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1  mediated by HO using the chemical inhibitor tin protoporphyrin.
2 ity by EFS was inhibited by the HO inhibitor Tin protoporphyrin (1 x 10(-4) mol/L).
3                 Inhibition of HO activity by tin protoporphyrin 9 reverses the component of endotheli
4             Enzyme studies demonstrated that tin protoporphyrin-9 is a selective inhibitor of HO with
5                                              Tin protoporphyrin, a selective inhibitor of HO, reverse
6                  In contrast, treatment with tin protoporphyrin abolished the benefit of BC1 gene tra
7                          Inhibition of HO by tin protoporphyrin abrogated the impairment of resistanc
8 amycin and, more importantly, the effects of tin protoporphyrin, an inhibitor of HO activity, on the
9 toporphyrin, an inducer of HO-1 activity, or tin protoporphyrin, an inhibitor of HO-1.
10                                 In contrast, tin protoporphyrin and tin mesoporphyrin did not display
11                          The HO-1 inhibitor, tin protoporphyrin, blocked MP4CO protection, consistent
12  Inhibition of HO activity by treatment with tin protoporphyrin blunted survival advantage in Tg mice
13 closporin A), inhibition of HO-1 activity by tin protoporphyrin, caused graft rejection in 3--7 days.
14 treatment of Ad-HO-1 with the HO-1 inhibitor tin protoporphyrin completely restored each morphometric
15                 Heme oxygenase inhibition by tin protoporphyrin exacerbates stasis in sickle mice.
16         Under inhibition of HO-1 activity by tin protoporphyrin, exogenous CO suppressed graft reject
17 nhibition of heme oxygenase via injection of tin protoporphyrin IX (20 micromol/kg intraperitoneally)
18 Th3/(+) We observed that HO inhibition using tin protoporphyrin IX (SnPP) decreased heme-iron recycli
19 ence and absence of neurohumoral inhibitors (tin protoporphyrin IX [SnPP IX] for CO synthesis, N(omeg
20 cts of PMA are prevented by the HO inhibitor tin protoporphyrin IX and in cultures from mice with del
21               The heme oxygenase-1 inhibitor tin protoporphyrin IX completely blocked the effect of S
22             Administration of HO-1 inhibitor tin protoporphyrin IX dichloride in infected BALB/c mice
23                                              Tin protoporphyrin IX did not affect heme oxygenase-1 ex
24 n vivo heme oxygenase enzyme inhibition with tin protoporphyrin IX in common bile duct ligation anima
25 ase or heme oxygenase-1 inhibitors (1400W or tin protoporphyrin IX).
26                 MGd used in combination with tin protoporphyrin IX, an inhibitor of HO1, resulted in
27                         HO-1 inhibition with tin-protoporphyrin IX and silencing with RNA interferenc
28 y bilirubin and abolished by incubation with tin protoporphyrin-IX and knock down of nuclear factor-E
29 s potentiated by the HO inhibitors, zinc and tin protoporphyrin-IX as well as by the CO scavenger, he
30                Treatment of the animals with tin protoporphyrin-IX, a global HO inhibitor, or HO-1 sm
31 ent of sheared SMCs with the HO-1 inhibitor, tin protoporphyrin-IX, blocked the antiaggregatory effec
32 th DHCR24 and HO-1 small interfering RNA and tin-protoporphyrin-IX treatment abolished these effects.
33 al cells with HO-1 small interfering RNA and tin-protoporphyrin-IX treatment did not inhibit the (A-I
34 CR24 and HO-1 and systemic administration of tin-protoporphyrin-IX, an HO inhibitor, abolished these
35 acological inhibition of HO-1 activity using tin protoporphyrin or knockdown of HO-1 prevents the ind
36  the induction of HO-1 and was attenuated by tin protoporphyrin (SnPP) IX, an inhibitor of HO-1 activ
37                        Inhibition of HO-1 by tin protoporphyrin (SnPP) or siRNA downregulated Pax3/7-
38 O inhibitors (zinc protoporphyrin (ZnPP) and tin protoporphyrin (SnPP)) suppressed the channel in a m
39 proliferation was inhibited by ZnPP, whereas tin protoporphyrin (SnPP), another equally potent HO-1 i
40 sing a competitive inhibitor of HO activity, tin protoporphyrin (SnPP), in protocols affording a comp
41             The effects of the HO inhibitor, tin protoporphyrin (SnPP), on brain electrical activity
42 ondiabetic and diabetic animals treated with tin protoporphyrin (SnPP, a heme oxygenase-1 enzyme inhi
43   When HO-1 enzymatic activity is blocked by tin protoporphyrin (SnPPIX) or the action of CO is inhib
44 decreased in the presence of HO-1 inhibitor, tin protoporphyrin (SnPPIX).
45 n vitro with the heme oxygenase-1 inhibitor, tin protoporphyrin, substantially decreased survival and
46                                              Tin protoporphyrin treatment normalized carboxyhemoglobi