ライフサイエンスコーパス: tibial nerve

1 diabetic peripheral neuropathy (DPN) of the tibial nerve.

2 cause macrophage infiltration of the distal tibial nerve.

3 conducted via electrical stimulation of the tibial nerve.

4 nd TA and stimulating cuffs on the posterior tibial nerve.

5 y reduced after capsaicin application on the tibial nerve.

6 APs of C-fibers could not be detected in the tibial nerve.

7 l sciatic nerve, mid-tibial nerve and distal tibial nerve.

8 , and demonstrates high expression levels in tibial nerve.

9 m/s) measured either in vivo or in isolated tibial nerves.

10 after stimulating ulnar, median, fibular and tibial nerves.

11 Tibial nerve action potentials were weaker, and recovery

12 les induced by electrical stimulation of the tibial nerve activated these dorsal horn neurons.

13 how morphological differences in the sciatic tibial nerve after 5 months of hyperglycemia when compar

14 Stimulation of the tibial nerve after paralysis with pancuronium had no eff

15 mal sciatic nerve, distal sciatic nerve, mid-tibial nerve and distal tibial nerve.

16 After severing the tibial nerve and subsequent transplantation of hair foll

17 interosseus muscles after stimulation of the tibial nerve and the low-frequency-dependent depression

18 ctrical stimulation was applied to the right tibial nerve, and standardized low-resolution brain elec

19 its branches such as the peroneal nerve, the tibial nerve, and the sural nerve.

20 Images of rat sciatic and human cadaver tibial nerves are presented, illustrating the applicabil

21 by applying an electrical stimulation to the tibial nerve as adolescents (Age = 14.8 +/- 2.5 yrs.) an

22 ciatic nerve at the sciatic notch and of the tibial nerve at the ankle were significantly reduced in

23 iatic nerve at the sciatic notch or near the tibial nerve at the ankle.

24 e at the middle level of the thigh or on the tibial nerve at the lower level of the lower hindlimb al

25 otential evoked calcium signals in mammalian tibial nerve axons using an in vitro mouse model with a

26 caused by electrical stimulation of afferent tibial nerve axons.

27 and lower limb mechanics with and without a tibial nerve block that prevented contraction of these m

28 rgery to treat foot drop entails rerouting a tibial nerve branch to the denervated common fibular ner

29 g, stimulation of the central end of the cut tibial nerve, brief repeated carotid occlusions and caro

30 Tibial nerve CES prior to DNT significantly improved fun

31 Negative correlations were found for the tibial nerve conduction velocity (r = -0.23; 95% CI: -0.

32 Negative correlations were found for the tibial nerve conduction velocity (r = -0.24; 95% CI: -0.

33 Mean tibial nerve cross-sectional area was lower in the metfo

34 Topical application of capsaicin (1%) on the tibial nerve did not affect SCS-induced vasodilation at

35 ither the proximal sural nerve nor the motor tibial nerve exhibit axon loss.

36 ele at this locus was associated with higher tibial nerve expression of an adjacent gene (SCN2A) codi

37 e likely to reflect pathology in sciatic and tibial nerve fibers.

38 chondria of small, medium and large diameter tibial nerve fibers.

39 e injury by direct electrical stimulation of tibial nerve fibres, confirming that centrally mediated

40 trating strong associations with sciatic and tibial nerve findings.

41 beta, mRNA expression in the rat sciatic and tibial nerves following ischemia-reperfusion (IR) injury

42 ment transport in mature myelinated axons of tibial nerves from male and female mice of this strain e

43 ed by applying electrical stimulation to the tibial nerve in 22 persons with CP and 25 neurotypical c

44 ), True Blue (TB) or Fluoro-Ruby (FR) in the tibial nerve in adult Spague-Dawley rats.

45 The reflex was evoked by stimulating the tibial nerve in decerebrated rats whose femoral artery w

46 ibular nerve crush, CES was delivered to the tibial nerve in half the animals, and at 2 weeks, all an

47 Common fibular or tibial nerves in thy-1-YFP-H mice were cut bilaterally a

48 cle contraction evoked by stimulation of the tibial nerve increased mean arterial pressure (MAP) and

49 female rats with neuropathic pain induced by tibial nerve injury (TNI).

50 ycytidine) model of AIDS therapy-induced and tibial nerve injury-related peripheral neuropathy.

51 HDAC6 inhibitor normalized cisplatin-induced tibial nerve mitochondrial deficits.

52 easurements included peroneal nerve skin and tibial nerve muscle sympathetic activities; the electroc

53 r, combined interventions improved sural and tibial nerve myelin thickness, hind paw epidermal innerv

54 Na, K, Cl, Ca, Mg, P and S) in rat posterior tibial nerve myelinated axons and Schwann cells.

55 nduction velocity deficits, and reduction in tibial nerve myelinated fiber diameter, but not intraepi

56 d not prevent morphometric changes in distal tibial nerve myelinated fibers.

57 nificant cis-eQTLs for PMF1 (P = 1 x 10-4 in tibial nerve), NBEAL1, FAM117B and CARF (P < 2.1 x 10-7

58 covered proximal (sciatic nerve) and distal (tibial nerve) nerve segments of the lower extremity.

59 eceptive properties of regenerated cutaneous tibial nerve nociceptors, and to obtain evidence for cou

60 Histological examination of the tibial nerve of EAN animals revealed that flecainide pro

61 sites adjacent to the sciatic, peroneal, and tibial nerves of one leg.

62 arge myelinated fibers were decreased in the tibial nerves of the hyperglycemic animals.

63 ach of the supplying arteries to the sciatic-tibial nerves of the right hind limb was ligated and the

64 rats the effects of ligating and cutting one tibial nerve on sensory function and on density of inner

65 , 20 s, C-fiber strength) was applied to the tibial nerve or the neuronal receptive field.

66 ) amplitudes of median, ulnar, peroneal, and tibial nerves (P < 0.001), but was not related to cortic

67 nally, iPSCMNs grafted into transected mouse tibial nerve projected axons to denervated gastrocnemius

68 scle action potentials ratio of peroneal and tibial nerves (split index, SI) and semi-quantitative sc

69 Conclusion Tibial nerve stiffness measurements appear to be highly

70 ssess the long-term efficacy of percutaneous tibial nerve stimulation (PTNS) in fecal incontinence (F

71 Percutaneous tibial nerve stimulation (PTNS) is a new ambulatory ther

72 Using tibial nerve stimulation at the knee as a screening test

73 Percutaneous tibial nerve stimulation continues to display superiorit

74 e of sacral neuromodulation and percutaneous tibial nerve stimulation in the treatment of men with ur

75 Posterior tibial nerve stimulation is not covered here.

76 Both sacral neuromodulation and percutaneous tibial nerve stimulation prove to be viable, durable opt

77 ight-sided (i) handgrip; and (ii) median and tibial nerve stimulation were assessed using functional

78 eal nerve; test reflex elicited by posterior tibial nerve stimulation) was used during: (1) rest, (2)

79 l injection of botulinum toxin, percutaneous tibial nerve stimulation, sacral neuromodulation, and su

80 (MAP) and heart rate (HR) following a 2-min tibial nerve stimulation-evoked static muscle contractio

81 trols, but no increased activation following tibial nerve stimulation.

82 ve stimulation, but reduced activations with tibial nerve stimulation.

83 observed for the soleus H-reflex elicited by tibial nerve stimulation.

84 ld produced qualitatively similar results to tibial nerve stimulation.

85 the femoral, common peroneal, and posterior tibial nerves (targeting the quadriceps femoris, tibiali

86 n, Merkel cells are lost from the denervated tibial nerve territory but are relatively preserved in n

87 g initial loss of cutaneous afferents in the tibial nerve territory, we observed progressive centripe

88 ernodal length (0.95-1.3 mm) in axons of the tibial nerve that varied in proportion to the mechanical

89 sociated with SEC31B expression in heart and tibial nerve tissue.

90 ese possibilities in rats after crushing the tibial nerve (TN), and using Vesicular GLUtamate Transpo

91 data from skeletal muscle, whole blood, and tibial nerve to test the effects of disease-associated p

92 Adult rats underwent complete tibial nerve transection followed by microsurgical reatt

93 e soft tissue injury and three had posterior tibial nerve transection; the average MESS was 7.6.

94 The tibial nerve was examined at 4 cm proximal to the medial

95 emained at rest for the entire study, or the tibial nerve was stimulated, as in the contraction group

96 FA of the sciatic and tibial nerves was lowest in the sDPN group.

97 mic compound action potentials (CAPs) of the tibial nerve were recorded in response to SCS.

98 At that time, entire sciatic-tibial nerves were harvested and fixed in situ.

99 Motor nerve conduction velocities of the tibial nerves were slowed.

100 s, a stimulus was delivered to the posterior tibial nerve while the ipsilateral leg was moving either

101 in the rat, C nociceptors isolated from the tibial nerve with receptive fields (RFs) on the plantar