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1 of six eyes presenting with a PUK and proven systemic vasculitis.
2 tion in rheumatic diseases including SLE and systemic vasculitis.
3 ly reported but poorly understood paediatric systemic vasculitis.
4 involvement, and is more often a form of non-systemic vasculitis.
5 zing ocular signs as potential indicators of systemic vasculitis.
6 al blood eosinophilia and symptoms caused by systemic vasculitis.
7 granulomatosis with polyangiitis, a form of systemic vasculitis.
8 se features and treatments of these forms of systemic vasculitis.
9 e, and tolerable therapies for children with systemic vasculitis.
10 recommendations in the management of primary systemic vasculitis.
11 ease should be integral to the management of systemic vasculitis.
12 vascular morbidity for children with primary systemic vasculitis.
13 in finding other therapies to treat primary systemic vasculitis.
14 tis is defined within the context of primary systemic vasculitis.
15 the use of biologic agents as treatments for systemic vasculitis.
16 of endothelial injury during ANCA-associated systemic vasculitis.
17 i-neutrophil cytoplasmic antibody-associated systemic vasculitis.
18 (ANCA) are implicated in the pathogenesis of systemic vasculitis.
19 e response from tolerance to autoimmunity in systemic vasculitis.
20 (ANCA) are implicated in the pathogenesis of systemic vasculitis.
21 play an important role in clinical trials of systemic vasculitis.
22 dels have been proposed for various forms of systemic vasculitis.
23 of morbidity and mortality in patients with systemic vasculitis.
24 arious infectious syndromes in patients with systemic vasculitis.
25 und in the circulation of many patients with systemic vasculitis.
26 rkedly improved the outcome in patients with systemic vasculitis.
27 sa (PAN) has been used as a generic term for systemic vasculitis.
28 t not the ACR criteria for any other type of systemic vasculitis.
29 nuate the consequences of CCE like that with systemic vasculitis.
30 al tract, peripheral nerves, and skin due to systemic vasculitis.
32 on included connective tissue disease (38%), systemic vasculitis (36%), and autoimmune cytopenia (22%
33 ), systemic autoimmune disorders (7472), and systemic vasculitis (6283) and in 373 851 matched contro
34 ovasculitis was significantly related to non-systemic vasculitis (71% versus 23%, odds ratio, 7.9; 95
35 tineutrophil cytoplasmic antibody-associated systemic vasculitis (AASV) as a model of systemic inflam
36 i-neutrophil cytoplasmic antibody-associated systemic vasculitis (AASV) as a model of systemic inflam
37 trophil cytoplasm antibody (ANCA)-associated systemic vasculitis (AASV) remain limited and hampered b
38 tineutrophil cytoplasmic antibody-associated systemic vasculitis (AASV), a disease not associated wit
39 They are common in patients with primary systemic vasculitis and are seen in vasculitis secondary
40 teinase-3 (PR3) antibodies in development of systemic vasculitis and granulomatosis with polyangiitis
41 periodontitis, inflammatory bowel diseases, systemic vasculitis and HIV infection, as well as chemot
42 tant role in vascular injury associated with systemic vasculitis and in models of autoantibody- and i
43 inicians should perform extended work-up for systemic vasculitis and keep a high index of suspicion f
44 rats induced marked autoantibody production, systemic vasculitis and lymphocytic infiltration of the
45 in 88% of patients with antibody-associated systemic vasculitis and permitted reduction in steroid d
46 ion underscores the importance to assess for systemic vasculitis and to maintain high suspicion for i
47 tinguish it from atherosclerotic AAA or from systemic vasculitis and to treat it with the appropriate
48 of agents commonly used in the treatment of systemic vasculitis, and 2) a framework for the diagnost
49 n important complication of several forms of systemic vasculitis, and it may result in significant mo
50 Autoimmune rheumatic diseases may present as systemic vasculitis, and systemic vasculitis may simulat
51 of this activity have long been attempted in systemic vasculitis, and, more recently, in atherosclero
52 cent developments in the nosology of primary systemic vasculitis are placed in the context of an hist
55 granulomatosis with polyangiitis (EGPA) is a systemic vasculitis associated with chronic rhinosinusit
56 tion of hepatitis C virus (HCV) infection is systemic vasculitis associated with type II cryoglobulin
57 The study cohort comprised 39 children with systemic vasculitis at various stages of disease activit
58 to an unselected cohort of 130 patients with systemic vasculitis attending a single district hospital
59 scopic polyangiitis is a subgroup of primary systemic vasculitis, but diagnostic problems remain with
60 tis, are the connective tissue disorders and systemic vasculitis, but systemic infections and maligna
61 e-phase reactant levels in the patients with systemic vasculitis, but there was a poor correlation ov
63 ents with a new diagnosis of ANCA-associated systemic vasculitis confirmed by renal biopsy and serum
65 e specific factors involved in thrombosis in systemic vasculitis could help explain the role of infla
66 is affecting the nervous system includes (1) Systemic vasculitis disorders (necrotizing arteritis of
67 expression on neutrophils from patients with systemic vasculitis, Fc gammaRIIIb is likely to play a c
69 ments in the classification and treatment of systemic vasculitis have led the European League Against
70 Temporal arteritis, the most common form of systemic vasculitis in adults, is a panarteritis that ch
71 ell arteritis (GCA), the most common form of systemic vasculitis in adults, preferentially involves l
72 ayasu arteritis) are the most common form of systemic vasculitis in adults, they remain incompletely
77 ience with these agents for the treatment of systemic vasculitis is with antitumor necrosis factor ag
78 ene (previously known as CECR1) results in a systemic vasculitis known as deficiency of ADA2 (DADA2).
80 tis can occur in various conditions, such as systemic vasculitis like Anti-Neutrophil Cytoplasmic Ant
81 ry information suggesting that the impact of systemic vasculitis may be significant and far greater t
83 ases may present as systemic vasculitis, and systemic vasculitis may simulate autoimmune rheumatic di
85 och Schonlein Purpura (HSP) is the commonest systemic vasculitis of childhood typically presenting wi
89 MC clinical manifestations are determined by systemic vasculitis of low/medium sized vessels (mixed c
90 saki disease is an acute febrile illness and systemic vasculitis of unknown aetiology that predominan
92 upus erythematosus (SLE) and 4 patients with systemic vasculitis on the expression of adhesion molecu
93 bsence of clinical or laboratory evidence of systemic vasculitis or a condition predisposing to such,
94 sitive SLE patients and 3 of 3 AECA-positive systemic vasculitis patients up-regulated adhesion molec
96 ng non-systemic vasculitic neuropathy have a systemic vasculitis presenting with neuropathy; in other
98 alignant hypertension, may signal underlying systemic vasculitis rather than isolated hypertensive da
101 erived from the largest genetic study of any systemic vasculitis, suggest a central role of CCR5-CCL3
104 uppressive treatments for conditions such as systemic vasculitis, systemic lupus erythematosus, or ma
107 hcet's disease (BD) is a chronic, relapsing, systemic vasculitis that can involve both arteries and v
108 l arteritis (GCA) is the most common form of systemic vasculitis that causes visual loss in the elder
109 he rate of renal recovery in ANCA-associated systemic vasculitis that presented with renal failure wh
111 ally based assessment of disease activity in systemic vasculitis, the Vasculitis Integrated Assessmen
112 icroparticle (PMP) profiles in children with systemic vasculitis to test the hypothesis that EMPs may
113 e 1990 ACR criteria and CHCC definitions for systemic vasculitis were applied to an unselected cohort
114 of tolerance to neutrophil components during systemic vasculitis, which is marked by autoantibodies a
116 lomatosis with polyangiitis (EGPA) is a rare systemic vasculitis with a prevalence rate of seven per