戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 iamine transporter ThTR-2, which may lead to thiamine deficiency.
2 lsive status epilepticus, endocrinopathy, or thiamine deficiency.
3 chanism, which appears to be up-regulated in thiamine deficiency.
4  the diagnosis, treatment, and prevention of thiamine deficiency.
5  cofactors, particularly a low-protein diet, thiamine deficiency, alcoholism, and hypothyroidism.
6 occur during the acute and chronic phases of thiamine deficiency and describe how rodent models of We
7 extending the focus from lethal to sublethal thiamine deficiency, and by linking biochemical alterati
8                    Undiagnosed and untreated thiamine deficiencies are often fatal or lead to severe
9 y, accurate data on the global prevalence of thiamine deficiency are still lacking due to the difficu
10                   Unfortunately, symptoms of thiamine deficiency are variable and clinical determinat
11 us salmonids will continue to be impacted by thiamine deficiency as long as their ocean forage base a
12 mical and cognitive deficits associated with thiamine deficiency as well as proven useful toward grea
13 us on day 10 of PTD treatment, a duration of thiamine deficiency associated with perivascular edema i
14 ley rats were assigned to one of 4 stages of thiamine deficiency based on behavioral symptoms: pre-sy
15 ignificantly, these results demonstrate that thiamine deficiency causes selective cholinergic dysfunc
16                                              Thiamine deficiency disorders, such as thiamine-responsi
17 rrier (BBB) breakdown in the pathogenesis of thiamine deficiency encephalopathy was investigated in R
18  RBE4 cells may be important early events in thiamine-deficiency encephalopathy.
19 rugs inhibit ThTR-2, which may contribute to thiamine deficiency, especially in at-risk populations.
20  Primary and secondary conditions leading to thiamine deficiency have overlapping features in childre
21                             Rodent models of thiamine deficiency have yielded significant insight int
22 ent enzymes, revealed episodically occurring thiamine deficiency in all three animal classes.
23  supplementation may be necessary to correct thiamine deficiency in breastfed infants.
24  and oxidative stress hypothesis for causing thiamine deficiency in California salmon.
25 ently investigated the impacts and causes of thiamine deficiency in California's anadromous salmonids
26 gg surveillance effort that found widespread thiamine deficiency in CCV Chinook salmon in 2020 and 20
27 derstanding sources of thiaminase leading to thiamine deficiency in fisheries and other organisms, in
28 hinook salmon in 2020 and 2021, and emerging thiamine deficiency in Klamath River and Trinity River c
29                  Currently, the diagnosis of thiamine deficiency in remote regions is hampered due to
30 defect of TRMA may shed light on the role of thiamine deficiency in such common diseases.
31 iovascular traits previously associated with thiamine deficiency, including elevated cardiac stroke v
32 ces from granulocytes may be responsible for thiamine deficiency-induced vascular breakdown and periv
33                           One major cause of thiamine deficiency is an overreliance on diet items con
34                      It is unlikely that the thiamine deficiency is caused by impaired phosphorylatio
35                                              Thiamine deficiency is common in parts of Asia and cause
36  effects, we demonstrate that the problem of thiamine deficiency is considerably more widespread and
37 ative role of the BBB in the pathogenesis of thiamine deficiency is discussed.
38 dle income countries are at a higher risk of thiamine deficiency, mainly due to the lack of dietary d
39                       In those with baseline thiamine deficiency, patients in the thiamine group had
40                               The effects of thiamine deficiency produced by addition of pyrithiamine
41               Using the pyrithiamine-induced thiamine deficiency (PTD) animal paradigm of WKS, our la
42 ith the thalamus of the pyrithiamine-induced thiamine deficiency (PTD) rat model of Wernicke's enceph
43                  In the pyrithiamine-induced thiamine deficiency (PTD) rat model of WKS, there are si
44                         Pyrithiamine-induced thiamine deficiency (PTD) was used to produce a rodent m
45 f diencephalic amnesia, pyrithiamine-induced thiamine deficiency (PTD), was used to investigate dienc
46 iberi, a potentially fatal disease caused by thiamine deficiency, remains a public health concern in
47 ied several known (Down syndrome RR = 116.1, thiamine deficiency RR = 76.1, and Parkinson's disease R
48                                 Experimental thiamine deficiency (TD) is a classical model of a nutri
49                                 Experimental thiamine deficiency (TD) is a model of impaired oxidativ
50                                              Thiamine deficiency (TD) models the cellular and molecul
51 n gut contents, and stable isotope data link thiamine deficiency to an ocean diet dominated by a boom