ライフサイエンスコーパス: tissue transglutaminase

1 tin recognition site within this sequence of tissue transglutaminase.

2 dependent on excess activity of cell surface tissue transglutaminase.

3 l digestion and that have been deamidated by tissue transglutaminase.

4 cells requires deamidation of the protein by tissue transglutaminase.

5 ucts of physiological digestion of gluten by tissue transglutaminase.

6 highly specific substrate for deamidation by tissue transglutaminase.

7 bly occurring as a result of cell leakage of tissue transglutaminase.

8 s to immobilized maltose-binding protein and tissue transglutaminase.

9 ts with a monoclonal antibody raised against tissue transglutaminases.

10 lization of antibodies against epidermal and tissue transglutaminases.

11 830 children tested positive for IgA against tissue transglutaminase (1.0%; 95% confidence interval,

12 e (14%), and anti-IgA autoantibodies against tissue transglutaminase (12%).

13 tory of celiac disease or antibodies against tissue transglutaminase 2 (controls), healthy family mem

14 (~30%) to both human Factor XIII (FXIII) and tissue transglutaminase 2 (hTG2).

15 Tissue transglutaminase 2 (TG2) has been of particular i

16 Tissue transglutaminase 2 (TG2) is a multifunctional pro

17 Tissue transglutaminase 2 (TG2) is overexpressed in epit

18 igratory cells release microvesicles rich in tissue transglutaminase 2 (Tg2) which activate murine fi

19 progression by interacting with its ligand, tissue transglutaminase 2 (TG2), but the molecular basis

20 While H3Q5ser is known to be installed by tissue transglutaminase 2 (TGM2), the substrate characte

21 We demonstrate that tissue transglutaminase 2 can serotonylate histone H3 tr

22 isease patients, who have antibodies against tissue transglutaminase 2 in the absence of villous atro

23 st results (for levels of antibodies against tissue transglutaminase-2 and deamidated gliadin peptide

24 , distinct from the two CD hallmark antigens tissue transglutaminase-2 and deamidated gliadin, exhibi

25 ymes (lysyl oxidase, lysyl oxidase-like 2-4, tissue transglutaminase-2), and ECM turnover genes/enzym

26 s between fibrillar collagens, introduced by tissue transglutaminase-2, are necessary for the self-as

27 le as 60 microU of purified guinea pig liver tissue transglutaminase (4.2 ng or 54 fmol of enzyme).

28 ed nonmitochondrial autoantibody was against tissue transglutaminase (57.1% of ALF patients).

29 Reducing tissue transglutaminase activity in collagen gels contai

30 /-0.35 to 13.93+/-4.21 U/mg DNA in cytosolic tissue transglutaminase activity was seen.

31 of 60 to 120 kJ per m2 resulted in increased tissue transglutaminase activity when measured either in

32 en and transforming growth factor beta1, and tissue transglutaminase activity.

33 zing agent maitotoxin to increase endogenous tissue transglutaminase activity.

34 induced in cortical neurons (ie, cyclin D3, tissue transglutaminase, alpha1-antichymotrypsin, and ST

35 These effects are specific for tissue transglutaminase and are not shared by its functi

36 These include elevated tissue transglutaminase and consequent inactivation thro

37 uding serologic tests for antibodies against tissue transglutaminase and deamidated gliadin peptide,

38 strates and/or modulators of enzymes such as tissue transglutaminase and ecto-protein kinases that mo

39 ntion of the profibrotic cleavage substrates tissue transglutaminase and endoglin accompanied MMP-14

40 lder were tested for CD based on analysis of tissue transglutaminase and endomysial antibodies.

41 ed on results of serum tests for IgA against tissue transglutaminase and endomysium or on both a heal

42 pgli), as well as for IgA antibodies against tissue transglutaminase and endomysium.

43 The number of cells showing increases in tissue transglutaminase and its cross-link product, epsi

44 It was shown recently that tissue transglutaminase and presumably plasma transgluta

45 In conclusion, IGFBP-1 is a substrate for tissue transglutaminase and Tg leads to the formation of

46 Sera were tested for tissue transglutaminase and, if abnormal, for endomysial

47 ee or four antibody tests including IgA anti-tissue transglutaminase and/or IgA anti- endomysium perm

48 est results (IgA/IgG gliadin, endomysium, or tissue transglutaminase) and compared them with 213,208

49 st three of which, namely, TGase 1, TGase 2 (tissue transglutaminase), and TGase 3, are present in th

50 ad antibodies against gliadin, 1.22% against tissue transglutaminase, and 0.61% against endomysium (P

51 d peroxidase, gastric parietal cells (PCAs), tissue transglutaminase, and 21-hydroxylase was tested u

52 slinking of VEGF165, catalysed by the enzyme tissue transglutaminase, and associates with MVs through

53 entromere, chromatin, soluble liver antigen, tissue transglutaminase, and deaminated gliadin peptides

54 s (IgA anti-dpgli, IgG anti-dpgli, IgA anti- tissue transglutaminase, and IgA anti-endomysium) yielde

55 d factors such as age, sex, diabetes status, tissue transglutaminase, and smoking status have emerged

56 though high levels of autoantibodies against tissue transglutaminase (anti-TG2) are strongly indicati

57 d of 10 years and analyzed for antibodies to tissue transglutaminase (anti-TG2A) using a radiobinding

58 Serum samples were analyzed for anti-tissue transglutaminase (anti-tTG) concentrations at age

59 iations between levels of antibodies against tissue transglutaminase (anti-tTG, a marker of celiac di

60 (ACPAs), antinuclear antibodies (ANAs), anti-tissue transglutaminase antibodies (AGTAs), and anti-thy

61 by positive results from serologic tests for tissue transglutaminase antibodies (anti-TG2) but normal

62 oped and optimized for determination of anti-tissue transglutaminase antibodies (anti-tTG) in human s

63 th positive results from serologic tests for tissue transglutaminase antibodies (tTGA) without endosc

64 nd specific screening serologic tests (anti- tissue transglutaminase antibodies IgA [anti-TTG] and ed

65 disease autoimmunity was defined as positive tissue transglutaminase antibodies in two consecutive se

66 Positivity for IgA tissue transglutaminase antibodies was detected in 97%.

67 els of soluble E-selectin (sE-sel), IgA anti-tissue transglutaminase antibodies, and serum IL-8 level

68 Simultaneously, anti-tissue transglutaminase antibodies, questionnaire result

69 he sensitivities of Simtomax, endomysial and tissue-transglutaminase antibodies were compared in 133

70 , P = 0.026) LC density also correlated with tissue transglutaminase antibody (Anti-TtG) levels asses

71 It captures the target tissue transglutaminase antibody (anti-tTG), and finally

72 c screening for celiac disease using the IgA tissue transglutaminase antibody (IgA tTG) and, if posit

73 were younger (P = .03), had lower titers of tissue transglutaminase antibody (P = .001), and less fr

74 ed for markers of CD autoimmunity using anti-tissue transglutaminase antibody (tTG) and anti-endomysi

75 estigations revealed a slightly elevated IgA tissue transglutaminase antibody level in the setting of

76 Tissue transglutaminase appears to have a significant ro

77 t domain revealed that amino acids 81-140 of tissue transglutaminase are involved in fibronectin bind

78 In addition the levels of tissue transglutaminase, as determined by quantitative i

79 By age 3-years, persistent tissue transglutaminase autoantibodies (tTGA), i.e., cel

80 wed up for up to 20 years for development of tissue transglutaminase autoantibodies (tTGA).

81 Screening for CD was performed with tissue transglutaminase autoantibodies (tTGA).

82 llowed from birth were screened annually for tissue transglutaminase autoantibodies (tTGAs).

83 were defined as being positive for islet or tissue transglutaminase autoantibodies at 2 consecutive

84 ac disease autoimmunity, defined as positive tissue transglutaminase autoantibodies found in 2 consec

85 Screening for celiac disease with tissue transglutaminase autoantibodies was performed ann

86 he TEDDY study who were tested for islet and tissue transglutaminase autoantibodies, respectively.

87 ed by intestinal biopsy or persistently high tissue transglutaminase autoantibody levels.

88 hese results suggest that a risk of islet or tissue transglutaminase autoimmunity need not influence

89 Tissue transglutaminase belongs to the multigene transgl

90 Transglutaminase 2 (TG2, a.k.a. tissue transglutaminase) belongs to a family of transglu

91 The protein cross-linking enzyme tissue transglutaminase binds in vitro with high affinit

92 Thus, induction of tissue transglutaminase by all-trans-RA and, surprisingl

93 Both factor XIIIa and tissue transglutaminase catalyzed the polymerization of

94 The results suggest that tissue transglutaminase-catalyzed covalent linkages invo

95 The glutamate at P6, which is formed by tissue transglutaminase-catalyzed deamidation, is an imp

96 alysis, showing a parallel increase in renal tissue transglutaminase content.

97 nt upon transglutamination, we determined if tissue transglutaminase could catalyze this reaction and

98 -linking pattern similar to that observed in tissue transglutaminase cross-linked fibrin(ogen) with m

99 ell death with dying cells showing extensive tissue transglutaminase cross-linking of intracellular p

100 Adhesive function of tissue transglutaminase does not require its cross-linki

101 nsor based on the covalent immobilization of tissue transglutaminase enzyme in its open conformation

102 roduce cornified envelope precursors and the tissue transglutaminase enzyme that cross-links them.

103 New diagnostic options include the tissue transglutaminase enzyme-linked immunosorbent assa

104 This observed association between tissue transglutaminase, epsilon-(gamma-glutamyl) lysine

105 Retinoid and interleukin-6 inductions of tissue transglutaminase expression are mediated by speci

106 Furthermore, simvastatin decreased tissue-transglutaminase expression and IMV inward remode

107 r and extracellular compartments elicited by tissue transglutaminase following exposure to ultraviole

108 to Ala significantly reduced the affinity of tissue transglutaminase for fibronectin, indicating that

109 cular mechanisms mediating the regulation of tissue transglutaminase gene expression by TGF-beta fami

110 GF-beta1, BMP2, and BMP4 regulation of mouse tissue transglutaminase gene expression requires a compo

111 t retinoid-dependent expression of the mouse tissue transglutaminase gene is mediated by a versatile

112 oids regulate the transcription of the mouse tissue transglutaminase gene via activation of regulator

113 he regulation of the expression of the human tissue transglutaminase gene.

114 lating the tissue-specific expression of the tissue transglutaminase gene.

115 t G protein, Gh, that can also function as a tissue transglutaminase, has been described.

116 estinal inflammatory disease, and studies on tissue transglutaminase have generated significant new i

117 sitive serology for immunoglobulin A against tissue transglutaminase, health care provider diagnosis,

118 en in coeliac disease, and to explore if the tissue transglutaminase homologues found in other organi

119 Human tissue transglutaminase (hTG2) is a multifunctional enzy

120 nducted, including serological tests such as Tissue transglutaminase IgA (tTG-IgA), Endomysium Antibo

121 By comparing salivary and serum levels of tissue transglutaminase IgA (tTG-IgA), this study aims t

122 ease had detectable gluten in faeces, whilst tissue transglutaminase IgA antibodies (TGA) continued t

123 Her positive anti-tissue transglutaminase IgA antibodies and ensuing duode

124 patients with concentrations of IgA against tissue transglutaminase (IgA-TTG) >10-fold the upper lim

125 red; patients should also be tested for anti-tissue transglutaminase, IgA against deamidated gliadin

126 ational modification of proteins mediated by tissue transglutaminase II (TGase), a GTP-binding protei

127 Tissue transglutaminase II (TGase-II), which is capable

128 mboxane receptor (TP), but also functions as tissue transglutaminase II (tTG).

129 ed formation of noncovalent MK multimers nor tissue transglutaminase II covalent multimerization of M

130 coefficient between the CDAT score and anti-tissue transglutaminase immunoglobulin A (anti-t-TG-IgA)

131 detection and diagnosis of CD, particularly tissue transglutaminase-immunoglobulin A (TG2-IgA), IgA

132 the role of the protein cross-linking enzyme tissue transglutaminase in changes associated with the e

133 n between gluten, immune responsiveness, and tissue transglutaminase in coeliac disease.

134 ainty evidence for an increased risk of anti-tissue transglutaminase in patients with IBD vs controls

135 udy, we have investigated the involvement of tissue transglutaminase in the development of kidney fib

136 eriments clearly demonstrate localization of tissue transglutaminase in the nucleus that can be activ

137 dehydrogenase is less strongly inhibited by tissue transglutaminase in the presence of constructs co

138 3-phosphate dehydrogenase is inactivated by tissue transglutaminase in the presence of glutathione S

139 patients have circulating antibodies against tissue transglutaminase; in children, European guideline

140 disease assay using an assay for IgA against tissue transglutaminase; in subjects with positive test

141 Overexpression of tissue transglutaminase increases its amount on the cell

142 nied by the cross-linking of fibronectin and tissue transglutaminase into nonreducible high molecular

143 Tissue transglutaminase is a calcium-dependent enzyme th

144 Tissue transglutaminase is a calcium-dependent transamid

145 Tissue transglutaminase is a calcium-dependent, protein

146 Gluten deamidation by human tissue transglutaminase is critical to activate celiac d

147 Tissue transglutaminase is expressed at low levels in th

148 stoma SH-SY5Y cells demonstrated that 93% of tissue transglutaminase is localized to the cytosol.

149 The expression of tissue transglutaminase is regulated by a variety of mol

150 Competing autoantibodies against tissue transglutaminase (marking coeliac disease autoimm

151 rring in rats submitted to SNx suggests that tissue transglutaminase may play an important role in th

152 Together our results demonstrate that tissue transglutaminase mediates the interaction of inte

153 nt with earlier reports indicating that host tissue transglutaminase modification of gliadin enhances

154 s regulation was paralleled by a decrease in tissue transglutaminase mRNA in MC3T3 E1 cells.

155 the lower limit of detection and IgG against tissue transglutaminase or deamidated gliadin peptide, o

156 GTP-binding protein/transglutaminases (tissue transglutaminases or TGases) have been implicated

157 dehydrogenase complex also is inactivated by tissue transglutaminase plus glutathione S-transferase c

158 Among tests for celiac disease, IgA tissue transglutaminase presented the best diagnostic te

159 Retinoid-dependent activation of the tissue transglutaminase promoter depends on both a proxi

160 rs in CV-1 cells demonstrated that the mouse tissue transglutaminase promoter is activated by ligand

161 - (BMP) and BMP4-dependent inhibition of the tissue transglutaminase promoter.

162 ly conserved between the human and the mouse tissue transglutaminase promoters and a distal region th

163 s been the discovery of a potential role for tissue transglutaminase, recently found to be the autoan

164 ology with total and food-specific IgE, anti-tissue transglutaminase, skin prick testing, food intole

165 hances gliadin-specific CD T-cell responses, tissue transglutaminase specifically deamidated Q65 in t

166 molecular methodologies to demonstrate that tissue transglutaminase (TG) activates downstream NF-kap

167 Tissue transglutaminase (TG) is a Ca2+-dependent acyltra

168 Tissue transglutaminase (TG) is an enzyme that stabilize

169 Following incubation with pure tissue transglutaminase (Tg), IGFBP-1 formed covalently

170 g for the inducible enzyme/adhesion molecule tissue transglutaminase (TG), we demonstrate coordinate

171 major necessary genetic predisposition, and tissue transglutaminase (TG2) as mechanistically involve

172 lcium-dependent protein crosslinking enzyme, tissue transglutaminase (TG2) but no direct link between

173 The ubiquitous cross-linking enzyme tissue transglutaminase (TG2) has been implicated in irr

174 Gluten lacks such peptides, but tissue transglutaminase (TG2) introduces negatively char

175 Tissue transglutaminase (TG2) is a multifunctional Ca(2+

176 Tissue transglutaminase (TG2) is a multifunctional enzym

177 Tissue transglutaminase (TG2) is a widely distributed mu

178 Tissue transglutaminase (TG2) is involved in Ca(2+)-depe

179 The multifunctional protein tissue transglutaminase (TG2) is known to be secreted in

180 We tested the hypothesis that tissue transglutaminase (TG2) modifies FXIII-directed fi

181 c expression of the pro-inflammatory protein tissue transglutaminase (TG2) reprograms the transcripti

182 e activity of the matrix crosslinking enzyme tissue transglutaminase (TG2) via S-nitrosylation in you

183 GPR56 inhibits melanoma growth and binds to tissue transglutaminase (TG2), a major crosslinking enzy

184 Tissue transglutaminase (TG2), an enzyme involved in cel

185 Tissue transglutaminase (TG2), an enzyme that catalyzes

186 two natural GPR56 ligands, collagen III and tissue transglutaminase (TG2), and one small-molecule ag

187 c breast cancer cells express high levels of tissue transglutaminase (TG2), but established no direct

188 te the performance of antibody tests against tissue transglutaminase (TG2), deamidated gliadin peptid

189 associated with the sulfhydryl-rich protein tissue transglutaminase (TG2), thereby endowing the memb

190 of circulating autoantibodies to the enzyme tissue transglutaminase (TG2).

191 umor cell lines expressed elevated levels of tissue transglutaminase (TG2).

192 We show that GPR56 binds specifically to tissue transglutaminase, TG2, a widespread component of

193 ptoms and levels of immunoglobulin A against tissue-transglutaminase (TGA-IgA) 10-fold or more the up

194 Tissue transglutaminase (TGase) catalyzes transfer of ga

195 Tissue transglutaminase (TGase) exhibits both a GTP bind

196 Retinoic acid (RA) is a potent activator of tissue transglutaminase (TGase) expression, and it was r

197 Tissue transglutaminase (TGase) has been implicated in n

198 Tissue transglutaminase (TGase) is a Ca(2+)-dependent en

199 Tissue transglutaminase (TGase) is a Ca2+-dependent enzy

200 Tissue transglutaminase (TGase) is a dual function enzym

201 Tissue transglutaminase (TGase) is involved in the regul

202 However, tissue transglutaminase (TGase) provides an interesting

203 disease (AD), we investigated the ability of tissue transglutaminase (TGase) to convert human recombi

204 a model system for investigating the role of tissue transglutaminase (TGase), a protein that has been

205 to the enzyme (transamidase) active site of tissue transglutaminase (TGase), eliminated RA protectio

206 lar aggregates that may be formed in part by tissue transglutaminase (Tgase).

207 Tissue transglutaminase (TGase-2), which binds GTP and c

208 RA induces expression of tissue-transglutaminase (TGase) and promotes migration a

209 We demonstrate amplification of tissue transglutaminase (TGC), keratinocyte transglutami

210 lipase Cdelta1 (PLCdelta1) is an effector in tissue transglutaminase (TGII)-mediated alpha1B-adrenore

211 Tissue transglutaminase (TGM2) belongs to a family of ca

212 In vitro tissue transglutaminase (Tgm2) cross-linking was monitor

213 By gene expression screening, tissue transglutaminase (TGM2) was identified as one of

214 ifferent extents on stiffer substrates; TAZ, tissue transglutaminase (TGM2), and soluble frizzled-rel

215 sensitive fluorometric well plate assay for tissue transglutaminase that is suitable for multiple ki

216 The peptide reacted with tissue transglutaminase, the major autoantigen in Celiac

217 RTX toxin cross-links actin independently of tissue transglutaminase, thus eliminating an indirect mo

218 e as predicted after GC-1g, and the mean IgA-tissue transglutaminase titers declined, contrary to the

219 Addition of exogenous tissue transglutaminase to cultured cells mimicking exte

220 silon-(gamma-glutamyl) lysine cross-link and tissue transglutaminase took place predominantly in the

221 Expression of tissue transglutaminase (transglutaminase II, tTG) was s

222 Tissue transglutaminase (transglutaminase type II) is an

223 undergoing short-term antigen challenge and tissue transglutaminase-treated, overlapping synthetic p

224 e dissimilar with known CD-specific antigens tissue transglutaminase (tTG) and deamidated gliadin, an

225 )((app))) of FXIIIa and the guinea pig liver tissue transglutaminase (tTG) and reactivities of Gln su

226 ce suggests that immunoglobulin A (IgA) anti-tissue transglutaminase (tTg) antibody levels >=10 times

227 l transglutaminase (TGe) and closely related tissue transglutaminase (tTG) are considered to be autoa

228 Human factor XIII (FXIII) and tissue transglutaminase (tTG) are homologous proteins.

229 Immunoglobulin A (IgA) antibodies to tissue transglutaminase (tTG) are the serologic test of

230 Tissue transglutaminase (tTG) assay was available, but o

231 d to proteolytic degradation of cell surface tissue transglutaminase (tTG) at the leading edge of mot

232 core protein dimer was determined to be the tissue transglutaminase (tTG) because, first, tTG could

233 Tissue transglutaminase (tTG) catalyzes a Ca(2+)-depende

234 Tissue transglutaminase (tTG) catalyzes a Ca2+-dependent

235 Tissue transglutaminase (tTG) exhibits a magnesium-depen

236 (TIMP) expression, stellate cell apoptosis, tissue transglutaminase (tTg) expression, and matrix cro

237 Tissue transglutaminase (tTG) functions as a GTPase and

238 se (CD) often relies on the presence of anti-tissue transglutaminase (tTG) IgA autoantibodies.

239 The possible involvement of tissue transglutaminase (tTG) in apoptosis during photor

240 r studies have suggested a possible role for tissue transglutaminase (tTG) in this process.

241 for this defect, we analyzed the activity of tissue transglutaminase (tTG) in TSP-2-null dermal fibro

242 Crosslinking of Abeta peptides by tissue transglutaminase (tTg) indicates that Gln15 of on

243 Our previous work showed that cell surface tissue transglutaminase (tTG) induces clustering of inte

244 Tissue transglutaminase (tTG) is a calcium-dependent enz

245 Tissue transglutaminase (tTG) is a multifunctional enzym

246 Tissue transglutaminase (tTG) is a multifunctional prote

247 Tissue transglutaminase (tTG) is a sulfhydryl rich prote

248 Tissue transglutaminase (tTG) is a unique member of the

249 Tissue transglutaminase (tTG) is an acyltransferase/GTP-

250 The recognition scaffold is the following: tissue transglutaminase (tTG) is immobilized as a captur

251 Here, we show that tissue transglutaminase (tTG) is one such protein.

252 Tissue transglutaminase (tTG) is present in the human ne

253 , duodenal biopsy, and serologic analysis of tissue transglutaminase (tTg) levels; endomysial antibod

254 Experimental renal scarring indicates that tissue transglutaminase (tTg) may be associated with the

255 d gliadin peptide above 10.0 U/mL and/or IgA-tissue transglutaminase (TTG) or IgG-TTG above 7.0 U/mL)

256 Tissue transglutaminase (tTG) serves as a potent and ubi

257 hological crosslinking of alpha-synuclein by tissue transglutaminase (tTG) using immunohistochemistry

258 ometry, glycated haemoglobin (HbA1c) and IgA tissue transglutaminase (tTg) were collected prior to, a

259 ndomysial antibodies (EMA) and antibodies to tissue transglutaminase (tTG) were developed to screen f

260 ne expression for transglutaminase (TGase 2; tissue transglutaminase (tTG)) in hippocampus and isocor

261 n cell lines stably overexpressing wild type tissue transglutaminase (tTG), a mutant inactive tTG, or

262 irected to different antigens, in particular tissue transglutaminase (tTG), gliadin (Glia), and endom

263 The protein-crosslinking enzyme, tissue transglutaminase (tTg), has recently been implica

264 ty to bind the protein cross-linking enzyme, tissue transglutaminase (tTG), in cancer cell migration.

265 Tissue transglutaminase (tTG), involved in PTM of gluten

266 e repeats are glutaminyl-donor substrates of tissue transglutaminase (tTG), it has been hypothesized

267 sforming growth factor-beta1 (TGF-beta1) and tissue transglutaminase (tTG), two proteins with documen

268 isease, such as increased antibody levels to tissue transglutaminase (tTG).

269 rface adhesion/signaling receptors including tissue transglutaminase (tTG).

270 l activity of the retinoid-response gene rat tissue transglutaminase (tTG).

271 celiac disease have serum antibodies against tissue transglutaminase (tTG).

272 transfer of the protein cross-linking enzyme tissue transglutaminase (tTG).

273 The IgA endomysium (EMA-IgA) and tissue transglutaminase (TTG-IgA) tests were both highly

274 first documented diagnosis or positive serum tissue transglutaminase (tTG-IgA).

275 Ca2+-dependent protein cross-linking enzyme tissue transglutaminase (tTGase) is involved in THG-indu

276 Previously, tissue transglutaminase (tTGase) was found to contribute

277 they are recognized and deamidated by human tissue transglutaminase (tTGase) with high selectivity.

278 Recently, tissue transglutaminase (tTGase)-catalyzed deamidation o

279 The positive predictive value of tissue transglutaminase type 2 (tTG) antibodies performe

280 nt increase in liver retinoid concentration, tissue transglutaminase type II (TGaseII) activity, tran

281 These studies revealed that tissue transglutaminase was activated in the nucleus, a

282 2 to more than 12000 participants) found IgA tissue transglutaminase was associated with high accurac

283 es, the nuclear localization and activity of tissue transglutaminase was examined.

284 tin, hepatocyte growth factor activator, and tissue transglutaminase was quantitated.

285 These oligomers and those prepared with tissue transglutaminase were used to establish a mechani

286 cellular matrix is stabilized by the enzyme, tissue transglutaminase, which we demonstrated to be ove

287 ification of the fibronectin-binding site on tissue transglutaminase will help to dissect the role of

288 QQDCTLSLQLTT106 inhibited the interaction of tissue transglutaminase with fibronectin and decreased t

289 Substitution of individual domains of tissue transglutaminase with those from homologous facto