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1 nst multidrug-resistant Plasmodium yoelii in Swiss mice.
2 nized limit of toxicity to adult Sv128/Black Swiss mice.
3 g-resistant Plasmodium yoelii nigeriensis in Swiss mice.
4 C. rodentium- and DBZ-induced colitis in NIH:Swiss mice.
5 at affect high-frequency hearing loss in NIH Swiss mice.
6 nd fungal persistence in both BALB/c and NIH Swiss mice.
7 nd Staphylococcus aureus septic arthritis in Swiss mice.
8  between the X-receptors of M. dunni and NIH Swiss mice.
9 ro in SC-1 cells and in vivo in neonatal NIH-Swiss mice.
10 ers from BALB/C, CBA/J, C57BL/6, and outbred Swiss mice.
11 uconeogenic genes in the liver of young male Swiss mice.
12 lism and glycaemic homeostasis in young male Swiss mice.
13 d in GF in comparison with conventional (CV) Swiss mice.
14 cisional skin wounds by using germ-free (GF) Swiss mice.
15 aluated in vivo in a time series study using Swiss mice.
16 min on nineteen isoflurane-anesthetized male Swiss mice after femoral artery (n = 7), femoral vein (n
17 o study the role of microglia in drug abuse, Swiss mice aged 8-9 weeks were treated with the CSF1R in
18  peptide in 5-fluorouracil (5-FU)-challenged Swiss mice and IEC-6 cell monolayers.
19                                          NIH Swiss mice appear to be resistant in susceptibility to r
20 n that juvenile audiogenic seizures in Black Swiss mice are unlikely to be due to abnormalities of HC
21 rsus group housing) affects the phenotype of SWISS mice as measured by a range of behavioral and phys
22 ion was induced unilaterally in 13 NIH Black Swiss mice by laser photocoagulation of the limbus.
23 nst multidrug-resistant Plasmodium yoelii in Swiss mice by oral route.
24 N2 protein expression in the brains of Black Swiss mice, compared to control mice.
25 ined with AE (TRF+Exe) were compared in male Swiss mice fed a high-fat diet, with evaluation of the e
26 event weight gain and metabolic disorders in Swiss mice fed a high-fat diet.
27                   Here, we report that Black Swiss mice have a heretofore unrecognized specific susce
28  sequencing genomic DNA, we found that Black Swiss mice have a single polymorphism in exon 2 within t
29                Lymphomas were induced in NIH Swiss mice infected as neonates with tissue culture-prop
30 n parasitemia and increased survival time of Swiss mice infected with Plasmodium yoelii (strain N-67)
31  be activated to cause lymphoma, whereas NIH Swiss mice lack proviral sequences that can be expressed
32                                       Female Swiss mice lactating at 30 degrees C had lower asymptoti
33 essed MATSAP performance with videos of male Swiss mice moving in an open field box and in an elevate
34                                          NIH/Swiss mice neonatally inoculated with MoFe2 developed T-
35  neuroinvasiveness after i.p. inoculation of Swiss mice or the more permissive SCID mice with 10(5) o
36                In a masked study design, NIH Swiss mice received a 2-microL topical instillation of 0
37                        After anesthesia, NIH Swiss mice received intracameral injection of 1.5 microL
38 ction of TGF-beta and CTGF into neonatal NIH Swiss mice resulted in a large stimulation of granulatio
39       Furthermore, acute toxicity test using Swiss mice showed all mice survived without any signs of
40 ee other strains, the nu/nu, 129E, and Black Swiss mice, showed in most parameters intermediate pheno
41 ration of lactoferrin into S aureus-infected Swiss mice significantly suppressed paw inflammation and
42 21 degrees C and 5 degrees C suggest that in Swiss mice sustained energy intake (SusEI) and reproduct
43 r (2 log) initial infectivity in newborn NIH Swiss mice than that of wild-type virus, and revertants
44 ring the third month of the study, a few NIH Swiss mice that died had granulosa cell tumors of the ov
45 gh frequency hearing loss (HFHL) line of NIH Swiss mice to three different inbred strains and perform
46 d for 30 days in wild-type and Msx2 knock-in Swiss mice using Porphyromonas gingivalis infected ligat
47 ) infection in thymus and bone marrow of NIH/Swiss mice was performed to assess changes that occur du
48 48 hr postischemia, renal dysfunction in NIH Swiss mice was significantly reduced, compared with othe
49 al mucosa from National Institutes of Health Swiss mice was stripped of seromuscular layers and mount
50                                              Swiss mice were acutely administered by oral route (p.o.
51                                     Male NIH Swiss mice were exposed to HBO2 (100% oxygen at 3.5atm a
52                                              Swiss mice were fed a HFD (59% kcal from fat) or standar
53                                      Outbred Swiss mice were infected with the E3 mutant viruses to d
54                                 Neonatal NIH/Swiss mice were inoculated intraperitoneally with ca. 10
55                                 Neonatal NIH/Swiss mice were inoculated subcutaneously (s.c.) with wi
56                     Healthy corneas of Black Swiss mice were inoculated with 10(7) or 10(9) CFU of in
57  chambers of the eyes of 64 anesthetized NIH Swiss mice were perfused with various fluorescent dextra
58                                     Male NIH Swiss mice were subjected to a 5-min HBO(2) treatment (1
59             In these studies, adult male NIH Swiss mice were trained to discriminate 0.3 mg/kg MDPV f
60                                          ICR Swiss mice were treated with the following protocols: 1)
61                             Twenty-two black Swiss mice were used.
62                               Studying black Swiss mice, which inherently exhibit proclivity for rewa
63             Nonetheless, i.p. inoculation of Swiss mice with 10 or 10(3) PFU of either chimeric virus
64 combinant viruses following infection of NIH/Swiss mice with the murine retrovirus SL3-3 murine leuke