ライフサイエンスコーパス: surface expression

1 lation of major histocompatibility complex I surface expression.

2 ed to absolute eosinophil counts or Siglec-8 surface expression.

3 variation in expression and confirming cell surface expression.

4 y, loss of ion selectivity, and reduced cell-surface expression.

5 (IIb)beta(3) activation without altering its surface expression.

6 e CT abolished synaptic trafficking, but not surface expression.

7 total K(V) 2.1 expression or deficient cell-surface expression.

8 urther upregulate receptor assembly and cell surface expression.

9 ng variant rs6967330 increases CDHR3 protein surface expression.

10 e current amplitude, consistent with reduced surface expression.

11 to CD20xCD3 bsAb that is independent of CD20 surface expression.

12 cytotoxicity of mutant TRPC6 and reduced its surface expression.

13 17.1 currents, presumably caused by impaired surface expression.

14 of p-aminohippuric acid and an enhanced OAT1 surface expression.

15 modulating its intracellular trafficking and surface expression.

16 cellular mechanisms that acutely control DAT surface expression.

17 es NF-kappaB activation and reduces CD4 cell surface expression.

18 Golgi complex and show markedly reduced cell-surface expression.

19 o the mechanism by which cocaine alters AMPA surface expression.

20 0 in LPS-mediated decrease in MC FcepsilonRI surface expression.

21 y, whereas radioimmunolabeling measured cell-surface expression.

22 ulum seems to be the cause of the lower cell-surface expression.

23 was reduced, reflecting a reduction in ENaC surface expression.

24 lding of HFE, CD1, and MHC class I and their surface expression.

25 estrophin-1 confirmed reduction of Ca(V) 1.3 surface expression.

26 increased major histocompatibility complex I surface expression.

27 tive regulator of both MHC-I and MHC-II cell surface expression.

28 tes to the inhibition of platelet P-selectin surface expression.

29 ponsible for GSH recycling, promoted ULBP2/5 surface expression.

30 r protein glycosylation and reduce JAG1 cell surface expression.

31 ty by modulating pore-function or decreasing surface expression.

32 imethyl fumarate (DMF) also promotes ULBP2/5 surface expression.

33 activity independently of effects on channel surface expression.

34 ositively and negatively modulate MHC-I cell surface expression.

35 d Ca(V) 1.3 channel activity, and changes in surface expression.

36 nel trafficking also regulates Na(+) channel surface expression.

37 Rab11b-mediated control of integrin beta1 surface expression allows efficient engagement with the

38 gene products are characterized by low cell-surface expression and a highly conserved peptide-bindin

39 The newly formed RBC had reduced CD47 surface expression and a reduced life span and were phag

40 nd investigated how glycosylation alters its surface expression and activity.

41 ous TCRs compete with the transgenic TCR for surface expression and allow mixed dimer formation.

42 tism, human GluN2B S1415L, displayed reduced surface expression and binding to PSD-95.

43 onjugated to ion channels to influence their surface expression and biophysical properties.

44 air channel folding or assembly prevent cell surface expression and cause congenital hyperinsulinism.

45 ion in inflammatory chemokine receptor CXCR3 surface expression and cellular activation of lipid phos

46 ullin 3, which was revealed to regulate CD22 surface expression and clathrin-dependent CD22 internali

47 ted (HCN) ion channels, alters both the cell surface expression and cyclic nucleotide dependence of t

48 hic mutation results in diminished IL-2Rbeta surface expression and dysregulated IL-2/15 signaling, w

49 ecycling of AMPA receptors to increase their surface expression and elicits structural changes in spi

50 that an SNX3-retromer complex regulates the surface expression and function of PC1 and PC2.

51 e in DAT(+) neurons produced increased AMPAR surface expression and function that lead to impaired in

52 stem in which IL2RB mutations caused reduced surface expression and IL-2 binding.

53 TDM downregulated MHC class II cell surface expression and impaired T cell activation by pep

54 n 3 in B lymphocytes, namely regulating CD22 surface expression and internalization after B cell acti

55 AB receptors (GABABRs) to control their cell surface expression and intracellular trafficking via a d

56 required for UM7F8's capacity to reduce CD98 surface expression and its capacity to inhibit the proli

57 During this phase, CTLA4 surface expression and its complexation with CD80/CD86 c

58 The mutation did not affect surface expression and ligand binding but changed the su

59 cal models, poziotinib upregulated HER2 cell-surface expression and potentiated the activity of T-DM1

60 Upon verifying mutant cell surface expression and proteolytic cleavage, we tested t

61 NA into knockout BLCLs fully restored HLA-DP surface expression and recognition by alloreactive human

62 tide caused an opposing increase in Na(V)1.8 surface expression and repetitive firing.

63 Blockade of ICOSL rescues T cell ICOS surface expression and rescues, at least in part, T foll

64 g proteins (KChIPs), which leads to enhanced surface expression and shapes the inactivation gating of

65 Alterations in both cell-surface expression and single-channel properties account

66 loss of function, and increasing TREM2 cell surface expression and thereby its function(s) might hav

67 o changes in spine morphology, AMPA receptor surface expression and trafficking, and AMPA receptor-me

68 ccr5 RNA levels, reduced CCR2 and CCR5 cell-surface expression, and decreased levels of secreted che

69 egments, with consequences for the assembly, surface expression, and function of the polycystin compl

70 trast, tracking of IL7Ralpha message levels, surface expression, and Il7r-Cre-mediated labeling acros

71 otein structure, in vitro assessment of cell surface expression, and in vitro knockdown, revealed pot

72 GN4Y displays severe deficits in maturation, surface expression, and synaptogenesis regulated by one

73 neered and evaluated for GP1-GP2 processing, surface expression, and the ability to mediate cell-to-c

74 nce resonance energy transfer-based and cell-surface expression approaches, we comprehensively screen

75 targets that could modulate microglial TREM2 surface expression are not known.

76 features of SAMs and indicate that increased surface expression, as observed for APLP1, is essential

77 on levels on the somatic surface but reduced surface expression at the AIS of cultured rat embryonic

78 6L, located in the TM3-4 loop, showed normal surface expression but reduced chloride currents, and ac

79 d T cell subsets did not correlate with PD-1 surface expression but was inversely correlated with int

80 at cannot be S-acylated show attenuated cell surface expression, but expression was restored by co-ex

81 slit diaphragm molecules can influence their surface expression, but it is unknown whether tyrosine p

82 se C (PKC) activation acutely diminishes DAT surface expression by accelerating DAT internalization.

83 , while antagonizing inhibition reduces KCC2 surface expression by increasing the lateral diffusion a

84 the absence of CD95 expression and increased surface expression CCR7, CD27, the activation markers T-

85 E-associated variants of STAB2 had a reduced surface expression compared with reference STAB2.

86 beta or undergoing EMT upregulated CD73 cell-surface expression, confirming roles for these pathways.

87 ll lines revealed that increased IGF-1R cell surface expression correlates with decreased sensitivity

88 , and consequently subunit assembly and cell surface expression, critically impacting on the efficacy

89 cities, as these depend on glycoprotein cell surface expression (CSE) levels, we inserted identical e

90 Crucially this reduced surface expression did not cause the reduced agonist res

91 nges, calculated stabilities correlated with surface-expression differences, but neither parameter co

92 An alteration in alpha2delta surface expression during critical developmental windows

93 t tyrosine 319 (Y319) led to increased CD226 surface expression, enhanced anti-tumor immunity and imp

94 In addition, we observed a deficit in surface expression for GluN2B S1413L.

95 e as well as promoter region that alter cell surface expression have been associated with disease pro

96 ures we found were lower CD3 and higher CD28 surface expression in AGMs compared to RMs.

97 aying the effect on insulin-stimulated GLUT4 surface expression in differentiated L6 rat myocytes.

98 as enhanced, but was not due to enhanced DAT surface expression in either dorsal or ventral striatum.

99 of cisplatin responses correlated with EGFR surface expression in head and neck cancer cells.

100 10(-13)) and was associated with HLA-DP cell surface expression in healthy individuals (p = 2.04 x 10

101 rus 2 (SARS-CoV-2), demonstrates its highest surface expression in the lung, small bowel, and vascula

102 Thus, HDAC inhibition restored HLA class-I surface expression in vitro and in a mouse xenotransplan

103 ation and reduced nephrin signaling and cell surface expression in vitro In a rat model of reversible

104 of BCMA+ tumor cells, and the levels of BCMA surface expression in vivo.

105 enes: US18 and US20, to interfere with B7-H6 surface expression, in a mechanism involving endosomal d

106 This did not affect surface expression, inhibitor binding, and substrate inf

107 These functions include surface expression, internalization, and forward and rev

108 This reduced HLA class-I surface expression is caused by an impaired expression o

109 This suggests that surface expression is not directly indicative of the exp

110 We report that MET surface expression is reduced by MARCH1, 4, or 8-mediate

111 is IgM(+), but due to low/negative IgD cell surface expression, it was named B-cell IgD low (BD(L)).

112 anchoring of CaValpha2delta1 averts its cell-surface expression, its interaction with CaValpha1, and

113 Despite the fact that the cell surface expression level of HLA-C on both uninfected and

114 by integrin alphavbeta3 depended on the cell surface expression level.

115 ch can be quantified indirectly by measuring surface expression levels of CD5.

116 there are substantial variations in the cell surface expression levels of human TCRs, which can impai

117 domain (TM3-4 loop) of the GlyRalpha1 impair surface expression levels of the receptors.

118 expression, both at the message level and at surface expression, mainly mediated through FcgammaRIIa.

119 Cell surface expression of ACE2 determines the tissue suscept

120 eceptive to Notch ligands via Taok3-mediated surface expression of ADAM10.

121 nusual Ag processing pathways, which reduces surface expression of Ag-derived peptides while selectiv

122 We show that cell surface expression of Ail produces Y. pestis virulence p

123 e Beta-2 Microglobulin (B2M) gene eliminates surface expression of all class I molecules, but leaves

124 -wide screening determined that assembly and surface expression of alpha9alpha10 require ligand bindi

125 upregulating GLT-1 and resulted in increased surface expression of AMPA receptor subunits GluA1 and G

126 manipulation of xCT expression increases the surface expression of AMPA receptor subunits, providing

127 nhibited the adverse effects of Abeta on the surface expression of AMPARs in neurons.

128 se-3/7, cleavage of PARP and increase in the surface expression of Annexin-V.

129 , Vgamma9Vdelta2 T cells presented increased surface expression of APC-associated markers HLA-DR and

130 In MVID, decreased surface expression of apical anion channels involved in

131 ) and membrane traffic, resulting in reduced surface expression of apical membrane proteins.

132 CMA); inhibition of gamma-secretase enhanced surface expression of BCMA and reduced the release of sB

133 Human and mouse ILC2s were assessed for surface expression of beta1 and beta2 integrin adhesion

134 T-6:beta2M interaction and could rescue cell surface expression of beta2M and HLA in human macrophage

135 currents, and 3) LINGO1 reduced the membrane surface expression of BK channels.

136 First, TSLP downregulated surface expression of bone marrow (BM) retention recepto

137 full-length and cleaved TLR3, demonstrating surface expression of both forms of TLR3.

138 RAP alone produced a small increase in cell-surface expression of CaV2.2, alpha2delta-1 and the asso

139 We observed preferential cell surface expression of CCR10 and CXCR3 by HSV-specific CD

140 infected with P. gingivalis and controls for surface expression of CD11b, Ly6G, and Ly6C.

141 Surface expression of CD138 increased heparan sulfate le

142 e subsets are defined by their relative cell surface expression of CD14 and CD16.

143 acid D(205) also regulates the turnover and surface expression of CD16A in the absence of FcepsilonR

144 n but stimulates TNF production, and reduces surface expression of CD206 and CD115, markers of immuno

145 e that select analogs potently increase cell surface expression of CD22, a promising CAR T cell targe

146 arge proportion of CD8(+) TILs had decreased surface expression of CD226 and exhibited features of dy

147 n monocytes demonstrated significantly lower surface expression of CD31 and CD11b, and mechanistic ex

148 ells by creating conditions that induce cell surface expression of CD39, an immunosuppressive molecul

149 NK) cells as defined by accumulation of cell-surface expression of CD57 is associated with increased

150 with distinct functions defined according to surface expression of CD69 and CD45RB.

151 ls and eosinophil activation, as assessed by surface expression of CD69 and serum levels of eosinophi

152 anti-PD-L1), but not anti-PD-1, reduced cell surface expression of CD80 on APCs, and this effect was

153 eukin 6 (IL-6), IL-23, Arginase1, as well as surface expression of CD86 and programmed death ligand 1

154 self neurites is mediated by stochastic cell-surface expression of combinations of about 60 isoforms

155 L lactis G121-induced cytokine release and surface expression of costimulatory molecules by untreat

156 However, a significant reduction in cell surface expression of CRTh2 was observed between the pla

157 ne (CXC motif) ligand 12 stimulation reduced surface expression of CXCR4 and PAR1:CXCR4 heteromers, b

158 ssion is required for the stability and cell-surface expression of discoidin domain-containing recept

159 key role for ALK2 in the BMP9/BMP10-induced surface expression of E-selectin, and both ALK1 and ALK2

160 on of Bcl-2 member Mcl-1 in neurons enhanced surface expression of endogenous alpha7 nAChRs, while a

161 Here, we demonstrate that cell-surface expression of endogenous Notch1 in HEK293T cells

162 he core retromer protein VPS35 increased the surface expression of endogenous PC1 and PC2 in vitro an

163 This increases permeability, surface expression of endothelial adhesion molecules and

164 ells were localised to adventitia and lacked surface expression of endothelial markers (<1% for CD31,

165 nd interleukin-27 (IL-27) also increases the surface expression of Env and thus boosts the ability of

166 In vitro Thpo supported the surface expression of Epcr on primary murine hematopoiet

167 ingly, we found that these mutations reduced surface expression of full-length G1 but not G1-DeltaNT

168 during development by the modulation of cell surface expression of Fz receptor.

169 eric GIRK1/3 channels, without affecting the surface expression of GIRK or Gbeta.

170 ecrease in AMPAR function was due to reduced surface expression of GluA1 subunits through dynamin-dep

171 cate that the HCND is necessary for the cell-surface expression of HCN channels and provides a functi

172 Deletion of the HCND prevented surface expression of HCN2 channels.

173 Leveraging the unique surface expression of heat shock protein 90 (Hsp90) in b

174 HFE in endoplasmic reticulum, causing poorer surface expression of HFE and HFE:TFR1 complex (nonfunct

175 ive light chains (LCs) resulting in the cell surface expression of HIV specific B cell receptors (BCR

176 beta-Cell surface expression of HLA Class II was detected on a por

177 fused to B2M and a peptide antigen), without surface expression of HLA-A, B or C.

178 Likewise, we documented the frequent loss of surface expression of HLA-DR, -DQ and -DP on leukemia ce

179 a manner that confers inducible, regulated, surface expression of HLA-E single-chain dimers (fused t

180 ent tissue samples revealed significant cell surface expression of HSPG2 in both primary tumors and m

181 ration induces MR1 refolding and upregulates surface expression of human MR1, forms MR1 tetramers tha

182 Accordingly, surface expression of hyperpolarization-activated cyclic

183 tory cytokine stimulation increased the cell surface expression of ICAM-1 and induced cell surface ex

184 -BMEC-like cells exhibited constitutive cell surface expression of ICAM-1, ICAM-2, and E-selectin.

185 In the absence of FcmuR, the surface expression of IgM-BCR was increased, which resul

186 ology by constraining the transport and cell-surface expression of IgM-BCR.

187 mination of circulating ILC subsets revealed surface expression of IL-10Ralpha and mRNA expression of

188 ion toward the M(2) phenotype, and increased surface expression of IL-1R8, diminishing activation med

189 Patient T lymphocytes lacked surface expression of IL-2Rbeta and were unable to respo

190 found that IL-4 significantly increases the surface expression of IL-3R and also increases the numbe

191 blood Th cells in resting state do not show surface expression of IL-3R; however, its expression was

192 t of IL5RA and IGHG4 or IGHE, with confirmed surface expression of IL-5Ralpha and functional IL-5 sig

193 ns with high LSC activity, and that the cell surface expression of IL1RL1 is dynamic, implying that t

194 es activate human monocytes, leading to cell-surface expression of immune stimulatory natural killer

195 Macrophage subpopulation cell-surface expression of immunological markers and phagocyt

196 hils and monocytes by 60% to 80% and lowered surface expression of integrin CD11b on monocytes by 20+

197 euronal properties including the feature and surface expression of ionotropic receptors.

198 ses against infected cells, HIV-1 limits the surface expression of its envelope glycoprotein (Env).

199 lar retention elements that control the cell surface expression of Kv1.3 and immune system physiology

200 Infection with HHV-8 did not alter the cell surface expression of langerin on LC but downregulated t

201 used a licensing defect in NK cells, but the surface expression of Ly49A was unaltered.

202 t anti-PD-1 resistant tumours that have lost surface expression of major histocompatibility complex c

203 filtration rate but alter the total and cell-surface expression of major Na(+) transporters all along

204 chymal stem cells are positively selected by surface expression of markers CD90 and CD105.

205 on decreases PD-L1 without compromising cell surface expression of MHC class I.

206 uman beta(2) -microglobulin, which increases surface expression of MHC-I and suggests a role for recy

207 rsed by depleting CD8(+) T cells or reducing surface expression of MHC-I.

208 Each TAP inhibitor reduced surface expression of MHC-Ia molecules but did not reduc

209 f soluble proteins, these data indicate that surface expression of MICB*002 with B44 supertype allele

210 mIgE-ITT motif was required for optimal cell surface expression of mIgE B-cell antigen receptors but

211 In this study we found that surface expression of mutant beta3 subunits is variable.

212 ature protein, alpha1(Lys374Serfs)*(25) Cell surface expression of mutant murine GABA(A)Rs is severel

213 f the NLGN1 and PSD-95 interaction decreases surface expression of NLGN1 in cultured neurons.

214 We demonstrate that N9 recognizes surface expression of Notch1 on the plasma membrane and

215 urface expression of ICAM-1 and induced cell surface expression of P-selectin and VCAM-1.

216 Mechanistically, TLR4-dependent surface expression of P-selectin triggered an unconventi

217 Whereas thrombin stimulation reduced surface expression of PAR1, CXCR4, and PAR1:CXCR4 hetero

218 However, it was consistent that surface expression of partnering gamma2 subunits was low

219 and showed that 1,25D treatment induces cell-surface expression of PD-L1 in epithelial and myeloid ce

220 demonstrated that the epithelial cells with surface expression of PD-L1, E-cadherin, CD24, and VEGFR

221 EG-01 cells showed a marked reduction in the surface expression of platelet markers (CD41, CD42a, and

222 Secretion and surface expression of pro-inflammatory proteins was quan

223 CUS also increased surface expression of RAGE protein on hippocampal microg

224 CRISPR-Cas9 disruption of either gene, cell surface expression of sialic acid was diminished.

225 te that Cmas and Slc35a1, which mediate cell surface expression of sialic acid, are required in murin

226 bset of functional CD8(+) T cells defined by surface expression of SIRPalpha, a protein not previousl

227 unophenotyping of single cells based on cell surface expression of specific proteins together with si

228 ealed that chlamydial infection induced cell surface expression of T-cell homing and activation prote

229 metry analyses indicated significantly lower surface expression of T66M TREM2 variant than wild type

230 ATP6V0C plays a role in down-regulating cell-surface expression of tetherin and thereby contributes t

231 t with this, loss of the BBSome reduced cell surface expression of the 5-HT(2C)R, interfered with ser

232 t the associated beta2 subunits promote cell surface expression of the alpha subunit.

233 el currents independently of changes in cell surface expression of the alpha-subunit.

234 Cell surface expression of the alpha1 subunits and L-type cal

235 tes from JAK2V617F+ mice have increased cell surface expression of the alpha5 subunit of the alpha5be

236 induce the expression of ITGB3 and increase surface expression of the alphaVbeta3 integrin heterodim

237 -1 plasmid, where gene conversion results in surface expression of the antigenically variable VlsE pr

238 roportion of human neutrophils that mediates surface expression of the antineutrophil cytoplasmic ant

239 ), but not the NSs-A46 mutant, increased the surface expression of the CD36 scavenger receptor, resul

240 Additionally, cell surface expression of the CML stem cell markers CD25, CD

241 18 and US20 work in concert to suppress cell surface expression of the critical NKp30 ligand B7-H6 th

242 After extinction training, we assessed surface expression of the glutamate transporter GLT-1 an

243 In vitro, G-1 reduced surface expression of the Hla receptor, ADAM10, in a hum

244 from host cells by down-regulating the cell-surface expression of the host restriction factor tether

245 This can be explained by the low neutrophil surface expression of the IL-4 receptor alpha-chain (IL-

246 endogenous fumarate accumulation upregulates surface expression of the immune stimulatory NK group 2,

247 mice an HCD resulted in upregulated MZB cell surface expression of the immunoregulatory ligand PDL1 i

248 e a novel approach to efficiently inhibiting surface expression of the inhibitory receptor CD94/NK gr

249 Moreover, iron supplementation increased surface expression of the iron-efflux complex, and coppe

250 m (P=0.0013), and there was 1.79-fold higher surface expression of the LDL receptor than in noncarrie

251 ore, CD11b(+)CD45(+) macrophages with a high surface expression of the M1-like markers CD38 and CD86

252 However, the reduced AIS surface expression of the MAP1B mutant was restored to W

253 endogenous or exogenous ligands induced the surface expression of the metalloproteinase ADAM10 on T1

254 (HCMV) UL138 protein downregulates the cell surface expression of the multidrug resistance-associate

255 However, surface expression of the P. falciparum virulence protei

256 Branching deficiency markedly reduced surface expression of the pre-BCR/BCR coreceptor CD19 an

257 els of active TRKB, along with enhanced cell-surface expression of the receptor in cultured hippocamp

258 lls is accompanied by the down-regulation of surface expression of the short form of membrane IgE (mI

259 lls (TCR-T cells) are not restricted by cell surface expression of their targets and are therefore po

260 ns and that HIV-1 infection reduces the cell surface expression of these proteins.

261 pread.IMPORTANCE HIV infection modulates the surface expression of Tim-3, but the molecular determina

262 tion and facilitates the maturation and cell surface expression of TIM-3.

263 cule inhibition of MEK led to increased cell surface expression of TNF receptor-1 (TNFR1) and sensiti

264 a and lung cancer cells, MEKi increased cell-surface expression of TNFalpha receptor 1 (TNFR1), which

265 l extracellular trap generation and elevated surface expression of toll-like receptor 2 and CD11b on

266 ment strategy resulted in markedly increased surface expression of transgenic alphabeta and gammadelt

267 port is not sufficient to enable normal cell-surface expression of TREM2.

268 roblast cell line (MG87.TRKB) increased cell-surface expression of TRKB and facilitated its activatio

269 gly, FH-deficient renal cancer cells had low surface expression of ULBP2/5 and were unresponsive to D

270 stigated the biophysical properties and cell-surface expression of variant K(V) 2.1 channels expresse

271 es of New Zealand White rabbits, despite the surface expression of VlsE.

272 nvolutional neural network (DCNN) to map the surface expressions of fractures in satellite imagery ac

273 onnection between lithospheric evolution and surface expressions of plateau uplift and volcanism.

274 artment upon immunization, had reduced MHCII surface expression on GC cells, and developed accelerate

275 umoral effects but also restores HLA class I surface expression on MCC cells, therefore, restoring su

276 n, presented with suppressed levels of CD11b surface expression on neutrophils and lower myeloperoxid

277 anti-Clr-g mAb detected only residual Clr-g surface expression on splenic monocytes, whereas many he

278 CR trafficking, pathway inhibitors, and cell-surface expression or functional desensitization as indi

279 nd, in some cases, have been shown to affect surface expression or ligand specificity of G-protein-co

280 ylation and even more markedly reduced Gp130 surface expression, potentially explaining the importanc

281 for UL20 membrane targeting and thus gK cell surface expression, providing new mechanistic insights i

282 downregulation between the two varied, with surface expression reducing earlier than the message.

283 ed that PKC activation acutely decreased DAT surface expression selectively in ventral, but not dorsa

284 Endothelial VCAM-1 surface expression stimulated by TNFalpha provided a rea

285 its for flow dictated by geology, leading to surface expression that can be greater or less than the

286 ved a withdrawal-dependent decrease in mGlu1 surface expression that precedes and enables CP-AMPAR ac

287 Abrogation of HLA-II surface expression was achieved in five different HLA-ty

288 Low CCR7 surface expression was associated with high expression o

289 d this discrepancy revealed that the loss of surface expression was because of internalization, which

290 iculocytes was consistently reduced when CR1 surface expression was reduced through enzymatic cleavag

291 Reduced surface expression was reflected by smaller IPSCs, which

292 Integrin alphaVbeta3 surface expression was specifically upregulated in stimu

293 Clr-g surface expression was strongly upregulated on splenic C

294 of beta1-subunits, S-acylation promotes cell surface expression, whereas in its presence, S-acylation

295 -nAChR function principally by lowering cell-surface expression, whereas single-channel effects were

296 als that all six variants decreased receptor surface expression, which may underline some shared clin

297 ndent calcium channels, promoted alpha6beta4 surface expression while IRE1alpha, an unfolded protein

298 ing a mutant gamma2 subunit had reduced cell surface expression with altered subunit stoichiometry or

299 romised and differential alpha1 subunit cell surface expression with beta subunits resulting in sever

300 cific blocking of the proximal 3'UTR reduced surface expression without decreasing mRNA expression.