ライフサイエンスコーパス: tooth loss

1 was feasible only for the objective measure 'tooth loss'.

2 d a significantly higher periodontal-related tooth loss.

3 changes and, ultimately, bone resorption and tooth loss.

4 tive tissue and alveolar bone and results in tooth loss.

5 iable significantly related to risk ratio of tooth loss.

6 t lead to the breakdown of alveolar bone and tooth loss.

7 ween patient compliance with regular SPT and tooth loss.

8 I, higher gingival inflammation, and greater tooth loss.

9 D was inversely associated with incidence of tooth loss.

10 hetic resolution for patients suffering from tooth loss.

11 ective factor for periodontitis, caries, and tooth loss.

12 ing on probing (BOP), plaque index (PI), and tooth loss.

13 t were used to determine treatment costs and tooth loss.

14 eolar bone loss around teeth, and subsequent tooth loss.

15 Vitamin D might be a protective factor for tooth loss.

16 ral hygiene, gingival bleeding, and bone and tooth loss.

17 the beneficial effect of protecting against tooth loss.

18 onal beneficial effects on periodontitis and tooth loss.

19 veolar bone damage and resorption, promoting tooth loss.

20 and microbial burden and is a major cause of tooth loss.

21 evel (CAL), alveolar crest height (ACH), and tooth loss.

22 ble plaque, supragingival calculus, and mean tooth loss.

23 eriodontal maintenance therapy in preventing tooth loss.

24 based on measures of ACH in combination with tooth loss.

25 ival inflammation, oral bone resorption, and tooth loss.

26 s associated with increased odds of complete tooth loss.

27 Periodontitis can ultimately result in tooth loss.

28 on, alveolar bone resorption, and ultimately tooth loss.

29 e that leads to local bone deterioration and tooth loss.

30 inflammation, bone erosion, severe pain, and tooth loss.

31 ad less recurrence of periodontitis and less tooth loss.

32 of the tooth-supporting tissues, leading to tooth loss.

33 fluenced the recurrence of periodontitis and tooth loss.

34 e of the tooth-supporting tissues leading to tooth loss.

35 bone supporting a tooth, and terminates with tooth loss.

36 xtent of loss of periodontal attachment, and tooth loss.

37 ls and periodontitis progression or incident tooth loss.

38 ty of causes, such as infection, trauma, and tooth loss.

39 tionship between socio-economic position and tooth loss.

40 at maintenance visits may result in greater tooth loss.

41 odontitis of the primary dentition and early tooth loss.

42 ntium, which, in severe cases, can result in tooth loss.

43 d was associated with a marginal increase in tooth loss.

44 T) versus increase, and no tooth loss versus tooth loss.

45 l epithelium was associated with cypriniform tooth loss.

46 dontitis patients had a beneficial impact on tooth loss.

47 y cervical resorption leading to significant tooth loss.

48 e not significantly associated with incident tooth loss.

49 lead to the periodontal pocket formation and tooth loss.

50 , outcomes of dental caries, and outcomes of tooth loss.

51 ESCC risk with exposure to unpiped water and tooth loss.

52 at leads to destruction of alveolar bone and tooth loss.

53 those who smoke, suffer from a high rate of tooth loss.

54 up data (n = 375) was evaluated for incident tooth loss.

55 ecrosis, arrested tooth-root development and tooth loss.

56 including both periodontal measurements and tooth loss.

57 abetes can increase the risk and severity of tooth loss.

58 ect size relative to microbiome shifts after tooth loss.

59 eteriorating socioeconomic circumstances and tooth loss.

60 ssues, leading to severe bone resorption and tooth loss.

61 an association between vitamin D status and tooth loss.

62 ing one in five individuals that can lead to tooth loss.

63 teeth/gum health, loose teeth and history of tooth loss.

64 eriodontitis progression and determinant for tooth loss.

65 orted for the interaction of combinations of tooth loss (0, 1 to 5, 6 to 31, or all) and cigarettes s

66 Diabetes Federation increased the hazards of tooth loss (1.39; 1.08 to 1.79), pocket depth >/=5 mm (1

67 cts had a significantly higher prevalence of tooth loss (90.2% versus 40.4% and 86.1% versus 43.4%, P

68 However, compared with no report of tooth loss, a report of losing teeth within the past 2 y

69 s and periodontal disease defined by ACH and tooth loss (adjusted OR = 0.96, 95% CI = 0.68 to 1.35).

70 or more days was not associated with reduced tooth loss [Adjusted rate ratio (RR) = 1.0; 95% Confiden

71 in the new classification were predictive of tooth loss after a long-term follow-up (>10 years) in pa

72 sociation of the state Gini coefficient with tooth loss after sequential adjustment for state- (media

73 The risk of tooth loss also increased 2.5 times for each millimeter

74 Probing depths (PDs), tooth loss, alveolar bone levels, and systemic health we

75 tween state income inequality and individual tooth loss among 386,629 adults in the United States who

76 It is the most common cause of tooth loss among adults in the United States, and recent

77 status and progression of periodontitis and tooth loss among individuals during PMT.

78 proved dental prognosis through reduction of tooth loss among molars and minimization of alveolar bon

79 f tetracyclines were associated with reduced tooth loss among persons receiving periodontal care, and

80 , and penicillin was associated with reduced tooth loss among persons with more severe disease.

81 c factors contributed to 14% of variation in tooth loss among women, and 39% among men.

82 ACH) measures from intraoral radiographs and tooth loss and 2) Centers for Disease Control and Preven

83 nce and periodontal maintenance intervals on tooth loss and alveolar bone loss, respectively.

84 th significantly more calculus formation and tooth loss and an increased extent and severity of perio

85 periodontal diseases are a leading cause of tooth loss and are linked to multiple systemic condition

86 s the multiplicative effect of self-reported tooth loss and cigarette smoking on COPD among United St

87 No significant association was found between tooth loss and cognitive impairment.

88 Traditional systems are based on tooth loss and may have limited use for patient manageme

89 e of the outcome, including risk factors for tooth loss and measures of cholesterol metabolism.

90 nian patients with PLS experienced premature tooth loss and palm plantar hyperkeratosis.

91 Studies suggest that tooth loss and periodontal disease might increase the ri

92 ease is a significant health burden, causing tooth loss and poor oral and overall systemic health.

93 flammatory bone loss, potentially leading to tooth loss and systemic complications.

94 tatistically significant correlation between tooth loss and the proportion of patients with low prote

95 cial for understanding how subjects adapt to tooth loss and their prosthetic replacement.

96 lay a role in adaptive mechanisms related to tooth loss and their replacement with dental implants.

97 deregulated immune response and resulting in tooth loss and various systemic conditions.

98 etermine whether metabolic syndrome predicts tooth loss and worsening of periodontal disease in a coh

99 baseline, 152 females reported no history of tooth loss, and 628 were categorized as reporting a hist

100 l caries (tooth decay), periodontal disease, tooth loss, and cancers of the lips and oral cavity.

101 who had complete data on cigarette smoking, tooth loss, and covariates.

102 riodontal status, periodontitis progression, tooth loss, and influence of predictable risk variables

103 y may play a role in periodontal disease and tooth loss, and insufficient vitamin D status is common

104 isms, resulting in dentognathic pathologies, tooth loss, and loss of masticatory function.

105 Dental caries, tooth loss, and periodontal attachment loss (AL) were re

106 the case-definition of periodontal disease, tooth loss, and prevalence and severity of the disease.

107 d by bacteria, resulting in bone resorption, tooth loss, and systemic inflammation.

108 duction in tooth loss risk, with the risk of tooth loss approaching that of never smokers after appro

109 se burden, as both periodontal pathology and tooth loss are associated with both functional impairmen

110 Periodontitis, alveolar bone loss, and tooth loss are associated with low BMD.

111 odontitis, progression of periodontitis, and tooth loss are common in older men, they were not associ

112 her oral hygiene, gingival inflammation, and tooth loss are correlated with stress biomarkers in pati

113 treatment strategies may influence long-term tooth loss are hard to find.

114 effects of smoking and smoking cessation on tooth loss are scarce.

115 Cigarette smoking and tooth loss are seldom considered concurrently as determi

116 tors, baseline clinical status, and incident tooth loss are significantly associated with attachment

117 nd oral diseases (periodontitis, caries, and tooth loss) are highly prevalent in Germany.

118 ses, the distribution of affected teeth, and tooth loss as a function of age.

119 tle data, often conflicting, that pertain to tooth loss as a function of patient compliance.

120 ith statins should also result in diminished tooth loss as a long-term response.

121 8 were categorized as reporting a history of tooth loss as a result of periodontal disease (n = 70) o

122 by sex and age, to estimate familial risk of tooth loss as well as estimates of heritability.

123 of the tooth supporting apparatus leading to tooth loss; as such, it is a major public health issue.

124 The data also showed reduced tooth loss associated with the 5-year reduction in LDL-c

125 We used risk-based approaches to test tooth loss association with 1 vs. 2 annual preventive vi

126 ing was associated with higher prevalence of tooth loss at baseline as well as higher incidence of to

127 An increased odds of the history of tooth loss attributable to caries was observed with incr

128 Tooth loss attributable to periodontitis occurred in 13%

129 ne in the prevalence and incidence of severe tooth loss between 1990 and 2010 at the global, regional

130 to as the burden of periodontal diseases and tooth loss (BPT).

131 Prevention reduces tooth loss, but little evidence supports biannual preven

132 (ORs) and 95% confidence intervals (CIs) for tooth loss by category of baseline 25(OH)D (nmol/L) conc

133 on and housing damage due to the disaster on tooth loss by fitting an instrumental variable probit mo

134 Tooth loss can be a consequence of the natural history o

135 Tooth loss categories were based on the number of missin

136 ween 25(OH)D and the history or incidence of tooth loss caused by periodontal disease.

137 rporated in the collection were examined for tooth loss, cavity occurrence, average and maximum lingu

138 cantly less progression of periodontitis and tooth loss compared to AG.

139 th a higher progression of periodontitis and tooth loss compared to NDC and GGC individuals.

140 a 20% (95% CI, 1.11, 1.30) increased risk of tooth loss compared with never- and former smokers of pi

141 thin the United States adult population, and tooth loss correlates to severity and risk.

142 The present study further emphasizes that tooth loss could be an easily obtained risk indicator fo

143 However, the exact reason for tooth loss could not be identified.

144 oporotic females, who are at greater risk of tooth loss, could minimize the potential effects of bone

145 reated caries increased, those due to severe tooth loss decreased.

146 g sociodemographic characteristics, smoking, tooth loss, dental caries, periodontal status, and OHRQo

147 Tooth loss, dental caries, worse periodontal status, and

148 use of antihypertensive medication; smoking; tooth loss; dental caries; periodontal status (bleeding

149 whose expression loss parallels cypriniform tooth loss, Dlx2b, retains the capacity for expression i

150 l destruction, and an increased frequency of tooth loss due to periodontitis.

151 Assessment of tooth loss during follow- up was assessed clinically by

152 and the baseline prevalence and incidence of tooth loss during follow-up, respectively.

153 s at baseline as well as higher incidence of tooth loss during follow-up.

154 ntitis and osteoporosis at baseline and with tooth loss during follow-up.

155 ase severity based on alveolar bone loss and tooth loss during follow-up.

156 general anesthesia including 4 deaths and 1 tooth loss during intubation.

157 and 1 case each for numbness, diplopia, and tooth loss during intubation.

158 trol in the progression of periodontitis and tooth loss during periodontal maintenance therapy (PMT)

159 del with the recurrence of periodontitis and tooth loss during periodontal maintenance therapy (PMT).

160 To assess the effect of compliance on tooth loss during SPT, pooled risk ratio of tooth loss (

161 For studies reporting tooth loss during the 'observational period' (excluding

162 nd sex, statins were associated with reduced tooth loss during the follow-up period (incidence risk r

163 number of natural teeth at baseline and any tooth loss during the previous 2 years was reported on t

164 decades of decline in prevalence of complete tooth loss (edentulism), the trend continues to be misin

165 e individuals affected by cancer separately, tooth loss/edentulism is associated with SNPs in AXIN2 (

166 oefficient was significantly associated with tooth loss even after adjustment for state- and individu

167 ss than erratic compliers, with the greatest tooth loss exhibited by complete compliers under the def

168 ication of periodontitis severity, risk, and tooth loss exists within the United States adult populat

169 ing depth, bleeding index, plaque index, and tooth loss for 505 patients in a long-term period of obs

170 extractions at initial therapy), the average tooth loss for AgP was 0.09 per patient-year.

171 The average tooth loss for all AgP cases was 0.09 (95% C.I. = 0.06-0

172 prevalence and incidence estimates of severe tooth loss for all countries, 20 age groups, and both se

173 ad an approximately 30% reduction in risk of tooth loss for complete compliance, with 2-year complian

174 At the 5-y follow-up visit, the average tooth loss for flossers was ~1 tooth compared to ~4 teet

175 robing depth (PD), attachment loss (AL), and tooth loss from 584 HIV-seropositive and 151 HIV-seroneg

176 Smoking and tooth loss from tooth decay or gum disease were associat

177 Periodontitis is a major cause of tooth loss globally.

178 ombinations of smoking status categories and tooth loss had a higher likelihood of COPD, with adjuste

179 Tooth loss (hazard ratio (HR) = 1.2, 95% confidence inte

180 BOP in >30% of sites (OR = 4.1); and 2) for tooth loss, HbA1c >/=6.5% (OR = 3.1), smoking (OR = 4.1)

181 PPC stage V (Mild Tooth Loss/High Gingival Inflammation) was significant f

182 ables (HR 1.62; 95% CI 1.03-2.56), excessive tooth loss (HR 1.66; 95% CI 1.04-2.64), drinking unpiped

183 up of gram-negative microbes, which leads to tooth loss if untreated.

184 ted caries, severe periodontitis, and severe tooth loss in 2010 and compares those figures with new e

185 ongitudinal data on tobacco use and incident tooth loss in 43,112 male health professionals, between

186 for medical or dental reasons to subsequent tooth loss in a cohort of 12,631 persons with destructiv

187 eroids are associated with periodontitis and tooth loss in a cohort of 1210 older dentate men followe

188 n and housing damage due to the disaster and tooth loss in a cohort of community-dwelling residents (

189 tions and prevalence and 5-year incidence of tooth loss in a cohort of postmenopausal females was exa

190 essation and the prevalence and incidence of tooth loss in a large cohort study in Germany.

191 ressive periodontitis resulting in premature tooth loss in adolescents.

192 inflammation accounting for the majority of tooth loss in adult population worldwide.

193 Periodontitis is the major cause of tooth loss in adults and is linked to systemic illnesses

194 Periodontitis is a leading cause of tooth loss in adults and occurs in about 50% of the US p

195 sue and bone support and is a major cause of tooth loss in adults.

196 tory bone disorder and the greatest cause of tooth loss in adults.

197 ically investigating disease progression and tooth loss in AgP.

198 d that family background importantly affects tooth loss in both the middle-aged and the older populat

199 or an association of statin use with reduced tooth loss in chronic periodontitis patients.

200 ssess risk for periodontitis progression and tooth loss in dental patients.

201 eolar vestiges and indicate that ontogenetic tooth loss in Limusaurus is a gradual, complex process.

202 was associated with >3 times higher risk of tooth loss in men (odds ratio, 3.6; 95% confidence inter

203 riodontal disease as a predictor of incident tooth loss in postmenopausal women has not been determin

204 trong and independent predictor for incident tooth loss in postmenopausal women.

205 The risk of tooth loss in the RC group was significantly lower than

206 tween state income inequality and individual tooth loss in the United States.

207 l, 3.0, 4.4) and more than twice the risk of tooth loss in women (odds ratio, 2.5; 95% confidence int

208 has both dose- and time-dependent effects on tooth loss incidence.

209 aries among treated patients and factors for tooth loss include CP severity and risk.

210 Tooth loss independently predicts low energy and protein

211 l health, and COPD, particularly the role of tooth loss, infection, and subsequent inflammation, is e

212 Tooth loss is a common health concern in older adults.

213 med to assess whether periodontal disease or tooth loss is associated with cancer risk.

214 Tooth loss is associated with increased cardiovascular d

215 We aimed to assess whether tooth loss is associated with specific CVD mortality end

216 Tooth loss is common, and exploring the neuroplastic cap

217 that a substantial part of the variation in tooth loss is explained by genetic as well as environmen

218 Tooth loss is generally considered the final outcome of

219 Tooth loss is more precisely and accurately predicted by

220 Tooth loss is the ultimate negative consequence of perio

221 y induced inflammatory disease that leads to tooth loss, is believed to result from infection by a se

222 and periodontal disease is a major cause of tooth loss, it was necessary to account for edentulousne

223 Alterations following tooth loss, itself a major risk factor for oral cancer,

224 c inflammation, increased probing depth, and tooth loss likely attributable to the direct effects of

225 reated dental caries, caries experience, and tooth loss (<20 teeth) on NAFLD while controlling for cl

226 rs hypothesize that among patients with CKD, tooth loss may affect nutritional status, using the Nati

227 data heterogeneity and affecting the risk of tooth loss may have been present.

228 Incident tooth loss occurred in 22% of the cohort.

229 ience (odds ratio, 1.40; 95% CI, 1.19-1.65), tooth loss (odds ratio, 1.66; 95% CI, 1.48-1.86), and tr

230 (ALI) and 2) quantify the effect of incident tooth loss on conclusions made about ALI.

231 nce assessing the magnitude of the effect of tooth loss on malnutrition in CKD populations.

232 n analysis confirmed the significant role of tooth loss on serum albumin and protein and energy intak

233 Hazards ratios (95% confidence intervals) of tooth loss or a periodontitis event were estimated from

234 Tooth loss or periodontal disease is associated with sys

235 We estimate that tooth loss, or at least the loss of enamel caps that pro

236 as a positive association of severe CAL with tooth loss (P = 0.000), BOP (P = 0.004), and heavy smoke

237 h periodontal disease have increased risk of tooth loss, particularly in cases with associated loss o

238 alues by diagnosis were 0.05, 0.14, and 0.12 tooth loss per patient-year, respectively, for LAgP, GAg

239 NAFLD was associated with tooth loss, periodontitis, and, for some NAFLD measures,

240 Periodontitis was evaluated by measuring tooth loss, plaque and bleeding indexes, probing depths,

241 .1% and 1.7% increases in the probability of tooth loss (probit coefficients were 0.469 (95% confiden

242 iations between serum 25OHD and incidence of tooth loss, progression of CAL >/= 3 mm, and progression

243 5-hydroxy vitamin D (25OHD) and incidence of tooth loss, progression of clinical attachment loss (CAL

244 464, P <0.01), BOP (r = 0.401, P <0.05), and tooth loss (r = 0.245, P <0.05).

245 c factors between cardiovascular disease and tooth loss (r(G) = 0.18) and periodontal disease (r(G) =

246 loss (RDTL) and weighted mean difference of tooth loss rate (WDTLR) were used as secondary outcomes.

247 ents in the RC group had significantly lower tooth loss rate during SPT than did patients in the EC g

248 ssociated with a non-significant 37% reduced tooth loss rate in the year subsequent to the 3-year per

249 n use during 3 years was not associated with tooth loss rate in the year subsequent to the 3-year per

250 tal exam was associated with a 48% decreased tooth loss rate in year 4 and subsequent years (RR = 0.5

251 risk (P = 0.00129) scores predicted the mean tooth loss rate.

252 that disease and risk scores predicted mean tooth loss rate.

253 Pooled risk difference of tooth loss (RDTL) and weighted mean difference of tooth

254 HR, 3.05; 95% CI, 2.38, 3.90) higher risk of tooth loss, respectively, compared with never-smokers.

255 ber of missing teeth and 5-year incidence of tooth loss, respectively.

256 rend = <0.05 for the history (prevalence) of tooth loss resulting from periodontal disease or caries

257 o 1.85), P-trend = 0.11 for the incidence of tooth loss resulting from periodontal disease or caries.

258 LDL-c, statins were associated with reduced tooth loss, resulting in IRR = 0.89 (95% CI = 0.44 to 1.

259 consistently associated with a reduction in tooth loss risk, with the risk of tooth loss approaching

260 was associated with a 13% decreased risk of tooth loss (risk ratio: 0.87; 95% confidence interval: 0

261 orst AL (RR, 1.01; 95% CI, 0.94 to 1.07), or tooth loss (RR, 1.02; 95% CI, 1.0 to 1.05).

262 tooth loss during SPT, pooled risk ratio of tooth loss (RRTL) was used as the primary outcome.

263 le quality literature on the epidemiology of tooth loss shows a significant decline in the prevalence

264 Tooth loss status significantly modifies the association

265 lower rates of periodontitis progression and tooth loss than did the AG.

266 showed less recurrence of periodontitis and tooth loss than ECs (P <0.05).

267 mplete compliers were more likely to exhibit tooth loss than erratic compliers, with the greatest too

268 list practice in Norway had a higher rate of tooth loss than patients who were compliant.

269 number of children) reportedly is related to tooth loss, the relationship between parity and dental c

270 nificant decrease in the prevalence of total tooth loss throughout the world from 1990 to 2010.

271 nificant decrease in the prevalence of total tooth loss throughout the world from 1990 to 2010.

272 ought to contribute to oral disease, such as tooth loss, tissue changes in the gums and throat, and p

273 77.4%, 22.0%, and 97.4%, respectively, when tooth loss to periodontitis (prevalence of 7%) was the c

274 as >=2 teeth with >=1 mm ACH loss or >=1 new tooth loss to periodontitis.

275 : 1) mean ACH >/=3 mm, two sites >/=5 mm, or tooth loss to periodontitis; 2) >/=2 sites with CAL >/=6

276 nto the aforementioned categories related to tooth loss (total n = 472).

277 odontal exam were evaluated as predictors of tooth loss using negative binomial regression models wit

278 However, tooth loss varies among treated patients and factors for

279 filled teeth (DMFT) versus increase, and no tooth loss versus tooth loss.

280 Vitamin D is hypothesized to reduce risk for tooth loss via its influence on bone health, inflammatio

281 Five-year incident tooth loss was also evaluated.

282 was not associated with risk of POAG, recent tooth loss was associated with an increased risk of POAG

283 Tooth loss was common, but actual number of teeth lost,

284 In contrast, tooth loss was more strongly associated with coronary he

285 ciation between preventive dental visits and tooth loss was not significantly different whether the f

286 A 5-year population-based follow-up study of tooth loss was performed comparing participants treated

287 ciation between smoking and the incidence of tooth loss was stronger in men than women and stronger i

288 fully adjusted model, PPC stage VII (Severe Tooth Loss) was moderately significantly related to inci

289 To investigate the mechanism of cypriniform tooth loss, we compared the oral expression of seven gen

290 Persons with incident tooth loss were also at increased risk for ALI, and teet

291 ted, and the recurrence of periodontitis and tooth loss were analyzed using univariate and multivaria

292 table CAD, the presence of periodontitis and tooth loss were associated with a poor dietary intake of

293 The associations with recent tooth loss were not significantly different for the POAG

294 Progression of periodontitis and tooth loss were significantly higher among PGC compared

295 they generally showed that systems based on tooth loss were unpredictable over the long term.

296 most involved teeth (periodontal pathology, tooth loss) were weighted more heavily.

297 to provide evidence that antibiotics reduce tooth loss when used in the management of destructive pe

298 Grade A) at baseline and periodontal related tooth loss, whereas no differences were detected for the

299 periodontal disease characterized by severe tooth loss, while none of the categories of the CDC/AAP

300 red community composition and function after tooth loss, with smaller alterations in current tobacco