ライフサイエンスコーパス: thyroiditis

1 h SLE and AITD (Graves' disease or Hashimoto thyroiditis).

2 in human autoimmune thyroiditis (Hashimoto's thyroiditis).

3 Hashimoto disease is a chronic autoimmune thyroiditis.

4 like structures is a hallmark of Hashimoto's thyroiditis.

5 ion of granulomatous experimental autoimmune thyroiditis.

6 ation of susceptibility genes for autoimmune thyroiditis.

7 and amelioration of experimental autoimmune thyroiditis.

8 ded our understanding of the pathogenesis of thyroiditis.

9 igation that have been applied to autoimmune thyroiditis.

10 e the NOD.H-2(h4) mouse model of spontaneous thyroiditis.

11 es that resemble those observed in Hashimoto thyroiditis.

12 before developing hypothyroidism, suggesting thyroiditis.

13 y present in the pathogenesis of Hashimoto's thyroiditis.

14 these families: T1D, RA, SLE, and Hashimoto thyroiditis.

15 sites and in the development of spontaneous thyroiditis.

16 s allowed for the development of spontaneous thyroiditis.

17 sue from patients diagnosed with Hashimoto's thyroiditis.

18 such as the autoimmune disease, Hashimoto's thyroiditis.

19 most often caused by autoimmune (Hashimoto) thyroiditis.

20 ase-limiting role of IFN-gamma in autoimmune thyroiditis.

21 T cell response to mTg and failed to develop thyroiditis.

22 thyrocytes, but did not develop spontaneous thyroiditis.

23 nt autoimmune diseases including Hashimoto's thyroiditis.

24 autoimmune disorders, particularly Hashimoto thyroiditis.

25 hyroid dysfunction that occurs in autoimmune thyroiditis.

26 tiation between Graves' disease and painless thyroiditis.

27 ized by the sera of patients with autoimmune thyroiditis.

28 ti-RT6.1 mAb induced autoimmune diabetes and thyroiditis.

29 -versus-host disease, multiple sclerosis and thyroiditis.

30 ce with mouse or human Tg resulted in severe thyroiditis.

31 t none of the patients actually had subacute thyroiditis.

32 B rats develop spontaneous hyperglycemia and thyroiditis.

33 pe 1 diabetes: celiac disease and autoimmune thyroiditis.

34 de toxic nodules and the thyrotoxic phase of thyroiditis.

35 nodular goiter, hypothyroidism, and subacute thyroiditis.

36 tis, Sjogren's-like syndrome, urticaria, and thyroiditis.

37 ystemic lupus erythematosus, and Hashimoto's thyroiditis.

38 celiac disease and 1000 cases of autoimmune thyroiditis.

39 betes, rheumatoid arthritis, and Hashimoto's thyroiditis.

40 ated with sex or the presence of lymphocytic thyroiditis.

41 reased risk for celiac disease or autoimmune thyroiditis.

42 c patterns for GD, TMNG, TA, and destructive thyroiditis.

43 ng and presentation to T-cells in autoimmune thyroiditis.

44 y correlated with the progress of autoimmune thyroiditis.

45 ht prevent missing a diagnosis of autoimmune thyroiditis.

46 lphian lymph node during different stages of thyroiditis.

47 Eight patients had a history of Hashimoto thyroiditis.

48 f hyperthyroidism consistent with autoimmune thyroiditis.

49 uitary gland), features found in Hashimoto's thyroiditis.

50 pithelial cells of patients with Hashimoto's thyroiditis.

51 h other abnormalities of hyperthyroidism and thyroiditis.

52 s anaemia (0.79 [0.72-0.86]) and Hashimoto's thyroiditis (0.81 [0.75-0.86]) significantly decreased i

53 -3) mm(2)/sec, and in patients with painless thyroiditis 1.46+/-0.22x10(-3) mm(2)/sec, respectively.

54 vs. 1%), sarcoidosis (1% vs. 1%), autoimmune thyroiditis (1% vs. 0%), type 1 diabetes (1% vs. 0%), or

55 enile rheumatoid arthritis, 1 with Hashimoto thyroiditis, 1 with psoriasis and iritis, 1 with diabete

56 The most common were Hashimoto thyroiditis (10%), psoriasis (6%), inflammatory bowel di

57 5.2-32.0]), and thyroid disease (Hashimoto's thyroiditis 13.3 [11.8-14.9] and Graves' disease 6.7 [5.

58 Immune-related AEs included grade 1 or 2 thyroiditis (13%), grade 1 rash (6%), and grade 3 esopha

59 showed autoimmune disorders, i.e. autoimmune thyroiditis (26.3%), dermatitis herpetiformis (4%) and d

60 d CD (29%) developed ADs (mainly Hashimoto's thyroiditis, 29 cases), compared with a smaller proporti

61 enced ongoing autoimmune problems, including thyroiditis (3), hemolysis (1), thrombocytopenia (4), an

62 ic thyroiditis that mimics human Hashimoto's thyroiditis, a disease where iodine, IFN-gamma, and adhe

63 , the study suggests that MYMD-1 ameliorates thyroiditis acting on specific lymphoid subsets.

64 a (PTC) and thyroid epithelia in Hashimoto's thyroiditis activates nuclear factor-kappa B (NF-kappaB)

65 iabetes, rheumatoid arthritis, lupus, Graves thyroiditis, Addison disease and other autoimmune disord

66 nce of rheumatoid arthritis (RA), autoimmune thyroiditis (AIT), multiple sclerosis (MS), and insulin-

67 hypothyroidism, hyperthyroidism, autoimmune thyroiditis (AIT), serum concentrations of thyroid-stimu

68 ence of type 1 diabetes (T1D) and autoimmune thyroiditis (AITD).

69 chronic autoimmune thyroiditis (Hashimoto's thyroiditis), although other causes, including drugs (su

70 Hashimoto thyroiditis (an autoimmune disease) is the cause of prim

71 ic stimulation in the setting of Hashimoto's thyroiditis and aberrant somatic hypermutation may play

72 between the ultrasonography (US) results of thyroiditis and characteristics of the Delphian lymph no

73 ET scan of 2 patients, ICI-related toxicity (thyroiditis and colitis) was detected.

74 iated with chronic lymphocytic (Hashimoto's) thyroiditis and does not seem to be affected by thyroid

75 ction, type 1 diabetes mellitus, Hashimoto's thyroiditis and Graves' disease, Sjogren's syndrome, der

76 me to target tissues involved in Hashimoto's thyroiditis and Graves' disease, we performed ex vivo an

77 diseases, chronic lymphocytic (Hashimoto's) thyroiditis and Graves' disease.

78 hism determines susceptibility to autoimmune thyroiditis and implicate Tg as an important autoantigen

79 es showed coexistent, biopsy-proven Riedel's thyroiditis and infiltrative soft tissue along the right

80 ifestations include, among others, vitiligo, thyroiditis and paraneoplastic syndromes, concurrent wit

81 story included hypothyroidism from Hashimoto thyroiditis and pneumonia with left pleural effusion.

82 is an excellent animal model for Hashimoto's thyroiditis and provides a unique opportunity to investi

83 from the metabolic biomarkers in Hashimoto's thyroiditis and psoriasis (METHAP) clinical trial.

84 hyroid-infiltrating T cell of a patient with thyroiditis and specific for a cryptic thyroid-peroxidas

85 evaluate the association between autoimmune thyroiditis and the Delphian lymph node during different

86 The association of Hashimoto's thyroiditis and thyroid cancer remains an active focus o

87 ort no linkage between serologic Hashimoto's thyroiditis and thyroid cancer, yet they are limited by

88 Eleven other patients with thyroiditis and two with Graves' disease recognized only

89 children had autoimmune comorbidity, such as thyroiditis and type 1 diabetes.

90 that SjD patients diagnosed with autoimmune thyroiditis and/or hypothyroidism were significantly mor

91 both autoimmune hypothyroidism (Hashimoto's thyroiditis) and autoimmune thyrotoxicosis (Graves' dise

92 gkin's lymphoma [NHL]); endocrine (diabetes, thyroiditis); and rheumatologic (Sjogren's syndrome).

93 , 2 had multiple sclerosis, 2 had autoimmune thyroiditis, and 7 had other conditions.

94 rats, including insulin-dependent diabetes, thyroiditis, and experimental autoimmune uveitis.

95 lin-dependent diabetes mellitus, Hashimoto's thyroiditis, and primary ovarian failure).

96 seases such as systemic lupus erythematosus, thyroiditis, and rheumatic autoimmune diseases.

97 dal lymphocytes isolated from a patient with thyroiditis, and unexpectedly, thyroid follicular cells

98 hip between thyroid lymphoma and Hashimoto's thyroiditis appears to be well established.

99 ronic inducible urticaria (>10%), autoimmune thyroiditis (approximately 20%), metabolic syndrome (6%-

100 e regulatory T cells that prevent autoimmune thyroiditis are generated in vivo only when the relevant

101 Hypothyroidism and Hashimoto's thyroiditis are more common than hyperthyroidism and Gra

102 We used thyroiditis as a model to dissect these possibilities.

103 thyroxine at position 2553 (T4p2553) induces thyroiditis as well as strong specific T and B cell resp

104 ed the incidence and severity (p < 0.001) of thyroiditis, as assessed by histopathology.

105 Tg-cleaving activity in IgG from autoimmune thyroiditis (ATh) and systemic lupus erythematosus (SLE)

106 , thymus hyperplasia, autoimmune lymphocytic thyroiditis, autoimmune hemolytic anemia, and colitis.

107 matoid arthritis, Graves' disease, Hashimoto thyroiditis, autoimmune thyroid disease, and systemic lu

108 on self-Ag presentation, not only suppressed thyroiditis but also prevented reemergence of the diseas

109 lls (effector CD8(+) T cells) present in ICI-thyroiditis but not HT or healthy controls.

110 s chronic lymphocytic thyroiditis (Hashimoto thyroiditis), but there are many other causes.

111 rther diagnostic work-up revealed autoimmune thyroiditis, but no signs of inflammatory bowel disease.

112 cells offset the suppression of H2E-mediated thyroiditis by H2A.

113 Previous studies have shown that autoimmune thyroiditis can be induced in normal laboratory rats aft

114 Subacute thyroiditis can be unilateral or focal (1,2).

115 Postpartum thyroiditis can occur up to 1 year after delivery and mu

116 roiditis cases; Group 4: advanced-late stage thyroiditis cases.

117 2: indeterminate cases; Group 3: established thyroiditis cases; Group 4: advanced-late stage thyroidi

118 utonomously functioning thyroid nodules, and thyroiditis caused by inflammation, which results in rel

119 ase (TPO), a region frequently recognized in thyroiditis, cDNA sequences coding for peptide fragments

120 )) and seven autoimmune diseases (autoimmune thyroiditis, celiac disease, inflammatory bowel disease

121 mice treated with Flt3-L showed more severe thyroiditis characterized by enhanced lymphocytic infilt

122 TD), Graves' disease and chronic lymphocytic thyroiditis (CLT) are amongst the most common endocrine

123 blication of a paper showing that autoimmune thyroiditis could be induced in animals.

124 umatoid arthritis, Graves disease, Hashimoto thyroiditis, Crohn disease, ulcerative colitis, systemic

125 Thyroiditis developed in aged CD7/CD28-double-deficient

126 etes, ICI-induced arthritis, and ICI-induced thyroiditis due to the wealth of knowledge about the dev

127 Less common etiologies include destructive thyroiditis (e.g., amiodarone-induced thyroid dysfunctio

128 eptide when inducing experimental autoimmune thyroiditis (EAT) in NOD mice expressing human DRbeta1-A

129 i-B7.2 had decreased experimental autoimmune thyroiditis (EAT) severity compared with recipients of c

130 model (A(-)E(+)) of experimental autoimmune thyroiditis (EAT) that permits disease induction with he

131 to rat neu and mTg with resultant autoimmune thyroiditis (EAT) were both enhanced.

132 Experimental autoimmune thyroiditis (EAT) with granulomatous histopathological f

133 LA-DR) predispose to experimental autoimmune thyroiditis (EAT), a classical mouse model of HT.

134 e other hand, murine experimental autoimmune thyroiditis (EAT), a model for HT, presents a clear link

135 ent of granulomatous experimental autoimmune thyroiditis (EAT), DBA1 mice with a disrupted IFN-gamma

136 t the development of experimental autoimmune thyroiditis (EAT), experimental autoimmune myasthenia gr

137 ent of granulomatous experimental autoimmune thyroiditis (EAT), IL-4 gene-disrupted mice expressing t

138 diseases, as well as experimental autoimmune thyroiditis (EAT).

139 ut also to suppress, experimental autoimmune thyroiditis (EAT).

140 elops in up to 15% of patients with subacute thyroiditis, even more than 1 y after presentation.

141 he cases with a sonographic finding matching thyroiditis formed Group 2.

142 ion of granulomatous experimental autoimmune thyroiditis (G-EAT) at least in part through regulation

143 Granulomatous experimental autoimmune thyroiditis (G-EAT) is induced by mouse thyroglobulin-se

144 Granulomatous experimental autoimmune thyroiditis (G-EAT) is induced by mouse thyroglobulin-se

145 Granulomatous experimental autoimmune thyroiditis (G-EAT) is induced by mouse thyroglobulin-se

146 When granulomatous experimental autoimmune thyroiditis (G-EAT) was induced in CBA/J or DBA/1 mice,

147 ion of granulomatous experimental autoimmune thyroiditis (G-EAT) was promoted when thyroid epithelial

148 del of granulomatous experimental autoimmune thyroiditis (G-EAT) was used to determine the role of TG

149 erse events (myositis in addition to grade 3 thyroiditis, grade 3 hepatitis, grade 3 pneumonia, and g

150 have linked an increased risk of autoimmune thyroiditis, Graves disease and goitre to low selenium s

151 Fifty percent of the strains susceptible to thyroiditis had a unique SNP haplotype at exons 10 and 1

152 of the mouse strains that were resistant to thyroiditis had this SNP haplotype (P = 0.01).

153 ommon cause in adults is chronic lymphocytic thyroiditis (Hashimoto thyroiditis), but there are many

154 ause of hypothyroidism is chronic autoimmune thyroiditis (Hashimoto's thyroiditis), although other ca

155 oral autoimmune response in human autoimmune thyroiditis (Hashimoto's thyroiditis).

156 entation in patients with chronic autoimmune thyroiditis have generally resulted in reduced thyroid a

157 expression of ICAM-1 in this mouse model of thyroiditis, highlighting the complex interplay present

158 49, 1.01], p-value = 0.053), and Hashimoto's thyroiditis (HR 1.06, 95% CI [0.91, 1.23], p-value = 0.4

159 95, 2.26], p-value = 0.087), and Hashimoto's thyroiditis (HR 1.14, 95% CI [0.97, 1.33], p-value = 0.1

160 Graves' disease (GD) and Hashimoto's thyroiditis (HT) are common autoimmune diseases of the t

161 s of orbitopathy associated with Hashimoto's thyroiditis (HT) have rarely been described.

162 Hashimoto's thyroiditis (HT) is associated with HLA, but the associa

163 One such gene implicated in Hashimoto's thyroiditis (HT) is HLA-DR3, but the association is weak

164 uals with spontaneous autoimmune Hashimoto's thyroiditis (HT) or no thyroid disease.

165 comprising Graves disease (GD) and Hashimoto thyroiditis (HT), develop as a result of a complex inter

166 cluding Graves' disease (GD) and Hashimoto's thyroiditis (HT), is one of the most common of the immun

167 Hashimoto's thyroiditis (HT), one of the most common autoimmune dise

168 imately 30% of CIU patients have Hashimoto's thyroiditis (HT).

169 include Graves disease (GD) and Hashimoto's thyroiditis (HT); although these diseases contrast clini

170 ed one grade 1 reversible immune-related AE (thyroiditis, hyperthyroidism).

171 Thus, interferon induced thyroiditis (IIT) is a major clinical problem for patien

172 1, may mediate the occurrence of Hashimoto's thyroiditis in AIS1-susceptible family members.

173 We report a case of Coccidioides thyroiditis in an HIV-infected patient with a history of

174 ted their effects on experimental autoimmune thyroiditis in CBA/J mice.

175 xistence of CD4(+)CD25(+) T cells regulating thyroiditis in E(+)B10.Ab(0) (A(-)E(+)) and B10 (A(+)E(-

176 A two-locus analysis of Hashimoto thyroiditis in family members segregating an AIS1 suscep

177 r similar to what is observed in Hashimoto's thyroiditis in humans.

178 with a modestly increased risk of autoimmune thyroiditis in infancy analysis.

179 more frequently caused by chronic autoimmune thyroiditis in iodine-replete areas.

180 P = 7.5 x 10(-10)), which is associated with thyroiditis in other cohorts.

181 differentiate Graves' disease from painless thyroiditis in patients with untreated thyrotoxicosis.

182 ajor role in the expression of insulitis and thyroiditis in the BB rat, that Th1 lymphocytes may pred

183 ore the challenge to determine their role in thyroiditis in the presence of both H2A and H2E genes.

184 Moreover, mTg1677 induced mild thyroiditis in Treg-depleted B10 mice, and in E(+)B10 mi

185 r, whether treatment can prevent post-partum thyroiditis in women who are or have been antibody posit

186 follicular cells in experimental autoimmune thyroiditis, in a manner similar to what is observed in

187 regions is most commonly caused by Hashimoto thyroiditis, in pregnancy is important for the health of

188 disorders, Graves disease (GD) and Hashimoto thyroiditis, in which perturbations of immune regulation

189 Experimental autoimmune thyroiditis, induced in mice after challenge with thyrog

190 depletion of CD4(+)CD25(+) T cells enhanced thyroiditis induction in the context of either H2E or H2

191 ditionally resistant B10 (H2(b)) mice permit thyroiditis induction with mouse thyroglobulin (mTg) aft

192 molecules (E(+)B10 mice), are susceptible to thyroiditis induction without Treg depletion.

193 sed risk for autoimmune disorders, including thyroiditis, inflammatory bowel disease, rheumatoid arth

194 H2A genes is protective against H2E-mediated thyroiditis, inhibiting EAT development.

195 dermatomyositis, Graves' disease, Hashimoto thyroiditis, insulin-dependent diabetes mellitus, inflam

196 dence and severity of spontaneous autoimmune thyroiditis [iodide-accelerated spontaneous autoimmune t

197 r important causes of thyrotoxicosis include thyroiditis, iodine-induced and drug-induced thyroid dys

198 Subacute thyroiditis is a self-limited inflammatory disorder of t

199 Hashimoto's thyroiditis is associated with mitochondrial dysfunction

200 prevalent in older women, in whom autoimmune thyroiditis is common.

201 en than men, and post partum exacerbation of thyroiditis is common.

202 ty and resistance to experimental autoimmune thyroiditis is encoded by MHC H2A genes.

203 The lymphocytic infiltration of Hashimoto's thyroiditis is frequently associated with papillary thyr

204 The lymphocytic infiltration of Hashimoto's thyroiditis is frequently encountered in thyroid glands

205 Subacute thyroiditis is generally felt to have a viral etiology,

206 results suggest that spontaneous autoimmune thyroiditis is inhibited in mice expressing transgenic T

207 dioactive iodine, and thyroidectomy, whereas thyroiditis is managed symptomatically or with glucocort

208 Although subacute thyroiditis is often related to a viral infection, no in

209 Thyroiditis is one of the most common IrAEs, but the cau

210 Since subacute thyroiditis is self-limited, treatment is directed towar

211 Post-partum thyroiditis is substantially more frequent in women who

212 s, which suffers from spontaneous autoimmune thyroiditis, is an excellent animal model for Hashimoto'

213 op iodine-accelerated spontaneous autoimmune thyroiditis (ISAT) with chronic inflammation of the thyr

214 s [iodide-accelerated spontaneous autoimmune thyroiditis (ISAT)] in NOD.H2(h4) mice.

215 opment of lymphocytic spontaneous autoimmune thyroiditis (L-SAT) in NOD.H-2h4 mice and inhibits the d

216 h develop lymphocytic spontaneous autoimmune thyroiditis (L-SAT), all TGF-beta transgenic (Tg) mice g

217 ch tolerance is typically lost in autoimmune thyroiditis leading to hypothyroidism.

218 -requiring diabetes mellitus and lymphocytic thyroiditis (LT).

219 Subacute thyroiditis may be caused by a viral infection in geneti

220 Thyrotoxicosis from thyroiditis may be observed if symptomatic or treated wi

221 pressive effect of H2A genes on H2E-mediated thyroiditis mirrors previous reports of H2E suppression

222 4), neutropenia (n = 4), mucositis (n = 2), thyroiditis (n = 1), and rash (n = 1).

223 mmune neutropenia, n = 1) and other tissues (thyroiditis, n = 3; psoriasis, n = 2; Graves disease, n

224 ld block the continuous T-cell activation in thyroiditis needed to maintain the autoimmune response t

225 mily cluster in which vitiligo and Hashimoto thyroiditis occur in numerous individuals.

226 Post-partum thyroiditis occurs in 5-10% of women, and many of those

227 One serious adverse event-autoimmune thyroiditis of grade 2 severity-was reported in the vite

228 or diagnosis of hypothyroidism or autoimmune thyroiditis, of whom 56 were receiving thyroxine therapy

229 effective in relieving symptoms of subacute thyroiditis, often within 24 hr (3).

230 thyroid specimens from individuals with ICI-thyroiditis, one of the most common IRAEs, and compared

231 n-autoimmune IIT can manifest as destructive thyroiditis or as hypothyroidism with negative thyroid a

232 (MS), autoimmune thyroid disease (Hashimoto thyroiditis or Graves disease), juvenile RA, inflammator

233 Nineteen were found with autoimmune thyroiditis or hypothyroidism, and replacement therapy w

234 causes and comorbidities, such as autoimmune thyroiditis or infections.

235 ce, which developed an extensive lymphocytic thyroiditis or insulitis that nevertheless did not elimi

236 cytes could transfer experimental autoimmune thyroiditis or L-SAT to Tg mice, indicating that the tra

237 ied associations with additional phenotypes: thyroiditis (OR = 0.58, p = 1.4 x 10(-5)), nodular (OR =

238 such as pneumonitis, colitis, hypophysitis, thyroiditis, or myocarditis.

239 values between Graves' disease and painless thyroiditis (P=0.001).

240 al Ig-gene libraries derived from autoimmune thyroiditis patients and specific for the main autoantig

241 otes are absent from a cohort of Hashimoto's thyroiditis patients.

242 C3H/gld mice, lupus patients, and autoimmune thyroiditis patients.

243 odine increases the incidence of Hashimoto's thyroiditis, perhaps by augmenting the antigenicity of t

244 autoimmune diseases, including scleroderma, thyroiditis, primary biliary cirrhosis, Sjogren syndrome

245 family history of thyroid cancer, autoimmune thyroiditis, prior radiation exposure, cancer predisposi

246 Similar to index cases, Hashimoto thyroiditis, psoriasis, and inflammatory bowel disease w

247 ng thyroidectomy with coexisting Hashimoto's thyroiditis report an increased prevalence of papillary

248 ve been published in the field of autoimmune thyroiditis (represented by Graves' disease and Hashimot

249 We previously reported that in wild-type, thyroiditis-resistant BALB/c mice that underwent regress

250 Although thyroiditis risk was slightly increased, risk for thyroi

251 OD.H-2h4 mice develop spontaneous autoimmune thyroiditis (SAT) and anti-mouse thyroglobulin (MTg) aut

252 OD.H-2h4 mice develop spontaneous autoimmune thyroiditis (SAT) and produce anti-mouse thyroglobulin a

253 ed for development of spontaneous autoimmune thyroiditis (SAT) in NOD.H-2h4 mice where they function

254 Spontaneous autoimmune thyroiditis (SAT) is an organ-specific autoimmune diseas

255 OD.H-2h4 mice develop spontaneous autoimmune thyroiditis (SAT) when given 0.05% NaI in their drinking

256 in a murine model of spontaneous autoimmune thyroiditis (SAT), B cells were depleted from adult NOD.

257 presented by Graves' disease and Hashimoto's thyroiditis) since January 2005.

258 significant difference in the prevalence of thyroiditis, Sjogren's syndrome, or Hodgkin's or NHL.

259 yroid lymphomas in patients with Hashimoto's thyroiditis strongly suggests a pathogenetic link betwee

260 row transplantation, patient 1 had Hashimoto thyroiditis, suggesting that organ-specific autoimmunity

261 icularly either Graves' disease or Hashimoto thyroiditis, suggesting the possibility of different pat

262 gland during the early stages of autoimmune thyroiditis suggests a possible effector function of CD1

263 spontaneously develop autoimmune lymphocytic thyroiditis that mimics human Hashimoto's thyroiditis, a

264 ifferentiating Graves' disease from painless thyroiditis, the best result was obtained with area unde

265 ogenetic process of autoimmune (Hashimoto's) thyroiditis, the most common cause of hypothyroidism in

266 cluded atopy, granulomatous rash, autoimmune thyroiditis, the presence of antinuclear antibodies, sin

267 ed where the initial impression was subacute thyroiditis, there was a clinical response to prednisone

268 gest that sunitinib may induce a destructive thyroiditis through follicular cell apoptosis.

269 Autoimmune postpartum and subacute thyroiditis, tumours that secrete thyrotropin, and drug-

270 severe infections and autoimmunity including thyroiditis, type 1 diabetes, coeliac disease and alopec

271 es' disease or toxic nodular goitre, whereas thyroiditis (typically autoimmune, viral, or drug induce

272 reated PVG donors were still able to prevent thyroiditis upon adoptive transfer.

273 oth peptides induced experimental autoimmune thyroiditis upon direct challenge of CBA/J mice with pep

274 portantly, treatment of mice with autoimmune thyroiditis using mouse thyroglobulin (mTg)-pulsed anti-

275 minority of CS patients, include Hashimoto's thyroiditis, vascular malformations and mental retardati

276 Diagnosis of Coccidioides thyroiditis was made based on histopathologic examinatio

277 Neck pain due to thyroiditis was reported in up to 18% of patients, but i

278 The prevalence of Hashimoto thyroiditis was significantly higher in the relatives of

279 Risk of autoimmune thyroiditis was similar by vaccination status.

280 One serious adverse event (thyroiditis) was noted.

281 In a 23-year-old patient with thyroiditis, we identified a novel rearrangement leading

282 In a humanized model of autoimmune thyroiditis, we investigated the mechanism underlying br

283 rom macroglobulinemia, cryoglobulinemia, and thyroiditis were all <.0038, the Bonferroni threshold fo

284 , and might be the only treatment needed for thyroiditis, which is transient.

285 and mice spontaneously developed destructive thyroiditis with histological, clinical and hormonal sig

286 12 induced severe, destructive granulomatous thyroiditis with neutrophil inflammation, fibrin deposit