ライフサイエンスコーパス: superior mesenteric artery

1 isolated rings of endothelium-denuded rabbit superior mesenteric artery.

2 h after release of the occluded (for 15 min) superior mesenteric artery.

3 pletion of transmitter (CGRP) content of the superior mesenteric artery.

4 tment of the celiac axis, common hepatic and superior mesenteric artery.

5 One patient died of thrombosis in the superior mesenteric artery.

6 vein (LRV) lodged between the aorta and the superior mesenteric artery.

7 n, right hepatic vein, common bile duct, and superior mesenteric artery.

8 giography and Doppler ultrasonography of the superior mesenteric artery.

9 showed uniform patterns of branching at the superior mesenteric artery.

10 cells derived from the adult rat (or mouse) superior mesenteric artery.

11 epatic artery arising independently from the superior mesenteric artery.

12 of postburn endotoxemia on blood flow in the superior mesenteric artery.

13 ter release of the occluded (for 15 minutes) superior mesenteric artery.

14 ere only found in thoracic aortas but not in superior mesenteric arteries.

15 the coeliac trunk (50), hepatic artery (29), superior mesenteric artery (35), and other segments (4).

16 gh obstruction of the distal branches of the superior mesenteric artery (60 minutes) and reperfusion

17 nteric vein (94%), hepatic artery (93%), and superior mesenteric artery (93%) in these patients.

18 ependent relaxation of microvessels from the superior mesenteric artery after I/R was significantly a

19 umented with an ultrasonic flow probe on the superior mesenteric artery and a catheter into the super

20 ultrasonic flow probe was inserted into the superior mesenteric artery and a catheter into the super

21 h a magnetic flowprobe was placed around the superior mesenteric artery and an ileal tonometer was in

22 minutes) and reperfusion (60 minutes) of the superior mesenteric artery and assess the effects of neu

23 he right half of the nerve plexus around the superior mesenteric artery and celiac axis.

24 Lower blood flow in the superior mesenteric artery and CT was correlated with HF

25 s clearance of [14C]lactate infused into the superior mesenteric artery and direct measurements of bl

26 teric-retroperitoneal D3 located between the superior mesenteric artery and the aorta was seen on US

27 ock was induced in rats by clamping both the superior mesenteric artery and the celiac trunk for 45 m

28 ock was induced in rats by clamping both the superior mesenteric artery and the celiac trunk for 45 m

29 ow probes were placed around a branch of the superior mesenteric artery and the right femoral artery.

30 ssion was differentially enhanced in the PHT superior mesenteric artery and thoracic aorta during the

31 djacent structures, relative position of the superior mesenteric artery and vein.

32 nflow is given by the terminal branch of the superior mesenteric artery and venous outflow by a proxi

33 on was observed in an artery with white fat (superior mesenteric artery) and in aorta from both male

34 k into C (for common hepatic artery), S (for superior mesenteric artery), and L (for left gastric art

35 creas, pancreatic adenocarcinoma, celiac and superior mesenteric arteries, and superior mesenteric an

36 flow volume were measured in celiac artery, superior mesenteric artery, and main portal vein (MPV).

37 loops, hurricane eye, small bowel behind the superior mesenteric artery, and right-sided anastomosis.

38 Superior mesenteric artery aneurysm (SMAA) is an uncommo

39 tive and less invasive option for rupture of superior mesenteric artery aneurysm.

40 umors involving both roots of the celiac and superior mesenteric artery are deemed unresectable by co

41 Reports of dissection of the celiac and/or superior mesenteric artery are rare; as far as we know,

42 les of 30-sec reperfusion and reocclusion of superior mesenteric artery at the initiation of reperfus

43 ch was the result of a threefold increase in superior mesenteric artery BFV (P < .0001).

44 t elevations in gastric volume (P < 0.0001), superior mesenteric artery blood flow (P < 0.0001), and

45 indigestion score that correlated with peak superior mesenteric artery blood flow (P=0.017).

46 s concomitant with a significant decrease in superior mesenteric artery blood flow (Qsma) after 15 da

47 stric volume, small bowel water content, and superior mesenteric artery blood flow and velocity were

48 Bowel wall thickness and superior mesenteric artery blood flow were measured by u

49 ributed ~22% of the postprandial increase in superior mesenteric artery blood flow.

50 Blood flows decreased in the iliac and superior mesenteric arteries, but not in the renal arter

51 32, Cx40 and Cx43 was detected in the rabbit superior mesenteric artery by reverse transcriptase-poly

52 und GIP(1-42) to stimulate blood flow in the superior mesenteric artery by ~10% in the fasting state.

53 ucose alone increased mean blood flow in the superior mesenteric artery by ~70% and portal vein by ~4

54 lucose ingestion increased blood flow in the superior mesenteric artery by ~70%, and the increase was

55 No tumor-vessel interface was noted at the superior mesenteric artery, celiac artery, or common hep

56 , or mannitol (osmotic control), followed by superior mesenteric artery clamping for 60 minutes and 3

57 rtal vein) contact (r = -0.38), and post-CRT superior mesenteric artery contact (r = 0.34).

58 regression analysis, lower blood flow in the superior mesenteric artery, CT (p < 0.04), and inferior

59 he pericardial space until blood flow in the superior mesenteric artery decreased to half of baseline

60 namically significant (>70%) stenosis of the superior mesenteric artery developed 7-14 days after sur

61 ection, and 71 cases of spontaneous isolated superior mesenteric artery dissection have been reported

62 rare, spontaneous isolated celiac artery and superior mesenteric artery dissections must be kept in m

63 0.45 [SD, 0.62] L.min(-1); P=3.8x10(-8)) and superior mesenteric artery flow (Deltamean, 0.76 [SD, 0.

64 superior mesenteric artery flow and PP were measured in

65 nduced nonocclusive intestinal ischemia, the superior mesenteric artery flow and RBC velocity correla

66 icardial tamponade (n = 12), which decreased superior mesenteric artery flow from 351 +/- 55 to 182 +

67 ar resistance, reduced portal pressure (PP), superior mesenteric artery flow, mesenteric vascular den

68 bes were placed around the iliac, renal, and superior mesenteric arteries for measurement of MAP, hea

69 ntestinal I/R injury induced by clamping the superior mesenteric artery for 100 min with tissue analy

70 Intestinal I/R was induced by occluding the superior mesenteric artery for 30 min followed by reperf

71 a-reperfusion by occlusion (clamping) of the superior mesenteric artery for 30 min, followed by uncla

72 nal I/R injury by transient occlusion of the superior mesenteric artery for 30 min.

73 ry was induced by temporary occlusion of the superior mesenteric artery for 30 mins, followed by 2 hr

74 Ischemia was induced by clamping the superior mesenteric artery for 60 min, followed by 60 mi

75 rats (six animals/group) by occlusion of the superior mesenteric artery for 90 min and subsequent rep

76 Direct infusion of urokinase into the superior mesenteric artery for treatment of mesenteric v

77 e was continuously infused into the cranial (superior) mesenteric artery for 48 hours.

78 t when the [14C]lactate was infused into the superior mesenteric artery, indicating increased first-p

79 The patient was treated with selective superior mesenteric artery infusion of urokinase resulti

80 higher precision than GPT-3.5 for extracting superior mesenteric artery involvement (100% vs 88.8%, r

81 gic (eg, tumor grade, lymph node positivity, superior mesenteric artery involvement), or treatment fa

82 pentobarbital and subjected to 30 minutes of superior mesenteric artery ischemia, followed by 4 hours

83 fter occlusion (35 min) and reopening of the superior mesenteric artery, MC3R-null mice displayed a h

84 Both iNOS+/+ and iNOS-/- mice subjected to superior mesenteric artery occlusion (SMAO) in which bac

85 mechanical ventilation (CMV) over 60 mins of superior mesenteric artery occlusion and 60 mins of repe

86 subjected to a sham operation or 30 mins of superior mesenteric artery occlusion followed by reperfu

87 erated or ischemia-reperfusion groups, where superior mesenteric artery occlusion was maintained for

88 After superior mesenteric artery occlusion, intestinal permeab

89 ET receptor expression was determined in the superior mesenteric artery of sham and PHT rats by in si

90 and 30 mins of ischemia by occlusion of the superior mesenteric artery or 30 mins of ischemia follow

91 eatic disease, 2) no tumor encasement of the superior mesenteric artery or celiac axis, and 3) a pate

92 The transplant hepatic artery, celiac trunk, superior mesenteric artery, portal vein, superior mesent

93 lso was used for 417 renal and 50 celiac and superior mesenteric artery reconstructions.

94 Doppler ultrasonography of the superior mesenteric artery revealed a twofold increase i

95 igher celiac RI (0.78 versus 0.73, P = 0.04) superior mesenteric artery RI (0.89 versus 0.84, P = 0.0

96 al analysis of CGRP-containing nerves in the superior mesenteric artery showed no differences in dens

97 ents) were placed in 692 renal arteries, 156 superior mesenteric arteries (SMA), and 50 celiac arteri

98 origin and branching pattern, including the superior mesenteric artery (SMA) and inferior phrenic ar

99 Blood pressure, superior mesenteric artery (SMA) and skeletal muscle blo

100 Two 4D flow datasets were acquired, over the superior mesenteric artery (SMA) and the main portal ven

101 e fasting and postprandial blood flow in the superior mesenteric artery (SMA) and vein (SMV) in 22 pa

102 Aortic and superior mesenteric artery (SMA) blood flow was monitore

103 thoracic aorta by approximately 60 % and the superior mesenteric artery (SMA) by approximately 90 %.

104 Duodenal obstruction by compression from the superior mesenteric artery (SMA) can be managed using mi

105 IIR was established by clamping the superior mesenteric artery (SMA) for 45 minutes followed

106 Vasoconstriction of the superior mesenteric artery (SMA) is the earliest hemodyn

107 ysis of the final bile duct, pancreatic, and superior mesenteric artery (SMA) margins.

108 hepatic artery (CHA) arising from either the superior mesenteric artery (SMA) or the aorta.

109 Superior mesenteric artery (SMA) syndrome describes vasc

110 rience in preoperative embolization of graft superior mesenteric artery (SMA) to facilitate intestina

111 died adhesion of isolated neutrophils to rat superior mesenteric artery (SMA) vascular segments stimu

112 effect of octreotide on vascular tone in the superior mesenteric artery (SMA) was studied in portal-h

113 An aneurysm of the superior mesenteric artery (SMA) with a diameter of 2.2

114 ia either the splenic artery (SA)(n = 47) or superior mesenteric artery (SMA)(n = 51) in 98 patients,

115 agnetic flow probe was positioned around the superior mesenteric artery (SMA), and cannulation of the

116 origin, hepatic artery (HA) arising from the superior mesenteric artery (SMA), and increasing donor B

117 cer patients with arterial encasement of the superior mesenteric artery (SMA), the hepatic artery (HA

118 , orifice of celiac truncus (CT), orifice of superior mesenteric artery (SMA), vena cava inferior con

119 OS) pathways was analyzed by western blot in superior mesenteric artery (SMA).

120 ks, a catheter was placed selectively in the superior mesenteric artery (SMA).

121 B1 and B2 kinin receptors on cultured rabbit superior mesenteric artery smooth muscle cells with des-

122 vated fat diet had catheters placed into the superior mesenteric artery so that the visceral adipose

123 core at all assessed anatomic locations- the superior mesenteric artery (spearman correlation coeffic

124 r mesenteric vein thrombosis, and 4 (3%) had superior mesenteric artery stricture or spasm.

125 ve identified the occurrence of an allograft superior mesenteric artery-superior mesenteric vein (SMA

126 ditions, annular pancreas, duplication cyst, superior mesenteric artery syndrome, midgut volvulus, an

127 retic hormone; pancreatitis; cholelithiasis; superior mesenteric artery syndrome; ileus; pnemothorax;

128 Seventy-four (65%) had a superior mesenteric artery thromboembolism, 25 (22%) had

129 nalogue methanandamide relax rings of rabbit superior mesenteric artery through endothelium-dependent

130 d rings were surgically implanted around the superior mesenteric arteries to create gradual stenosis.

131 significantly reduced the blood flow in the superior mesenteric artery to 53% of baseline.

132 to 17 (85%) of 20 MR angiograms obtained in superior mesenteric artery trunks, 15 (75%) in celiac ar

133 In the porcine experiments, the superior mesenteric artery was gradually obstructed duri

134 nt in whom surgical revascularization of the superior mesenteric artery was necessary and in one in w

135 line) was administered 60 minutes before the superior mesenteric artery was occluded for 90 minutes a

136 rrent infusion of GIPR-An, blood flow in the superior mesenteric artery was ~22% lower.