ライフサイエンスコーパス: tracheobronchial

1 irway epithelia (HAE) derived from nasal and tracheobronchial airway regions, we generated recombinan

2 were induced by mechanically stimulating the tracheobronchial airway.

3 sion intensified and was confined around the tracheobronchial airways, while it lessened during the p

4 Despite its localized nature, tracheobronchial amyloid deposition may be asymptomatic

5 Three of 4 patients with localized tracheobronchial amyloidosis required Nd:YAG (neodymium:

6 Recognition of tracheobronchial anatomy and familiarity with the use of

7 This review is focused on tracheobronchial anatomy and the use of flexible fiberop

8 A complete knowledge of tracheobronchial anatomy is a key factor in determining

9 In addition, changes occur in tracheobronchial anatomy with age; therefore, it is very

10 previous bronchial resection, recognition of tracheobronchial anatomy with the fiberoptic bronchoscop

11 a doses and dose relative intensities in the tracheobronchial and alveolar regions of lungs were calc

12 essential for goblet cell differentiation in tracheobronchial and gastrointestinal epithelium of mice

13 osition of nanomaterial(s) will occur in the tracheobronchial and head airways--not in the alveolar r

14 ted in the pulmonary region, followed by the tracheobronchial and head regions.

15 The tracheobronchial ASL hypotonicity was hypothesized to re

16 Rationale: Invasive tracheobronchial aspergillosis (ITBA) is an uncommon but

17 iagnostic and prognostic differences between tracheobronchial aspergillosis and pulmonary aspergillos

18 Tracheobronchial aspirates and blood samples were collec

19 alizes in adults to a discrete population of tracheobronchial basal cells.

20 who underwent postmortem examination and/or tracheobronchial biopsy during ICU admission from Septem

21 c activity, promoted transformation in human tracheobronchial cells and growth of xenografted human L

22 CBF of basolaterally permeabilized human tracheobronchial cells, re-differentiated at the air-liq

23 d with a significantly greater difference in tracheobronchial damage at the carina and main bronchus.

24 th antiviral properties are present in human tracheobronchial epithelial (HTBE) cell culture secretio

25 duced mucosecretory differentiation in human tracheobronchial epithelial (HTBE) cells and compared wi

26 drug-activated gene (NAG-1) in normal human tracheobronchial epithelial (HTBE) cells and several lun

27 Human tracheobronchial epithelial (HTBE) cells from donors wit

28 d trigger beta-defensin (hBD2) mRNA in human tracheobronchial epithelial (hTBE) cells through a CD14-

29 erentiated squamous metaplastic normal human tracheobronchial epithelial (NHTBE) and mucous NHTBE cel

30 uction in normal, well differentiated, human tracheobronchial epithelial (NHTBE) cell cultures, witho

31 EB) in a nonclassical manner in normal human tracheobronchial epithelial (NHTBE) cells.

32 mucous cell differentiation of normal human tracheobronchial epithelial (NHTBE) cells.

33 hly differentiated cultures of primary human tracheobronchial epithelial (TBE) cells with a panel of

34 sfer technique on passage 1 culture of human tracheobronchial epithelial (TBE) cells, the Flag-SPRR1

35 yes were used with well-differentiated human tracheobronchial epithelial cell cultures exhibiting spo

36 We characterized passage 2 human tracheobronchial epithelial cell cultures morphologicall

37 In experiments using primary culture human tracheobronchial epithelial cells (hTBECs) and each of t

38 and tumor specimens than in the normal human tracheobronchial epithelial cells and adjacent normal lu

39 ance regulator (CFTR) complementation and in tracheobronchial epithelial cells from patients with ver

40 n A is able to suppress the proliferation of tracheobronchial epithelial cells in culture.

41 Human CF tracheobronchial epithelial cells in primary culture rel

42 CBF and pHi of single human tracheobronchial epithelial cells in submerged culture w

43 xpression of the novel gene in primary human tracheobronchial epithelial cells in vitro.

44 nstrate that RSV infection of A549 and human tracheobronchial epithelial cells increases the amounts

45 ons were determined in primary culture human tracheobronchial epithelial cells transduced with gene t

46 In primary human tracheobronchial epithelial cells, OSCN(-) is generated

47 and growth of selected reassortants in human tracheobronchial epithelial cells.

48 sociated with this effect using normal human tracheobronchial epithelial cells.

49 transcription signaling components in human tracheobronchial epithelial cells.

50 h was developed from a cDNA library of human tracheobronchial epithelial cells.

51 aling pathways that mediate the induction in tracheobronchial epithelial cells.

52 he PMA-inducible expression of the SPRR1B in tracheobronchial epithelial cells.

53 Polarized tracheobronchial epithelial cultures from normal and cys

54 's), yet the appearance of mature MCs in the tracheobronchial epithelial surface is a characteristic

55 Furthermore, HBD2 with CD14 in human tracheobronchial epithelium can complex with "toll-like

56 gs demonstrate a novel role for Duox1 in the tracheobronchial epithelium, in addition to its proposed

57 identified NADPH oxidase homolog within the tracheobronchial epithelium, in airway epithelial cell m

58 NAG-1 expression was observed in the normal tracheobronchial epithelium, whereas no expression was f

59 and the Nkx2.8-/- mice exhibited generalized tracheobronchial hyperplasia.

60 89 trauma patients, 561 of whom had suffered tracheobronchial injuries (0.4%).

61 9), emergency esophagectomy (P = 0.013), and tracheobronchial injuries (P = 0.0011) were independent

62 incidence, therapy and outcome of traumatic tracheobronchial injuries (TTBI) in trauma patients with

63 The mortality of patients with tracheobronchial injuries was higher: 24.6%, versus 13.7

64 We compared tracheobronchial injury following short-term intratrache

65 The combined tracheobronchial injury scores for all samples were sign

66 Located at the tracheobronchial junction, the syrinx is responsible for

67 Located close to the heart at the tracheobronchial junction, vocal folds or membranes atta

68 gillosis and pulmonary aspergillosis without tracheobronchial lesions among patients admitted to the

69 PURPOSE OF REVIEW: Tracheobronchial lesions requiring significant resection

70 han invasive pulmonary aspergillosis without tracheobronchial lesions.

71 ted for removal were partly eroding into the tracheobronchial lumen and 23 were free.

72 Viral RNA was also detected in tracheobronchial lymph node and myocardium, together wit

73 specific lymphoproliferative responses from tracheobronchial lymph node cells, immunoglobulin M (IgM

74 -FLU vaccine induced weaker BAL and stronger tracheobronchial lymph node responses and a lesser reduc

75 body-secreting cells (ASC) in the spleen and tracheobronchial lymph node.

76 nterferon (IFN-gamma)-secreting cells in the tracheobronchial lymph nodes as determined by enzyme-lin

77 ecognized by antibody-secreting B cells from tracheobronchial lymph nodes isolated immediately follow

78 positive (DP) T cells in the mediastinal and tracheobronchial lymph nodes of RALT.

79 ever, relatively high activity levels in the tracheobronchial lymph nodes of the beagles indicated th

80 ions while no alpha activity was seen in the tracheobronchial lymph nodes of this individual.

81 roduction of IFN-gamma by lymphocytes of the tracheobronchial lymph nodes was also detected.

82 at gene expression patterns in the lungs and tracheobronchial lymph nodes would fit into a coherent a

83 ate and trachea, followed by tonsils, lungs, tracheobronchial lymph nodes, and stomach.

84 ophages and type II epithelial cells, and in tracheobronchial lymph nodes.

85 in blood, bronchoalveolar lavage (BAL), and tracheobronchial lymph nodes.

86 nhibited the binding activity of CD to human tracheobronchial mucin in a serum concentration-dependen

87 We have recently shown that the tracheobronchial mucin MUC5B is a major component of MG1

88 her lectin was involved in adhesion to human tracheobronchial mucin.

89 as to describe the management and outcome of tracheobronchial necrosis (TBN) after caustic ingestion.

90 day p.i. by TSA-ISH and in retropharyngeal, tracheobronchial, or external iliac lymph nodes and some

91 tter defines conditions under which relevant tracheobronchial proxies of human respiratory diseases c

92 stem resembling the epithelial lining of the tracheobronchial region of the porcine respiratory tract

93 f human airway tissues derived from nasal or tracheobronchial regions, suggesting that SARS-CoV may i

94 The lack of other Mesozoic tracheobronchial remains, and the poorly mineralized con

95 Tracheobronchial replacement remains a surgical and biol

96 Tracheobronchial replacement was indicated for extensive

97 02, to July 1, 2024, a total of 137 cases of tracheobronchial replacement were published.

98 gnant lesions are the primary indication for tracheobronchial replacement, with cryopreserved aortic

99 July 1, 2024, to identify studies examining tracheobronchial replacement.

100 Histopathologic testing of tracheobronchial samples showed PSMA expression in bronc

101 pecific gene expression is stable across all tracheobronchial samples.

102 d cells) from the nose (MUC4, PI3, SIX3) and tracheobronchial (SCGB1A1, TFF3) airways.

103 strongly express Nkx2.8 seem to function as tracheobronchial stem cells.

104 ful adjuncts for management of select benign tracheobronchial stenoses.

105 Attrition of tracheobronchial stent patency is most rapid during the

106 The tracheobronchial submucosal glands secrete liquid that i

107 tribution accurately reflected that in human tracheobronchial tissue.

108 nstrated a specimen consistent with sloughed tracheobronchial tissues and inflammatory cells in a bac

109 cleus); 2) soft palate, pharynx, larynx, and tracheobronchial tree (e.g., dorsal, intermediate, and i

110 Twelve movies of the thoracic aorta (n=3), tracheobronchial tree (n=4), colon (n=3), paranasal sinu

111 air leaks - any extrusion of air outside the tracheobronchial tree - have been attributed to high ven

112 ng evidence suggesting that formation of the tracheobronchial tree and alveoli results from heterogen

113 ary tissue that lacks communication with the tracheobronchial tree and has an aberrant non-pulmonary

114 d within the large conducting airways of the tracheobronchial tree being primarily responsible for oz

115 of partly eroded or free broncholiths in the tracheobronchial tree can be considered safe and effecti

116 m 11.5-day-old LKLF(-/-) embryos show normal tracheobronchial tree formation.

117 of dye placed in the subglottic space to the tracheobronchial tree in a rigid tracheal model and a be

118 l investigation, direct visualization of the tracheobronchial tree might be useful in determining the

119 The entire tracheobronchial tree obtained at autopsy was embedded i

120 No invasive tumor was found in the tracheobronchial tree or any other location.

121 Keywords: CT-Quantitative, Tracheobronchial Tree, Chronic Obstructive Pulmonary Dis

122 Keywords: CT-Quantitative, Tracheobronchial Tree, Chronic Obstructive Pulmonary Dis

123 This "colonization" of the tracheobronchial tree, currently believed to be innocuou

124 Keywords: Thorax, Lung, Mediastinum, Pleura, Tracheobronchial Tree, Fungus, Coccidioidomycosis, Spher

125 ition.Keywords: Lymphography, Lymphatic, CT, Tracheobronchial Tree, Thorax(C) RSNA, 2022See also comm

126 egion, or the more peripheral aspects of the tracheobronchial tree.

127 f systemic and pulmonary vasculature and the tracheobronchial tree.

128 f the ears, nose, peripheral joints, and the tracheobronchial tree.

129 (15.5%) needles did not fully penetrate the tracheobronchial tree.

130 is entirely or predominantly confined to the tracheobronchial tree.Objectives: To analyze the diagnos

131 ated the incidence and potential etiology of tracheobronchial uptake in patients being evaluated by (

132 PCa patients undergoing (18)F-DCFPyL PET/CT, tracheobronchial uptake occurred in 31% of patients.

133 The presence or absence of physiologic tracheobronchial uptake on PET/CT was recorded.