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1 V, eliminating over 90% of latent virus from superior cervical ganglia.
2 ia, but was absent in sympathetic neurons of superior cervical ganglia.
3 during naturally occurring cell death in the superior cervical ganglia.
4 ntly reduces developmental cell death in the superior cervical ganglia.
5 sympathetic postganglionic neurons from the superior cervical ganglia.
6 anches of the facial nerve and resecting the superior cervical ganglia.
7 wo to three times greater in dorsal root and superior cervical ganglia.
8 radrenergic innervation originating from the superior cervical ganglia.
9 central structures, and projections from the superior cervical ganglia; activation of this pathway re
12 ies on cultured rat sympathetic neurons from superior cervical ganglia (and also insect neurons) have
14 cholinergic fibers likely originate from the superior cervical ganglia because unilateral ganglionect
16 t a novel interaction between JLP and SCG10 (superior cervical ganglia clone 10), which is a microtub
19 nt levels of cellular proliferation in mouse superior cervical ganglia during the first 18 days after
20 ral sympathetic fibers, originating from the superior cervical ganglia, grow into the cholinergically
21 llular physiological measurements of control superior cervical ganglia identified two distinct types
22 rents was severely reduced in the neurons of superior cervical ganglia in -/- mice with five physiolo
24 doubling of neuron number in trigeminal and superior cervical ganglia, many components of the sensor
26 The microtubule-destabilizing protein SCG10 (superior cervical ganglia, neural specific 10) was found
27 to be determined; however, infection of rat superior cervical ganglia neurons in vitro indicates tha
28 e calcium channels in mammalian Purkinje and superior cervical ganglia neurons with similar IC50 valu
32 In contrast, the peripheral neurons of the superior cervical ganglia of PKR(-/-) x RL(-/-) mice sho
34 Comparable changes failed to develop in the superior cervical ganglia of the NOD mouse or in the SMG
35 hannel activity of neurons isolated from the superior cervical ganglia of young (12 weeks), middle-ag
36 bryonic rat trigeminal, dorsal root, nodose, superior cervical ganglia or retina with a variety of na
37 alternative splice variant expression in rat superior cervical ganglia revealed multiple variant isof
39 mporal expression of BMP5, -6, and -7 in rat superior cervical ganglia (SCG) is consistent with their
40 In patch-clamp studies, nodose, coeliac and superior cervical ganglia (SCG) neurones from wild-type
41 rmed microgels, to culture individual murine superior cervical ganglia (SCG) neurons or epithelial ce
43 ur types of G(q/11)-coupled receptors in rat superior cervical ganglia (SCG) sympathetic neurons.
45 of mud genes was analyzed in developing rat superior cervical ganglia (SCG) undergoing programmed ce
46 de from rat sympathetic neurones in isolated superior cervical ganglia (SCG), coeliac ganglia (CG), a
48 nAChRs in several rat autonomic ganglia: the superior cervical ganglia (SCG), sensory nodose ganglia,
49 ition of neuronal nicotinic receptors in rat superior cervical ganglia (SCG), their single-channel pr
50 NA sequencing traced this defect back to the superior cervical ganglia (SCG), which responded to card
54 n sympathetic neurons cultured from traf6+/+ superior cervical ganglia (SCGs), there was an increase
56 ed in sympathetic neurons, isolated from rat superior cervical ganglia, using whole-cell voltage clam
58 n a nearly total loss of SN-LI fibers in the superior cervical ganglia, whereas immunoreactivity in t