ライフサイエンスコーパス: systolic pressure

1 than or equal to two-thirds of the systemic systolic pressure.

2 ic dimension, LV mass, and right ventricular systolic pressure.

3 ht ventricular afterload by pulmonary artery systolic pressure.

4 Sildenafil had no effect on pulmonary artery systolic pressure.

5 blood pressure as well as right ventricular systolic pressure.

6 on abdominal aortic aneurysm than did raised systolic pressure.

7 ely 60% of the increase in right ventricular systolic pressure.

8 re, diastolic pressure, and left ventricular systolic pressure.

9 y vascular remodeling, and right ventricular systolic pressure.

10 reased systemic as well as right ventricular systolic pressure.

11 elated with an increase in right ventricular systolic pressure.

12 200 mug/kg) decreased right ventricular peak systolic pressure.

13 A secondary endpoint was nocturnal dip in systolic pressure.

14 h reversible increments of right ventricular systolic pressure.

15 PAH as judged by elevated right ventricular systolic pressure.

16 enous iloprost in reducing right ventricular systolic pressure.

17 ure with treatment but greater reductions in systolic pressure.

18 ated with elevation of the right ventricular systolic pressure.

19 rate lowering consistently increases central systolic pressure.

20 ontrol were unchanged after adjusting for RV systolic pressure.

21 fference between the left and right brachial systolic pressures.

22 nce of methods to quantify right ventricular systolic pressures.

23 0.4 to 23.6 + or - 0.4 kg), left ventricular systolic pressure (137.0 + or - 3.4 to 124.0 + or - 6.7

24 DASH/SRD reduced clinic and 24-hour brachial systolic pressure (155 +/- 35 to 138 +/- 30 and 130 +/-

25 Patients with hypertension (systolic pressure 160-200 mm Hg; diastolic pressure <110

26 ne, patients had severe hypertension (aortic systolic pressure, 176+/-26 mm Hg), pulmonary hypertensi

27 1% (P < 0.01) and increased pulmonary artery systolic pressure (19.6 +/- 4.3 vs. 26.0 +/- 5.4, P < 0.

28 nd 53%, respectively), and right ventricular systolic pressure (32 +/- 11, 45 +/- 15, and 50 +/- 14 m

29 th a 46% reduction in right ventricular peak systolic pressure (38 mm Hg), suggesting significant pul

30 were associated with higher pulmonary artery systolic pressure: 39 +/- 9 mm Hg with thrombi versus 33

31 olume index (44 mL/m2), and pulmonary artery systolic pressure (41 mm Hg) were consistent with chroni

32 with control intake, peripheral and central systolic pressures [-5.7 mm Hg (P = 0.007) and -5.4 mm H

33 atients on PDE5i had higher pulmonary artery systolic pressure (53.4 mm Hg versus 49.5 mm Hg) and pul

34 mean reduction in estimated pulmonary artery systolic pressure (63.9 +/- 13 to 54.2 +/- 12 mm Hg, p =

35 2 men; mean, 77 years; mean pulmonary artery systolic pressure, 69.4+/-10.5 mm Hg), of which PH was l

36 ary hypertension (estimated pulmonary artery systolic pressure, 71+/-23 mm Hg) and in 44 age- and gen

37 o 21.7 mm Hg, P<0.001) and right ventricular systolic pressure (72.8 to 47.3 mm Hg, P<0.001) at cathe

38 stenting included significantly decreased RV systolic pressure (89+/-18 to 65+/-20 mm Hg, P<0.001) an

39 putamen radioactivity correlated with supine systolic pressure across all subjects and among PD patie

40 es were -0.9 (95% CI: -6.4, 4.6) mmHg/kg for systolic pressure and -0.2 (95% CI: -4.1, 3.7) mmHg/kg f

41 es were -0.1 (95% CI: -4.0, 3.8) mmHg/kg for systolic pressure and -0.4 (95% CI: -2.9, 2.2) mmHg/kg f

42 monotonically decreasing association between systolic pressure and adjusted probability of death acro

43 ompared with control, right ventricular (RV) systolic pressure and arterial elastance (measure of vas

44 >= 15%), fluid administration increased end-systolic pressure and decreased effective arterial elast

45 ne acetate mice had similar left ventricular systolic pressure and fractional shortening but more hyp

46 n, judged by regression of right ventricular systolic pressure and hypertrophy and pulmonary artery o

47 At 17 weeks postinfection, right ventricular systolic pressure and liver and lung egg counts were mea

48 ral wall CS correlated with pulmonary artery systolic pressure and LV eccentricity index, after adjus

49 iography-estimated elevated pulmonary artery systolic pressure and LV lateral E/e' ratio were indepen

50 statistically supportable threshold between systolic pressure and mortality emerges from the data a

51 re used to determine the association between systolic pressure and probability of death, adjusting fo

52 estimated vagal baroreflex sensitivity with systolic pressure and R-R interval cross-spectra measure

53 The strong correlation between systolic pressure and R-R intervals at respiratory frequ

54 ed modest increases in the right ventricular systolic pressure and right ventricle hypertrophy.

55 ly attenuated elevation of right ventricular systolic pressure and right ventricular hypertrophy and

56 00A4/Mts1 mice had greater right ventricular systolic pressure and right ventricular hypertrophy at b

57 acute hypoxia (10% O2) or the increase in RV systolic pressure and RV hypertrophy following 3 weeks i

58 ced PH, including attenuated increases in RV systolic pressure and RV hypertrophy, decreased platelet

59 The similar elevation in RV systolic pressure and RV hypertrophy, despite the attenu

60 termined by measuring right ventricular (RV) systolic pressure and RV hypertrophy.

61 nol on relaxation velocity, left ventricular systolic pressure and stroke volume were blunted in dysf

62 ed significantly increased right ventricular systolic pressure and substantial pulmonary vascular rem

63 eatment with candesartan lowered (P<0.05) LV systolic pressure and the first derivative of LV pressur

64 nt significantly decreased right ventricular systolic pressure and total pulmonary vascular resistanc

65 sure, end-diastolic pressure and volume, end-systolic pressure and volume, and ratio of systole to di

66 characteristics caused a decrease in aortic systolic pressure and wasted LV pressure energy.

67 +) mice exhibited elevated right ventricular systolic pressures and right ventricular hypertrophy wit

68 n fraction, LV dimensions, right ventricular systolic pressure) and exercise variables (metabolic equ

69 del, A-17 and A-21 reduced right ventricular systolic pressure, and all antagomirs decreased pulmonar

70 ence, association with high pulmonary artery systolic pressure, and attributable mortality remain unk

71 diastolic LV volume, augmentation index, end-systolic pressure, and cardiovascular disease risk facto

72 er adjustment for age, sex, pulmonary artery systolic pressure, and comorbidities, the presence of an

73 at the start of LVP assessments, heart rate, systolic pressure, and double product fell below baselin

74 despite similar E/e' ratio, pulmonary artery systolic pressure, and event rates.

75 The TRI, calculated from baseline age, systolic pressure, and heart rate, was established in pa

76 edure RV/LV diameter ratio, pulmonary artery systolic pressure, and modified Miller Score were 1.59 (

77 , and baseline RV/LV ratio, pulmonary artery systolic pressure, and modified Miller Score, patients w

78 and cardiac output, higher pulmonary artery systolic pressure, and more severe RV enlargement and tr

79 ed pulmonary hypertension (right ventricular systolic pressure approximately 100 mm Hg) and severe pu

80 diponectin, and had higher right ventricular systolic pressure associated with right ventricular hype

81 ed MR severity and reduced right ventricular systolic pressure at 30 days are associated with a long-

82 MR grade and estimated right ventricular systolic pressure at 30 days were improved to a greater

83 at peak exercise and higher pulmonary artery systolic pressure at rest and at peak exercise, and lowe

84 the abdominal vasculature is associated with systolic pressure augmentation in the ascending aorta an

85 ading conditions in the ascending aorta, and systolic pressure augmentation may be a more useful guid

86 Wave-reflection-induced systolic pressure augmentation was determined using the

87 Keeping systolic pressure below 120 mm Hg before age 35 years ma

88 (beta=-0.22, P=0.002), and pulmonary artery systolic pressure (beta=-0.14, P=0.047).

89 ity score of 3 or greater) and a prehospital systolic pressure between 40 and 119 mm Hg were included

90 (30%) and depends weakly on pulmonary artery systolic pressure but mainly on left ventricular remodel

91 oximately 118% increase in right ventricular systolic pressure) but not polycythemia and was associat

92 dose of norepinephrine necessary to increase systolic pressure by 33 and 100 mm Hg (pressor dose 33 a

93 0 mm Hg were associated with larger falls in systolic pressure by 4.3/2.9 mm Hg in Gabon/Bastrop per

94 o 27 mL/min), causing right ventricular peak systolic pressure/cardiac output to increase from 0.6 co

95 bited significantly higher right ventricular systolic pressure compared with wild-type littermates un

96 was associated with risk for CHF, pulse and systolic pressure conferred greater risk than diastolic

97 sing doses of norepinephrine (norepinephrine-systolic pressure curve) were assessed during a baseline

98 ith LV hypertrophy were older and had higher systolic pressure, damage index scores, C-reactive prote

99 dP/dt (dP/dt(Min)), mean arterial pressure, systolic pressure, diastolic pressure, and left ventricu

100 Subclavian stenosis, diagnosed by a brachial systolic pressure difference (BSPD) > or =15 mm Hg, is a

101 Pulmonary artery systolic pressure dropped more in HFpEF than in HFrEF de

102 rements revealed complete restoration of end-systolic pressure, ejection fraction, end-systolic volum

103 th heart function including heart rate, peak-systolic pressure, end-diastolic pressure and volume, en

104 stroke volume index, ejection fraction, peak systolic pressure/end-systolic volume ratio) to endotoxi

105 The estimated pulmonary artery systolic pressure (ePASP) was >30 mm Hg in 4 participant

106 distribution of estimated right ventricular systolic pressure (eRVSP) was examined in 157,842 men an

107 eters, including estimated right ventricular systolic pressure (ERVSP), and a full review of medical

108 An increase in pulmonary artery systolic pressure, estimated noninvasively by echocardio

109 For systolic pressure, estimates were -12.7 mm Hg (95% confi

110 end-systolic pressure/SVI, and E(LV)I = end-systolic pressure/ESVI, at rest and during exercise in 2

111 Systolic pressure exhibited similar relationships with A

112 7 mm Hg (P<0.001), and the right ventricular systolic pressure fell from 71.6 +/- 21.7 to 46.7 +/- 15

113 re was a 0.2-mm Hg reduction (0.0 to 0.3) in systolic pressure for each 3 months of breast-feeding.

114 ry embolism increased right ventricular peak systolic pressure (from 28 to 47 mm Hg) and decreased ca

115 4.9 to 10.5 +/- 3.1 mm Hg), pulmonary artery systolic pressure (from 60.6 +/- 14.2 to 33.8 +/- 10.7 m

116 In addition, cardiac systolic pressure generation at a diastolic pressure of

117 .7 to 5.6+/-9.6 mm Hg) and the invasive peak systolic pressure gradient (34+/-12 to 11+/-9 mm Hg).

118 Overall, tricuspid regurgitation estimated systolic pressure gradient (PG) and QOL score were signi

119 ak longitudinal global systolic strain rate, systolic pressure gradient between RV and right atrium (

120 Preoperative transvalvular peak systolic pressure gradients across stenotic aortic valve

121 1 g to >6 g), the average transvalvular peak systolic pressure gradients progressively increased.

122 data, pHTN was defined as right ventricular systolic pressure greater than or equal to two-thirds of

123 uncomplicated systolic hypertension (supine systolic pressure > or =140 mm Hg off medication) and 30

124 ine blood pressure in patients with PD + SH (systolic pressure >/= 180 mm Hg, n = 8), patients with P

125 an SVEF </= 40%, a subpulmonary ventricular systolic pressure >/= 50 mm Hg, atrial fibrillation, and

126 Doppler-echocardiography estimated pulmonary systolic pressure >/=45 mm Hg (n=692) and those without

127 erate PH, defined here as a pulmonary artery systolic pressure >/=60 mm Hg detected echocardiographic

128 mptomatic patients with hypertension (aortic systolic pressure >140 mm Hg) and low-gradient (mean gra

129 A subset of mice with right ventricular systolic pressure >30 mm Hg exhibited right ventricular

130 ion (defined as an elevated pulmonary artery systolic pressure >40 mm Hg on echocardiogram).

131 mean transaortic gradient, pulmonary artery systolic pressure >60 mm Hg; p < 0.05 for all) and 2 pro

132 2 to 2.51; p = 0.012), and right ventricular systolic pressure >=50 mm Hg (HR: 2.27; 95% CI: 1.50 to

133 TMG >=8 mm Hg and right ventricular systolic pressure >=50 mm Hg were independently associat

134 patients with an estimated right ventricular systolic pressure>35 mm Hg, suggestive of pulmonary vasc

135 tio 1.55), higher baseline right ventricular systolic pressure (hazard ratio 1.11), more abnormal LV-

136 17-0.50; P<0.001), resting right ventricular systolic pressure (hazard ratio, 1.03; 95% confidence in

137 d ratio, 1.29), and higher right ventricular systolic pressure (hazard ratio, 1.3) were associated wi

138 Baseline systolic pressures higher by 10 mm Hg were associated wi

139 2), and higher peak-stress right ventricular systolic pressure (HR, 1.35), was associated with higher

140 Association class IV (HR: 5.88), and aortic systolic pressure (HR: 0.99) as independent correlates f

141 : 1.02 to 1.06; p < 0.001), pulmonary artery systolic pressure (HR: 1.51 per 10 mm Hg; 95% CI: 1.29 t

142 ardiographic assessments of pulmonary-artery systolic pressure in 195 consecutive patients (82 men an

143 e found in 78% and elevated pulmonary artery systolic pressure in 67%.

144 ypoxia-induced increase in right ventricular systolic pressure in anesthetized mice.

145 udil also markedly reduced right ventricular systolic pressure in late-stage rats.

146 c profile except for a marginal reduction in systolic pressure in old carrier mice.

147 thrombi and to measure the pulmonary artery systolic pressure in patients with a cardiovascular impl

148 a significant increase in right ventricular systolic pressure in Prkg1(-/-) mice in the absence of s

149 d DBL(PKA-) mice displayed depressed maximum systolic pressure in response to dobutamine as measured

150 m) did not rise directly in proportion to RV systolic pressure in Rosa26(R899X) but did in Sm22(R899X

151 right ventricular systolic pressures in BERK-SS were higher than WT at mod

152 ed rates of haemolysis and right ventricular systolic pressures in mice with SCD compared to healthy

153 resence of SS, easily diagnosed by comparing systolic pressures in the left and right arm, predicts t

154 and a significantly increased LV afterload (systolic pressure increase, P<0.001).

155 ne) attenuated MCT-induced right ventricular systolic pressure increase, right ventricular hypertroph

156 In the 15 fluid nonresponders, end-systolic pressure increased (p < 0.05), whereas effectiv

157 aortic regurgitation, and right ventricular systolic pressure), increased the c-statistic from 0.57

158 prorenalase becomes maximally activated when systolic pressure increases by >5 mm Hg.

159 on in patients in whom the right ventricular systolic pressure is calculated to be 50 mmHg or greater

160 tension, but estimation of right ventricular systolic pressure is often inaccurate.

161 volume [LAV], and estimated pulmonary artery systolic pressure), lead to the presence and severity of

162 Consistently, ejection duration and aortic systolic pressure load were significantly diminished, in

163 Participants were exposed to intensive (goal systolic pressure < 120 mm Hg) versus standard (<140 mm

164 Controls had a right ventricular systolic pressure < 40 mm Hg (if estimable) and normal r

165 vanced liver disease, right ventricular (RV) systolic pressure <40 mm Hg, and normal RV function by e

166 .009), while in those with right ventricular systolic pressure<35 mm Hg, a lower value for the percen

167 ardiography with estimated right ventricular systolic pressure, <35 mm Hg; n = 122).

168 =692) and those without PH (n=692; pulmonary systolic pressure, <45 mm Hg) for age, sex, LV ejection

169 increased heart rate (HR), left ventricular systolic pressure (LVSP), the maximum first derivative o

170 intraperitoneally) to mice, left ventricular systolic pressure, maximum first derivative of ventricul

171 ces in age, country, hospital location, era, systolic pressure, mean arterial pressure, lactate, bund

172 ary hypertension judged by right ventricular systolic pressure measurement, right ventricular hypertr

173 olic pressure, peak stress right ventricular systolic pressure, metabolic equivalents achieved, and h

174 lute change in RV/LV ratio, pulmonary artery systolic pressure, modified Miller Score was 0.71, 0.57,

175 Participants with repeated measurements of systolic pressure of 130 to 139 mm Hg and diastolic pres

176 diastolic pressure of 89 mm Hg or lower, or systolic pressure of 139 mm Hg or lower and diastolic pr

177 an, 10% lean, or 20% lean to maintain aortic systolic pressure of 80-90 mm Hg.

178 n = 10; compression depth titrated to aortic systolic pressure of 90 mm Hg, vasopressors titrated to

179 Perfusion was maintained at an average systolic pressure of 93 +/- 2 mm Hg, flow 310 +/- 20 mL/

180 Hg diastolic and a relatively flat curve for systolic pressures of 110 to 130 mm Hg and diastolic pre

181 The curve was relatively flat for systolic pressures of 110 to 130 mm Hg and diastolic pre

182 veloped PH with respective right ventricular systolic pressures of 40.2 +/- 1.5 and 39.6 +/- 1.5 mm H

183 heral atherosclerosis was assessed using the systolic pressures of the dorsal pedal, posterior tibial

184 associated with an elevated pulmonary artery systolic pressure on echocardiogram, may identify an at-

185 at 6 months was defined by right ventricular systolic pressure or MPAP as significant (<35 mm Hg), pa

186 t of PH because we found no difference in RV systolic pressure or RV hypertrophy in wild-type versus

187 y increased cardiac output, pulmonary artery systolic pressure or sympathetic nervous system activity

188 mes (p = 0.005) and higher right ventricular systolic pressure (p < 0.0001).

189 (P = .92), diastolic pressure (P = .31), or systolic pressure (P = .06) before and after US-triggere

190 n fraction (p = 0.013), and pulmonary artery systolic pressure (p = 0.047) were associated with in-ho

191 retinopathy and hypertension (P = 0.037 for systolic pressure; P = 0.019 for diastolic pressure).

192 orse renal function, higher pulmonary artery systolic pressure (PAP), abnormal left ventricular (LV)

193 s the presence of estimated pulmonary artery systolic pressure (PASP) >35 mmHg and/or tricuspid regur

194 about age-related change in pulmonary artery systolic pressure (PASP) and its prognostic impact in th

195 n is associated with higher pulmonary artery systolic pressure (PASP) and prevalent echocardiographic

196 imary outcome measures were pulmonary artery systolic pressure (PASP) and the PASP response to acute

197 heart failure (HF) whether pulmonary artery systolic pressure (PASP) assessed by Doppler echocardiog

198 urements of ventilation and pulmonary artery systolic pressure (PASP) assessed by Doppler echocardiog

199 esised that the increase in pulmonary artery systolic pressure (PASP) at HA would be associated with

200 Baseline pulmonary artery systolic pressure (PASP) estimated from echocardiography

201 able echocardiogram-derived pulmonary artery systolic pressure (PASP) from the Jackson Heart Study (N

202 to assess exercise-induced pulmonary artery systolic pressure (PASP) increase by means of stress Dop

203 Although elevated pulmonary artery systolic pressure (PASP) is associated with heart failur

204 t failure who have elevated pulmonary artery systolic pressure (PASP) on echocardiography.

205 to 1 mo (P < 0.001); median pulmonary artery systolic pressure (PAsP) was 45.9 mm Hg and decreased si

206 Pulmonary artery systolic pressure (PASP) was derived from the tricuspid

207 Pulmonary artery systolic pressure (PASP) was determined using Doppler ec

208 Pulmonary artery systolic pressure (PASP) was serially assessed with Dopp

209 ADR cardiac output (QT) and pulmonary artery systolic pressure (PASP) were significantly increased; h

210 Echocardiography-derived pulmonary artery systolic pressure (PASP), pulmonary vascular resistance

211 lation and dysfunction, and pulmonary artery systolic pressure (PASP).

212 n fraction) with PH (HF-PH; pulmonary artery systolic pressure [PASP] >/=40 mm Hg) were compared to n

213 elicit a change of 20 mm Hg in radial artery systolic pressure (PD20) defined the vasopressor respons

214 ak oxygen uptake, resting pulmonary arterial systolic pressure, peak exercise heart rate, and quality

215 ystolic dimension, resting right ventricular systolic pressure, peak stress right ventricular systoli

216 fraction, and high resting right ventricular systolic pressure predicted worse outcomes.

217 urgeons score and baseline right ventricular systolic pressure) provided incremental prognostic utili

218 Dynamic changes in systolic pressure, pulse pressure, and stroke volume in

219 n inversely correlated with pulmonary artery systolic pressure (r=-0.39, P<0.01) and LV eccentricity

220 al wall LS, correlated with pulmonary artery systolic pressure (r=0.56, P<0.01; r=0.32, P<0.01) and L

221 The median ventricular-to-aortic systolic pressure ratio decreased from 1.7 (1.3-2.6) to

222 y associated with the TAPSE/pulmonary artery systolic pressure ratio.

223 nt in RV-PA coupling (TAPSE/pulmonary artery systolic pressure) ratio.

224 as evidenced by decreased right ventricular systolic pressure, ratio of right ventricular weight to

225 clib reverses the elevated right ventricular systolic pressure, reduces right heart hypertrophy, rest

226 This group had a more effective PA systolic pressure reduction after PTE (49 +/- 20 mm Hg v

227 d) were identified based on the RVESRI to RV systolic pressure relationship.

228 multivariable analysis, age, sex, pulmonary systolic pressure, right atrial minimal volume, as well

229 with no differences in right ventricular end-systolic pressure, right ventricular dP/dt, bromodeoxyur

230 ension, judged by elevated right ventricular systolic pressure, right ventricular hypertrophy, and lo

231 in prevention of increased right ventricular systolic pressure, right ventricular hypertrophy, as wel

232 ia rat model, by improving right ventricular systolic pressure, right ventricular hypertrophy, cardia

233 th established PH improved right ventricular systolic pressures, right ventricular function, and surv

234 s below 40% and the subpulmonary ventricular systolic pressure rises above 50 mm Hg.

235 y developed PAH, as indicated by elevated RV systolic pressure, RV hypertrophy, and increased muscula

236 in central venous pressure, pulmonary artery systolic pressure, RV/left ventricular ratio, and RV fra

237 Right ventricular systolic pressure (RVSP) (mean +/- SD) on the two-dimens

238 atio for prediction of the right ventricular systolic pressure (RVSP) in patients clinically known to

239 The rise in right ventricular systolic pressure (RVSP) normally observed following chr

240 t ventricular function and right ventricular systolic pressure (RVSP) was performed.

241 olic diameter (LVESD), and right ventricular systolic pressure (RVSP) were 62 +/- 2%, 0.56 +/- 0.30 c

242 o have the same life span, right ventricular systolic pressure (RVSP), and lung histology as those of

243 gurgitant orifice, resting right ventricular systolic pressure (RVSP), exercise metabolic equivalents

244 graphy and measurements of right ventricular systolic pressure (RVSP).

245 ocardiography to determine right ventricular systolic pressure (RVSP).

246 (PHT) (RVEF 31.4 +/- 9.6%, right ventricular systolic pressure [RVSP] 76.5 +/- 26.2 mm Hg) and 60 hea

247 h exaggerated elevation of right ventricular systolic pressure, significant right ventricular hypertr

248 imates of effective arterial elastance = end-systolic pressure/stroke volume in critically ill patien

249 lowering of systemic arterial elastance (end-systolic pressure/stroke volume) and systemic vascular r

250 dex (SVI) and its two determinants EaI = end-systolic pressure/SVI, and E(LV)I = end-systolic pressur

251 Baseline systolic pressures tended to be higher in space than on

252 gs of these mice with high right ventricular systolic pressure, the expression of proteins involved i

253 ciated with an increase in right ventricular systolic pressure, thickening of the pulmonary artery me

254 of the right ventricular to left ventricular systolic pressure-time area during inspiration versus ex

255 significantly reduced the right ventricular systolic pressure to 30.1 +/- 1.3 mm Hg.

256 re exerted by the contracting ventricle (end systolic pressure) to its volume (end systolic volume).

257 +/- 18 mm Hg; both P=0.02), and central end-systolic pressure trended lower (116 +/- 18 to 111 +/- 1

258 pressure variation, stroke volume variation, systolic pressure variation, and the change in stroke/ca

259 perating characteristic between the baseline systolic pressure variation, stroke volume variation, an

260 pleural pressure; pulse pressure variations, systolic pressure variations, and stroke volume variatio

261 The slope of the end-systolic pressure volume relationship (i.e., contractili

262 volume [EDV]); contractile function (the end-systolic pressure volume relationship slope [Eessb] and

263 fect on systolic function, improving the end-systolic pressure-volume relation (+0.98 +/- 0.41 mm Hg/

264 The end-systolic pressure-volume relation was increased by isopr

265 ad-independent indexes of contractility (end-systolic pressure-volume relation, preload-recruitable s

266 lopment (p = 0.002), and slope of the LV end-systolic pressure-volume relationship (p = 0.04).

267 one (P<0.01), and a steeper slope of the end-systolic pressure-volume relationship (P=0.01).

268 <0.05) and induced systolic dysfunction (end systolic pressure-volume relationship =24.86+/-2.46 in w

269 Ischemia shifted the end-systolic pressure-volume relationship and cardiac output

270 p < .05), and significantly improved the end-systolic pressure-volume relationship and preload recrui

271 al, 13-24]% versus 12 [10-14]%, P=0.008; end-systolic pressure-volume relationship slope 2.4 [1.9-3.2

272 The end-systolic pressure-volume relationship was increased in D

273 IQR, 21-46 mm Hg]; P=0.005), whereas the end-systolic pressure-volume relationship was not significan

274 systolic stress-shortening relationship, end-systolic pressure-volume relationship, and peak (+)dP/dt

275 on functional class and on right ventricular systolic pressure, volumes, and dimensions.

276 Mean systolic pressure was 100 versus 107 mm Hg in those not

277 Each 10-point increase of systolic pressure was associated with a decrease in the

278 End-systolic pressure was calculated as 0.9 x systolic arter

279 According to the "cardiocentric" view, end-systolic pressure was considered the classic index of le

280 Pulmonary artery systolic pressure was higher in AF.

281 nths of altitude exposure, right ventricular systolic pressure was measured (solid-state transducer).

282 Systolic pressure was not a useful metric in the vasopre

283 Systolic pressure was related to AF (HR, 1.14 per 20-mm

284 aortic regurgitation, and right ventricular systolic pressure) was 0.64 (95% confidence interval 0.5

285 regurgitant velocity, a measure of pulmonary systolic pressure, was predictive of events in a multiva

286 dence interval, 1.4-2.3), pulmonary arterial systolic pressure (weighted mean difference, -3.7 mm Hg;

287 eak-stress MV gradient and right ventricular systolic pressure were 17+/-7 and 61+/-14 mm Hg, respect

288 horacic Surgeons score and right ventricular systolic pressure were 2+/-3 and 15+/-16 mm Hg, respecti

289 horacic Surgeons score and right ventricular systolic pressure were 3.3+/-3 and 31+/-7 mm Hg, respect

290 end-diastolic volume, and right ventricular systolic pressure were 4+/-1%, 62+/-3%, 0.55+/-0.2 cm(2)

291 ortic valve gradients, and right ventricular systolic pressure were 7+/-6, 58+/-6%, 54+/-10 mm Hg and

292 an resting MV gradient and right ventricular systolic pressure were 8.5+/-3 and 39+/-13 mm Hg.

293 ment of leaflet coaptation, and estimated PA systolic pressure were determined on pre- and post-PTE e

294 ge, renal dysfunction, and right ventricular systolic pressure were independently associated with the

295 Wide ranges of right ventricular systolic pressure were observed in mice with heterozygou

296 for sham; P<0.001), whereas left ventricular systolic pressures were significantly reduced (ligated 8

297 ministration increased stroke volume and end-systolic pressure, whereas effective arterial elastance

298 ts, and higher peak-stress right ventricular systolic pressure, while invasive MV procedures were ass

299 Despite higher right ventricular systolic pressures with chronic hypoxia, S100A4/Mts1 mic

300 The LV was able to generate twice the LV systolic pressure without an increase in LV end-diastoli