ライフサイエンスコーパス: trachomatis

1 the trpRBA tryptophan salvage pathway in C. trachomatis.

2 r rectal), recent, and ever infection with C trachomatis.

3 r outer membrane protein (MOMP) of Chlamydia trachomatis.

4 h pharyngeal and rectal N. gonorrhoeae and C.trachomatis.

5 ence of circulating genomic resistance in C. trachomatis.

6 is probably longer than for NGU caused by C. trachomatis.

7 mission is probably lower than for Chlamydia trachomatis.

8 r investigating the human pathogen Chlamydia trachomatis.

9 lopmentally controlled ompA expression in C. trachomatis.

10 m each child and tested for antibodies to C. trachomatis.

11 he obligate intracellular bacteria Chlamydia trachomatis.

12 pharyngeal and rectal N. gonorrhoeae and C. trachomatis.

13 .1]), HSV-2 excretion (26.7 [2.9-244.3]), C. trachomatis (11.7 [2.3-58.9]), and M. genitalium infecti

14 seen with T. vaginalis (9.0%; P = 0.005), C. trachomatis (6.2%), and N. gonorrhoeae (1.4%).

15 ence by gold standard testing was 8.5% for C trachomatis, 7.1% for N gonorrhoeae, 16.1% for T vaginal

16 nalyzed data from 655 patients tested for C. trachomatis (887 specimens) and N. gonorrhoeae (890 spec

17 In the class I-c beta subunit from Chlamydia trachomatis, a heterodinuclear Mn(II)/Fe(II) complex rea

18 Chlamydia trachomatis, a leading bacterial cause of sexually trans

19 Chlamydia trachomatis, a leading infectious cause of tubal inferti

20 fication tests (NAATs) that detect Chlamydia trachomatis AC2 also detects Neisseria gonorrhoeae Stora

21 on with bacterial burden, suggesting that C. trachomatis actively suppresses CXCL10.

22 nd changes in sexual behaviors and Chlamydia trachomatis, an infection with similar epidemiology to a

23 ysis were vaccine status, positive Chlamydia trachomatis and >/=4 partners in the preceding year.

24 of 300 colony forming units (CFU)/mL for C. trachomatis and 1500CFU/mL for N. gonorrhoeae.

25 Importantly, C. trachomatis and C. pneumoniae are Trp auxotrophs and are

26 Chlamydia trachomatis and Chlamydia muridarum are intracellular ba

27 steps in the polarized division of Chlamydia trachomatis and Chlamydia muridarum.

28 ch to other host cytokines in response to C. trachomatis and found evidence that RANTES, another T-ce

29 up to 84 days and (ii) swabs seeded with C. trachomatis and N. gonorrhoeae and then placed in transp

30 Participants were tested for C. trachomatis and N. gonorrhoeae at three sites (anorectum

31 pheid Xpert CT/NG assay (Xpert) to detect C. trachomatis and N. gonorrhoeae in rectal and pharyngeal

32 itical for the comprehensive detection of C. trachomatis and N. gonorrhoeae in the pediatric populati

33 viors impact the anatomic distribution of C. trachomatis and N. gonorrhoeae infection is needed to op

34 The burden of C. trachomatis and N. gonorrhoeae infection was significant

35 We assessed the positivity of C. trachomatis and N. gonorrhoeae infections at different a

36 entity factors may influence detection of C. trachomatis and N. gonorrhoeae infections at specific an

37 lar large sample of data on the burden of C. trachomatis and N. gonorrhoeae infections by anatomic si

38 th men, have shown that the prevalence of C. trachomatis and N. gonorrhoeae infections is much higher

39 l to aid public health efforts to control C. trachomatis and N. gonorrhoeae infections.

40 Specimens were tested for C. trachomatis and N. gonorrhoeae using a Gen-Probe Aptima

41 ree sources from 148 and 154 patients for C. trachomatis and N. gonorrhoeae, respectively.

42 study displayed the clinical features of C. trachomatis and N. gonorrhoeae, the 2 organisms that dri

43 score had point-of-care tests for Chlamydia trachomatis and Neisseria gonorrhoea (nucleic acid ampli

44 rtility and ectopic pregnancy, and Chlamydia trachomatis and Neisseria gonorrhoeae are recognized mic

45 Chlamydia trachomatis and Neisseria gonorrhoeae are the two most c

46 Chlamydia trachomatis and Neisseria gonorrhoeae cases reached a re

47 stems (Cobas) for the detection of Chlamydia trachomatis and Neisseria gonorrhoeae was established in

48 is the preferred method to detect Chlamydia trachomatis and Neisseria gonorrhoeae, but no commercial

49 ociations, which parallel those of Chlamydia trachomatis and Neisseria gonorrhoeae, the mechanisms by

50 most common bacterial infections: Chlamydia trachomatis and Neisseria gonorrhoeae.

51 th highest diagnostic yield is rectum for C. trachomatis and rectum and oropharynx for N. gonorrhoeae

52 d young adults for Chlamydia trachomatis (C. trachomatis) and Neisseria gonorrhoeae (N. gonorrhoeae)

53 were infected with a clinical isolate of C. trachomatis, and inclusions containing chlamydial develo

54 justed odds ratio [AOR], 1.75; P = .103), C. trachomatis (AOR, 1.43; P = .247), and T. vaginalis (AOR

55 of the proportion of PID cases caused by C. trachomatis are 35% (95% credible interval [CrI], 11%-69

56 ugh most individuals infected with Chlamydia trachomatis are initially asymptomatic, symptoms can ari

57 e obligate intracellular bacterium Chlamydia trachomatis arrives at a physiologically similar 'persis

58 for further interventions to prevent future trachomatis blindness.

59 ificantly reduced IFN-gamma and increased C. trachomatis burden in the endometrial tissue, confirming

60 n of chlamydia might have been exposed to C. trachomatis but not infected.

61 g adolescents and young adults for Chlamydia trachomatis (C. trachomatis) and Neisseria gonorrhoeae (

62 the membrane of the human pathogen Chlamydia trachomatis (C.t.).

63 Chlamydia trachomatis can cause reproductive morbidities after asc

64 Chlamydia trachomatis can enter a viable but nonculturable state i

65 However, C. trachomatis can productively infect mice when the lower

66 fecal-oral route; (2) in the modern era, C. trachomatis causes "opportunistic" infection at non-GI s

67 Chlamydia trachomatis causes sexually transmitted infections and t

68 assembles at the pole of dividing Chlamydia trachomatis cells where daughter cell formation occurs,

69 mptoms is used to manage anorectal Chlamydia trachomatis (chlamydia) and Neisseria gonorrhoeae (gonor

70 Chlamydia trachomatis (CT) and Mycoplasma genitalium (MG) are two

71 Rectal infections with Chlamydia trachomatis (CT) are prevalent in women visiting a sexua

72 The Chlamydia trachomatis (CT) bacterial load could have impact on tra

73 fertility in women with a previous Chlamydia trachomatis (CT) diagnosis compared with women who teste

74 Chlamydia trachomatis (Ct) has been associated with miscarriage bu

75 he obligate-intracellular pathogen Chlamydia trachomatis (Ct) has undergone considerable genome reduc

76 tected in 25 (17.2%) participants, Chlamydia trachomatis (CT) in 13 (9.0%), Mycoplasma genitalium (MG

77 d symptomatic and asymptomatic women with C. trachomatis (CT) infection and asymptomatic, uninfected

78 tricts and a considerable level of Chlamydia trachomatis (Ct) infection was evident.

79 for Neisseria gonorrhoeae (NG) and Chlamydia trachomatis (CT) infections in all men who have sex with

80 Rectal Chlamydia trachomatis (CT) is common among clinic-attending women,

81 Chlamydia trachomatis (Ct) is the leading cause of bacterial sexua

82 The frequency and duration of Chlamydia trachomatis (Ct) ocular infections decrease with age, su

83 tal Neisseria gonorrhoeae (NG) and Chlamydia trachomatis (CT) so universal extragenital sampling is r

84 V, Neisseria gonorrhoeae (NG), and Chlamydia trachomatis (CT) transmission dynamics among MSM in the

85 An example is infection with Chlamydia trachomatis (Ct), which is the most common sexually tran

86 We tested 30 potentially Chlamydia trachomatis (CT)-infected patients in a hospital emergen

87 Trachoma is caused by Chlamydia trachomatis (Ct).

88 tal Neisseria gonorrhoeae (NG) and Chlamydia trachomatis (CT).

89 Although a C. trachomatis cteG mutant did not display a defect in intr

90 Human C. pneumoniae (Cpn) and C. trachomatis (Ctr) seroreactivity was 54% biased towards

91 ted HLA-DR4 transgenic mice with 5 x 10(5)C. trachomatis D inclusion forming units (IFU) induced a si

92 were more susceptible to a transcervical C. trachomatis D infection than WT mice.

93 be protected by vaccination, 10(4) IFU of C. trachomatis D was delivered intranasally, and mice were

94 cally 6 weeks later with 5 x 10(5) IFU of C. trachomatis D.

95 The PEFs of PID due to C. trachomatis decline steeply with age by a factor of arou

96 ate intracellular bacteria such as Chlamydia trachomatis depend on metabolites of the host cell and t

97 and conjunctival swabs were collected for C. trachomatis detection and to analyze the expression of 4

98 MA (12,999 specimens) on the basis of the C. trachomatis detection rate, specimen source distribution

99 erichia coli was toxic to cells, LOS from C. trachomatis did not induce any appreciable cell death, s

100 aginal squamous epithelial cells, whereas C. trachomatis did not.

101 Here, we identified CteG as the first C. trachomatis effector associated with the Golgi.

102 Chlamydia trachomatis elementary body enzyme-linked immunosorbent

103 Wild-type C. trachomatis entry into HeLa cells resulted in host cell

104 mannii, Burkholderia pseudomallei, Chlamydia trachomatis, Escherichia coli, Klebsiella pneumoniae, Le

105 al fragmentation prevented replication of C. trachomatis even in p53-deficient cells.

106 latives, the oculogenital pathogen Chlamydia trachomatis evolved as a commensal organism of the human

107 comparison, mice vaginally infected with C. trachomatis exhibited transient low-burden infections, p

108 Chlamydia trachomatis exits host epithelial cells through two equa

109 ement for detection of N. gonorrhoeae and C. trachomatis for 3 investigational assays compared to a c

110 n of Cdu1 led to increased sensitivity of C. trachomatis for IFNgamma and impaired infection in mice.

111 ement for detection of N. gonorrhoeae and C. trachomatis for three investigational assays compared to

112 Chlamydia trachomatis forms inclusions that are decorated with poo

113 that the putative OppABCDF transporter of C. trachomatis functions in both oligopeptide transport and

114 on of reproductive damage attributable to C. trachomatis Further studies using modern assays in conte

115 findings and to advance understanding of C. trachomatis genetic expression.

116 Chlamydia trachomatis genital tract infection is a major cause of

117 olates appear to be recombinants with UGT C. trachomatis genome backbones, in which loci that encode

118 The Chlamydia trachomatis genome encodes multiple bifunctional enzymes

119 ve PCR analysis showed that expression of C. trachomatis glycolytic enzymes inversely correlated with

120 he obligate intracellular pathogen Chlamydia trachomatis, grow within a membrane-bound bacterium-cont

121 , and Neisseria gonorrhoeae and/or Chlamydia trachomatis had 92% lower odds of any adverse birth outc

122 In this work, we are showing that C. trachomatis has an active respiratory metabolism that se

123 For decades C. trachomatis has been considered an "energy parasite" tha

124 We show that a LipL2 enzyme from Chlamydia trachomatis has similar activity, demonstrating conserva

125 One such pathogen, Chlamydia trachomatis, has a limited capacity to synthesize amino

126 netii, Listeria monocytogenes, and Chlamydia trachomatis have developed bipartite metabolism within t

127 ection induces partial immunity to Chlamydia trachomatis Identification of chlamydial antigens that i

128 lity of the chlamydial Opp transporter in C. trachomatis Importantly, we found that one chlamydial SB

129 support the full infectious life cycle of C. trachomatis in a manner that mimics the infection of hum

130 type III secreted effector used by Chlamydia trachomatis In aggregate, existing data suggest a role o

131 sensitivity of Cobas for the detection of C. trachomatis in female specimens was 95.6% (95% confidenc

132 ng the potential role of gastrointestinal C. trachomatis in human chlamydial pathogenesis.

133 y estimates of Cobas for the detection of C. trachomatis in male urine samples were 100% (96.8% to 10

134 miscarriage suggests a potential role for C trachomatis in poor reproductive health outcomes in this

135 ntributing to the increased prevalence of C. trachomatis in the human GI tract.

136 The epidemiology of Chlamydia trachomatis in the Middle East and north Africa is poorl

137 voids the characteristic low virulence of C. trachomatis in the mouse, we previously demonstrated a s

138 actate dehydrogenase, are enriched at the C. trachomatis inclusion membrane during infection.

139 major suppressor of metabolite supply in C. trachomatis-infected cells.

140 ; P < 0.0001) and higher than that of the C. trachomatis-infected females (mean, 23.8 years; P = 0.00

141 (miRNAs) in maintaining the viability of C. trachomatis-infected primary human cells.

142 hat CVM was significantly associated with C. trachomatis infection (odds ratio [OR], 4.2 [95% confide

143 Women who tested positive for Chlamydia trachomatis infection after having been contact-traced b

144 , which replicates many features of human C. trachomatis infection and avoids the characteristic low

145 We did a systematic review of C trachomatis infection as well as a meta-analysis and met

146 a cohort of 149 women with genital Chlamydia trachomatis infection at baseline who were followed quar

147 rge to diagnose N. gonorrhoeae and Chlamydia trachomatis infection in certain populations by nucleic

148 care test for detecting urogenital Chlamydia trachomatis infection in nonpregnant women and men at re

149 omprehensive epidemiological assessment of C trachomatis infection in the Middle East and north Afric

150 etrial resistance to sexually transmitted C. trachomatis infection in women.

151 Finally, C. trachomatis infection interferes with the SNX5:CI-MPR in

152 oductive tract pathology caused by Chlamydia trachomatis infection is an important global cause of hu

153 eport that levels of CXCL10 change during C. trachomatis infection of cultured cells in a manner depe

154 Primary C. trachomatis infection of mice also causes no genital pat

155 Chlamydia trachomatis infection of the female genital tract can le

156 Chlamydia trachomatis infection of the human fallopian tubes can l

157 Characteristics associating with C. trachomatis infection were examined using bivariable and

158 LOS synthesis was inhibited during Chlamydia trachomatis infection, HeLa cells regained susceptibilit

159 In response to C. trachomatis infection, IL23A and PDGF were significantly

160 ve been associated with increased risk of C. trachomatis infection, suggesting that the impact of ant

161 rvivin levels was also detected following C. trachomatis infection, which was reversed by blocking LO

162 innate immunity in endometrial control of C. trachomatis infection.

163 omen notified by a sex partner for Chlamydia trachomatis infection.

164 Genital Chlamydia trachomatis infections in women typically are asymptomat

165 Currently, a vaccine to prevent C. trachomatis infections is not available.

166 o thiazolino 2-pyridones which attenuated C. trachomatis infectivity without affecting host cell or c

167 ), and IL-4Ralpha(-/-) mice with low-dose C. trachomatis inoculums.

168 at interact with Chlamydia, we introduced C. trachomatis into mouse endometrium via transcervical ino

169 dence for the significant role of Tarp in C. trachomatis invasion of host cells.

170 Chlamydia trachomatis is a bacterial pathogen causing ocular and g

171 The bacterial pathogen Chlamydia trachomatis is a global health burden currently treated

172 Chlamydia trachomatis is a global health burden due to its prevale

173 Although Chlamydia trachomatis is a human genital tract pathogen, chlamydia

174 relatively little is known regarding how C. trachomatis is able to hijack host cell metabolism.

175 Plasmid-free C. trachomatis is also attenuated in both the mouse genital

176 Chlamydia trachomatis is an important human pathogen that undergoe

177 Chlamydia trachomatis is an obligate intracellular bacteria that u

178 Chlamydia trachomatis is an obligate intracellular human pathogen

179 Chlamydia trachomatis is an obligate intracellular pathogen that c

180 Chlamydia trachomatis is an obligate intracellular pathogen that r

181 With a reduced genome, C. trachomatis is dependent on its host for survival, in pa

182 The genital tract pathogen Chlamydia trachomatis is frequently detected in the gastrointestin

183 common, but ocular infection with Chlamydia trachomatis is not.

184 The trp operon of Chlamydia trachomatis is organized differently from other model ba

185 long-term infections of humans by Chlamydia trachomatis is poorly understood.

186 al and ocular clinical isolates of Chlamydia trachomatis is that only the former express a functional

187 Chlamydia trachomatis is the leading cause of bacterial sexually t

188 Chlamydia trachomatis is the leading cause of infection-induced in

189 he obligate intracellular pathogen Chlamydia trachomatis is the leading cause of noncongenital blindn

190 Chlamydia trachomatis is the leading cause of sexually transmitted

191 Chlamydia trachomatis is the most common sexually transmitted bact

192 Chlamydia trachomatis is the most common sexually transmitted bact

193 Chlamydia trachomatis is the most common sexually transmitted bact

194 Chlamydia trachomatis is the most commonly reported bacterial sexu

195 Chlamydia trachomatis is the world's most prevalent bacterial sexu

196 nducted whole-genome sequence analysis on C. trachomatis isolates collected from a previously describ

197 is, Incs fused to APEX2 were expressed in C. trachomatis L2.

198 he complete deletion of specific genes in C. trachomatis L2.

199 ing a novel cis complementation approach, C. trachomatis lacking tarP demonstrated significant attenu

200 n some women, sexually transmitted Chlamydia trachomatis may ascend to infect the endometrium, leadin

201 exually transmitted infection with Chlamydia trachomatis may lead to fibrotic blockage in women's upp

202 C. trachomatis multiplies exclusively inside host cells wit

203 pathic when Neisseria gonorrhoeae, Chlamydia trachomatis, Mycoplasma genitalium, Trichomonas vaginali

204 H algorithms identified similar numbers of C trachomatis, N gonorrhoeae, and T vaginalis infections,

205 Nucleic acid amplification tests for C trachomatis, N gonorrhoeae, T vaginalis, bacterial vagin

206 Incidence of any bacterial STI (C. trachomatis, N. gonorrhoeae, or M. genitalium infection)

207 52 tested C. trachomatis positive and 41 C. trachomatis negative.

208 he intervention effect on incident Chlamydia trachomatis, Neisseria gonorrhoeae, and Mycoplasma genit

209 Positivity rates for Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vagi

210 st-void female urine specimens for Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vagi

211 ghts concerning the concurrence of Chlamydia trachomatis, Neisseria gonorrhoeae, Mycoplasma genitaliu

212 ens were tested for M. genitalium, Chlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginali

213 For C. trachomatis, neither system was >95% sensitive from the

214 atics analysis suggests that the putative C. trachomatis oligopeptide transporter OppABCDF (OppABCDF

215 We previously showed that DCs infected by C trachomatis or C muridarum present epitopes from a limit

216 hra/cervix) testing identified 92-100% of C. trachomatis or N. gonorrhoeae infections in participants

217 adation in the AC2 assay for detection of C. trachomatis or N. gonorrhoeae was observed, although som

218 ifferent urine samples spiked with either C. trachomatis or N. gonorrhoeae, and also containing both

219 eported recent sexual contact with either C. trachomatis or N. gonorrhoeae, or had symptoms of an STI

220 The exploitation of genetically labeled C. trachomatis organisms with P3-driven GFP allows for the

221 ; HSV-2, P = .001; and a trend for Chlamydia trachomatis, P = .06).

222 darum, a model pathogen for investigating C. trachomatis pathogenesis, readily spreads from the mouse

223 inx in mice and is used for investigating C. trachomatis pathogenicity.

224 riplasmic domain of the RsbU protein from C. trachomatis (PDB 6MAB) displays close structural similar

225 A common feature of C. trachomatis persistence models is that reticulate bodies

226 er paper to test for antibodies to Chlamydia trachomatis pgp3 using a multiplex bead assay.

227 nd dried to test for antibodies to Chlamydia trachomatis pgp3 using the Luminex platform.

228 ead being placed within UGT clades of the C. trachomatis phylogenetic tree.

229 d rectal Neisseria gonorrhoeae and Chlamydia trachomatis play important roles in infection and antiba

230 acterized for 93 women, of whom 52 tested C. trachomatis positive and 41 C. trachomatis negative.

231 luded 98 women who were contact-traced by C. trachomatis-positive sex partners at the STI outpatient

232 in the pharynx and 7.9% in the rectum and C.trachomatis positivity of 2.0% in the pharynx and 8.7% i

233 in the pharynx and 7.9% in the rectum and C. trachomatis positivity of 2.0% in the pharynx and 8.7% i

234 C trachomatis prevalence in the Middle East and north Afri

235 s, of which 255 reports contributed to 552 C trachomatis prevalence measures from 20 countries.

236 r report including biological measures for C trachomatis prevalence or incidence was eligible for inc

237 as a meta-analysis and meta-regression of C trachomatis prevalence.

238 Although the Chlamydia trachomatis protein Cta1 has been identified to be a dom

239 arator assays included BD ProbeTec Chlamydia trachomatis Q(x) (CTQ)/Neisseria gonorrhoeae Q(x) (GCQ),

240 Here, we determined the ability of a C. trachomatis recombinant major outer membrane protein (rM

241 Live C. trachomatis recovered from vaginal swabs or endometrial

242 Chlamydia trachomatis remains a leading cause of bacterial sexuall

243 Obligate intracellular Chlamydia trachomatis replicate in a membrane-bound vacuole called

244 We show that C. trachomatis require mitochondrial ATP for normal develop

245 The genomic data suggest that C. trachomatis respiratory chain could produce a sodium gra

246 excess l-glutamate or meso-DAP (mDAP) to C. trachomatis resulted in 90% reduction in bacterial titer

247 ys, respectively) in two studies: (i) dry C. trachomatis-seeded swabs were used with ACT after storag

248 tions compared with gold standard testing: C trachomatis sensitivity 58.3%, specificity 44.7%; N gono

249 good sensitivity and high specificity for C trachomatis (sensitivity 71.7%, specificity 100%), N gon

250 ns that can be applied to humans, we used C. trachomatis serovar D (strain UW-3/Cx) to induce inferti

251 nated by mucosal and systemic routes with C. trachomatis serovar D (UW-3/Cx) rMOMP and challenged in

252 Chlamydia trachomatis serovar L2 and Chlamydia muridarum, which do

253 h positive cultures) mice challenged with C. trachomatis serovars of the same complex were protected

254 logy, and infertility caused by different C. trachomatis serovars.

255 ansmission of Chlamydia closely parallels C. trachomatis sexual transmission in humans and may be a g

256 region verifies that P3 is a new class of C. trachomatis sigma(66)-dependent promoter, which requires

257 ithin a few days, while a CPAF-sufficient C. trachomatis strain (L2-5) survived in the lower genital

258 We now report that a Chlamydia trachomatis strain deficient in expression of CPAF (L2-1

259 anipulation have allowed us to generate a C. trachomatis strain expressing a heterologous CD4(+) T ce

260 Genital C. trachomatis strains can counter tryptophan limitation be

261 For this, C. trachomatis strains expressing candidate effectors fused

262 , we screened a population of mutagenized C. trachomatis strains for mutants that failed to reactivat

263 rivatives de-repress the trpRBA operon of C. trachomatis strains with trpA or trpB mutations, no ammo

264 susceptible to endometrial infection with C. trachomatis, suggesting a role for group 3-like ILCs in

265 The lack of resistance in Chlamydia trachomatis suggests that azithromycin might remain effe

266 3 deficiency also significantly increased C. trachomatis susceptibility to lactic acid.

267 The human pathogen Chlamydia trachomatis targets epithelial cells lining the genital

268 he obligate intracellular bacteria Chlamydia trachomatis, the causative agent of trachoma and sexuall

269 y reduces the prevalence of ocular Chlamydia trachomatis, the causative organism of trachoma.

270 lity (TFI) that is attributable to Chlamydia trachomatis, the population excess fraction (PEF), can b

271 ri-diaminopimelic acid) in E. coli and in C. trachomatis These findings suggest that Chlamydia evolve

272 ctivity of DapF compromises the growth of C. trachomatis Thus, a substrate competition strategy can b

273 comparison to a 6-month audit of clinical C. trachomatis TMA (12,999 specimens) on the basis of the C

274 ected with Chlamydia muridarum and Chlamydia trachomatis to determine if there were differences betwe

275 n mammalian cells, themselves hijacked by C. trachomatis to sustain their own metabolic needs.

276 mate reduced in vivo synthesis of mDAP by C. trachomatis to undetectable levels, thus confirming that

277 trol strategies on the dynamics of Chlamydia trachomatis transmission are difficult to quantify.

278 rrhoeae, Streptococcus agalactiae, Chlamydia trachomatis, Trichomonas vaginalis, and Candida spp., as

279 es function by acting as de-repressors of C. trachomatis TrpR.

280 nt in serum, rapidly induce expression of C. trachomatis tryptophan synthase, even under conditions o

281 ound in the intracellular pathogen Chlamydia trachomatis, uses YhhQ and tRNA guanine transglycosylase

282 mmon Ag in Chlamydia muridarum and Chlamydia trachomatis Using an adoptive-transfer approach, we show

283 ion excess fractions (PEFs) of PID due to C. trachomatis, using routine data, surveys, case-control s

284 Our results indicate that C. trachomatis utilizes functionally diverse genes to media

285 ansgenic mouse model for evaluating human C. trachomatis vaccine antigens are discussed.

286 e obligate intracellular bacterium Chlamydia trachomatis, variation in immune activation and disease

287 and for the Tarp effector as a bona fide C. trachomatis virulence factor.

288 The specificity for the detection of C. trachomatis was >=98.8% for all female sample types.

289 C. trachomatis was detected from 59 rectal swabs and 8 phar

290 ity with a high prevalence of STI, Chlamydia trachomatis was detected in 8.7% and Neisseria gonorrhoe

291 The positivity rate for C. trachomatis was highest for rectal (16.2%), followed by

292 demonstrate DapF (Ct) function in vivo in C. trachomatis We reasoned that, because DapF (Ct) utilizes

293 n be challenging, as mice naturally clear C. trachomatis when it is deposited in the lower genital tr

294 es were associated with susceptibility to C. trachomatis, whereas cytokines involved in Th1 polarizat

295 peated conjunctival infection with Chlamydia trachomatis, which causes a chronic inflammatory respons

296 cans, Streptococcus agalactiae and Chlamydia trachomatis with a single biochip, enabling a quick scre

297 By tagging proteins expressed in C. trachomatis with OVA(323-339), we can begin to understan

298 everal analogues can impair the growth of C. trachomatis without affecting host cell viability.

299 g infectivity across multiple serovars of C. trachomatis without host cell toxicity.

300 For C. trachomatis, Xpert was 95% sensitive (95% CI, 86 to 99%)