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1  such as malocclusions and delayed or failed tooth eruption.
2 trosis such as high bone mass and failure of tooth eruption.
3 ntoblastic processes until the completion of tooth eruption.
4 HrP-PPR system during root morphogenesis and tooth eruption.
5 llicle and bone remodeling are essential for tooth eruption.
6 have been associated with root formation and tooth eruption.
7 tic mechanisms underlying root formation and tooth eruption.
8 n tooth tissue growth, but on the process of tooth eruption.
9 n opaque enamel that fails prematurely after tooth eruption.
10 re observed in the periostin-null mice after tooth eruption.
11 null mice dramatically deteriorate following tooth eruption.
12  rare genetic disorder exclusively affecting tooth eruption.
13 ates the alveolar bone resorption needed for tooth eruption.
14  on osteoclastogenesis, root resorption, and tooth eruption.
15 cause the enamel secreting cells are lost at tooth eruption.
16 , and in the junctional epithelium following tooth eruption.
17 esorption and restores the normal program of tooth eruption.
18 evalence and severity change over time after tooth eruption.
19 ng cranial chondrodystrophy and a failure of tooth eruption.
20  and children starting at the age of primary tooth eruption.
21 affecting enamel structure, size, shape, and tooth eruption.
22 are required for normal bone development and tooth eruption.
23  that the critical initial cellular event of tooth eruption, an influx of mononuclear cells into the
24 me at 3 critical time points: at the time of tooth eruption and 2 and 6 weeks after eruption.
25 port the discovery of an agent that inhibits tooth eruption and also show that tooth eruption require
26 s for the periodontal ligament (PDL) include tooth eruption and anchorage, force absorption, and prov
27 nsgenic mice cause osteopetrosis with normal tooth eruption and bone elongation and inhibit the devel
28           Also, PTHLH, a hormone involved in tooth eruption and invasive growth, was one of the most
29 networks in physiological conditions such as tooth eruption and movement and also for periodontal dis
30                     "Rescued" animals lacked tooth eruption and showed premature epiphyseal closure,
31 nes and axial skeleton but apparently normal tooth eruption and skull plate development, indicating a
32              The reproductive, hematopoietic tooth eruption and tissue macrophage defects of CSF-1-de
33 al progenitor cell populations that regulate tooth eruption and tooth root formation are beginning to
34 iodontium, play a role in physiologic (e.g., tooth eruption) and pathologic (e.g., periodontitis) eve
35 ce, including growth retardation, failure of tooth eruption, and abnormal male and female reproductiv
36 osis, with no osteoclasts, marrow spaces, or tooth eruption, and exhibited profound growth retardatio
37  strategy for detecting variants involved in tooth eruption, and potentially craniofacial growth and
38 h enamel surface, early loss of enamel after tooth eruption, and severe attrition.
39                         Root development and tooth eruption are very important topics in dentistry.
40 ary glands, placental calcium ion transport, tooth eruption, bone formation and bone remodeling, and
41           Root development accompanies rapid tooth eruption, but roots are required for the movement
42 ently corrected Csf1(op)/Csf1(op) defects of tooth eruption, eyelid opening, macrophage morphology, a
43    Experiments in vivo have established that tooth eruption fails in the absence of parathyroid hormo
44 tus, tooth root malformation, and failure of tooth eruption in molars, which essentially recapitulate
45 ony-stimulating factor-1 (CSF-1) accelerates tooth eruption in rats and is localized in the dental fo
46                                              Tooth eruption is a continuous biological process with d
47                                              Tooth eruption is a unique biological process by which h
48                                              Tooth eruption is defined as the movement of a tooth fro
49  studies indicate that the timing of primary tooth eruption is highly heritable, with estimates typic
50 s that the down-regulation of OPG needed for tooth eruption is mediated by CSF-1.
51                 A critical cellular event in tooth eruption is the formation of osteoclasts that are
52 severe enamel hypoplasia, delayed and failed tooth eruption, misshapen teeth, intrapulpal calcificati
53  inhibit the gene expression of the putative tooth eruption molecules, colony-stimulating factor-1 an
54 nd if its secretion is enhanced by potential tooth eruption molecules.
55 hat this bisphosphonate inhibits the time of tooth eruption of both rat molars and incisors.
56  bone, whether through the normal process of tooth eruption or by strains generated by orthodontic ap
57 teum of long bones, nor were they present in tooth eruption pathways.
58 t inhibits tooth eruption and also show that tooth eruption requires alveolar bone resorption.
59 cted and coincides better with estimates for tooth eruption times in Homo erectus.
60 t mammals(5), the relative sequence of adult tooth eruption was already established in Krusatodon and
61 ntroduction of new nutrient sources, but not tooth eruption, was associated with increasing complexit
62     To identify variants involved in primary tooth eruption, we performed a population-based genome-w
63 rent-nurse conversation about child's future tooth eruption, with advice given to visit a general den