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1 children and young adults using pulsed-wave tissue Doppler imaging.
2 ere studied using echocardiography including tissue Doppler imaging.
3 and Brain Lesions) study by traditional and tissue Doppler imaging.
4 tween baseline and 38 weeks as determined by tissue doppler imaging.
5 adjusted diastolic impairment as measured by tissue Doppler imaging.
6 7.6+/-1.2 versus 8.5+/-1.2 cm/s, P=0.003) by tissue Doppler imaging.
7 al myocardial contraction measured by pulsed tissue Doppler imaging.
8 tinely measured left atrial volume index and tissue-doppler-imaging a' allowed LACI calculation as (l
10 emental to that of left atrial volume index, tissue-doppler-imaging a', or any other characteristic i
11 nsive cardiac function assessment, including tissue Doppler imaging and 2-dimensional (2D) strain ech
15 e technical features of strain imaging using tissue Doppler imaging and speckle tracking, their stren
16 exercise testing, echocardiography including tissue-Doppler imaging and speckle tracking, and cardiov
17 es obtained from ventricular time intervals, tissue Doppler imaging, and color M-mode echocardiograph
18 her mitral annular velocities as assessed by tissue Doppler imaging are associated with invasive meas
19 We identified all diabetic patients with a tissue Doppler imaging assessment of diastolic function
21 on during sepsis, as demonstrated by in vivo tissue Doppler imaging, better-preserved left ventricle
23 imary abnormalities in HCM, we reasoned that tissue Doppler imaging could identify contraction and re
24 echocardiographic techniques (predominantly tissue Doppler imaging) currently in the process of stan
26 tolic pressure correlated significantly with tissue Doppler imaging-derived systolic:diastolic durati
27 were independent predictors of tau, whereas tissue Doppler imaging-derived systolic:diastolic durati
28 e diastolic tricuspid annular velocity (A'), tissue Doppler imaging-derived systolic:diastolic durati
29 relates of the change in the transmitral and tissue Doppler imaging diastolic indexes included sex, a
31 biventricular dysfunction using pulsed-wave tissue Doppler imaging early after HT with most signific
32 onal (2D) imaging echocardiography, Doppler, tissue Doppler imaging echocardiography, and speckle-tra
34 c (A') velocities obtained using pulsed-wave tissue Doppler imaging in 380 healthy children were used
35 the mitral annular velocities (e' and a') by tissue Doppler imaging in 650 participants (mean age, 50
36 ons and the resultant S:D ratio using pulsed tissue Doppler imaging in children with idiopathic or fa
37 scores for normalized pulse wave Doppler and tissue Doppler imaging in pediatric echocardiography.
38 ssed by two-dimensional echocardiography and tissue Doppler imaging, including the load-independent s
39 sion, and systolic and diastolic pulsed-wave tissue Doppler imaging indices were similar to controls
42 y affect long-axis ventricular function, and tissue Doppler imaging is the most sensitive test to ide
44 time intervals and the MPI assessed by color tissue Doppler imaging M-mode through the mitral leaflet
46 a complete echocardiographic study including tissue Doppler imaging, magnetic resonance imaging (MRI)
51 ed with exercise intolerance and (2) whether tissue Doppler imaging of the early diastolic mitral ann
56 went dobutamine stress echocardiography with tissue Doppler imaging on 2 separate occasions: the firs
60 ent echocardiographic examination, including tissue Doppler imaging, simultaneously with left heart c
61 ement of transthoracic echocardiography with tissue Doppler imaging, speckle tracking, and three-dime
64 c assessment of longitudinal dyssynchrony by tissue Doppler imaging (TDI) and radial dyssynchrony by
70 chocardiography-derived variables, including tissue Doppler imaging (TDI) parameters and strain rate
71 eveloped echocardiographic modality based on tissue Doppler imaging (TDI) that allows quantitative as
72 using 2-dimensional echocardiography (2DE), tissue Doppler imaging (TDI), and 3-dimensional echocard
73 c function, including color M-mode (CMM) and tissue Doppler imaging (TDI), and of right ventricular (
74 echocardiography, Doppler echocardiography, tissue Doppler imaging (TDI), and strain rate imaging.
75 and annular early diastolic velocity (Ea) by tissue Doppler imaging (TDI), T(E-Ea), which is well rel
76 l longitudinal strain, and showed elevated E/tissue Doppler imaging (TDI)-e', markedly diminished pea
77 left ventricular (LV) diastolic function via tissue-Doppler imaging (TDI) as well as Doppler flow var
78 ontinuous association of diastolic measures (tissue Doppler imaging [TDI] e', E/e', and left atrial s
79 cardiac parameters by echocardiography (eg, Tissue Doppler Imaging [TDI] S-wave R=0.52, P<0.001) and
80 time as a ratio of cycle length, and several tissue Doppler imaging techniques to assess intraventric
82 ic echocardiography, pulse wave Doppler, and tissue Doppler imaging velocities are widely used to ass
84 ral left ventricular (LV) inflow velocity to tissue Doppler imaging velocity of the medial mitral ann
85 he passive transmitral LV inflow velocity to tissue Doppler imaging velocity of the medial mitral ann
86 he passive transmitral LV inflow velocity to tissue Doppler imaging velocity of the medial mitral ann
91 M-mode, 2D, Doppler echocardiography and tissue Doppler imaging were performed in nontransgenic (
92 Shortening fraction and myocardial strain by tissue doppler imaging were quantified by echocardiograp