ライフサイエンスコーパス: synaptic potential

1 ance, leading to a striking reshaping of the synaptic potential.

2 threshold for action potential generation by synaptic potential.

3 ne potential and the temporal integration of synaptic potentials.

4 ficant increase in the field excitatory post-synaptic potentials.

5 ed by the timing of arriving signals such as synaptic potentials.

6 ritic space constant for the auditory-evoked synaptic potentials.

7 ynamics, spontaneous firing, and tone-evoked synaptic potentials.

8 ol also displayed large-amplitude inhibitory synaptic potentials.

9 ynaptic currents to generate rapid and small synaptic potentials.

10 5-HT inhibited spontaneous synaptic potentials.

11 es may contribute to repolarization of large synaptic potentials.

12 over which they effectively summate incoming synaptic potentials.

13 was followed by a suppression of inhibitory synaptic potentials.

14 partments but might also electrically modify synaptic potentials.

15 cellular excitability and the integration of synaptic potentials.

16 onsiderable complexity to the integration of synaptic potentials.

17 tional barrages of excitatory and inhibitory synaptic potentials.

18 ptic inputs from other neurons by generating synaptic potentials.

19 t barrages of both excitatory and inhibitory synaptic potentials.

20 nction is to generate outputs in response to synaptic potentials.

21 bitory barrages and narrower visually evoked synaptic potentials.

22 ) as a function of the amplitude of the post-synaptic potentials.

23 membrane potential, evoked quantal release, synaptic potentials, acetylcholine receptor channel kine

24 -B synaptic potentials and suppressed single synaptic potentials after the cessation of the artificia

25 followed by persistent synaptic depression (synaptic potential amplitude reduced by 22% at 30 min).

26 ficant decrease of the field excitatory post-synaptic potentials amplitude in 60-day animals, but not

27 riable depolarization that summates with the synaptic potential and can act as a trigger for action p

28 ar Ca(2+) transients, associated with the CF synaptic potential and colocalized with the PF Ca(2+) in

29 roach capable of resolving sequentially both synaptic potentials and action potential firing in large

30 ording with the ability to detect both small synaptic potentials and action potentials, we developed

31 e found in the spine, resulting in increased synaptic potentials and Ca transients.

32 We measured synaptic potentials and complex spike signals across the

33 late currents) and the kinetic properties of synaptic potentials and currents were similar to control

34 at spines have an electrical role, filtering synaptic potentials and electrically isolating inputs fr

35 fluid percussion injury (FPI) on hippocampal synaptic potentials and excitability.

36 Spine sodium channels could boost synaptic potentials and facilitate action potential back

37 pond to stimuli by integrating generator and synaptic potentials and generating action potentials.

38 in synaptic potency reduced the duration of synaptic potentials and limited temporal summation of af

39 esis of l-and d-serine and reduced the NMDAR synaptic potentials and long-term potentiation (LTP) at

40 ndrites modulate the summation of excitatory synaptic potentials and prevent the generation of dendri

41 intrinsic jitter in spike triggering, evoked synaptic potentials and spikes had surprisingly similar

42 synaptic transmission, properties of evoked synaptic potentials and spontaneous synaptic currents we

43 attern: DSI strongly enhanced summated VSI-B synaptic potentials and suppressed single synaptic poten

44 ce of sodium action potential (AP)-dependent synaptic potentials and synaptic currents in >50% of the

45 bserved a monotonic decline in extracellular synaptic potentials and their initial slope over the tem

46 eurons minimizes spike-induced distortion of synaptic potentials and thus preserves the accuracy of b

47 Some neurons also showed modulation of synaptic potentials and/or spike firing locked to the os

48 lutamate receptors (mGluRs) can modify field-synaptic-potentials and protein kinase activity in rat a

49 d excitatory components of the bidirectional synaptic potentials are dependent upon AMPA (GluA) recep

50 isting electrophysiological data on group Ia synaptic potentials are discussed.

51 he depolarizing drive, and the amplitudes of synaptic potentials are generally thought to reflect the

52 Synaptic potentials are likely amplified at the spine he

53 Furthermore, NMDAR synaptic potentials are preserved in SR-KO mice and are

54 . find that spontaneously released miniature synaptic potentials arise from a pool of vesicles distin

55 Many of these synaptic potentials arise from the activity of nearby ne

56 e translation of propriosensory signals into synaptic potentials as a result of defective mechanisms

57 primarily onto their dendrites, which filter synaptic potentials as they spread toward the soma.

58 ctive desynchronization reduces summation of synaptic potentials at target AOS cells and thus provide

59 Synaptic potentials attenuate dramatically, however, as

60 Simulations suggest that synaptic potentials attenuate enormously as they propaga

61 the presence of 1% halothane, the excitatory synaptic potential between VD4 and the PeA(E) cells was

62 completely abolished, whereas the inhibitory synaptic potential between VD4 and the PeA(I) cells was

63 he first characterization of visually evoked synaptic potentials between the visual thalamus and visu

64 The processing of synaptic potentials by neuronal dendrites depends on bot

65 Two recent studies now show that synaptic potentials can also spread far down the axon an

66 om CA3 neurons revealed bursts of excitatory synaptic potentials coincident with the network bursts r

67 larly with higher frequency signals (such as synaptic potentials compared with steady-state signals),

68 ynaptic transmission such as multi-component synaptic potentials, cotransmission, and neuromodulation

69 s in the auditory cortex of mice and measure synaptic potentials/currents, membrane potentials, and s

70 0 sec after DSI, then the amplitude of VSI-B synaptic potentials decreased.

71 dulla that receive increased excitatory post-synaptic potentials during inspiration, but also have sp

72 were recorded as field potentials and focal synaptic potentials during systematic exploration of the

73 ntrast, mono- and disynaptic excitatory post-synaptic potentials elicited from the medial longitudina

74 trains of brief, fast-rising excitatory post-synaptic potentials (EPSCs) to the model.

75 c but not dendritic post-synaptic excitatory synaptic potential (EPSP) integration in CA1 pyramidal c

76 sible reduction of the field excitatory post-synaptic potential (EPSP) slope in response to low-frequ

77 After 6 hr to 12 d of survival, synaptic potentials (EPSP) elicited by two synaptic inpu

78 and increased amplitudes of excitatory post-synaptic potentials (EPSPs) in adult rat hippocampal sli

79 All showed monosynaptic excitatory post-synaptic potentials (EPSPs) to both TMS and TES with no

80 d at layer 6 (L6), whereas evoked excitatory synaptic potentials (EPSPs) were recorded from L5 neuron

81 in long-term depression (LTD) of excitatory synaptic potentials (EPSPs), long-term decrease of popul

82 tic response, slope of field excitatory post-synaptic potentials (EPSPs), or the input/output functio

83 spiking, and slowed decay of excitatory post-synaptic potentials (EPSPs).

84 Roundup depressed excitatory synaptic potentials(EPSPs) in a dose-dependent manner wi

85 h the same reversal potential as that of the synaptic potential evoked by cortical stimulation.

86 (15 microM) facilitated the excitatory field synaptic potential evoked in the BLA by stimulation of t

87 (10 microM) facilitated the excitatory field synaptic potential evoked in the BLA by stimulation of t

88 DSI spike trains heterosynaptically enhanced synaptic potentials evoked by another CPG neuron, ventra

89 electrodes in the accumbens shell to record synaptic potentials evoked by electrical stimulation of

90 about half the cells demonstrated excitatory synaptic potentials evoked by extracellular stimulation.

91 heart muscle contractility, or amplitudes of synaptic potentials evoked by stimulating postganglionic

92 PSPs occurred coincident with fast nicotinic synaptic potentials evoked by the myenteric projections

93 Synaptic potentials evoked by the same stimulus intensit

94 sensory maps to synaptogenesis, we recorded synaptic potentials evoked by whisker deflection in laye

95 ressed as smaller than normal stretch-evoked synaptic potentials evoked monosynaptically at Ia-motone

96 axonal pattern of arborization, spontaneous synaptic potentials, evoked postsynaptic currents, patte

97 We tested this by recording CST-evoked focal synaptic potentials, extracellularly, in the cervical en

98 a lasting increase in field excitatory post-synaptic potential (fEPSP) slope in area CA1 following t

99 only small effects on field excitatory post-synaptic potentials (fEPSP) in slices from 60-day animal

100 , by monitoring evoked field excitatory post-synaptic potentials (fEPSP), and slice nicotinamide aden

101 Recordings of hippocampal field excitatory synaptic potentials (fEPSPs) showed that prenatal exposu

102 Field excitatory post-synaptic potentials (fEPSPs) were recorded in stratum ra

103 7 per 1 mV in a 1-kHz bandwidth (n = 42 post-synaptic potentials from 5 cells, data are mean +/- s.d.

104 Summation of small synaptic potentials from many fibers is required to brin

105 Preventing synaptic potentials from overshooting their optimal rang

106 viding 122 regions of maximum negative focal synaptic potentials (FSPs) of mean amplitude 7.51 microV

107 ement of terminal potentials (TPs) and focal synaptic potentials (FSPs), recorded with spike-triggere

108 Inhibitory synaptic potentials generated from single interneurons a

109 action potential duration, dampen excitatory synaptic potentials, impede temporal summation, and rais

110 indicating that they represent synchronized synaptic potentials impinging on layer II-III neurons.

111 Glutamate produces a hyperpolarizing synaptic potential in On bipolar cells by binding to the

112 A pharmacologically isolated excitatory synaptic potential in the superficial layers depended on

113 activated during subthreshold glutamatergic synaptic potentials in a principal cell generate an exci

114 nd patch-clamp recordings of inhibitory post synaptic potentials in an L2-3 neuron induced by electri

115 eir activity reduces the amplitude of evoked synaptic potentials in an NMDA receptor (NMDAR)-dependen

116 sed Ih in corticocollicular neurons, whereas synaptic potentials in L2/3-->corticocallosal connection

117 Synaptic potentials in L2/3-->corticocollicular connecti

118 re the predominant entry site for excitatory synaptic potentials in most types of central neurons.

119 axonal conduction velocity and elicit faster synaptic potentials in Purkinje cells.

120 ns in the sensory layers revealed excitatory synaptic potentials in response to glutamate application

121 hese models we have recorded visually evoked synaptic potentials in simple cells while cooling the co

122 ide but not 9-phenanthrol reduced excitatory synaptic potentials in slices from EAE mice, an effect t

123 enhances dendritic integration of excitatory synaptic potentials in SPNs and enhances - not antagoniz

124 ion while recording the resulting volley and synaptic potentials in target motoneurons.

125 ewed as occurring through the interaction of synaptic potentials in the dendrite and soma, followed b

126 The recordings of field excitatory post-synaptic potentials in the hippocampal CA1 subfield demo

127 When rats learn a motor skill, synaptic potentials in the motor cortex are enhanced.

128 agonists depressed the field excitatory post-synaptic potentials in the SC-CA1 pathway, but had no si

129 ciated Ca(2+) influx following a train of PF synaptic potentials in two cases: (1) when the dendritic

130 rrents in dorsal horn neurons and population synaptic potentials in ventral roots provided evidence t

131 Our characterization of synaptic potentials in vivo disagreed with prior finding

132 r optimal range requires both SKIP to temper synaptic potentials (incoherent feedforward loop) and mi

133 the facilitating effects of field excitatory synaptic potential induced by the agonist were reduced t

134 the facilitating effects of field excitatory synaptic potential induced by the agonist were reduced t

135 tes, where they are important for regulating synaptic potential integration and plasticity.

136 pyramidal neurons modulate spike firing and synaptic potential integration by influencing the membra

137 ) can be mediated by GABA(B) inhibitory post-synaptic potentials (IPSPs) or slow after-hyperpolarizat

138 Inhibitory synaptic potentials (IPSPs) originating from nRt cells a

139 Presumed inhibitory synaptic potentials (IPSPs) recorded from principal cell

140 Inhibitory post-synaptic potentials (IPSPs) were studied in neurons of p

141 Since HCN1 channels also affect post-synaptic potential kinetics and integration, our results

142 Focal synaptic potentials (mean maximal amplitudes 135-200 mic

143 that intrinsic properties can override fast synaptic potentials, mediate circuit reconfiguration, an

144 However, ephedrine did not reduce fast synaptic potentials mediated by GABAA or ionotropic glut

145 Electrically evoked inhibitory synaptic potentials mediated by GABAB receptors were red

146 supraoptic nucleus (SON) were used to study synaptic potentials mediated by nicotinic acetylcholine

147 Both kinds of synaptic potentials occurred in neurons with uniaxonal m

148 Synaptic potentials originating at distal dendritic loca

149 to the presynaptically mediated decreases in synaptic potentials previously demonstrated in vitro wit

150 rmation and its circuit translation into the synaptic potentials produced in motoneurons.

151 with a foot-shock resulted in enhanced post-synaptic potential (PSP) responses to the odour and incr

152 Post-synaptic potential (PSP) variability is typically attrib

153 DeltaF/F) and SNR of optically recorded post-synaptic potentials (PSPs) as a function of the amplitud

154 Once the summated synaptic potentials reach threshold for action potential

155 induced a transient depression of the evoked synaptic potential recorded in the rat hippocampal CA1 r

156 In addition, primary afferent-evoked synaptic potentials recorded in a subpopulation of lamin

157 ron somata, the amplitude of afferent-evoked synaptic potentials recorded in motor neurons was reduce

158 ent 1.7-fold enhancement in the amplitude of synaptic potentials recorded intracellularly in basolate

159 urons well beyond the duration of excitatory synaptic potentials resulting from activation of the tri

160 ify dendritic intrinsic excitability as well synaptic potential shapes and integration.

161 Examination of the post-synaptic potential size has suggested, however, that sum

162 pulation spike amplitude and excitatory post-synaptic potential slope compared to control mice.

163 eshold, enhancing intrinsic excitability and synaptic potential-spike coupling.

164 t, the effect of somatic K(V)7/M-channels on synaptic potential summation was dependent upon the neur

165 rd high-frequency bursting activity and slow synaptic potentials that are hard to resolve by multicel

166 These oscillations reflect synchronized synaptic potentials that entrain the discharge of neuron

167 ansmit visual information by means of graded synaptic potentials that spread to the synaptic terminal

168 terconnections can generate giant excitatory synaptic potentials that support the bursting behaviour

169 als generated, but also the amplitude of the synaptic potentials that these action potentials initiat

170 developing and sustained depression of field synaptic potentials that was quantitatively similar in b

171 lso drastically decreases the isolated NMDAR synaptic potentials, these responses are still enhanced

172 y of the pyramidal neurons and the spread of synaptic potentials throughout the dendrites.

173 gic agonist carbachol suppressed each of the synaptic potentials to different degrees.

174 rons increases excitability and enables fast synaptic potentials to drive reliable firing.

175 es the resistance of Schaffer collateral-CA1 synaptic potentials to further, longer periods of hypoxi

176 1-43 imaging and intracellular recordings of synaptic potentials to measure the time course of endocy

177 ocytosis), intracellular recording of evoked synaptic potentials (to measure acetylcholine release),

178 the detection of either action potentials or synaptic potentials, to follow high versus low rates of

179 ious components of excitatory and inhibitory synaptic potentials, using intracellular current- and vo

180 of EtOH, the amplitude of dorsal root-evoked synaptic potentials was reduced, the frequency of sponta

181 ed, temporal summation at the soma of distal synaptic potentials was similar to that seen with curren

182 To achieve this, synaptic potential were evoked monosynaptically by near

183 depolarization persisted in TTX or when fast synaptic potentials were blocked, indicating that it rep

184 sociated with spatially uniform subthreshold synaptic potentials were comparable but not equal in amp

185 Synaptic potentials were evoked either by auditory or el

186 an early (2 min) consequence of OGD, evoked synaptic potentials were lost, and K, Cl, P, and Mg conc

187 Synaptic potentials were recorded from developing rat NM

188 During spontaneous episodes, rhythmic synaptic potentials were recorded from the VLF and from

189 he lateral geniculate nucleus (LGN) generate synaptic potentials, which sometimes produce spikes sent

190 with tetrodotoxin and by blocking inhibitory synaptic potentials with bicuculline and strychnine.

191 scillations consisted of compound inhibitory synaptic potentials with initial IPSPs with slow kinetic

192 mma-aminobutyric acidergic and glutamatergic synaptic potentials with moderate neuronal firing.

193 to the spinal cord elicited Ca(2+)-dependent synaptic potentials with short latencies in reticulospin

194 Theory predicts that nonlinear summation of synaptic potentials within dendrites allows neurons to p