ライフサイエンスコーパス: tert-butylhydroquinone

1 igargin (Tg), cyclopiazonic acid, and 2,5-di-tert-butylhydroquinone.

2 lasmic reticulum Ca(2)(+) ATPase with 2,5-di-tert-butylhydroquinone.

3 hibited by prototypical inducers arsenic and tert-butylhydroquinone.

4 activation was induced with the electrophile tert-butylhydroquinone.

5 human UGT1 locus (Tg-UGT1) were treated with tert-butylhydroquinone.

6 and this activation was further increased by tert-butylhydroquinone.

7 but this activation was no longer induced by tert-butylhydroquinone.

8 2 activator prodrugs of 4-methylcatechol and tert-butylhydroquinone.

9 ured cells treated with pro-oxidants such as tert-butylhydroquinone, 2,3-dimethoxy-1, 4-naphthoquinon

10 tert-Butylhydroquinone and a mixture containing ascorbic

11 epG2 cells were treated with the prooxidants tert-butylhydroquinone and beta-naphthoflavone, cellular

12 ntenal) with hydroquinones (hydroquinone and tert-butylhydroquinone) and benzoquinones (benzoquinone,

13 pump (thapsigargin, cyclopiazonic acid, and tert-butylhydroquinone), and prolonged incubation of cel

14 The reduced forms of quinone, tert-butylhydroquinone, and 5-imino-daunorubicin do not

15 variety of oxidants, including sulforaphane, tert-butylhydroquinone, and H2O2, could effectively indu

16 utic potential of a combination of apocynin, tert-butylhydroquinone, and salubrinal to prevent oxidat

17 ed-hydroxyanisole, butylated-hydroxytoluene, tert-butylhydroquinone, ascorbyl palmitate, tocopherol,

18 Known inducers of phase II enzymes, such as tert-butylhydroquinone, beta-naphthoflavone, and sulfora

19 protein kinase C in HepG2 cells treated with tert-butylhydroquinone, beta-naphthoflavone, or 12-O-tet

20 Thapsigargin and 2,5-di-tert-butylhydroquinone (BHQ) blocked insulin signaling,

21 ion of a model hydroquinone compound, 2,5-di-tert-butylhydroquinone, by LPO/H(2)O(2) is also dependen

22 f2-ARE pathway by the known chemical inducer tert-butylhydroquinone can protect against 6-OHDA in vit

23 demonstrate that a common food preservative, tert-butylhydroquinone, can activate Nrf2 in T cells, as

24 ased basal NQO1 activity and no induction by tert-butylhydroquinone compared with Nrf2(+/+) astrocyte

25 cleus, arsenic, but not phenolic antioxidant tert-butylhydroquinone, dissociated Nrf2 from Keap1 and

26 ere increased in hepatoma cells treated with tert-butylhydroquinone for 2 h as measured by flow cytom

27 SN50 also blocked tert-butylhydroquinone-induced nuclear fluorescence of G

28 knockdown of CHD6 reduced both the basal and tert-butylhydroquinone-inducible expression of NQO1, a p

29 hepatoblastoma (HepG2) cells to antioxidant tert-butylhydroquinone led to induction of Bcl-2.

30 of Nrf2 were increased in cells treated with tert-butylhydroquinone or beta-naphthoflavone by a post-

31 atment of Hepa cells with hydrogen peroxide, tert-butylhydroquinone, or zinc suggested a rapid increa

32 tails formation of reactive oxygen species), tert-butylhydroquinone (promotes disposal of reactive ox

33 Oxidative stress (tert-butylhydroquinone) rapidly induced metallothionein-

34 ic reticulum Ca2+ reuptake inhibitor (2,5-di-tert-butylhydroquinone) rescued key phenotypic differenc

35 ng compounds, including 4-methylcatechol and tert-butylhydroquinone, show a markedly lower activity u

36 ement (ARE) that was positively regulated by tert-butylhydroquinone, sulforaphane, and hemin with res

37 er, a reported potential antiviral compound, tert-butylhydroquinone (t-b-HQ), showed no significant e

38 Treatment with the antioxidant tert-Butylhydroquinone (t-BHQ) leads to induction of Cul

39 itin H transcription was robustly induced in tert-butylhydroquinone (t-BHQ)-treated Jurkat cells via

40 50-fold following treatment with 50 mumol/L tert-butylhydroquinone (t-BHQ).

41 ion and induction in response to antioxidant tert-butylhydroquinone (t-BHQ).

42 tert-Butylhydroquinone (TBH) exerted a dose- and time-de

43 tert-Butylhydroquinone (TBH) induces human GCLC via Nrf2

44 (DEM), D,L-buthionine sulfoximine (BSO), or tert-butylhydroquinone (TBH).

45 cal sensor for the simultaneous detection of tert-butylhydroquinone (TBHQ) and butylated hydroxyaniso

46 In this study, tert-butylhydroquinone (tBHQ) and sulforaphane were used

47 the actions of the prototypical Nrf2 inducer tert-butylhydroquinone (tBHQ) and two biotin-tagged, thi

48 e (BHA), butylated hydroxytoluene (BHT), and tert-butylhydroquinone (TBHQ) are synthetic antioxidants

49 activation of Nrf2 by sulforaphane (SF) and tert-butylhydroquinone (tBHQ) depends upon Keap1-C151 an

50 ivation of the human NQO1-ARE (hNQO1-ARE) by tert-butylhydroquinone (tBHQ) is mediated by phosphatidy

51 Among these, tert-butylhydroquinone (TBHQ) is widely used but can pos

52 The enzyme-mediated grafting of tert-butylhydroquinone (TBHQ) onto chitosan and further

53 ci was enhanced by phenobarbital, but not by tert-butylhydroquinone (tBHQ) or paraquat.

54 ) and murine hepatoma (Hepa1c1c7) cells with tert-butylhydroquinone (tBHQ) or sulforaphane (SUL), two

55 resent study was designed to investigate how tert-butylhydroquinone (tBHQ) prevents hydrogen peroxide

56 pG2 and murine hepatoma Hepa1c1c7 cells with tert-butylhydroquinone (tBHQ) stimulated the activity of

57 Tert-butylhydroquinone (TBHQ) was tested for potential c

58 of the CuFe(2)O(4)@HNT for the detection of Tert-butylhydroquinone (TBHQ) were evaluated by the CV,

59 ted by antiestrogen to the same extent as by tert-butylhydroquinone (TBHQ), a known activator of EpRE

60 A major metabolite of BHA, tert-butylhydroquinone (tBHQ), also activated ERK2 but w

61 We examined the effect of tert-butylhydroquinone (tBHQ), an Nrf2-ARE signaling pat

62 llular fractionation revealed that PMA, like tert-butylhydroquinone (tBHQ), promoted the nuclear loca

63 how that phorbol myristate acetate (PMA) and tert-butylhydroquinone (tBHQ), which induce oxidative st

64 tion of both the CYP2C9 and CYP2C19 genes by tert-butylhydroquinone (tBHQ).

65 globin mRNA production by the NRF2 activator Tert-butylhydroquinone (tBHQ).

66 combinations (75:25, 50:50, and 25:75), and tert-butylhydroquinone (TBHQ).

67 er antioxidant activities than 0.02% (1.1mM) tert-butylhydroquinone (TBHQ).

68 perfused with vehicle or the AP-1 stimulator tert-butylhydroquinone (tBHQ).

69 n of widely used cytotoxic food preservative tert-butylhydroquinone (TBHQ).

70 In addition, antioxidant (tert-butylhydroquinone) treatment destabilized the Nrf2-

71 ated that H2O2 (hydrogen peroxide) or t-BHQ (tert-butylhydroquinone) treatment increased total protei

72 sponse to heme, cadmium, zinc, arsenite, and tert-butylhydroquinone was inhibited by 85-95%.

73 sensitive to tert-butylhydroperoxide but not tert-butylhydroquinone, whereas knockdown of Nrf2b did n

74 t from other electrophilic compounds such as tert-butylhydroquinone, which activates the antioxidant-

75 hat interaction of anthracyclines and 2,5-di-tert-butylhydroquinone with LPO/H(2)O(2)/NO(2)(-) genera

76 whereas Nrf2 activation by sulforaphane and tert-butylhydroquinone with subsequent HO-1 induction en