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1 sponses including development of conditioned taste aversion.
2 ) aversive effects as indexed by conditioned taste aversion.
3 take, induce pica, and produce a conditioned taste aversion.
4 d feeding and the formation of a conditioned taste aversion.
5 erone secretion, body weight, or conditioned taste aversion.
6 kness as icv TTR did not cause a conditioned taste aversion.
7 choice behavior when it was guided by innate taste aversion.
8 he development of a LiCl-induced conditioned taste aversion.
9 "good" to "bad" as occurs during conditioned taste aversion.
10 ince they successfully expressed conditioned taste aversion.
11 ns had no impact on LiCl-induced conditioned taste aversion.
12 d intake in rats without causing conditioned taste aversion.
13 oiding taste stimuli following a conditioned taste aversion.
14 nt used in laboratory studies of conditioned taste aversion.
15 pect to the neural substrates of conditioned taste aversion.
16 n of latent inhibition (LI) of a conditioned taste aversion.
17 tive to each monotherapy, without inducing a taste aversion.
18 nduces pica, and produces robust conditioned taste aversions.
19 dence that drugs of abuse induce conditioned taste aversions.
22 , nociception, and extinction of conditioned taste aversion and an appetitive instrumental response.
24 t genetic correlations were observed between taste aversion and ethanol-related behaviors measured in
28 ocked behavioral expression of a conditioned taste aversion and this was evident not only when lesion
31 conditioned fear responses and a conditioned taste aversion as well as enhanced performance in an att
34 atability, the current results indicate that taste aversions based on either lithium or activity redu
36 f paraquat at a dose sufficient to condition taste aversion, but produce no other signs of overt toxi
38 d that amygdala was absolutely necessary for taste aversions conditioned with the intraoral method bu
42 was then accomplished in 1 group by inducing taste aversion; controls received either saline or unpai
46 field is sufficient to induce a conditioned taste aversion (CTA) and induce brainstem expression of
47 g sodium depletion, and a normal conditioned taste aversion (CTA) for alanine when paired with lithiu
52 ucleus (PBN) failed to acquire a conditioned taste aversion (CTA) induced by lithium chloride (LiCl)
64 s widely regarded as integral to conditioned taste aversion (CTA) retention, a link that has been pri
71 nist, was infused into IC before conditioned taste aversion (CTA) training with a familiar taste.
72 correlate of the expression of a conditioned taste aversion (CTA) when conditioning occurs using tast
73 a herbicide capable of eliciting conditioned taste aversion (CTA), a behavioral response characterist
74 readily acquired a LiCl-induced conditioned taste aversion (CTA), the suppressive effects of sucrose
75 iCl) caused the development of a conditioned taste aversion (CTA), which rendered the typically rewar
95 thium chloride (referred to as a conditioned taste aversion, CTA); (2) access to a very palatable con
96 f permanent forebrain lesions on conditioned taste aversions (CTAs) and conditioned odor aversions (C
97 ) lesions attenuate LiCl-induced conditioned taste aversions (CTAs) by disruption of information abou
98 the acquisition and retention of conditioned taste aversions (CTAs) in rodents, but large lesions in
100 da tympani nerves (CTX) received conditioned taste aversions (CTAs) to the free fatty acids (FFAs), l
103 havioral experiments testing for conditioned taste aversion did not confirm that SEB challenge promot
104 e acquisition of ethanol-induced conditioned taste aversion, ethanol-induced conditioned place prefer
105 ncluding active place avoidance, conditioned taste aversion, fear conditioning and spatial learning.
109 rin taste developed a persistent conditioned taste aversion in both preference and taste reactivity t
111 tinction tests revealed rapid attenuation of taste aversions in all of the LiCl-injected groups.
112 parabrachial nucleus (PBN) failed to learn a taste aversion induced by lithium chloride (LiCl) toxico
114 lease of cholecystokinin whereas conditioned taste aversion is mediated by serotonin and substance P.
115 support the hypothesis that ethanol-induced taste aversion is mediated by the drug's rewarding prope
116 al amygdala (BLA) in response to conditioned taste aversion is necessary to form a memory for a taste
117 e basolateral amygdala (BLA) is critical for taste aversion learning and retrieval, suggesting this c
118 e GI tract suggests a new account of delayed taste aversion learning as well as learning about the po
119 ess this issue in the context of conditioned taste aversion learning by continuously tracking gustato
121 the light CS in the first training phase or taste aversion learning in the second training phase.
124 H3, c-Fos induction, and amygdalar-dependent taste aversion learning is constrained by endogenous his
127 rained first to reject it (i.e., conditioned taste aversion learning) and then to enjoy it again (i.e
128 itioning represents a molecular correlate of taste aversion learning, i.e. the formerly neutral CS no
129 details of the neural circuitry involved in taste aversion learning, including its anatomical distri
135 thdrawal with low drinking, high conditioned taste aversion, low tolerance to EtOH-induced hypothermi
137 abe and rural Latino Panamanians to study if taste aversion may be a limiting factor in chlorination
138 egulates ingestive behavior without inducing taste aversion may open the possibility of a therapeutic
140 lso attenuated the extinction of established taste aversion memory without altering the initial assoc
145 l training procedures and either conditioned taste aversion or satiation devaluation procedures.
146 ubstrates of LI as assessed in a conditioned taste aversion paradigm by comparing regional c-Fos acti
149 t study we subjected mice to the conditioned taste aversion paradigm, where animals learn to associat
153 elay in extinguishing an ethanol-conditioned taste aversion, suggesting that they drink less ethanol
155 xperiment 4) or consumption on a conditioned taste aversion test that does not elicit antipredator re
158 ransection (CTX) acquired a LiCl-conditioned taste aversion to 0.1 M NaCl at the same rate as control
161 lued the food unconditioned stimulus (US) by taste aversion to differentiate stimulus-stimulus(CS-US)
162 tration of GLP-1 did not cause a conditioned taste aversion to saccharin, suggesting that the anorexi
163 ate was confirmed in rats with a conditioned taste aversion to sucrose: after paired exposure to sucr
166 acquisition and expression of a conditioned taste aversion was assessed using two different conditio
168 l can induce memory given that a conditioned taste aversion was obtained for a novel taste, presented
171 y pairing it with lithium chloride (acquired taste aversion), while the other distinctive flavor was