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2 timulation technology called high-definition transcranial alternating current stimulation (HD-tACS).
5 cortical excitability.SIGNIFICANCE STATEMENT Transcranial alternating current stimulation (tACS) is a
6 sis on thirty-one healthy participants using transcranial Alternating Current Stimulation (tACS) to e
10 ephalography (EEG), we applied 20 minutes of transcranial alternating current stimulation (tACS) to t
12 rticospinal tES known so far, which is 20 Hz transcranial alternating current stimulation (tACS).
13 a combined transcranial magnetic stimulation-transcranial alternating current stimulation approach (i
17 characterizing the human sensation evoked by transcranial alternating-current stimulation, we observe
18 fields and to those produced by noninvasive transcranial brain stimulation, and therefore possibly a
24 inusoids that delivered maximally ~1% of the transcranial current to the auditory nerve, which was su
25 hat brief application of 2 mA (peak-to-peak) transcranial currents alternating at 10 Hz significantly
28 hether individually-targeted high-definition transcranial direct current stimulation (HD-tDCS) can re
29 this, we selectively applied high-definition transcranial direct current stimulation (HD-tDCS) to the
30 sions, we administered focal high definition transcranial direct current stimulation (HD-tDCS) while
31 in animals and humans provide evidence that transcranial direct current stimulation (tDCS) allows a
32 of the brain, and recent studies identified transcranial direct current stimulation (tDCS) as an eff
33 higher cognitive functions like reading with transcranial direct current stimulation (tDCS) can be ch
34 here report that application of non-invasive transcranial direct current stimulation (tDCS) during ad
36 interface-assisted motor imagery (MI-BCI) or transcranial direct current stimulation (tDCS) has been
40 ed to the aging brain.SIGNIFICANCE STATEMENT Transcranial direct current stimulation (tDCS) modulates
42 stion by applying double-blind bihemispheric transcranial direct current stimulation (tDCS) over both
44 that honesty can be increased in humans with transcranial direct current stimulation (tDCS) over the
46 sorders and functions reported responsive to transcranial direct current stimulation (tDCS) suggests
48 the possibility of suppressing the DLPFC by transcranial direct current stimulation (tDCS) to facili
52 nce supports application of one type of tCS, transcranial direct current stimulation (tDCS), for majo
55 we show that modulation of this region with transcranial direct current stimulation alters both acti
56 participants received 30 min of real or sham transcranial direct current stimulation applied to the l
58 we test the hypothesis that the response to transcranial direct current stimulation following trauma
59 ine working memory task performance, but the transcranial direct current stimulation group demonstrat
60 ced activation in the left cerebellum in the transcranial direct current stimulation group, with no c
61 cortex (PPC) using high-definition cathodal transcranial direct current stimulation impedes learning
62 PJ or dorsomedial PFC anodal high-definition transcranial direct current stimulation in a sham-contro
63 tory training, repetitive task training, and transcranial direct current stimulation may improve ADLs
64 nd randomized sham controlled pilot study of transcranial direct current stimulation on a working mem
65 However, only two studies on amantadine and transcranial direct current stimulation provided class I
67 alking speed and repetitive task training or transcranial direct current stimulation to improve activ
69 In the present study, we use high-definition transcranial direct current stimulation to provide evide
70 problem-solving, by applying High Definition Transcranial Direct Current Stimulation to the rATL (act
76 as is typical with transcranial magnetic or transcranial direct/alternating current electrical stimu
77 ty (FVsv), and methods derived from arterial transcranial Doppler (aTCD) on the middle cerebral arter
78 children with sickle cell anemia (SCA), high transcranial Doppler (TCD) velocities are associated wit
79 g optic nerve sheath diameter (ONSD), venous transcranial Doppler (vTCD) of straight sinus systolic f
80 f MR angiography was contraindicated, and by transcranial Doppler and carotid ultrasound if CT angiog
82 sion therapy, for SCD patients with abnormal transcranial Doppler results, there is appropriate anxie
83 iddle cerebral artery (MCAv) was obtained by transcranial Doppler sonography and arterial pressure in
84 li burden, assessed noninvasively by bedside transcranial Doppler ultrasonography, correlates with ri
86 ars) receiving chronic hemodialysis, we used transcranial Doppler ultrasound to measure cerebral arte
90 ings of MAP (Finometer), CBF velocity (CBFV; transcranial Doppler ultrasound), end-tidal CO(2) (capno
94 of blood transfusions, hydroxyurea therapy, transcranial Doppler-confirmed cerebral vasculopathy), g
96 lated vertebral artery injuries had positive transcranial Dopplers before stroke, and positive transc
97 I, 1.01-1.05) and with persistently positive transcranial Dopplers over multiple days (risk ratio, 16
98 cranial Dopplers before stroke, and positive transcranial Dopplers were not associated with delayed s
99 the following working hypothesis: in humans, transcranial electric stimulation (tES) with a time cour
101 rectly and objectively measure the amount of transcranial electric stimulation reaching the auditory
103 d evidence for activation of deep tissues by transcranial electric stimulation, its evoked human sens
109 nslation process, we used a novel technique, transcranial focused ultrasound stimulation, to reversib
110 presented here will benefit the multitude of transcranial FUS applications as well as future human ap
111 above described stimulation, which we named transcranial individual neurodynamics stimulation (tIDS)
112 tion and viral titers postinfection in vitro Transcranial inoculation of C57Bl/6 mice with RSA59 (PP)
113 vestigated this using two protocols based on transcranial magnetic brain stimulation (TMS) in healthy
115 lating cortical activity, as is typical with transcranial magnetic or transcranial direct/alternating
118 following adaptation, continuous theta-burst transcranial magnetic stimulation (cTBS) was delivered t
119 y study indicated beneficial effects of deep transcranial magnetic stimulation (dTMS) targeting the m
120 then temporarily inhibited PPC by repetitive transcranial magnetic stimulation (rTMS) and hypothesize
121 fusion tensor imaging, and online repetitive transcranial magnetic stimulation (rTMS) applied during
122 cs following low frequency (1 Hz) repetitive transcranial magnetic stimulation (rTMS) as an inhibitor
124 Despite growing evidence that repetitive transcranial magnetic stimulation (rTMS) can be used as
125 roposed therapeutic potential for repetitive transcranial magnetic stimulation (rTMS) in swallowing r
129 r, and 1 hour after low-frequency repetitive transcranial magnetic stimulation (rTMS) to the right PP
131 al-temporal cortex by delivering theta-burst transcranial magnetic stimulation (TBS) concurrent with
133 estingly, disrupting cerebellar activity via transcranial magnetic stimulation (TMS) abolished the ad
136 the right or left speech motor cortex using transcranial magnetic stimulation (TMS) and measured the
140 exposure group (N=17) underwent single-pulse transcranial magnetic stimulation (TMS) concurrent with
141 eceived brief patterns of rhythmic or random transcranial magnetic stimulation (TMS) delivered to the
142 plitude, and timing of beta events preceding transcranial magnetic stimulation (TMS) each significant
143 e feasibility, safety, and efficacy of 10-Hz transcranial magnetic stimulation (TMS) for adolescents
144 rts of patients who received left prefrontal transcranial magnetic stimulation (TMS) for treatment of
146 ese neural oscillations, we applied rhythmic transcranial magnetic stimulation (TMS) in either theta
148 ous, causal test by combining the FCM with a transcranial magnetic stimulation (TMS) intervention tha
149 order (MDD) has become a particular focus of transcranial magnetic stimulation (TMS) investigational
154 interval between trigeminal stimulation and transcranial magnetic stimulation (TMS) of fM1 was 15-30
155 xcitability and neural plasticity often used transcranial magnetic stimulation (TMS) of hand motor co
157 f motor evoked potentials (MEPs) obtained by transcranial magnetic stimulation (TMS) of M1 using an o
158 r or extensor muscle at the motor point with transcranial magnetic stimulation (TMS) of the motor cor
161 ere we explored this possibility by means of transcranial magnetic stimulation (TMS) over the hand ar
162 r (AP) and posterior-anterior (PA) pulses of transcranial magnetic stimulation (TMS) over the primary
163 11 healthy controls, we applied paired-pulse transcranial magnetic stimulation (TMS) protocols to eva
165 been at the forefront of advancing clinical transcranial magnetic stimulation (TMS) since the mid-19
167 In the current study, we used MRI-guided transcranial magnetic stimulation (TMS) to assess whethe
169 e areas and attentional modulations, we used transcranial magnetic stimulation (TMS) to briefly alter
170 halography (EEG) to record brain signals and transcranial magnetic stimulation (TMS) to deliver infor
172 ht be possible to use MI in conjunction with transcranial magnetic stimulation (TMS) to induce plasti
173 e and female participants using single-pulse transcranial magnetic stimulation (TMS) to interfere wit
175 ale and female) brain noninvasively, we used transcranial magnetic stimulation (TMS) to probe the exc
176 ity was modulated with 5 days of twice-daily transcranial magnetic stimulation (TMS) to the cerebella
178 ronometry of the process by combining online transcranial magnetic stimulation (TMS) with computation
181 f the first corticospinal volley elicited by transcranial magnetic stimulation (TMS), by interacting
182 itative real time polymerase chain reaction, transcranial magnetic stimulation (TMS), functional magn
183 increased resting motor threshold (RMT) with transcranial magnetic stimulation (TMS), is known to be
184 rk in prediction of response to both ECT and transcranial magnetic stimulation (TMS), offering a new
192 om neuropsychology [5], neuroimaging [6-11], transcranial magnetic stimulation [12, 13], single-unit
197 asive vagus nerve stimulation (nVNS), single-transcranial magnetic stimulation and external trigemina
199 efined electrophysiological method involving transcranial magnetic stimulation and peripheral nerve s
200 ked potentials (MEPs) evoked by single-pulse transcranial magnetic stimulation and short-interval int
201 itude of corticospinal responses elicited by transcranial magnetic stimulation and the magnitude of m
202 s that can be probed by the onset latency of transcranial magnetic stimulation applied to primary mot
206 plying a uniform spatial sampling procedure, transcranial magnetic stimulation can produce cortical f
208 that effective connectivity, as assessed by transcranial magnetic stimulation combined with electroe
210 ar inhibition (CBI): a conditioning pulse of transcranial magnetic stimulation delivered to the cereb
211 ssible region of the hippocampal network via transcranial magnetic stimulation during concurrent fMRI
217 nical neurophysiology of the brain employing transcranial magnetic stimulation has convincingly demon
218 nge to dIPL node with continuous theta-burst transcranial magnetic stimulation in a randomized, sham-
219 rticospinal conduction failure assessed with transcranial magnetic stimulation in the right upper lim
220 associative stimulation (cPAS) is a form of transcranial magnetic stimulation in which paired pulses
221 oups: piTBS monotherapy (n = 35), repetitive transcranial magnetic stimulation monotherapy (n = 35),
222 mpact the activity of brain regions, such as transcranial magnetic stimulation or rapid-acting antide
224 d motor-evoked potentials (MEPs) elicited by transcranial magnetic stimulation over the arm represent
226 d motor-evoked potentials (MEPs) elicited by transcranial magnetic stimulation over the leg represent
227 imed to have corticospinal volleys evoked by transcranial magnetic stimulation over the primary motor
229 nderlying plasticity induction by repetitive transcranial magnetic stimulation protocols such as inte
232 aging studies of phonological processing, or transcranial magnetic stimulation sites that did not use
233 ted by redefining the borders of each of the transcranial magnetic stimulation sites to include areas
236 nts and increased cortical excitability in a transcranial magnetic stimulation study in healthy volun
240 applying excitatory or inhibitory repetitive transcranial magnetic stimulation to a subject-specific
243 he conditional stop-signal task, and applied transcranial magnetic stimulation to the motor cortex, t
244 pre-SMA is a potential target for repetitive transcranial magnetic stimulation treatment in OCD, thes
245 y and related differentially to a repetitive transcranial magnetic stimulation treatment outcome.
246 g changes in motor-cortical excitability via transcranial magnetic stimulation up to 2 h after stimul
250 we measured motor cortical excitability with transcranial magnetic stimulation while female and male
251 tency of motor-evoked potentials elicited by transcranial magnetic stimulation with an anterior-poste
252 ency of motor-evoked potentials generated by transcranial magnetic stimulation with an AP orientation
253 combining inhibitory continuous theta-burst transcranial magnetic stimulation with model-based funct
254 peripheral nerve electrical stimulation and transcranial magnetic stimulation) combined with electro
255 rd protocol is an updated form of repetitive transcranial magnetic stimulation, and it is an effectiv
257 s should use individualized therapies (e.g., transcranial magnetic stimulation, intracerebral stem/pr
258 c options such as electroconvulsive therapy, transcranial magnetic stimulation, ketamine infusions, a
260 use of brain stimulation techniques such as transcranial magnetic stimulation, the therapeutic effic
263 sing a motoric incentive motivation task and transcranial magnetic stimulation, we studied the motor
264 lly compensate for the contribution that the transcranial magnetic stimulation-guided regions make to
266 ween those with and without damage to these 'transcranial magnetic stimulation-guided' regions remain
277 ose using region-specific therapies, such as transcranial magnetic stimulation.SIGNIFICANCE STATEMENT
279 te that relatively low power continuous wave transcranial MRgFUS in conjunction with 5-ALA can produc
280 o optimize the ultrasound parameters of SDT, transcranial MRI-guided focused ultrasound (MRgFUS) and
281 B) disruption using a low-frequency clinical transcranial MRI-guided focused ultrasound (TcMRgFUS) de
283 rons located in deep mouse brain regions via transcranial optical stimulation and elicit behavioral c
285 e potent channelrhodopsin ChRmine to achieve transcranial photoactivation of defined neural circuits,
286 als in mice following exposure to a train of transcranial pulses above normal clinical parameters.
287 at just 10 d of visual training coupled with transcranial random noise stimulation (tRNS) over visual
290 Plasticity-induction following theta burst transcranial stimulation (TBS) varies considerably acros
291 de first time evidence that slow oscillatory transcranial stimulation amplifies the functional cross-
292 es sensory adaptation.SIGNIFICANCE STATEMENT Transcranial stimulation has been claimed to improve per
296 to ascertain if a novel operator-independent transcranial ultrasound device delivering low-power high
297 are useful for describing the upper limit of transcranial ultrasound protocols, and the neurological