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1 ith no overlap between sensory stimuli (i.e. trace conditioning).
2 ed by food 15 seconds after its termination (trace conditioning).
3 imuli, reflecting the laboratory paradigm of trace conditioning.
4 ed with pretesting levels for both delay and trace conditioning.
5 terize neuronal responses mediating aversive trace conditioning.
6  of the US to enable associative learning in trace conditioning.
7 tate (NMDA) receptor antagonists also impair trace conditioning.
8 als with hippocampal lesions fail to acquire trace conditioning.
9 oning at a normal rate but failed to acquire trace conditioning.
10 ioning but was a prerequisite for successful trace conditioning.
11 of learning-related hippocampal activity for trace conditioning.
12  acquisition or retention of either delay or trace conditioning.
13 s is decreased by distractions solely during trace conditioning.
14 neuron activity during the trace interval of trace conditioning.
15 simultaneous conditioning, is dispensable in trace conditioning.
16 llar regions and mechanisms coding delay and trace conditioning.
17 red in delay conditioning and MWM but not in trace conditioning.
18 developed into a decrease in firing later in trace conditioning.
19 l gyrus, and inferior parietal lobule during trace conditioning.
20 ctivity in the hippocampus-dependent task of trace conditioning.
21 osure to 200 trials of paired stimuli during trace conditioning.
22 ntingency facilitates but does not guarantee trace conditioning.
23 s, which is required for mnemonic aspects of trace conditioning.
24  role of the learning mechanism in classical trace conditioning.
25 terference similar in slope to that found in trace conditioning.
26 timing of conditioned responses in classical trace conditioning.
27 n the hippocampal-dependent learning task of trace conditioning.
28  the training conditions include reversal or trace conditioning.
29 ormance on the hippocampal-dependent task of trace conditioning.
30  the stimulus and PPL1-01 punishment (better trace conditioning), (2) impairs learning when the stimu
31                                           In trace conditioning, a 'trace' interval separates the con
32                                              Trace conditioning, a form of classical conditioning in
33 ss is known about brain regions that support trace conditioning, a procedure in which an interval of
34                                           In trace conditioning, a short interval is interposed betwe
35  the adult rat impairs hippocampal-dependent trace conditioning, a task in which an animal must assoc
36 This study examined the effects of stress on trace conditioning, a task in which the CS and US were s
37                                           In trace conditioning, a variant of the standard paradigm,
38 ellar cortex and deep nuclei in delay versus trace conditioning add perspective on brain substrates o
39 because the hippocampus is not necessary for trace conditioning after a period of consolidation has e
40 wo-photon brain imaging, we show that visual trace conditioning and delay conditioning in Drosophila
41 ex in processing temporal information during trace conditioning and delayed matching- or nonmatching-
42 vent persistently facilitates acquisition of trace conditioning and enhances a bias toward acquiring
43                                              Trace conditioning and extinction learning depend on the
44 digm, but this impairment was greater during trace conditioning and in rabbits with mPFC lesions.
45 ppocampus (DHPC) in acquisition of Pavlovian trace conditioning and interval timing was examined in a
46 rtance of cholinergic modulation in mPFC for trace conditioning and show that the observed effects ca
47 training alcohol disrupted the expression of trace conditioning and that supplemental choline on PD 1
48 ns of delay conditioning and two versions of trace conditioning and then assessed for the extent to w
49                                              Trace conditioning appears to provide a simple model of
50 prefrontal cortex (mPFC) impaired appetitive trace conditioning at a 2 s trace interval.
51 ch from our laboratory and others shows that trace conditioning begins to emerge during the third pos
52 training has a dramatic facilitory effect on trace conditioning but also implicate theta activity in
53 roved to be more resilient than differential trace conditioning but does show a reduction due to task
54    Scopolamine infusions profoundly impaired trace conditioning but had no effect on delay conditioni
55 e memory, significantly enhances learning of trace conditioning, but leaves simultaneous conditioning
56  DHPC lesions did not disrupt acquisition of trace conditioning, but they selectively affected the di
57                                           In trace conditioning, calcium transients during the trace
58 onditioning can be hippocampal-dependent and trace conditioning can be spared following hippocampal l
59 e showed significantly poorer contextual and trace conditioning compared with young-adult mice.
60 ocampal dependent, we designed a "contiguous trace conditioning" (CTC) paradigm in which CS-US contig
61                 However, mPFC lesion-induced trace conditioning deficits were obtained only in the gr
62                                    Pavlovian trace conditioning depends on the temporal gap between t
63                                In this case, trace conditioning did not enhance the survival of new n
64 ng neurons have different roles in delay and trace conditioning; dopamine D1-like receptor 1 mediates
65  Aspects of such learning can be probed with trace conditioning, during which an animal learns to ass
66                                              Trace conditioning experiments can reveal the time over
67  conditioning and awareness for differential trace conditioning extinction.
68  infusion profoundly reduced freezing in the trace conditioning group but had no significant effect o
69  associative learning-delay conditioning and trace conditioning-have been widely investigated in huma
70 rmine whether the lack of contiguity renders trace conditioning hippocampal dependent, we designed a
71 delay conditioning, a procedure that renders trace conditioning hippocampal-independent.
72  shock makes consolidation of the memory for trace conditioning hippocampus dependent.
73 clarative memory since humans do not acquire trace conditioning if they are unable to become cognitiv
74 essions 2, 4, and 6) in the acquisition of a trace conditioning in behaving rabbits.
75  showed that humans can learn both delay and trace conditioning in parallel.
76 re did not affect retention or extinction of trace conditioning in rats that were stressed after acqu
77 ed previous findings of facilitated acoustic trace conditioning in the 17-day-old rat in a sensory-en
78 enhanced later recall of delay compared with trace conditioning in the MR-available groups as indexed
79 independent of the hippocampal system, while trace conditioning (in which the CS terminates before US
80    Classical conditioning paradigms, such as trace conditioning, in which a silent period elapses bet
81 osure to 800 trials of paired stimuli during trace conditioning increased the number of BrdU-labeled
82                                              Trace conditioning is a form of classical conditioning i
83             This is especially important, as trace conditioning is an experimentally tractable model
84                                              Trace conditioning is associated with increased neuronal
85                                 However, why trace conditioning is dependent on the hippocampus is un
86                                              Trace conditioning is hippocampus dependent because, as
87 aining DH lesions, hippocampus dependency of trace conditioning is not related to a bridging function
88  experiment supports a view that its role in trace conditioning is to maintain a memory trace between
89                                              Trace conditioning is valued as a simple experimental mo
90 mpal- and forebrain-dependent learning task (trace conditioning) is associative rather than activity
91                     Moreover, the absence of trace conditioning learning in D1 dopamine receptor muta
92                                       During trace conditioning, lesioned rabbits did, however, exhib
93                      These data suggest that trace conditioning may be useful for studying neural sub
94                                              Trace conditioning may provide a means for studying awar
95 TOR) signaling and impaired performance on a trace-conditioning memory task.
96 is a general tendency for animals to acquire trace conditioning more slowly than delay conditioning.
97 ning occurs for ISIs of up to 4 sec, whereas trace conditioning occurs only at shorter ISIs; mixed tr
98                           Rabbits were given trace conditioning of the nictitating membrane response
99 s) fed 2% cholesterol for 8 weeks were given trace conditioning of the nictitating membrane response
100 eptor-glycine site) treatment were tested on trace conditioning of young or aging rabbits using a con
101 onkeys and report behavioral results using a trace conditioning paradigm that is sensitive to hippoca
102                                       In the trace conditioning paradigm, significant conditioning de
103 rformance or learning in either a delay or a trace conditioning paradigm.
104 ved as naive, head-restrained mice learned a trace conditioning paradigm.
105 ed with an aversive air-puff to the eye in a trace-conditioning paradigm.
106 onditioned responses (CRs) in both delay and trace conditioning paradigms in the rabbit (Oryctolagus
107                                        After trace conditioning, percent freezing to the CS was posit
108                Here, we adapted an olfactory trace conditioning procedure in Drosophila melanogaster
109  test this, mice were trained on a Pavlovian trace conditioning procedure in which the presentation o
110 copolamine was without effect in an aversive trace conditioning procedure, but impaired appetitive co
111 oned eyeblink (EB) response was studied with trace conditioning procedures in rabbits (Oryctolagus cu
112                      Aversive and appetitive trace conditioning procedures were used to examine the e
113 parated in time are associatively learned in trace conditioning, recruiting more neuronal circuits an
114  delay and trace fear conditioning, although trace conditioning requires recruitment of additional br
115                                              Trace conditioning requires that a transient representat
116          The distinct molecular signature of trace conditioning revealed here shall contribute to the
117 ade of muscarinic receptors in mPFC impaired trace conditioning shows that these receptors are critic
118 d on a moment-by-moment basis as in eyeblink trace conditioning studies.
119                          Monkeys performed a trace conditioning task in which different novel abstrac
120  the ability of rabbits to learn a difficult trace conditioning task.
121 y during learning, animals were trained on a trace-conditioning task in which a visual conditioned st
122 torhinal neurons during the acquisition of a trace-conditioning task in which a visual conditioned st
123 la neural activity while monkeys performed a trace-conditioning task requiring fixation.
124  and entorhinal neurons during an appetitive trace-conditioning task.
125 y the discontiguity between the CS and US in trace conditioning that critically engages the hippocamp
126 ulus (US) contributes to the facilitation of trace conditioning that occurs when preweanling rats are
127 al ethanol produced a profound impairment in trace conditioning that was reversed by choline.
128 ere exposed to eight tone-shock pairings (in Trace conditioning the shock came 30 s after the tone),
129 necessary to condition their association in "trace conditioning." The present study used conditioning
130                                              Trace conditioning therefore relies on maintaining a neu
131 olesterol showed significant facilitation of trace conditioning to airpuff and conditioning-specific
132              Young and aged rats received 10 trace conditioning trials.
133 period, all animals were retested for 3 d on trace conditioning, using the same parameters used durin
134                                              Trace conditioning was associated with an increase in th
135 gh the involvement of the DHPC in appetitive trace conditioning was not found when a gross measure of
136                                              Trace conditioning was similarly impaired at the 2 s tra
137 e a role for serotonin that is pronounced in trace conditioning, weaker in delay conditioning, and ab
138 ippocampus codes temporal information during trace conditioning, whereas brain regions supporting wor
139 he MB as a crucial neuroanatomical locus for trace conditioning, which may harbor a Rac activity-sens
140 then blocking amygdalar mAChRs should impair trace conditioning, while sparing delay and context cond

 
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