ライフサイエンスコーパス: trabecular bone

1 ogenically-committed BMSC subset in human OA trabecular bone.

2 and no statistically significant changes in trabecular bone.

3 imals demonstrated a significant increase in trabecular bone.

4 ells and often, but not always, located near trabecular bone.

5 genetic knockdown of Nox4 mitigated loss of trabecular bone.

6 OA in the superior subchondral cortical and trabecular bone.

7 ereas in A. robustus it is a column of dense trabecular bone.

8 B number, serum human osteocalcin level, and trabecular bone.

9 completely blocked the tumor-induced loss of trabecular bone.

10 es and hindered accrual of peak cortical and trabecular bone.

11 expansion and were present in control non-OA trabecular bone.

12 astic bone consisting of mature cortical and trabecular bone.

13 l size and perfection in remnant metaphyseal trabecular bone.

14 gions are present in the disordered phase of trabecular bone.

15 d wide long bones, reduced bone collars, and trabecular bones.

16 exhibited severe osteopenia in cortical and trabecular bones.

17 ulation occurred in the epiphyseal plates of trabecular bones.

18 omography measurements were taken at the 4% (trabecular bone), 20% (cortical bone), and 66% (for meas

19 d the opposite bone phenotype, with 14% less trabecular bone, 22% fewer osteoblasts, and 10% thinner

20 KO mice compared WT controls, with 14% more trabecular bone, 35% more osteoblasts, 73% fewer osteocl

21 sity (+62%), accelerated age-related loss of trabecular bone (-61%), and markedly reduced B-cell numb

22 Tibial trabecular bone among participants completing 70% or mor

23 becular bone mass and attenuated PTH-induced trabecular bone anabolism, supporting the positive funct

24 d with marrow from KO kindred showed loss of trabecular bone analogous to KO mice, consistent with in

25 However, loss of trabecular bone and bone strength were most severe at th

26 These disorders result in excess growth of trabecular bone and collagen fibers that replace hematop

27 s important in bone remodeling, particularly trabecular bone and endogenous Epor expression in osteob

28 d with control knees, accompanied by loss of trabecular bone and erosion of subchondral bone surface.

29 at any anatomical site or alter cortical and trabecular bone and geometry.

30 the antibody does increase both cortical and trabecular bone and improves bone mechanical properties

31 and signaling were increased in Gja1(Jrt)/+ trabecular bone and osteogenic stromal cell cultures, wh

32 Increased trabecular bone and sinusoidal space and decreased arter

33 cular surface and is required for sustaining trabecular bone and skeletal growth.

34 pe characterized by osteopenia of epiphyseal trabecular bone and subchondral cortical plate.

35 The material behavior of the trabecular bone and teeth was always treated as isotropi

36 th Pyle's disease, have increased amounts of trabecular bone and unusually thin cortical bone, as a r

37 de that up-regulation of BMP2/4 signaling in trabecular bone and/or stromal cells increases osteoblas

38 s showed a decreased density of cortical and trabecular bone, and there was biochemical evidence for

39 Decreases in cortical bone, trabecular bone, and whole-bone failure strength were 7.

40 cro-finite-element models for cortical bone, trabecular bone, and whole-bone section were generated f

41 Decreases in cortical bone, trabecular bone, and whole-bone stiffness were 3.7% (P =

42 ensity, lower femoral bone mass, and altered trabecular bone architecture were observed in F508del Cf

43 d risedronate prevented the deterioration of trabecular bone architecture, reduced the degree of mine

44 ficiency on ligature-induced bone loss (BL), trabecular bone area (TBA), and postextraction bone heal

45 +) HSCs were more frequently detected in the trabecular bone area compared with compact bone area, an

46 SBP (subchondral bone plate) and B.Ar/T.Ar (trabecular bone area to total tissue area).

47 Trabecular bone, as compared with cortical bone, appeare

48 h DEX and Scl-ABI, a significant increase in trabecular bone at the femoral metaphysis (bone volume/t

49 nuation values (in Hounsfield units [HU]) of trabecular bone between the T12 and L5 vertebral levels,

50 In this case-control study, trabecular bone biopsies from iliac crest were collected

51 TG mice had progressively increased trabecular bone but decreased HSC function.

52 nges in the proximal tibia) was abolished in trabecular bone but not in the cortical component.

53 lds were generated from fully decellularized trabecular bone by using digitized clinical images, seed

54 Cellular isolates from trabecular bone cavities contained approximately 65-fold

55 hat CD45(low)CD271+ MSCs are abundant in the trabecular bone cavity and indistinguishable from aspira

56 The present study was aimed to evaluate the trabecular bone changes between healthy individuals and

57 type, with different effects on cortical and trabecular bone compartments.

58 s well as reduction of mineral bone density, trabecular bone content, and subcutaneous fat.

59 Assessment of microdamage within the trabecular bone core was performed using synchrotron X-r

60 es of aluminum and polycarbonate to simulate trabecular bone, cortical bone, and cartilage.

61 ne in humans may significantly underestimate trabecular bone damage sustained by ART.

62 ls, whereas the relationship between PTH and trabecular bone decreases was bimodal.

63 Besides increasing osteoblast number in the trabecular bone, deletion of Bmpr1a by Dmp1-Cre also not

64 We measured trabecular bone densities, cortical bone densities, VAT

65 gher trabecular connectivity density, higher trabecular bone density and thicker structures, somethin

66 ed with progressive declines in cortical and trabecular bone density at the peripheral skeleton.

67 The low trabecular bone density found in hemophilia is attribute

68 ing for changes in serum biochemical values, trabecular bone density using micro-computed tomography,

69 A have decreased skeletal size, muscle mass, trabecular bone density, cortical bone geometry, and str

70 All JRA patients had decreased tibia trabecular bone density, cortical bone size and strength

71 proliferation in the growth plate and lower trabecular bone density.

72 nctions to couple growth plate maturation to trabecular bone development in growing mice.

73 cy affected development of both cortical and trabecular bone differently, effects apparently dependen

74 The newly formed trabecular bone displayed similar biomechanical properti

75 intravenous injection of LLP2A-Ale increased trabecular bone formation and bone mass in both xenotran

76 conclusion, ERalpha in osteocytes regulates trabecular bone formation and thereby trabecular bone vo

77 parathyroidism, and simultaneously increased trabecular bone formation and trabecular connectivity, a

78 ic histomorphometry showed no differences in trabecular bone formation between WT and Col6alpha2-KO m

79 Trabecular bone formation is dramatically diminished in

80 and showed a dose dependent increase in the trabecular bone formation rate in ovariectomized rats fo

81 (Rs1) to address how the massive increase in trabecular bone formation resulting from increased G(s)

82 indicating that Bmpr1a signaling suppresses trabecular bone formation through effectors beyond Smad4

83 d locally over murine calvaria, and enhanced trabecular bone formation when administered systemically

84 increased osteoclastic activity and reduced trabecular bone formation.

85 simultaneously results in massively elevated trabecular bone formation.

86 Measurements included cortical and trabecular bone geometry, density, and strength at the d

87 deficiency of sFRP4, that cortical-bone and trabecular-bone homeostasis were governed by different m

88 ministration (1200 U/kg) for 10 days reduced trabecular bone in control mice but not in Tg mice.

89 e in male mice but it is dispensable for the trabecular bone in female mice and the cortical bone in

90 of muscle mass and strength, and the loss of trabecular bone in femurs and vertebrae following Folfir

91 Newly formed immature delicate trabecular bone in fibrovascular marrow filled the space

92 strogen response in NOER mice), cortical and trabecular bone in long bones, as well as uterus and thy

93 that ERalpha in osteocytes is important for trabecular bone in male mice and for cortical bone in bo

94 trogen in females, but it is dispensable for trabecular bone in male mice and for cortical bone in bo

95 results demonstrated an increased amount of trabecular bone in MULTI calluses at 21 days post-injury

96 the age-related changes in the cortical and trabecular bone in old age, and assessed whether we can

97 n response was highly tissue-dependent, with trabecular bone in the axial skeleton being strongly dep

98 with hypercholesterolemia lose cortical and trabecular bone in the femurs and vertebrae (bone minera

99 icroarchitecture deteriorated, mainly in the trabecular bone in the first 6 months and cortical bone

100 f the Hh effector Smoothened (Smo) increased trabecular bone in vivo and inhibited osteoclastogenesis

101 in vitro, suggesting that the restoration of trabecular bone in vivo was due to decreased bone resorp

102 s) that suppress bone remodeling will change trabecular bone in ways such that the size of the failur

103 ts that suppress bone remodeling will change trabecular bone in ways such that the size of the failur

104 resolution scans and histopathology revealed trabecular bones in two cases, hyaline cartilage in anot

105 med quantitative differences in cortical and trabecular bone, including decreased cortical thickness

106 of osteoclastogenesis, completely inhibited trabecular bone loss (-2.2+/-11.9%, P<0.01).

107 Additionally, LLP2A-Ale prevented trabecular bone loss after peak bone acquisition was ach

108 he field of osteoimmunology help explain the trabecular bone loss and generalized osteoporosis linked

109 -out mice, which are resistant to Pb-induced trabecular bone loss and maintain their mechanical bone

110 e protected against PTH-induced cortical and trabecular bone loss as well as from increases in serum

111 benefit from zoledronate); HR-pQCT detected trabecular bone loss at the peripheral skeleton, which z

112 our data imply that Col6a2 deficiency causes trabecular bone loss by enhancing osteoclast differentia

113 m supplementation on peripheral cortical and trabecular bone loss during pregnancy and bone gain post

114 CD8+ T cells may further contribute to trabecular bone loss in some patients with advanced AIDS

115 last activity and an overall TMJ subchondral trabecular bone loss in the UAC-treated rats.

116 Osteoporosis is characterised by trabecular bone loss resulting from increased osteoclast

117 not only muscle aging-like deficit but also trabecular bone loss, a feature of osteoporosis.

118 ntribute to post-transplant central skeleton trabecular bone loss, and zoledronate does not induce AB

119 uction, stimulate bone resorption, and cause trabecular bone loss, demonstrating that the gut microbi

120 nstrate that Sh3bp2 "cherubism" mice exhibit trabecular bone loss, TNF-alpha-dependent systemic infla

121 L-6 expression, hyperosteoclastogenesis, and trabecular bone loss, uncovering a pathological mechanis

122 caused by sex steroid deficiency, leading to trabecular bone loss.

123 lar joints (TMJs), displaying as subchondral trabecular bone loss.

124 ht be useful for treatment of postmenopausal trabecular bone loss.

125 ncreased osteoclast numbers, and significant trabecular bone loss.

126 formation, bone resorption, and cortical and trabecular bone loss.

127 tion of ART, causes significant cortical and trabecular bone loss.

128 is central in sex steroid deficiency-induced trabecular bone loss.

129 c BMD and bone microstructure indicated that trabecular bone mainly contributed to the positive assoc

130 cumulation of a hyaluronan matrix within the trabecular bone marrow, and adipocytes and macrophages e

131 n the endosteal surface in the calvarial and trabecular bone marrow.

132 eficient line, which included a reduction in trabecular bone mass and a functional deficit in bone st

133 is (MAFIA) mouse model] reduced cortical and trabecular bone mass and attenuated PTH-induced trabecul

134 entiated osteoblasts substantially increases trabecular bone mass and bone mineral density without af

135 an opposite effect was found with increased trabecular bone mass and increased PTH-induced anabolism

136 It acted as an ERalpha agonist on trabecular bone mass and uterine weight, whereas no effe

137 lamin A/C-KO mice diminishes the deficits in trabecular bone mass but not muscle.

138 ow that the F508del mutation in CFTR impacts trabecular bone mass by reducing bone formation.

139 ssion were increased in bones, and increased trabecular bone mass from pre-osteoblast specific Ezh2 d

140 b skeletogenic mesenchyme markedly increased trabecular bone mass in adolescent mice.

141 Dmp1-Cre, we observed a dramatic increase in trabecular bone mass in postnatal mice, which was due to

142 cted Hdac3 deficiency decreased cortical and trabecular bone mass parameters, suggesting that Hdac3 r

143 t in OC differentiation culminated in a high trabecular bone mass pathology.

144 omy (ovx) reduced the total body BMD and the trabecular bone mass to the same degree in Obl-Wnt16 mic

145 er, suppressed marrow adipogenesis, restored trabecular bone mass, and reduced bone resorption.

146 GC treatment reduced trabecular bone mass, microarchitecture, and the degree

147 y caused mainly by a substantial increase in trabecular bone mass, resulting in improved bone strengt

148 ssion of WNT16 surprisingly increases mainly trabecular bone mass.

149 whole body bone mineral density and reduced trabecular bone mass.

150 hes the osteoclastogenic deficit and reduces trabecular bone mass.

151 n wild type animals causes a 50% decrease in trabecular bone mass.

152 lls specifically in postnatal mice increased trabecular bone mass.

153 iously shown that MGUS patients have altered trabecular bone microarchitecture compared with controls

154 nt LT showed severe deficits in cortical and trabecular bone microarchitecture.

155 odifications of the aBMD and of cortical and trabecular bone microstructures.

156 d reduced bone volume and area, cortical and trabecular bone mineral content, and density.

157 A significant decrease in global trabecular bone mineral density (38.1%) and cortical thi

158 Trabecular bone mineral density (BMD) was determined in

159 ne mineral content and density, cortical and trabecular bone mineral density (BMD), BMC, and bone are

160 independent associations between volumetric trabecular bone mineral density (vBMD) of the lumbar spi

161 The local trabecular bone mineral density decreased in both high s

162 Total and trabecular bone mineral density were significantly lower

163 diabetic rat model, there is a large loss of trabecular bone mineral density without apparent proport

164 rs by intramembranous ossification forming a trabecular bone network that replaces the amputated cort

165 ese included fracture sites with predominant trabecular bone, not previously reported as being associ

166 odronate was less than the large increase in trabecular bone observed in a nonwounded long bone.

167 re decreased in the proximal tibia and spine trabecular bone of dKO-Hom mice compared to wild-type (W

168 antitative CT and microtomography to measure trabecular bone of limb epiphyses (long bone articular e

169 or phantomless in vivo BMD assessment of the trabecular bone of lumbar vertebrae and enables freely r

170 postprocessing dual-energy CT software, the trabecular bone of lumbar vertebrae L1-L4 were analyzed

171 .Dn), and by increased separation (Tb.Sp) in trabecular bone of the femur and vertebra.

172 mid-L5 level, the mean CT attenuation of the trabecular bone of the L5 vertebral body (L5HU) was meas

173 st activity in the tissue of TMJ subchondral trabecular bone of these UAC-treated rats was also enhan

174 anced osteoclast activity in TMJ subchondral trabecular bone of UAC-treated rats.

175 in the growth plate concomitant with radial trabecular bone orientation.

176 ese mutant mice exhibited continuous loss of trabecular bone over time, which was accompanied by redu

177 habitual tool manufacture, have a human-like trabecular bone pattern in the metacarpals consistent wi

178 SRK-015P treatment improves the cortical and trabecular bone phenotypes in these mice.

179 Pb and HFD each reduced trabecular bone quality and together had a further detri

180 distal femur cortical bone region but not at trabecular bone region at the 1.4 and 2.8 mg/kg/d GC dos

181 The trabecular bone region was divided into 37 volumes of in

182 oth PPR*Tg and control mice, suggesting that trabecular bone resorption was comparably suppressed by

183 pha(-/-) female mice displayed an attenuated trabecular bone response to supraphysiological E2 treatm

184 Further, muCT analysis of trabecular bone revealed that, compared with the vehicle

185 pha antagonist all protect cortical, but not trabecular bone, revealing complex effects of T-cell rec

186 micro-computed tomography of a human femoral trabecular bone sample, allowing full 3D reconstruction

187 neck BMD, lumbar spine BMD, and lumbar spine trabecular bone score (TBS) as secondary outcomes, all a

188 nd periodontitis remains unresolved; and the trabecular bone score (TBS) is a new index for assessing

189 Trabecular bone score and in vivo microindentation are n

190 Trabecular bone score was borderline lower (1.21 +/- 0.1

191 Trabecular bone score was measured by specific software

192 ed by the development of methods such as the trabecular bone score, which helps assess bone microarch

193 udy the association between KTR and BMD/BMSi/trabecular bone score.

194 total area ratios, thicker cortical bone and trabecular bone, significantly higher bone mineral densi

195 ERalpha in osteoclasts is crucial for the trabecular bone-sparing effect of estrogen in females, b

196 We propose that the physiological trabecular bone-sparing effect of estrogen is mediated v

197 In a rodent sepsis model, trabecular bone strength is functionally reduced within

198 ion of bortezomib (Bzb) reversed the loss of trabecular bone structure and strength in mice at 4 wk a

199 le of extant primates to assess variation in trabecular bone structure in the human hip joint.

200 at more highly mobile human populations have trabecular bone structure similar to what would be expec

201 Proximal femur trabecular bone structure was quantified from microCT da

202 Skinner and colleagues, based on metacarpal trabecular bone structure, argue that Australopithecus a

203 alyses were performed to assess cortical and trabecular bone structure, density, and rigidity.

204 ystemic bone loss in CIA mice by maintaining trabecular bone structure.

205 ut it failed in normalizing growth plate and trabecular bone structures.

206 essfully used in practice, especially in the trabecular bone studies because of high contrast between

207 logical analysis showed reduced cortical and trabecular bone, suggesting cell-autonomous functions of

208 ity in a subset of cells associated with the trabecular bone surface of long bones.

209 companied by increased osteoclast number per trabecular bone surface.

210 TRAP-positive osteoclast-covered trabecular bone surfaces also increased in microgravity

211 of TRAP(+) osteoclasts on distal metaphyseal trabecular bone surfaces were significantly decreased.

212 red a distinct cell type associated with the trabecular bone that appears to possess osteogenic poten

213 utant PTH1R exhibited a dramatic increase in trabecular bone that was dependent upon expression of Gs

214 ency of Crebbp included a marked decrease in trabecular bone that was predominantly caused by increas

215 In trabecular bone, the porosity of the mineralized matrix

216 her, MAGP1Delta mice have significantly less trabecular bone, the trabecular microarchitecture is mor

217 ronate treatment increased extraction socket trabecular bone thickness at 14 d, which correlated with

218 density and content, increased cortical and trabecular bone thickness, and greater net bone formatio

219 Femoral head trabecular bone tissue digests were sorted into CD45-CD2

220 Increased subchondral trabecular bone turnover due to imbalanced bone-resorbin

221 Analyses of mass-corrected trabecular bone variables reveal that the forager popula

222 -) mice displayed a substantial reduction in trabecular bone volume (-20%, P < 0.01) compared with co

223 Femoral bone mineral density (12 vs. 27%), trabecular bone volume (32 vs. 48%), and cortical thickn

224 d 22.2% and female Tg mice had 29.6% reduced trabecular bone volume (BV) compared to controls.

225 of the femur are associated with changes in trabecular bone volume (BV/TV) with altered estrogen sta

226 matically recognized compact bone volume and trabecular bone volume (IBV) in CT slices.

227 <.05) and resulted in a 2.5-fold increase in trabecular bone volume (p<.001).

228 hic analyses demonstrated markedly increased trabecular bone volume and bone mineral density in femor

229 th CLP for 2 weeks had significantly reduced trabecular bone volume and cortical bone thickness, asso

230 mice also have small skeletons with reduced trabecular bone volume and cortical thickness and that c

231 in human milk to this diet increased femoral trabecular bone volume and cortical thickness, reduced o

232 Zol treatment increased trabecular bone volume and decreased osteoclast paramete

233 ice, M-PTH(1-34) induced larger increases in trabecular bone volume and greater increases in cortical

234 ion activities, contributing to the enhanced trabecular bone volume and mineral density in these TG m

235 ultinucleated osteoclast-like cells and more trabecular bone volume and number in 26-wk-old male IL-2

236 ll (knockout, KO) mice, we observed that the trabecular bone volume and number of trabeculae were sig

237 Their prevalence correlated with decreased trabecular bone volume and osteoid and osteoblast surfac

238 S3 has been ablated in osteocytes, have high trabecular bone volume and poorly defined metaphyseal co

239 ing female mice, IgGs significantly improved trabecular bone volume and structure and increased both

240 e mass, with SRC-2 KO mice having 80% higher trabecular bone volume as compared with wild type mice.

241 g ACVR2A had significantly increased femoral trabecular bone volume at 6 weeks of age.

242 female mice had no significant reduction in trabecular bone volume but ovariectomized Dmp1-ERalpha(-

243 tomography revealed robust deterioration of trabecular bone volume by both subsets, while CD4+ T cel

244 in signaling in vivo and completely restored trabecular bone volume by increasing bone formation and

245 ale and female mice exhibited an increase in trabecular bone volume due to an increase in osteoblasts

246 -);Rosa(Notch) mice exhibited an increase in trabecular bone volume due to decreased bone resorption

247 grem1 null male mice displayed increased trabecular bone volume due to enhanced osteoblastic acti

248 T of the distal radius to determine apparent trabecular bone volume fraction (BV/TV), apparent trabec

249 me CT of distal radius to determine apparent trabecular bone volume fraction (BV/TV), apparent trabec

250 egative effects on bone, as shown by reduced trabecular bone volume fraction (BV/TV), thickness (Tb.T

251 Structural parameters (trabecular bone volume fraction [BV/TV, bone volume to t

252 the marrow, which correlated with increased trabecular bone volume fraction after 7 and 14 d.

253 rly Rhbdf2, whose close association with the trabecular bone volume fraction and number was strongly

254 treated only with loading showed the highest trabecular bone volume fraction at week 22.

255 lted in virtually identical losses in tibial trabecular bone volume fraction, BV/TV (24-28% reduction

256 All three agents similarly increased trabecular bone volume in both PPR*Tg and control mice,

257 ss phenotype with an approximate doubling of trabecular bone volume in both the tibia and femur.

258 ulates trabecular bone formation and thereby trabecular bone volume in male mice but it is dispensabl

259 The Lef1DeltaN transgenic mice had higher trabecular bone volume in the proximal tibias and L5 ver

260 ondral bone thickness and increased relative trabecular bone volume in the tibial epiphysis.

261 Trabecular bone volume increased in 3-month-old Osx-Cre(

262 olic effect of intermittent PTH treatment on trabecular bone volume is blunted by deletion of Gsalpha

263 In contrast, trabecular bone volume is not altered in these mice.

264 Trabecular bone volume of the distal femoral metaphysis

265 0 mug/kg/day) for 4 weeks failed to increase trabecular bone volume or cortical thickness in male and

266 showed a significantly decreased bone volume/trabecular bone volume ratio, decreased trabecular numbe

267 gnificant reduction in bone mineral density, trabecular bone volume, and cortical bone thickness comp

268 dependent reduction in bone mineral density, trabecular bone volume, and cortical thickness.

269 ed tomography analysis showed a reduction of trabecular bone volume, bone mineral density, and number

270 Cortical bone size, trabecular bone volume, bone mineralizing surfaces, and

271 creased bone size, bone mineral density, and trabecular bone volume, caused by impairment in osterix

272 to increase osteoclast numbers and decrease trabecular bone volume, cortical thickness and mechanica

273 rmation defect, fail to display increases in trabecular bone volume, cortical thickness, and bone for

274 t acid phosphatase staining revealed reduced trabecular bone volume, decreased cortical bone, and inc

275 Hes1 inactivation in osteoblasts increased trabecular bone volume, number, and connectivity due to

276 cts on bone as shown by higher bone mass and trabecular bone volume, number, and thickness and lower

277 d ACVR2B demonstrated sustained increases in trabecular bone volume, similar to those in ACVR2A singl

278 marrow fibrosis, despite similar effects on trabecular bone volume, suggests that marrow fibrosis wa

279 ed bone mineral density, cortical thickness, trabecular bone volume, thickness and number, and decrea

280 graphy analysis of femurs revealed increased trabecular bone volume, thickness, number, and connectiv

281 ase bone turnover, bone mineral density, and trabecular bone volume.

282 d thoracic spine, with an associated loss of trabecular bone volume.

283 and an increase in trabecular thickness and trabecular bone volume/tissue volume in U/NP and U/HP gr

284 In this study, PEDF delivery increased trabecular bone volume/total volume by 52% in 6-mo-old P

285 nt osteoporotic deficit, including decreased trabecular bone volumes and reduced trabecular thickness

286 ortical bone was -0.250, and between BMI and trabecular bone was -0.143 (all P < 0.001).

287 Trabecular bone was expanded in ACLL patients and occupi

288 ac bone sample from individual 2 showed that trabecular bone was hypermineralized on the material lev

289 Trabecular bone was likewise reduced and was accompanied

290 d densities of psoas muscle and cortical and trabecular bone were -0.460, -0.407, and -0.434, respect

291 raphy (muCT), and changes in subchondral and trabecular bone were assessed by standard muCT.

292 Osteoblasts, osteoclasts, and trabecular bone were increased in TG mice without change

293 e osteogenic benefits of co-treatment on the trabecular bone were lower than loading alone.

294 doubling in the number of osteoblasts lining trabecular bone, whereas osteoblast numbers in similarly

295 sed bone mineral density in the cortical and trabecular bone, whereas the bone mineral density of Fgf

296 ic-specific deletion of Epor exhibit reduced trabecular bone with age without change in marrow adipoc

297 d smaller bodies with shorter limbs, reduced trabecular bone with thinner cortices, and decreased ost

298 that accompany prostate cancer metastasis to trabecular bone, with potential implications to therapeu

299 s paralysis and profound loss of ipsalateral trabecular bone within days.

300 lly migrated and colonized tenascin-C-coated trabecular bone xenografts in a novel system that employ