggesting the presence of an a

73 Superfusion of 5-66 mM ethanol decreased compound glutam

74 Superfusion of 5-HI (100 microM) increased the mean freq

75 ed cardiomyocyte function improved after the superfusion of a PKCbeta selective inhibitor.

76 An increase in [H(+)]i, produced by superfusion of acetate (salt of membrane-permeant weak a

77 ponsiveness to cumulative (10(-10)-10(-4) M) superfusion of acetylcholine or phenylephrine.

78 arinic acetylcholine receptors), attained by superfusion of agonists or weak, sustained (approximatel

79 h aspartate iontophoresis were suppressed by superfusion of agonists.

80 Superfusion of ANCA IgG over rolling cells resulted in c

81 In vitro superfusion of bovine dental pulp was used to evaluate t

82 n-sensitive nociceptive neurons via in vitro superfusion of bovine dental pulp.

83 Superfusion of brain slices with PrRP (100-300 nm) produ

84 blocked by 30 nm IbTx but was unaffected by superfusion of Ca(2+)-free solution, nifedipine or Bay K

85 response to photorelease of caged IP(3) and superfusion of caffeine, respectively.

86 The superfusion of CeA slices with nociceptin/orphanin FQ pe

87 nsport and NO release were unaffected during superfusion of cells with a nominally Ca(2+)-free soluti

88 However, in the same ganglia, after superfusion of CO in the presence of ZnPP, tetanic stimu

89 ced L-glutathione mono-ethyl ester (GSH-MEE) superfusion of conjunctival tissues pre-exposed to mucos

90 ing HPLC with electrochemical detection from superfusion of corpus striatum fragments with Kreb's rin

91 Superfusion of CORT decreases evoked inhibitory postsyna

92 larizing current injections before and after superfusion of cPGI (1 microM and 10 microM).

93 Superfusion of diazoxide inhibited fast synaptic activit

94 e amplitude of depolarization in response to superfusion of different tachykinin agonists (neurokinin

95 n slice preparation for cellular recordings, superfusion of DynA onto pPVT neurons decreased the freq

96 This current was still present during superfusion of either IbTx (60 nm) or apamin (300 nm) bu

97 This effect was reversed with superfusion of excess L-arginine (1 mM).

98 Continuous superfusion of fMLP was also essential for maintenance o

99 Superfusion of GABA resulted in a concentration-dependen

100 In naive animals, superfusion of ghrelin increased the amplitude of evoked

101 tes excluded by the wall was very different; superfusion of growing hyphae with PEG-6000 or dextran-6

102 fect the propagation of SD induced by global superfusion of high [K(+)](e) containing artificial CSF

103 Superfusion of hippocampal slices isolated from rats chr

104 By contrast, kisspeptin superfusion of hypothalamic slices did not affect the ac

105 Superfusion of IL-1beta-stimulated rabbit mesentery with

106 Rapid superfusion of immobilized Ca2+- and calmodulin-dependen

107 Four rats received a localized superfusion of L-NAME (1 mmol/L) through the cranial win

108 al labyrinth in compensating animals through superfusion of local anaesthetic on the round window.

109 plied 5-HT was reproducibly inhibited by the superfusion of low concentrations of 5-HT which evoked l

110 In addition, superfusion of low micromolar concentrations of glutamat

111 on of mesenteric microvessels in response to superfusion of methacholine and bradykinin.

112 Superfusion of myocytes at 37 degrees C with Ca2+-free s

113 Superfusion of neurones with zero calcium (1 mM EGTA) Ri

114 Superfusion of NO donor (spermine NO, 100 mumol/L) incre

115 Superfusion of Noc robustly depressed spontaneous bursti

116 Superfusion of normoxic cells with H2O2 (25 microM) for

117 stsynaptic potential (IPSP) was abolished by superfusion of omega-conotoxin (omega-CTX) GVIA (3-300 n

118 Superfusion of omega-CTX MVIIC (500 nM) also suppressed

119 Superfusion of orexin A or B (10-300 nM) caused a slow d

120 Superfusion of other cyclic nucleotide analogues (8-Br-c

121 Superfusion of PASMCs or SASMCs with hypoxic media incre

122 Superfusion of PASMCs with hypoxic media increases the H

123 During superfusion of permeabilized hepatocytes, submaximal con

124 We demonstrated that superfusion of rat mesentery with LPS resulted in signif

125 and VIP, we developed assay systems based on superfusion of rat pituitary and pineal cells.

126 tric oxide was examined by using a method of superfusion of rat striatum slices in vitro.

127 Superfusion of S1P (0.001-10 muM) evoked concentration-d

128 Superfusion of SKCa and IKCa channel blockers UCL 1684 +

129 Moreover, superfusion of spinal slices from opiate-tolerant rats w

130 In the absence of A1R agonists, superfusion of the A1R antagonist, 8-cyclopentyl-1,3-dip

131 laced in a chamber that allowed for separate superfusion of the brainstem, spinal segments C(2)-C(4),

132 Superfusion of the cannabimimetics WIN55212-2 or methana

133 over a surface coated with platelets, until superfusion of the chemotactic peptide formyl-methionyl-

134 Superfusion of the GABAA agonist muscimol resulted in a

135 Superfusion of the GHS-R1A antagonists D-Lys3-GHRP-6 and

136 s that was reduced in amplitude by prolonged superfusion of the IMG with either PACAP27, maxidilan, P

137 Bath superfusion of the kappa-opioid agonists U69,593 (0.3-1

138 Superfusion of the lymphotoxin-stimulated neovasculature

139 mmatory action of D-glucose was inhibited by superfusion of the mesentery with 30 nmol/l bisindolylma

140 Superfusion of the muscle with NMA partially restored ar

141 in septic and control rats, before and after superfusion of the muscle with the nitric oxide synthase

142 Superfusion of the NK1R agonist (2 microM) elicited a pr

143 Topical superfusion of the nonselective NOS inhibitor N(G)-monom

144 Concomitant superfusion of the rat mesentery with 10 nmol/liter of e

145 Superfusion of the rat mesentery with 50 micromol/liter

146 Superfusion of the rat mesentery with either 0.5 U/ml th

147 Superfusion of the rat mesentery with either thrombin or

148 Thus, in vivo superfusion of the rat mesentery with stable lipoxin ana

149 It was not prevented by superfusion of the slices with alpha1 or alpha2 or beta-

150 Superfusion of the slices with N2,2'-O-dibutyrylguanosin

151 ypertonic solution (HS) was applied by local superfusion of the synaptic area.

152 Superfusion of the vas deferens with exogenous epsilon-A

153 Local superfusion of the ventral tegmental area with glutamate

154 Local superfusion of the ventral tegmental area with the D2 ag

155 Superfusion of tissue slices with 0.1 nM to 100 nM 2-p-(

156 However, superfusion of tissue slices with CGS 21680 over the sam

157 Superfusion of tolbutamide, a K(ATP) channel sulfonylure

158 In oocytes expressing GAT1 and syntaxin 1A, superfusion of transporter substrates increases the GAT1

159 Topical superfusion of vasoactive compounds on skeletal muscle r

160 Acute displacement of pH(i) (superfusion of weak acids/bases) triggered rapid pH(i) r

161 ignificant differences were obtained between superfusions of striatal tissue fragments stimulated wit

162 L-arginine methyl ester (L-NAME; 100 mumol/L superfusion or 10 mg/kg intravenously) significantly dec

163 scarinic receptor agonist, applied by either superfusion or iontophoresis, produced an atropine-sensi

164 Applied to the spinal cord slices by superfusion, PACAP-38 (10-30 nM) caused intense neuronal

165 s in DA recovery rates were obtained between superfusions performed in the presence or absence of TMX

166 automated pipets (subsequently called "dual superfusion pipet"), each providing the ability to dispe

167 n switching around the cell, additional dual superfusion pipets were inserted into the microchannel f

168 r ionic composition were unaltered after the superfusion procedures.

169 Increased [K+]o (5-10 mM) in the superfusion, producing modest depolarizations, consisten

170 Increasing the superfusion rate decreased the thickness of the surface

171 20 mM propionate after approximately 40 min superfusion resulted in an alkalinization of approximate

172 Intravenous infusion with IS confirmed the superfusion results and caused shedding of heparan sulfa

173 During acid superfusion, RX 77368 delayed acidification and enhanced

174 In group 4 (n=8), L-arginine pericardial superfusion significantly increased NO overflow measured

175 VEGF superfusion significantly increased permeability in both

176 teric arterioles in response to elevation of superfusion solution PO2 from approximately equal to 3 t

177 of assessing dopamine uptake using in vitro superfusion, striatal tissue from ovariectomized female

178 tus solitarius (NTS) were employed for slice superfusion studies of electrically evoked [3H]serotonin

179 Under pH 5 superfusion, surface pH was relatively acidic (pH 4.2 +/

180 These studies used a superfusion system to investigate the hypothesis that NO

181 A rapid superfusion system was used to apply various drugs for k

182 , RC-121, were compared in the rat pituitary superfusion system, both compounds were found to suppres

183 Using a superfusion system, we compared the effect of NPY on NMD

184 pituitary cells to 100 nM Cetrorelix in the superfusion system, which is devoid of LH-RH, did not ca

185 rated male rats as studied using an in vitro superfusion technique.

186 Using in vitro superfusion, the depolarization of spinal cord triggered

187 ncentration of 5-HT was not dependent on its superfusion time but on the number of activations of the

188 , in the case of the latter experiment), the superfusion was continued for another 115 min.

189 Rapid drug superfusion was used to study the mechanism of alcohol p

190 Rabbit corneal endothelial superfusions were performed under an in vitro specular m

191 causing fixed block after 50 minutes of drug superfusion, which prevented reentry at all delays.

192 Superfusion with 0.01 to 100 nM 11,12-EET caused graded

193 ed nicotinic EPSCs also were desensitized by superfusion with 1 microM nicotine, had extrapolated rev

194 Superfusion with 10 microM IP3 under conditions that min

195 ld be desensitized almost completely by bath superfusion with 100 nM nicotine.

196 etics; pH gating sensitivity was measured by superfusion with 100% CO2-saturated media.

197 .2-0.4 pH units of Helix neurones induced by superfusion with 3 mM concentrations of the weak bases t

198 mechanical stimulation of the soma, or brief superfusion with 300 nM capsaicin, resulted to [Ca(2+)](

199 while cells were progressively acidified by superfusion with a bicarbonate-buffered solution gassed

200 Intracellular acidification was induced by superfusion with a bicarbonate-buffered solution gassed

201 Neither superfusion with a nitric oxide synthase inhibitor, NG-m

202 After excision, superfusion with anti-PIP2 antibodies induced the appear

203 GPx-1((tg-)) mice showed vasoconstriction to superfusion with beta-methacholine and bradykinin (P < 0

204 mV in isolated type I cells recorded during superfusion with bicarbonate-buffered saline solution at

205 Superfusion with bicuculline (10 microM) and strychnine

206 cy induced by Bay K 8644 was not affected by superfusion with Ca2+-free solution (with 10 mmol/L EGTA

207 ed by 60 +/- 5% following a 10 min period of superfusion with caffeine (10 mM) to deplete the endopla

208 Superfusion with CCL21, but not CXC chemokine ligand 12,

209 or this idea comes from the observation that superfusion with cesium selectively reduces IR and elimi

210 Superfusion with Cl(-)-free solution also abolished the

211 In addition, superfusion with compound 48/80 (25 microg/ml) constrict

212 The clearance of FITC-dextran-10 K during superfusion with compound 48/80 in the presence of WT.3

213 ffer (no added Ca2+ + 1 mM EGTA) followed by superfusion with control (Ca2+-containing) buffer.

214 of AP frequency (max. dAP/dDeltamV), during superfusion with cPGI.

215 Simultaneous superfusion with either DHT or 3alpha-diol significantly

216 y to undergo vasoconstriction in response to superfusion with endothelin-1.

217 In contrast, superfusion with ethanol significantly enhanced the GABA

218 Conversely, superfusion with GABA receptor antagonists decreased the

219 ar effects could be demonstrated after brief superfusion with high concentrations of adenosine itself

220 Superfusion with high performance liquid chromatographic

221 Superfusion with IS induced strong leukocyte adhesion, e

222 sed during SS and was reduced by pericardial superfusion with L-arginine compared with control (6005.

223 o significant difference between control and superfusion with L-arginine, a NO precursor (222+/-20 ve

224 Superfusion with l-arginine, an NOS substrate, caused de

225 Superfusion with low-Ca(2+) bathing medium (no added Ca(

226 oamine-depleted ganglia, tetanus preceded by superfusion with m-CPBG readily induced LTP.

227 In wild-type mice, spinal superfusion with naloxone hydrochloride (10 mM, 30 micro

228 n measures of gastric defense during mucosal superfusion with neutral solutions.

229 Superfusion with pCS and pCG combined caused impaired bl

230 s release inhibition was prevented partly by superfusion with sulpiride (47% inhibition) and was redu

231 Superfusion with the agonists (GABA or glycine) produced

232 ists SKF 38393 or SKF 81297-C and blocked by superfusion with the D1 antagonist SCH 23390.

233 air cells by whole-cell voltage clamp during superfusion with the potassium channel antagonists, tetr

234 the CA3 region of rat hippocampal slices by superfusion with the potassium channel blocker 4-aminopy

235 he DMV neurones were also less responsive to superfusion with the satiety neuropeptides cholecystokin

236 Continuous superfusion with the uncoupling agent carbonyl cyanide-p

237 e capillaries were examined before and after superfusion with varying concentrations of adenosine (or

238 During superfusion with vehicle (saline), clearance of FITC-dex

239 During superfusion with vehicle (saline), clearance of FITC-dex

240 During superfusion with vehicle, clearance of FITC-albumin, FIT

241 During superfusion with vehicle, clearance of FITC-dextran-10K

242 During superfusion with zero [Ca2+]o and EGTA, about 73% of 40

243 Superfusion with zero calcium produced no effect on curr

244 l K(ATP) channel inhibition (5 mg kg(-1) GLI superfusion) would decrease blood flow (15 um microspher