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1 he peroneal nerve, the tibial nerve, and the sural nerve.
2 egment of the medial cutaneous branch of the sural nerve.
3 0.01) and mean axonal size (P < 0.05) in the sural nerve.
4 ers and profuse regenerative activity in the sural nerve.
5 on was also increased in the DRG, but not in sural nerve.
6 n nearby hairy skin innervated by the spared sural nerve.
7 (ulnar, deep peroneal) and sensory (median, sural) nerves.
9 nd the F-wave latency (p=0.03) decreased and sural nerve action potential amplitude increased (p=0.04
10 Changes in excitability of the terminals of sural nerve afferents were used to confirm that both loc
12 ith BIPN more frequently demonstrated absent sural nerve amplitudes and diminished distal sensation c
14 ical stimulation of afferent C fibers in the sural nerve and recorded from single neurons in the vent
17 was evoked by electrical stimulation of the sural nerve and was recorded in the ipsilateral hamstrin
18 pathies and compared these with normal human sural nerves and those from patients with Guillain-Barre
19 lation of a 54-kDa isoform of JNK in DRG and sural nerve, and this correlated with elevated c-Jun and
20 vestigated the surgical anatomy of the ovine sural nerve as a potential candidate for facial nerve re
25 ibres was a uniform feature in a total of 21 sural nerve biopsies and 'onion bulb' formations and/or
26 ith diabetic neuropathy progression in human sural nerve biopsies and describe their potential utilit
29 -4 (CD152) at the protein and mRNA levels in sural nerve biopsies of patients with chronic inflammato
30 eased PMP22 messenger RNA levels in skin and sural nerve biopsies of patients with CMT1A compared wit
31 and nuclear imaging, electroencephalography, sural nerve biopsies, sleep evaluation and neuropsychome
36 lectrophysiologic data were evaluated, and a sural nerve biopsy from one affected child was examined
37 tain circumstances replace the more invasive sural nerve biopsy in the morphological and molecular ev
43 area of the antidromic volley evoked in the sural nerve by intraspinal microstimulation in the L4/5
44 unmyelinated fiber function in the hind paw, sural nerve C-fiber morphometry, sciatic nerve neurotrop
46 iments were performed on 50 samples of human sural nerves collected during a 52-week clinical trial.
51 Nociceptive nerve function, unmyelinated sural nerve fiber and dorsal root ganglion (DRG) cell mo
52 urofilament distances (NNND) in the axons of sural nerves from patients with anti-MAG paraproteinaemi
56 can be achieved with minimal morbidity using sural nerve grafts, which surgeons commonly use to recon
57 e sensory latency of both the left and right sural nerves improved on the basis of faster median cond
68 f HIV-SN, freshly isolated mitochondria from sural nerves of macaques infected with a neurovirulent s
69 mtDNA common deletion mutation in postmortem sural nerves of patients with HIV-SN as compared to unin
72 s from four cryopreserved normal adult human sural nerves referenced to the Genome Reference Consorti
73 Stimulation of the gastrocnemius nerve and sural nerve revealed significant convergence of muscle a
75 staining of lateral plantar nerve (LPN) and sural nerve (SN) motor terminals, using the activity-dep
78 icantly lower C-fiber conduction velocity in sural nerves than uninfected animals and the magnitude o
86 The mean number of myelinated fibers in the sural nerve was significantly lower than that of the BB
87 were exposed and in situ recordings from the sural nerve were performed to determine compound C-fiber
89 omy and the number of fascicles of the ovine sural nerve were similar of those reported in humans.