ライフサイエンスコーパス: thyrotoxicosis

1 poorly with the mouse TSHR and do not cause thyrotoxicosis.

2 art disease, size of goiter, and severity of thyrotoxicosis.

3 mones that produce the symptoms and signs of thyrotoxicosis.

4 ly used to differentiate different causes of thyrotoxicosis.

5 ementia, but data are limited for iatrogenic thyrotoxicosis.

6 nless thyroiditis in patients with untreated thyrotoxicosis.

7 among 37 consecutive patients with untreated thyrotoxicosis (25 female and 12 male; mean age of 44 ye

8 ery, infection, acute myocardial infarction, thyrotoxicosis, acute alcohol consumption, acute pericar

9 goitre, because of the high relapse rate of thyrotoxicosis after discontinuation.

10 Amiodarone-induced thyrotoxicosis (AIT) is a difficult management problem a

11 respiratory failure, myocardial infarction, thyrotoxicosis, alcohol, pericarditis, pulmonary embolis

12 Amiodarone-associated thyrotoxicosis and Graves' disease represented the main

13 Treatment that safely alleviates peripheral thyrotoxicosis and reverses cerebral hypothyroidism is n

14 In this review, the causes of thyrotoxicosis and the treatment of syndromes with incre

15 id molecular imaging in patients affected by thyrotoxicosis and thyroid nodules.

16 beta blockers are used in symptomatic thyrotoxicosis, and might be the only treatment needed f

17 tening thyrotoxicosis in pregnancy, neonatal thyrotoxicosis, and post-partum hyperthyroidism.

18 Other types of thyrotoxicosis are monitored and treated with beta-block

19 urs, choriocarcinoma, and amiodarone-induced thyrotoxicosis are, more often than not, a challenge to

20 y peripheral vasodilatation during sepsis or thyrotoxicosis, blood shunting, or reduced blood oxygen

21 Gestational transient thyrotoxicosis can also occur during pregnancy and shoul

22 Iatrogenic thyrotoxicosis can cause symptoms, and, even when mild,

23 Thyrotoxicosis causes a variety of symptoms and adverse

24 Thyrotoxicosis, celiac disease, acquired hemolytic anemi

25 ts include age > or =75 years; hypertension; thyrotoxicosis; diabetes; cardiovascular disease; conges

26 ne therapy may be associated with iatrogenic thyrotoxicosis, especially in elderly patients, and ther

27 on the clinical context as due to endogenous thyrotoxicosis, exogenous thyrotoxicosis, or unknown cau

28 the lack of D3 function results in neonatal thyrotoxicosis followed later by central hypothyroidism

29 Thyrotoxicosis from thyroiditis may be observed if sympt

30 ism (Hashimoto's thyroiditis) and autoimmune thyrotoxicosis (Graves' disease).

31 Patients exposed to thyrotoxicosis had cognitive disorder incidence of 11.0%

32 Unlike thyrotoxicosis, hypothyroidism is related to a persisten

33 olished or reduced uptake whereas productive thyrotoxicosis (i.e., hyperthyroidism "sensu strictu") i

34 ate the consequences of untreated peripheral thyrotoxicosis in patients with MCT8 deficiency.

35 sm during pregnancy, severe life-threatening thyrotoxicosis in pregnancy, neonatal thyrotoxicosis, an

36 Common symptoms of thyrotoxicosis include anxiety, insomnia, palpitations,

37 Other important causes of thyrotoxicosis include thyroiditis, iodine-induced and d

38 Other causes of hyperthyroidism and thyrotoxicosis include toxic nodules and the thyrotoxic

39 Thyrotoxicosis is a common disorder, especially in women

40 Iatrogenic thyrotoxicosis is a common result of thyroid hormone the

41 Thyrotoxicosis is a general term for excess circulating

42 Destructive thyrotoxicosis is characterized by abolished or reduced

43 Clinical thyrotoxicosis is directly caused by autoantibodies that

44 Gestational transient thyrotoxicosis is typically reported in women with hyper

45 This chronic thyrotoxicosis leads to progressive deterioration in bod

46 Individuals with overt thyrotoxicosis (n = 4) were excluded because of small nu

47 , and might be the only treatment needed for thyrotoxicosis not caused by excessive production and re

48 Clinically significant thyrotoxicosis occurring in metastatic thyroid carcinoma

49 Thyrotoxicosis occurs in some patients and is related to

50 appeared to be a risk factor for developing thyrotoxicosis (odds ratio, 4.0; P=0.17), and amiodarone

51 acute attacks of weakness, hypokalemia, and thyrotoxicosis of various etiologies.

52 r (OR = 0.69, p = 3.9 x 10(-5)) goiters, and thyrotoxicosis (OR = 0.76, p = 1.5 x 10(-3)), but not Gr

53 due to endogenous thyrotoxicosis, exogenous thyrotoxicosis, or unknown cause, excluding those attrib

54 ies involving the thyroid (hypothyroidism or thyrotoxicosis), pituitary (hypophysitis), adrenal gland

55 This is a case of thyrotoxicosis, presumably due to Jod-Basedow syndrome,

56 nd secretion from the thyroid gland, whereas thyrotoxicosis refers to the clinical syndrome of excess

57 stratified by cause and severity, exogenous thyrotoxicosis remained a significant risk factor (adjus

58 Apart from the common causes of thyrotoxicosis, such as Graves' disease and functioning

59 en 1946 and 1964 in the original Cooperative Thyrotoxicosis Therapy Follow-up Study; 91 % had Graves

60 SH level from either endogenous or exogenous thyrotoxicosis was associated with higher risk of incide

61 After adjustment, all-cause thyrotoxicosis was significantly associated with risk of

62 Key features of peripheral thyrotoxicosis were alleviated in paediatric and adult p

63 toms or signs of potential hypothyroidism or thyrotoxicosis were seen.

64 Thyroid storm is the most severe form of thyrotoxicosis, with high mortality, and is treated with

65 y autoimmune, viral, or drug induced) causes thyrotoxicosis without hyperthyroidism.