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1 l skin inflammation at the injection site to systemic anaphylaxis.
2 sed using a mouse model of acute allergy and systemic anaphylaxis.
3 rgic reactions and inflammation and prevents systemic anaphylaxis.
4 hen challenged with peanut extract to induce systemic anaphylaxis.
5 lated to excessive vascular leakage, such as systemic anaphylaxis.
6 ive cutaneous anaphylaxis and protected from systemic anaphylaxis.
7 activity in an animal model of cutaneous and systemic anaphylaxis.
8 asses have been shown to enable induction of systemic anaphylaxis.
9 says ex vivo and in a mouse model of passive systemic anaphylaxis.
10 a leakage syndromes, including angioedema or systemic anaphylaxis.
11 f IgG1-, IgG2a-, and IgG2b-dependent passive systemic anaphylaxis.
12 are less sensitive to IgE-mediated, passive, systemic anaphylaxis.
13 ivo, and ROCK inhibitors protect from lethal systemic anaphylaxis.
14  that PAG is a positive regulator of passive systemic anaphylaxis.
15 tion and to ovalbumin (OVA) allergen induced systemic anaphylaxis.
16  airway inflammation, and active and passive systemic anaphylaxis.
17 esponsible for FcgammaRIIA-dependent passive systemic anaphylaxis.
18 ct OX40L contribution in an in vivo model of systemic anaphylaxis.
19 was demonstrated in a mouse model of passive systemic anaphylaxis.
20 on, and do not develop IgE-dependent passive systemic anaphylaxis.
21 axis in the ear and joint as well as passive systemic anaphylaxis.
22 osal tissues, thereby minimizing the risk of systemic anaphylaxis.
23 ntly greater and faster death rate in active systemic anaphylaxis.
24 gy, food allergy, conjunctivitis, and severe systemic anaphylaxis.
25  mice were resistant to IgE-mediated passive systemic anaphylaxis.
26 ce in both active and IgG1-dependent passive systemic anaphylaxis.
27 nists were used in a murine model of passive systemic anaphylaxis.
28 ining their fates in mice undergoing passive systemic anaphylaxis.
29 onRIalpha)-transgenic mouse model of passive systemic anaphylaxis.
30  human intravenous immunoglobulin and active systemic anaphylaxis after immunization and challenge.
31 he jejunum, increased serum IL-4 levels, and systemic anaphylaxis after oral challenge, as evidenced
32 ated pathophysiology in IgE-mediated passive systemic anaphylaxis and acute psychological restraint s
33 ary for the development of passive local and systemic anaphylaxis and airway inflammation.
34 and S1P(2) on mast cells are determinants of systemic anaphylaxis and associated pulmonary edema and
35 ine the capabilities of hFcgammaRs to induce systemic anaphylaxis and identify the cell types and med
36 ed that the IgG-mediated suppression of both systemic anaphylaxis and MC-driven tissue recruitment of
37 on FcepsilonRI-MC-mediated passive local and systemic anaphylaxis and on airway inflammation.
38 n shown to mediate an alternative pathway of systemic anaphylaxis and to participate in allergic skin
39 ergies, urticaria, nonhereditary angioedema, systemic anaphylaxis, and allergic conjunctivitis are as
40 sposes individuals to develop asthma, severe systemic anaphylaxis, and atopic dermatitis, is usually
41 cytopenia, neutrophils to be responsible for systemic anaphylaxis, and both cell types to be responsi
42 d IgE-mediated histamine release and passive systemic anaphylaxis, and Drebrin(-/-) mast cells also e
43 re known for their roles in allergy, asthma, systemic anaphylaxis, and inflammatory disease.
44 humanized mouse model, reducing IgE-mediated systemic anaphylaxis, and inhibits airway tryptase in As
45  Furthermore, in a low-dose model of passive systemic anaphylaxis, antigen-dependent decrease in body
46 rgic reactions such as passive cutaneous and systemic anaphylaxis are markedly impaired in Gab2-/- mi
47 d with PN, they exhibited mast-cell-mediated systemic anaphylaxis, as indicated by hypothermia and in
48  than their normal counterparts to a passive systemic anaphylaxis challenge.
49        We recently found in mice that active systemic anaphylaxis depends on IgG and IgG receptors (F
50                          In a mouse model of systemic anaphylaxis, fluoxetine reduced hypothermia and
51                     Mice with PSA and active systemic anaphylaxis had increased Fn14 and TWEAK expres
52  surface IgE and triggered degranulation and systemic anaphylaxis in allergen-naive WT but not in IL-
53 ation, IgE production by B cells and passive systemic anaphylaxis in an in vivo mouse model, ligelizu
54 t binding sites were able to mediate passive systemic anaphylaxis in FCepsilonRI transgenic mice.
55 phylaxis, and attenuated dansyl IgE-mediated systemic anaphylaxis in human FcepsilonRIalpha transgeni
56 aphylaxis; and attenuate dansyl IgE-mediated systemic anaphylaxis in human FcepsilonRIalpha transgeni
57                           Studies of passive systemic anaphylaxis in IL-10-transgenic mice showed tha
58                              We confirm that systemic anaphylaxis in mice can be mediated largely thr
59  and basophil activation, and severe passive systemic anaphylaxis in mice humanized for the IgE recep
60 granulated poorly in vitro and did not evoke systemic anaphylaxis in mice.
61  of an IgG1-dependent alternative pathway of systemic anaphylaxis in mice.
62 sensus guidelines for clinically recognizing systemic anaphylaxis in real time, regardless of whether
63 peanut allergy, as demonstrated by acute and systemic anaphylaxis in response to challenge with peanu
64  sensitized to exhibit IgE-dependent passive systemic anaphylaxis in vivo and to investigate the anti
65  cell degranulation and efficiently inhibits systemic anaphylaxis in vivo.
66 vascular hyperpermeability (AVH) and passive systemic anaphylaxis-induced hypothermia and edema.
67        Also, in 2 different models of active systemic anaphylaxis, inhibition of NOS, PI3K, or Akt or
68                                              Systemic anaphylaxis is generally recognized as a severe
69 ut neither IgG1- nor IgG2b-mediated, passive systemic anaphylaxis led to Ag retention in the lung.
70 d this was accompanied by down-regulation of systemic anaphylaxis mediators such as histamine and mas
71 g an AD-HIES mutation were hyporesponsive to systemic anaphylaxis models.
72 rum tryptase level is associated with severe systemic anaphylaxis, most notably caused by Hymenoptera
73                                         In a systemic anaphylaxis mouse model, single-dose administra
74 in passive cutaneous anaphylaxis and passive systemic anaphylaxis mouse models, and in ovalbumin-indu
75                                              Systemic anaphylaxis occurred in Was(-/-) mice (95%) wit
76  anti-IgE-CD33L suppresses anti-IgE-mediated systemic anaphylaxis of sensitized humanized mice and pr
77 ha-gal-specific IgE and IgG(1) and developed systemic anaphylaxis on challenge with both alpha-gal-co
78 nt specific IgE-mediated responses and drive systemic anaphylaxis on ingestion challenge.
79 endent degranulation of human mast cells and systemic anaphylaxis on peanut allergen challenge in hum
80 ral antigen dose determine whether diarrhea, systemic anaphylaxis, or both are induced, and ingested
81 lar permeability and mouse models of passive systemic anaphylaxis (PSA) and active systemic anaphylax
82  MrgprB2 contributes to IgE-mediated passive systemic anaphylaxis (PSA) in vivo, an MRGPRX2 inhibitor
83                                      Passive systemic anaphylaxis (PSA) induced HDAC3 expression and
84         Furthermore, antigen-induced passive systemic anaphylaxis (PSA) was analysed in PEP(+/+) and
85 aling in the context of IgG-mediated passive systemic anaphylaxis (PSA).
86 deficiency in mice leads to enhanced passive systemic anaphylaxis, reflected by increased drop in bod
87 lphaT was associated with increased risk for systemic anaphylaxis (relative risk = 9.5; P = .007).
88 t HaT was associated with increased risk for systemic anaphylaxis (relative risk = 9.5; P = .007).
89                       Mouse models of active systemic anaphylaxis rely predominantly on IgG Abs formi
90  and profound depletion of plasma S1P during systemic anaphylaxis rendered both platelet- and erythro
91  a human mast cell antigen-dependent passive systemic anaphylaxis response in primed mice.
92 ficacy, and mouse models of ear swelling and systemic anaphylaxis responses for in vivo efficacy.
93 d mast cell granularity and impaired passive systemic anaphylaxis responses.
94             IgG can mediate murine and human systemic anaphylaxis (SA).
95 tations of food allergy include diarrhea and systemic anaphylaxis (shock), which can occur together o
96                 Our in vivo model of passive systemic anaphylaxis showed that the residual exocytic f
97 of passive cutaneous anaphylaxis and passive systemic anaphylaxis that can be used to investigate the
98 allergen-specific IgG confers sensitivity to systemic anaphylaxis that relies on IgG Fc receptors (Fc
99 h trinitrophenyl-BSA intravenously to induce systemic anaphylaxis that was monitored by using rectal
100 n smooth muscle cells, also leads to reduced systemic anaphylaxis, the most severe form of allergic r
101                  In response to IgE-mediated systemic anaphylaxis, the symptoms and decreases in core
102 ce showed excellent responses during passive systemic anaphylaxis using human IgE to selectively evok
103                               Intestinal and systemic anaphylaxis was assessed, and the role of the h
104                                      Passive systemic anaphylaxis was induced by injection of heat-ag
105                                       Active systemic anaphylaxis was induced following immunization
106                                      Passive systemic anaphylaxis was induced in female and male mice
107                           Passive and active systemic anaphylaxis was induced, respectively, by injec
108                                  Severity of systemic anaphylaxis was measured by changes in body tem
109                                   A model of systemic anaphylaxis was studied in transgenic mice carr
110                                In a model of systemic anaphylaxis, we found no difference between WT
111 a of V3 mastocytosis mice undergoing passive systemic anaphylaxis, we used this in vivo model system
112 assive systemic anaphylaxis (PSA) and active systemic anaphylaxis were applied to wild-type (WT), TWE
113 ed passive cutaneous anaphylaxis and passive systemic anaphylaxis were reduced in NHERF1(+/-) mice an
114 th ovalbumin; models of allergic diarrhea or systemic anaphylaxis were studied.
115  has been proven to be IgE-independent acute systemic anaphylaxis, which may due to IgG immune comple

 
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