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1 ry, and chemokine expression, and normalized tissue survival.
2 tration resulted in the greatest decrease in tissue survival.
3 le factor 1alpha stabilization and increased tissue survival.
4  pathological TNF-alpha reaction and promote tissue survival.
5 lar networks in vertebrates are essential to tissue survival.
6 ry progression, suggesting an active role in tissue survival.
7 al activation of these receptors may promote tissue survival.
8  adversely affect microvascular function and tissue survival after an ischemic episode, and previous
9 l mitogenesis, a repair process important to tissue survival after ischemic damage, was not disrupted
10  deletion of CXCL12 (eKO) modulates ischemic tissue survival, altering tissue repair and tumor progre
11  suggests that sepsis disturbs post-ischemic tissue survival and brain remodeling.
12                       The promotion of brain tissue survival and inhibition of thromboinflammation id
13 e results highlight a pathway that preserves tissue survival and muscle function in the setting of is
14 wth, differentiation, homing to their target tissues, survival and activation are all controlled, to
15 al to evaluate implant integration with host tissue, survival, and functionality.
16  playing roles in signaling, cell migration, tissue survival, anti-inflammation, and T-cell-mediated
17 d energy is utilized to ensure that cell and tissue survival are maintained.
18 vity (firing rate and thermosensitivity) and tissue survival as a function of time and slice thicknes
19 ligonucleotide to CD47 or control agents and tissue survival assessed.
20  therapeutic peptide that increases cell and tissue survival by acting as a cofactor to PDGF-BB.
21 to assess endogenous cytokine expression and tissue survival comparable to undelayed TRAM flaps.
22 1%, P<0.005) that resulted in impaired gross tissue survival compared with young mice (2 to 6 months)
23 diated vascular smooth muscle relaxation and tissue survival following ischemic injury in skin flaps
24 cking TSP1-CD47 signaling increases ischemic tissue survival in random cutaneous porcine flaps.
25 ergetic resources in astrocytes help promote tissue survival in response to focal neuronal stress.
26                                 Furthermore, tissue survival of ischemic injury and acute recovery of
27 tanding of how Hh signaling governs cell and tissue survival remains incomplete.
28 pholino knock down of CD47 greatly increased tissue survival to ischemia.
29 imulated blood flow and growth and composite tissue survival to ischemia.
30             Cerebral blood flow required for tissue survival was higher in the mutants, leading to in
31 myocutaneous flap model for ischemic injury, tissue survival was significantly enhanced in thrombospo
32 rin loss may be incompatible with epithelial tissue survival, whereas partial compensation can result