ライフサイエンスコーパス: systolic velocity

1 lic velocity was inversely related to portal systolic velocity.

2 qual to 3:1, and intrastent doubling of peak-systolic velocity.

3 versus 65.7+/-8.0%; P<0.0001; mitral annular systolic velocity, 11.7+/-2.6 versus 10.9+/-2.3 cm/s; P=

4 6), despite increased blood flow (Delta peak systolic velocity, 6.3 cm/s, 3.5-9.07; P<0.001; Delta en

5 Peak systolic velocity after administration of topical atropi

6 measure endocardial (v(endo)) and epicardial systolic velocities and SR.

7 ssed to reconstruct mitral annulus (MA) peak systolic velocity and displacement.

8 During stress, the peak systolic velocity and early diastolic velocity increased

9 Myocardial systolic velocity and Em were also significantly higher

10 a values of the relationship between MA peak systolic velocity and LV end-diastolic volume and mass w

11 left ventricular function, assessed by peak systolic velocity and strain rate in 12 paired, nonapica

12 The best-performing parameters were peak systolic velocity and V(MCA)/V(ICA) ratio.

13 n area increased significantly, whereas peak systolic velocity and wall shear stress decreased.

14 Average volume of flow, peak systolic velocity, and diameter of residual lumen were r

15 atrial volume; tissue Doppler diastolic and systolic velocities; and serum levels of high-sensitivit

16 dneys and between regions was found for peak systolic velocity, but the magnitude of this variation w

17 Increasing age decreased peak systolic velocity by 0.34 cm/sec/y.

18 ths, defined as >/=2.5-fold increase in peak systolic velocity by duplex ultrasonography.

19 Internal carotid artery peak systolic velocities decrease with advancing age and incr

20 mplementation of middle cerebral artery peak systolic velocity Doppler measurements to detect fetal a

21 formation and function to reduce ventricular systolic velocity, elongates ejection time, and sustains

22 Children with SCA had higher peak systolic velocities, end-diastolic velocities, and blood

23 iagnostic US criteria for stenosis were peak-systolic velocity greater than 1.25 m/sec, internal caro

24 riteria for stenosis were also applied: peak-systolic velocity greater than 1.7 m/sec, ICA end-diasto

25 if flow approached 1,300 mL/min without peak systolic velocity greater than 400 cm/sec.

26 ents with isolated, asymptomatic AS and peak systolic velocity > or =4 m/s by Doppler echocardiograph

27 long-axis ratio <0.6, tricuspid annulus peak systolic velocity >/= 8 cm/s, and peak systolic longitud

28 asurement of the middle cerebral artery peak systolic velocity has led to a paradigm shift in antenat

29 Myocardial systolic velocity improved acutely (tricuspid 4.8 to 5.3

30 r imaging, viable segments show increases in systolic velocities in contrast to infarcted segments.

31 The SD in time to peak systolic velocity in the 12 left ventricular segments wa

32 ptal-posterior delay, and SD in time to peak systolic velocity in the 12 left ventricular segments) a

33 g-axis ratio >/= 0.6, tricuspid annulus peak systolic velocity <8 cm/s, and peak systolic longitudina

34 A systolic velocity <8.5 cm/s had an 86% sensitivity and 1

35 m s(-1) and -0.50 +/- 2.55 cm s(-1) for peak systolic velocity, mean flow velocity, and end diastolic

36 id annular plane systolic excursion and peak systolic velocity, myocardial performance (expressed as

37 Blood pressures and systolic velocities of the middle cerebral artery signif

38 Preimplantation diastolic velocity:systolic velocity of 0.6 0.16 dropped to 0.34 0.13 (P<0.

39 n TDI indices occurred with dobutamine: peak systolic velocity of 4.41 +/- 1.07 to 6.67 +/- 1.07 cm/s

40 ficant decreases occurred with esmolol: peak systolic velocity of 4.46 +/- 0.94 to 2.31 +/- 0.81 cm/s

41 artery of at least 70%, diagnosed by a peak systolic velocity of at least 3.0 m/s.

42 Duplex ultrasound peak systolic velocity of the cavernosal arteries increased f

43 7 mm; p < 0.05) and correlated with the peak systolic velocity of the second palmar digital artery (P

44 ( P=0.003) and RV tricuspid lateral annular systolic velocity ( P=0.02), and a higher RV Tei index (

45 Doppler-defined pre-procedural peak systolic velocities (PSV) and end-diastolic velocities (

46 (c) ICA peak systolic velocity (PSV) and presence of plaque on gray-s

47 Point, non-angle-corrected peak systolic velocity (PSV) and resistive index (RI) values

48 ty (MFV), end diastolic velocity (EDV), peak systolic velocity (PSV) and resistive index (RI).

49 ICA peak systolic velocity (PSV) and the ratio of the PSV in the

50 a focal twofold or higher elevation of peak systolic velocity (PSV) compared with the PSV immediatel

51 The range of peak systolic velocity (PSV) measurement (maximum minus minim

52 rvus tardus waveforms) with and without peak systolic velocity (PSV) thresholds (determined with rece

53 Early systolic acceleration (ESA), peak systolic velocity (PSV), end diastolic velocity (EDV), a

54 lly significant changes observed in the peak systolic velocity (PSV), end diastolic velocity (EDV), o

55 Peak systolic velocity (PSV), end-diastolic velocity (EDV), r

56 The range of CCA peak systolic velocities (PSVs) and end diastolic velocities

57 efined as primary patency at 12 months (peak systolic velocity ratio <2.4 by duplex ultrasound withou

58 e laboratory-assessed duplex ultrasound peak systolic velocity ratio <=2.4 in the absence of clinical

59 videnced by a higher mean graft/femoral peak systolic velocity ratio (1.6 vs. 0.90, P=0.006).

60 city (bAPV), hyperemic APV (hAPV), diastolic/systolic velocity ratio (DSVR), and coronary flow reserv

61 This may increase the accuracy of the systolic velocity ratio for assessment of internal carot

62 elocity greater than 0.4 m/sec, ICA/CCA peak-systolic velocity ratio greater than 2.0, and ICA/CCA en

63 ry (ICA) to common carotid artery (CCA) peak-systolic velocity ratio of greater than or equal to 3:1,

64 narrowing was assessed by graft/femoral peak systolic velocity ratio.

65 ardiography to calculate diastolic velocity: systolic velocity ratio.

66 ional area change [RVFAC], RV tissue Doppler systolic velocity [RV s'], and tricuspid annular plane s

67 s/length-area ratios, tricuspid annulus peak systolic velocity, RV peak longitudinal global systolic

68 .5 cm/s, P=0.02), with a significantly lower systolic velocity S' septal (7.6+/-1.2 versus 8.5+/-1.2

69 sed by measuring differences in time-to-peak systolic velocity (T(SV)) between the RV free wall, vent

70 The peak systolic velocity threshold for moderate (>=50%) stenosi

71 Reduced coronary forward wave energy and systolic velocity time integral imply a compromised syst

72 wave with TAVI was related to an increase in systolic velocity time integral.

73 m/s (p = .002), right ventricular myocardial systolic velocities to 15.0 (11.8-23) cm/s, (p = .003),

74 al admission to discharge; septal myocardial systolic velocities to 8.8 (7-11) cm/s (p = .002), right

75 The maximum systolic velocity (V(max)) was evaluated in the distal i

76 cal cohort, 1 in 10 (9.8%) patients had peak systolic velocity values that warranted the diagnosis of

77 pare (AI vs the reference standard, 4D flow) systolic velocity vector fields, peak velocity, wall she

78 s myocardial function was quantified by peak systolic velocity (Vs) and strain rate (SR) responses.

79 RAS was severe (60% to 99% stenosis) if peak systolic velocity was >200 cm/s.

80 Peak systolic velocity was additionally found to be inversely

81 Peak systolic velocity was influenced by age (P =.008), systo

82 Arterial systolic velocity was inversely related to portal systol

83 Peak systolic velocity was measured by Doppler velocimetry.

84 Preoperative portal peak systolic velocity was uniformly around 10 cm/sec.

85 Internal carotid artery peak systolic velocity was used by all centers to assess dise

86 pulsatility index, resistive index, or peak-systolic velocity, was associated with malignancy.

87 Septal myocardial systolic velocities were reduced (6.4 [4.8-10] vs. 8.1 [

88 Changes in TDI peak systolic velocity were correlated with changes in fracti

89 Resistive and pulsatility indexes and peak systolic velocity were documented.