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1 e molecular pathway of retinoic acid-induced teratogenesis.
2 ility of the embryo and developing organs to teratogenesis.
3 B1 appears to play a role in dioxin-mediated teratogenesis.
4 bit increased incidence of radiation-induced teratogenesis.
5 hanisms underlying copper deficiency-induced teratogenesis.
6 erapeutic strategies to prevent Zika-induced teratogenesis.
7 ct or sensitize offspring to alcohol-induced teratogenesis.
8 rucial variable in studies of ecotoxicology, teratogenesis, and developmental plasticity.
9 malidomide and Lenalidomide on angiogenesis, teratogenesis, and neurite outgrowth, known detrimental
10  are more sensitive to retinoic acid-induced teratogenesis, and retinoid target genes are expressed a
11  the genes regulating sensitivity to ethanol teratogenesis are largely unknown.
12        However, the nature and mechanisms of teratogenesis are poorly understood.
13 not by I6A-NAP, which does not block ethanol teratogenesis as potently.
14  resistant to the acute toxicity and cardiac teratogenesis caused by high levels of polycyclic aromat
15                              Neurobehavioral teratogenesis caused by prenatal nicotine exposure is as
16 is; some subpopulations resisted the cardiac teratogenesis caused by the challenges at doses that sti
17  mechanisms responsible for ethanol-mediated teratogenesis have not been resolved.
18 d embryo hatch to 3.7% +/- 1.95, and induced teratogenesis in 100% +/- 0 of hatched embryos.
19 ounteract the increased susceptibility to RA teratogenesis in embryos of diabetic mice.
20 vitamin A or retinoic acid causes widespread teratogenesis in rodents as well as humans.
21 se findings suggest that cyclopamine-induced teratogenesis is due to a more direct antagonism of Shh
22                         Susceptibility to RA teratogenesis is further potentiated in embryos with a p
23 en fetal sensitivity to RVFV vaccine-induced teratogenesis is highest.
24 ctively advance a mechanistic hypothesis for teratogenesis leading to microencephaly.
25  developing embryo; therefore, mechanisms of teratogenesis may be approached within the context of th
26  0.01) and designated as chemically mediated teratogenesis number 1 (Cmt1).
27 ice that differ in susceptibility to ethanol teratogenesis showed corresponding differences in MAPK a
28 always correlate strongly with resistance to teratogenesis; some subpopulations resisted the cardiac
29 onstrating the importance of maternal AHR in teratogenesis, these data may have implications that rea
30                   Overall, the resistance to teratogenesis was strongest in the subpopulations from s
31   To elucidate their roles in dioxin-induced teratogenesis, we compared Cyp1a1(-/-), Cyp1a2(-/-), and
32 ression may cause some aspects of RA-induced teratogenesis, we studied the mechanism underlying the i
33 tionship between p53-dependent apoptosis and teratogenesis, we subjected day 8 mouse embryos with dif