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1 of a large quantity of the Gag p27 into the supernatant fluid.
2 le amounts of the LspA1 protein into culture supernatant fluid.
3 that can be detected in concentrated culture supernatant fluid.
4 d in the release of virus particles into the supernatant fluid.
5 ibody was used to localize the enzyme in the supernatant fluid.
6 ount of NADase and SLO activities in culture supernatant fluids.
7 bstantially more H77-S RNA was secreted into supernatant fluids.
8 hanisms for regulating RTX toxin activity in supernatant fluids.
9 iation with the appearance of sVCAM-1 in the supernatant fluids.
10 surable IFN-gamma expression was detected in supernatant fluids.
11 or 6 h, and IL-1 was measured in the culture supernatant fluids.
12 ith RSV and determined nitrite levels in the supernatant fluids.
13 ol of product formed was readily detected in supernatant fluids.
14 the numbers of tachyzoites released into the supernatant fluids.
15 ion of LspA proteins from H. ducreyi culture supernatant fluid abolished this inhibitory effect, indi
16 ite presence of MGCs and infectious virus in supernatant fluids after apical infection, infected cell
18 by measuring alpha-toxin activity in culture supernatant fluids and by Northern analysis of the alpha
19 in, RTX (repeats-in-toxin) toxin, to culture supernatant fluids and that this toxin is responsible fo
20 onuclear cells after harvesting of cell-free supernatant fluids at peak antigen production (days 4-7)
21 vitro, human CCA cells were treated with MC supernatant fluids before evaluating angiogenesis and EM
24 ese cells, and because megakaryocyte-derived supernatant fluid can reproduce the EndMT switch in vitr
27 in cell-free concentrated H. ducreyi culture supernatant fluid could also be tyrosine phosphorylated
42 -ERK44/42 MAPK levels induced by C. albicans supernatant fluids in the IFN-gamma-plus-LPS-driven mono
44 nce of RTX toxin in stationary-phase culture supernatant fluids is also due to a lack of toxin secret
45 l inoculation with virions from concentrated supernatant fluid, JSRV-associated OPC lesions were foun
47 mmonium sulfate at 100% saturation by use of supernatant fluid of BCSP70 that had been precipitated w
48 type protein (CGCryV) has been isolated from supernatant fluids of Bacillus thuringiensis AB88 cultur
50 ked immunosorbent assay (ELISA) performed on supernatant fluids of LPS challenged MDM showed ImI-medi
53 . gingivalis CPS and assessed the ability of supernatant fluids produced by challenged macrophages to
55 obtained from S. cerevisiae, the C. albicans supernatant fluids significantly suppressed IL-12 produc
56 nditions, in concentrated H. ducreyi culture supernatant fluids that lacked detectable outer membrane
57 red necessary for H. ducreyi-derived culture supernatant fluid to exhibit cytotoxicity for HeLa cells
58 pressed in order for E. coli-derived culture supernatant fluids to demonstrate cytotoxicity for HeLa
59 otein that was readily detectable in culture supernatant fluid together with a barely detectable amou
60 dissected cytology slides and from discarded supernatant fluid was analyzed in 26 patients with atypi
62 2:1) to stimulate IL-12, concentrated fungal supernatant fluids were added and incubated for an addit
64 ht patients showed no evidence of fat in the supernatant fluid with either CT (n = 7) or CT and MR im