ライフサイエンスコーパス: tetralogy of Fallot

1 xtremities, and heart defects (predominantly tetralogy of Fallot).

2 rtic root dilatation in adults with repaired tetralogy of Fallot.

3 ole exposure may confer an increased risk of tetralogy of Fallot.

4 congenital heart defects such as in repaired tetralogy of Fallot.

5 in a concern in adult patients with repaired tetralogy of Fallot.

6 d murine dilated cardiomyopathy and repaired tetralogy of Fallot.

7 adoption of a RVOT-sparing strategy to treat tetralogy of Fallot.

8 rom 829 sporadic, nonsyndromic patients with Tetralogy of Fallot.

9 ome, aortic stenosis, pulmonic stenosis, and tetralogy of Fallot.

10 e in risk stratifying patients with repaired tetralogy of Fallot.

11 f the human congenital cardiac defect termed tetralogy of Fallot.

12 t late sequelae for patients after repair of tetralogy of Fallot.

13 rst-degree AV block, idiopathic AV block, or tetralogy of Fallot.

14 with atrioventricular septal defect or with tetralogy of Fallot.

15 or secondary outcome parameters in repaired tetralogy of Fallot.

16 t, respectively, of patients after repair of tetralogy of Fallot.

17 rtant determinant of outcome after repair of tetralogy of Fallot.

18 aused congestive failure than in hearts with tetralogy of Fallot.

19 rmal heart as well as in surgically repaired tetralogy of Fallot.

20 clinical outcomes in patients with repaired tetralogy of Fallot.

21 r risk stratification in adults with repared tetralogy of Fallot.

22 minant FLT4 mutations accounting for 2.3% of Tetralogy of Fallot.

23 isms and treatment of arrhythmia in repaired tetralogy of Fallot.

24 nary RV dysfunction in adults after repaired tetralogy of Fallot.

25 mortality after transannular patch repair of tetralogy of Fallot.

26 with transposition of the great arteries and tetralogy of Fallot.

27 vere congenital heart defects in general, or tetralogy of Fallot.

28 ain some of the clinical variability seen in tetralogy of Fallot.

29 cular performance in patients with corrected tetralogy of Fallot.

30 g in children and young adults with repaired tetralogy of Fallot.

31 s with LCR22C-D deletions had CTDs including tetralogy of Fallot.

32 h clinical outcome in patients with repaired tetralogy of Fallot.

33 hmuses improves ablation outcome in repaired Tetralogy of Fallot.

34 ology are found among patients with repaired tetralogy of Fallot.

35 ansposition of the great arteries; and 8 for tetralogy of Fallot.

36 as the prudent class to have offspring with tetralogy of Fallot.

37 ciated with smaller placental size at birth: tetralogy of Fallot, -0.45 (95% confidence interval, -0.

38 Four patients had tetralogy of Fallot, 1 had pulmonary atresia, and 1 had

39 titutional databases of adults with repaired tetralogy of Fallot: (1) for model development, a prospe

40 tro-transposition of the great arteries, 123 tetralogy of Fallot, 132 single ventricle, and 130 other

41 We found the following anomalies: tetralogy of Fallot (15 patients, 25%), tricuspid atresi

42 Most patients had tetralogy of Fallot (18/31).

43 The most common diagnoses were tetralogy of Fallot (19%) and hypertrophic cardiomyopath

44 dence of infective endocarditis was 1.3% for tetralogy of Fallot, 2.7% for isolated ventricular septa

45 The 3 most common CHDs were tetralogy of Fallot (20%), isolated ventricular septal d

46 patients (3%), including 9 of 201 (4%) with tetralogy of Fallot, 3 of 71 (4%) with a secundum ASD, o

47 ular septum, 7.8%; 2) paternal anesthesia in tetralogy of Fallot, 3.6%; 3) painting in atrioventricul

48 anian population (25%) or control cases with tetralogy of Fallot (30%).

49 Diagnoses included tetralogy of Fallot (51%), septal defects (20%), (congen

50 Cardiac diagnoses were tetralogy of Fallot (71%), valvular pulmonary stenosis (

51 anomaly (40%), pulmonary atresia (11%), and tetralogy of Fallot (8%).

52 The most prevalent CHD anatomy included tetralogy of Fallot (883 participants [22%]), transposit

53 defect seems to be a single-gene defect and tetralogy of Fallot a polygenic disorder with a small nu

54 ted into DNA during S-phase, in infants with tetralogy of Fallot, a common form of congenital heart d

55 the ADAM17 JMD identified in a patient with Tetralogy of Fallot, a serious heart valve defect, affec

56 Fifty-seven patients with repaired tetralogy of Fallot (age >/=16 y; mean age, 35.8+/-10.1

57 ized controlled pilot study in patients with tetralogy of Fallot aged 10 to 25 years.

58 % CI: 1.16-6.92) was associated with ALL and tetralogy of Fallot (aHR: 2.40, 95% CI: 1.27-4.55) was a

59 ralogy of Fallot with pulmonary atresia than tetralogy of Fallot alone.

60 der called Alagille syndrome, which includes tetralogy of Fallot among its more severe cardiac pathol

61 re not modified by supplement use except for tetralogy of Fallot; among supplement users, those in th

62 tion, transposition of the great arteries or tetralogy of Fallot, an event rate of 1/454 patients-yea

63 Eight patients with tetralogy of Fallot and 7 patients with transposition of

64 Of the 34 patients with tetralogy of Fallot and abnormal CA, 7 (21%) demonstrate

65 dden cardiac death remain common in repaired tetralogy of Fallot and affect even those with excellent

66 Patients with tetralogy of Fallot and an ICD experience high rates of

67 ogenic stop-gain variant co-segregating with tetralogy of Fallot and bicuspid aortic valve in materna

68 inst RV dilatation in patients with repaired tetralogy of Fallot and chronic pulmonary regurgitation

69 otal of 44 patients (40.3+/-19.2 years) with tetralogy of Fallot and CRT were enrolled.

70 aorta and coronary artery anomalies, such as tetralogy of Fallot and double outlet right ventricle.

71 two common human congenital heart diseases, tetralogy of Fallot and double outlet right ventricle.

72 ount for a clinically significant portion of tetralogy of Fallot and idiopathic AV block.

73 ents who underwent surgical intervention for tetralogy of Fallot and major aortopulmonary collateral

74 nts with both native and previously operated tetralogy of Fallot and major aortopulmonary collateral

75 pulmonary collaterals (APCs) in infants with tetralogy of Fallot and pulmonary atresia (TOF/PA) and t

76 ath occurred in 12 of 25 patients (48%) with tetralogy of Fallot and pulmonary atresia, and 4 of 54 (

77 up A lesions, which predominantly consist of tetralogy of Fallot and related variants.

78 Adults with repaired tetralogy of Fallot and RV dysfunction (RV ejection frac

79 ients aged median 12.0 years after repair of tetralogy of Fallot and similar lesions were studied ech

80 Neonates with tetralogy of Fallot and symptomatic cyanosis (sTOF) requ

81 so a subset of abnormalities associated with tetralogy of Fallot and the DiGeorge syndrome.

82 We detected associations for tetralogy of Fallot and the upper exposure categories fo

83 l assessment of PR in patients with repaired tetralogy of Fallot and warrants further investigation.

84 cts, 2 with patent ductus arteriosus, 2 with tetralogy of Fallot, and 1 with pulmonary valve dysplasi

85 efects, transposition of the great arteries, tetralogy of Fallot, and anomalous pulmonary venous retu

86 al septal defect, ventricular septal defect, tetralogy of Fallot, and transposition of the great arte

87 ations including ventricular septal defects, tetralogy of Fallot, and tricuspid atresia, defects that

88 ies, left-to-right shunts, valvular disease, tetralogy of Fallot, and truncus arteriosus.

89 and Fgf15 genes in mice are involved in the tetralogy of Fallot, and, using zebrafish phenotypes, pr

90 ; aortic stenosis; coarctation of the aorta; tetralogy of Fallot; and D-transposition of the great ar

91 opmental or neurological outcomes, including tetralogy of Fallot (AOR 2.4, 95% CI: 1.1, 5.4), anencep

92 defect (aOR = 1.19; 95% CI: 1.00, 1.43), and tetralogy of Fallot (aOR = 1.44; 95% CI: 1.01, 2.19) in

93 d incrementally 20 years after operation for tetralogy of Fallot, aortic stenosis and coarctation.

94 Patients with tetralogy of Fallot are at risk for ventricular arrhythm

95 nd sudden cardiac death late after repair of tetralogy of Fallot are devastating complications in adu

96 r (RV) dysfunction in patients with repaired tetralogy of Fallot are incompletely understood.

97 Patients with repaired tetralogy of Fallot are monitored for pulmonary regurgit

98 beyond 30 years after surgical correction of tetralogy of Fallot are nonexistent.

99 of BVGFI with clinical outcomes in repaired tetralogy of Fallot are unknown.

100 Long-term outcomes of transatrial repair of tetralogy of Fallot are unknown.

101 transposition of the great arteries, and 81 tetralogy of Fallot, as well as their unaffected parents

102 of a patient with Abernethy malformation and tetralogy of Fallot associated with nodular regenerative

103 he outcomes of PVR in adults after repair of tetralogy of Fallot at a single tertiary center were ret

104 patients undergoing a transatrial repair of tetralogy of Fallot between 1980 and 2005 were reviewed,

105 In all patients after correction for tetralogy of Fallot, BNP levels were elevated and correl

106 w tract; group 2 (n = 22) received repair of tetralogy of Fallot by the traditional technique with ve

107 ricular septal defect, atrial septal defect, tetralogy of Fallot, coarctation of the aorta, cleft lip

108 t are unlikely to escape clinical diagnosis (tetralogy of Fallot, coarctation of the aorta, transposi

109 55 years after diagnosis and first repair of Tetralogy of Fallot compared to natural progression.

110 adolescent and adult patients with repaired tetralogy of Fallot continues to grow as a result of exc

111 with a cerebellar infarct and a history of a tetralogy of Fallot corrected during childhood.

112 ent (PVR), the most common reintervention in tetralogy of Fallot, could potentially inform therapy to

113 Correction of tetralogy of Fallot (cTOF) often results in pulmonary va

114 aortic arch, aberrant subclavian artery and Tetralogy of Fallot, demonstrating that Hoxa1 is require

115 A growing number of patients with tetralogy of Fallot develop left ventricular systolic dy

116 Adults with repaired tetralogy of Fallot die prematurely from ventricular tac

117 en with transposition of the great arteries, tetralogy of Fallot, double-chamber right ventricle, int

118 The major conotruncal anomalies include tetralogy of Fallot, double-outlet right ventricle, tran

119 al septal defect, ventricular septal defect, tetralogy of Fallot, Ebstein anomaly, transposition of t

120 Patients with repaired tetralogy of Fallot experience variable outcomes for rea

121 of ventricular tachycardia (VT) in repaired Tetralogy of Fallot focuses on isthmuses in the right ve

122 cuspid valve abnormalities, five with severe tetralogy of Fallot, four with Ebstein's anomaly and two

123 t diameter < or =10 mm, diagnosis other than tetralogy of Fallot, Genesis stent, higher prestent RV:a

124 ute; P=0.01) and trended toward lower in the tetralogy of Fallot group (13.5+/-1.29 mL/kg per minute;

125 PVR after repair of tetralogy of Fallot has a low and improving mortality, w

126 ugh nearly one third of adults with repaired tetralogy of Fallot have an aortic root diameter >/=40 m

127 cardiovascular events (MACE) after repair of tetralogy of Fallot have been limited by modest predicti

128 nary valve replacement (PVR) after repair of tetralogy of Fallot have recently been broadened to incl

129 In tetralogy of Fallot, heart defects occurred in seven (3.

130 CHD were studied (atrial septal defect in 6, tetralogy of Fallot in 4, and Fontan procedure in 6).

131 One patient died after surgery for tetralogy of Fallot in situs inversus.

132 Relative to sex-specific controls, repaired tetralogy of Fallot in women had larger right ventricula

133 AGS (e.g., peripheral pulmonic stenosis and tetralogy of Fallot) in the absence of liver dysfunction

134 FINE trial (Right Ventricular Dysfunction in Tetralogy of Fallot: Inhibition of the Renin-Angiotensin

135 Tetralogy of Fallot is a congenital heart disease affect

136 Tetralogy of Fallot is a congenital heart disease that r

137 Complete surgical repair for neonates with tetralogy of Fallot is associated with a significantly h

138 nary valve replacement (PVR) after repair of tetralogy of Fallot is commonly required and is burdenso

139 Clinical outcome after repair of tetralogy of Fallot is determined by the adaptation of t

140 dium, common atrioventricular canal, and the tetralogy of Fallot malformation.

141 btopics identified included heart failure in tetralogy of Fallot, mechanical circulatory support/tran

142 included ventricular septal defect (n = 11), tetralogy of Fallot (n = 3), atrioventricular canal (n =

143 sition of the great arteries (TGA) (n = 69), tetralogy of Fallot (n = 66), hypoplastic left heart syn

144 of the aorta/hypoplastic aortic arch (n=5), tetralogy of Fallot (n=1), hypoplastic right heart (n=1)

145 terial hypertension (PAH; n=10) and repaired tetralogy of Fallot (n=10) underwent MR-augmented cardio

146 syndrome (n=346), tricuspid atresia (n=103), tetralogy of Fallot (n=127), pulmonary atresia (n=177),

147 Patients with repaired tetralogy of Fallot (n=143; 12.5+/-3.2 years) had an ech

148 great arteries after Mustard/Senning (n=2), tetralogy of Fallot (n=2), aortic valve disease (n=2), a

149 The majority had a variant of tetralogy of Fallot (n=36), or transposition of the grea

150 Fifty-four patients with tetralogy of Fallot (n=39), valvar pulmonary stenosis (n

151 es (n=14), anomalous pulmonary veins (n=13), tetralogy of Fallot (n=8), cor triatriatum (n=7), single

152 ive associations between carbon monoxide and tetralogy of Fallot (odds ratio = 2.04, 95% confidence i

153 r "total correction." Patients with repaired tetralogy of Fallot often have pulmonary regurgitation,

154 entricular septal defect was associated with tetralogy of Fallot or double-outlet right ventricle in

155 dults (>/=18 years) with surgically repaired tetralogy of Fallot or pulmonary atresia with ventricula

156 Alagille syndrome and some cases of isolated tetralogy of Fallot or pulmonic stenosis.

157 rmal CA anatomy, especially in patients with tetralogy of Fallot or transposition of the great arteri

158 ncy and specific malformations that resemble tetralogy of Fallot, overriding aorta with ventricular s

159 in contrast to coarctation of the aorta and tetralogy of Fallot (P=0.002; Fisher exact test).

160 Sixty-nine consecutive repaired tetralogy of Fallot patients (43 male, mean 40+/-15 year

161 We included 413 tetralogy of Fallot patients (age, 36 +/- 13 years; QRS

162 Heart Disease (ACHD) centres that follow up Tetralogy of Fallot patients and Great Ormond Street Hos

163 surgical era, this physiology is present in tetralogy of Fallot patients at mid-term follow-up and w

164 Records from 28 consecutive repaired Tetralogy of Fallot patients from 2 centers who underwen

165 Both the PAH and tetralogy of Fallot patients had blunted exercise-induce

166 Although tetralogy of Fallot patients had the largest increase in

167 Tetralogy of Fallot patients predicted to be at high ris

168 onductivity which, when defective, cause the tetralogy of Fallot phenotype.

169 s nearly universal in patients with repaired tetralogy of Fallot, precipitating a complex pathophysio

170 replacement (PVR) in patients with repaired tetralogy of Fallot provides symptomatic benefit and rig

171 ose with pulmonary artery stenosis (n = 94), tetralogy of Fallot/pulmonary atresia (n = 72), congenit

172 med ventricular stimulation in patients with tetralogy of Fallot referred for PVR from January 2020 t

173 med ventricular stimulation in patients with tetralogy of Fallot referred for PVR yields a high rate

174 ,149 infants undergoing 5 common operations: tetralogy of Fallot repair (n = 250), complete atriovent

175 In the first 24 hours after tetralogy of Fallot repair (n=11 patients), serial prosp

176 th adverse clinical outcomes in adults after tetralogy of Fallot repair (rTOF).

177 entricular (RV) dysfunction are common after tetralogy of Fallot repair (rTOF).

178 ren after total cavopulmonary connection and tetralogy of Fallot repair and may prove to be an import

179 1085 consecutive patients receiving standard tetralogy of Fallot repair in a single institution from

180 Thirteen patients, 3 to 35 years after tetralogy of Fallot repair or pulmonary valvotomy, had P

181 ng patients who underwent complete or staged tetralogy of Fallot repair prior to 30 days of age.

182 entricular (RV) restrictive physiology after tetralogy of Fallot repair results in low cardiac output

183 RLS alone explained 5% (tetralogy of Fallot repair) to 20% (Norwood) of variatio

184 After tetralogy of Fallot repair, acute restrictive RV physiol

185 (SCD) complicate the long-term outcome after tetralogy of Fallot repair, yet the diagnostic and predi

186 ry replicating a nontransannular approach to tetralogy of Fallot repair.

187 obal RV function and exercise capacity after tetralogy of Fallot repair.

188 tients undergoing Fontan-type operations and tetralogy of Fallot repair.

189 ysiology can be present early and late after tetralogy of Fallot repair.

190 roportion of patients require PVR late after tetralogy of Fallot repair.

191 Most patients with repaired tetralogy of Fallot require pulmonary valve replacement

192 g atrial and ventricular septal defects, and tetralogy of Fallot, resembling some of the most common

193 After repair of tetralogy of Fallot, right ventricular (RV) dilation has

194 In patients with repaired tetralogy of Fallot (rTOF) and spontaneous ventricular t

195 clinical outcomes in patients with repaired tetralogy of Fallot (rTOF) is unknown.

196 ulmonary valve replacement (PVR) in repaired tetralogy of Fallot (rTOF) rely on cardiovascular magnet

197 icular tachycardia (VT) late after repair of tetralogy of Fallot (rTOF).

198 In patients with tetralogy of Fallot, severe pulmonary stenosis and dimin

199 ally in patients with atrial septal defects, tetralogy of Fallot, single ventricle physiology, and fo

200 Neonates with symptomatic tetralogy of Fallot (sTOF) with hypoplastic pulmonary ar

201 s to management of neonates with symptomatic tetralogy of Fallot (sTOF).

202 We performed a cross-sectional study of tetralogy of Fallot subjects who were tested for 22q11.2

203 erived standard deviation scores in repaired tetralogy of Fallot suggest that women perform poorer th

204 on complex congenital heart defects such as tetralogy of Fallot, systemic right ventricle, and unive

205 f CHDs, ventricular septal defect (VSD), and tetralogy of fallot (TF) with increasing O3 exposure.

206 the effect of PVR in patients with repaired tetralogy of Fallot that developed pulmonary insufficien

207 trant monomorphic ventricular tachycardia in tetralogy of Fallot, the anatomy and histology of these

208 congenital heart disease, such as those with tetralogy of Fallot, the right ventricle (RV) is subject

209 athology has been described in patients with tetralogy of Fallot, the scope of the problem remains po

210 In patients with tetralogy of Fallot, the strongest correlations were obs

211 rom the mandatory follow-up of patients with Tetralogy of Fallot to model the health-related costs an

212 e contribute to cardiac defects ranging from tetralogy of Fallot to transposition of the great arteri

213 and central California carrying infants with tetralogy of Fallot (TOF) (n = 55), dextrotransposition

214 sociated with congenital heart diseases like tetralogy of Fallot (TOF) and pulmonary atresia (PA).

215 Adults late after total correction of tetralogy of Fallot (TOF) are at risk for major complica

216 termine if early primary repair of acyanotic tetralogy of Fallot (ToF) can be performed safely with l

217 Patients after repair of tetralogy of Fallot (ToF) frequently have right ventricu

218 Mortality in patients with tetralogy of Fallot (TOF) has decreased substantially si

219 ) for one-stage versus "two-stage" repair of tetralogy of Fallot (TOF) in infants younger than 1 year

220 ion-repolarization in patients with repaired tetralogy of Fallot (TOF) is a risk factor for malignant

221 e right surgical technique for correction of tetralogy of Fallot (TOF) is contentious for patients wi

222 Tetralogy of Fallot (TOF) is the commonest cyanotic form

223 Tetralogy of Fallot (TOF) is the most common cyanotic co

224 Tetralogy of Fallot (TOF) is the most common cyanotic co

225 Tetralogy of Fallot (TOF) is the most common cyanotic co

226 Tetralogy of Fallot (ToF) is the most common form of com

227 le (RV) to adapt to hypoxia in children with tetralogy of Fallot (TOF) is unknown.

228 Individuals with tetralogy of Fallot (TOF) now routinely survive to repro

229 n (TR) is common among adults with corrected tetralogy of Fallot (TOF) or pulmonary stenosis (PS) ref

230 ion of the aortic root is a known feature in tetralogy of Fallot (TOF) patients with pulmonary stenos

231 ulmonary valve replacement (PVR) in repaired tetralogy of Fallot (TOF) reduces pulmonary regurgitatio

232 The timing of repair of tetralogy of Fallot (TOF) remains controversial.

233 additional haemodynamic abnormalities after tetralogy of Fallot (TOF) repair, who had undergone card

234 mpaired clinical status in late survivors of tetralogy of Fallot (TOF) repair.

235 Tetralogy of Fallot (TOF) typically results in clinical

236 Diagnoses included tetralogy of Fallot (TOF) with pulmonary atresia (n=13),

237 myocardium from 16 infants with nonsyndromic tetralogy of Fallot (TOF) without a 22q11.2 deletion, 3

238 amined the 1q21.1 locus in 948 patients with tetralogy of Fallot (TOF), 1488 patients with other form

239 atrial appendage, ventricular septal defect, tetralogy of Fallot (TOF), and an aortic arch abnormalit

240 heart disease and 36% (n=326) of these have tetralogy of Fallot (TOF), comprising the largest subset

241 Disease entities included were tetralogy of Fallot (TOF), double outlet right ventricle

242 excess familial recurrence of non-syndromic Tetralogy of Fallot (TOF), implicating genetic factors t

243 Tetralogy of Fallot (TOF), the most common severe congen

244 y to search for risk alleles associated with Tetralogy of Fallot (TOF), using a northern European dis

245 Tetralogy of Fallot (TOF)-the most common cyanotic heart

246 ison with a cohort of patients with repaired tetralogy of Fallot (TOF).

247 mortality remain problematic after repair of tetralogy of Fallot (TOF).

248 etermine pregnancy outcomes in patients with tetralogy of Fallot (TOF).

249 vastating late complications after repair of Tetralogy of Fallot (ToF).

250 t ventricular outflow tract of postoperative tetralogy of Fallot (TOF).

251 he transcription factor NKX2.5 as a cause of tetralogy of Fallot (TOF).

252 a large group of preoperative patients with tetralogy of Fallot (TOF).

253 truction in patients after primary repair of tetralogy of Fallot (TOF).

254 with truncus arteriosus (TA), and 15.9% with tetralogy of Fallot (TOF).

255 Outcomes included complex heart defects (tetralogy of Fallot, transposition of the great arteries

256 uctus arteriosus, valvar pulmonary stenosis, tetralogy of Fallot, transposition of the great arteries

257 s, identified higher-risk substrates include tetralogy of Fallot, transposition of the great arteries

258 In patients with tetralogy of Fallot treated with CRT consistent improvem

259 congenital cardiovascular disease, including tetralogy of Fallot, truncus arteriosus and interruption

260 Severe CHD was defined as tetralogy of Fallot, truncus arteriosus, transposition c

261 h hypoplastic left heart syndrome (HLHS) and tetralogy of Fallot, two common forms of cyanotic CHD le

262 Infants with tetralogy of Fallot undergo surgical repair, which requi

263 icenter cohort of 252 patients with repaired tetralogy of Fallot undergoing programmed ventricular st

264 was performed on 205 patients with repaired tetralogy of Fallot undergoing PVR at our institution be

265 Patients with repaired tetralogy of Fallot undergoing PVR with history of ventr

266 3.3 years [range, 8-20 years]) with repaired tetralogy of Fallot underwent cardiac magnetic resonance

267 n age 22+/-11 years; range, 8.5 to 64.9; 72% tetralogy of Fallot) underwent PVR for severe pulmonary

268 Tetralogy of Fallot, univentricular heart, and dextro-tr

269 g long-term follow-up, transatrial repair of tetralogy of Fallot was associated with a minimal risk o

270 A significantly increased risk of tetralogy of Fallot was observed (7 cases in fluconazole

271 The average cost per patient of a repair for Tetralogy of Fallot was pound26,938 (SE = pound4,140).

272 Tetralogy of Fallot was present in 23 subjects and was a

273 Tetralogy of Fallot was present in 55.2%, with native/pa

274 he study population was 21.5+/-11 years, and tetralogy of Fallot was the cardiac condition in 59%.

275 a from 62 consecutive patients with repaired tetralogy of Fallot were analyzed (median age at follow-

276 pulmonary regurgitation following repair of tetralogy of Fallot were considered benign.

277 Autopsy hearts with tetralogy of Fallot were evaluated to clarify the pathol

278 Twenty-seven hearts with the diagnosis of tetralogy of Fallot were examined.

279 h-risk features such as QRS fragmentation in tetralogy of Fallot were identified.

280 Infants with tetralogy of Fallot were smaller in all measured dimensi

281 Her prior history was significant for tetralogy of Fallot which was repaired at nine months of

282 out the best strategy to treat patients with tetralogy of Fallot who are symptomatic in the neonatal

283 replacement (PVR) in patients with repaired tetralogy of Fallot who develop pulmonary insufficiency

284 Patients with repaired tetralogy of Fallot who had a CMR before the occurrence

285 hort study consists of the 144 patients with tetralogy of Fallot who underwent surgical repair at <15

286 nt, PBX1:c.551G>C p.R184P, in a patient with tetralogy of Fallot with absent pulmonary valve and extr

287 French Registry including all patients with tetralogy of Fallot with an ICD was initiated in 2010 by

288 vere form of the human birth defect syndrome tetralogy of Fallot with complete pulmonary artery atres

289 Four groups of tetralogy of Fallot with confluent central pulmonary art

290 Tetralogy of Fallot with cyanosis requiring surgical rep

291 Tetralogy of Fallot with diminutive pulmonary arteries a

292 a novel transcatheter management approach in tetralogy of Fallot with diminutive pulmonary arteries.

293 Tetralogy of Fallot with major aortopulmonary collateral

294 icular outflow tract stenting of symptomatic tetralogy of Fallot with poor anatomy (small pulmonary a

295 child with Alagille syndrome suffering from tetralogy of Fallot with pulmonary atresia and multiple

296 VCF) dysmorphic features, and more common in tetralogy of Fallot with pulmonary atresia than tetralog

297 ere pulmonary stenosis is rare and resembles tetralogy of Fallot with pulmonary atresia: There is a h

298 initially described as a phenotypic mimic of Tetralogy of Fallot with pulmonary atresia; however, sub

299 groups: group 1 (n = 20) received repair of tetralogy of Fallot with the modified technique with tra

300 these patients had undergone total repair of tetralogy of Fallot within 2 wks after admission for RSV