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1 d and cerebral blood flow was estimated with transcranial Doppler.
2 Preoperative embolization was recorded by transcranial Doppler.
3 measured in the middle cerebral artery using transcranial Doppler.
4 d significant right-to-left shunt defined by transcranial Doppler.
5 stroke risk in patients deemed high risk by transcranial Doppler.
6 measured in the middle cerebral artery using transcranial Doppler.
7 these fields to elucidate the exact role for transcranial Doppler.
8 disease, particularly the prognostic use of transcranial Doppler, (2) oral contraceptives as a strok
9 f MR angiography was contraindicated, and by transcranial Doppler and carotid ultrasound if CT angiog
13 n the middle cerebral artery was measured by transcranial Doppler at 12, 24, 36, 48, 60, 72, 84, 96,
14 ive of nine with delayed stroke had positive transcranial Dopplers (at least one microembolus detecte
15 ty (FVsv), and methods derived from arterial transcranial Doppler (aTCD) on the middle cerebral arter
16 ients with acute ischemic stroke, continuous transcranial Doppler augments t-PA-induced arterial reca
18 lated vertebral artery injuries had positive transcranial Dopplers before stroke, and positive transc
19 ers (at least one microembolus detected with transcranial Dopplers) before stroke, compared with 46 o
20 ma Score (beta = -0.201, P = 0.005) and peak transcranial Doppler (beta = 0.169, P = 0.016) explained
24 of blood transfusions, hydroxyurea therapy, transcranial Doppler-confirmed cerebral vasculopathy), g
25 n injury were the pressure reactivity index, transcranial Doppler-derived mean velocity index based o
27 cardiopulmonary exercise tests with contrast transcranial Doppler, esophageal, and gastric balloon ca
30 han 24-hour time frame provides a window for transcranial Doppler examinations and therapeutic interv
32 penicillin up to the age of 5 years, annual transcranial Doppler examinations from the ages of 2 to
37 surement of cerebral blood velocity (CBV) by transcranial Doppler has been used to identify patients
39 Although there are several settings in which transcranial Doppler has well established usefulness, th
40 e cerebral artery blood flow velocity (MCAv; transcranial Doppler), heart rate (ECG), blood pressure
43 oplethysmographic mean arterial pressure and transcranial Doppler middle cerebral artery blood flow v
44 Early recanalization (ER) was identified by transcranial Doppler monitoring during the first 2 hours
46 words including "cerebral autoregulation," "transcranial Doppler," "near-infrared spectroscopy," and
47 the microembolic signals (MESs) detected by transcranial Doppler on the use of different ablation te
48 I, 1.01-1.05) and with persistently positive transcranial Dopplers over multiple days (risk ratio, 16
49 without additional vessel injuries, positive transcranial Dopplers predicted stroke after adjusting f
54 sion therapy, for SCD patients with abnormal transcranial Doppler results, there is appropriate anxie
57 dren with sickle cell anemia, routine use of transcranial Doppler screening, coupled with regular blo
59 modynamic components of NVC were measured by transcranial Doppler sonography (TCD) during cognitive s
60 iddle cerebral artery (MCAv) was obtained by transcranial Doppler sonography and arterial pressure in
61 iddle cerebral artery (MCAv) was obtained by transcranial Doppler sonography and arterial pressure in
62 2 neither cerebral vasospasm prophylaxis nor transcranial doppler sonography is performed in 11 cente
63 velocity was measured in 6 patients through transcranial Doppler sonography of the middle cerebral a
64 aterality assessment using coarse functional transcranial Doppler sonography should be interpreted wi
66 erebral angiography, perfusion scintigraphy, transcranial Doppler sonography, CT angiography and MR a
69 history of stroke had to have undergone two transcranial Doppler studies that showed that the time-a
70 icroembolic signals to the brain detected by transcranial Doppler study and can predict the developme
71 ents (55 children) for indications including transcranial Doppler (TCD) abnormalities, AIS, or previo
73 on of asymptomatic embolic signals by use of transcranial doppler (TCD) could predict stroke risk in
75 ing/magnetic resonance angiography (MRA) and transcranial Doppler (TCD) exams were performed at entry
76 sickle cell anemia (SCA), but its effects on transcranial Doppler (TCD) flow velocities remain undefi
77 r children with sickle cell anaemia and high transcranial doppler (TCD) flow velocities, regular bloo
78 , computed tomographic angiography (CTA) and transcranial Doppler (TCD) have been advocated as altern
84 ic attack has not been compared with that of transcranial Doppler (TCD) using a comprehensive meta-an
85 children with sickle cell anemia (SCA), high transcranial Doppler (TCD) velocities are associated wit
86 sickle cell anemia (SCA), predicted by high transcranial Doppler (TCD) velocities, is prevented by t
88 oke prevention in children with SCA and high transcranial Doppler (TCD) velocities; after at least a
90 ng, white matter hyperintensities (WMHs) and transcranial doppler (TCD) were used as control conventi
100 ransesophageal echocardiography and contrast transcranial doppler to detect patent foramen ovale.
102 studies with transthoracic echocardiography/transcranial Doppler +/- transesophageal echocardiograph
103 investigated this proposal using functional transcranial Doppler ultrasonography (fTCD), which asses
104 tion Trial has confirmed that utilization of transcranial Doppler ultrasonography (TCD), which examin
105 andomly assigned to receive continuous 2-MHz transcranial Doppler ultrasonography (the target group)
106 ocardiogram with second harmonic imaging and transcranial Doppler ultrasonography during a standardiz
108 6 months over the following 18 months using transcranial Doppler ultrasonography in 144 patients wit
109 ic polysomnography for sleep apnea underwent transcranial Doppler ultrasonography of the middle cereb
110 t was complete recanalization as assessed by transcranial Doppler ultrasonography or dramatic clinica
111 T (including abdominal, thoracic, pubic, and transcranial Doppler ultrasonography scan), systematic c
113 imary stroke prevention has occurred through transcranial Doppler ultrasonography screening, but util
115 iddle cerebral arteries were insonated using transcranial Doppler ultrasonography to calculate mean m
120 ess contraindicated, and 82% underwent daily transcranial Doppler ultrasonography with embolic monito
121 rtery mean blood flow velocity (MCAVm) using transcranial Doppler ultrasonography, and expressed resp
122 li burden, assessed noninvasively by bedside transcranial Doppler ultrasonography, correlates with ri
124 markers, urine osmolality, neurodevelopment, transcranial Doppler ultrasonography, growth, and mutage
125 amentarium in Parkinson's disease, including transcranial Doppler ultrasonography, radiolabeled trace
133 s using magnetic resonance imaging (n = 26), transcranial Doppler ultrasound (n = 35) and perfusion c
135 atic microembolic signals (MES), detected by transcranial Doppler ultrasound (TCD), are markers of fu
136 ith a neurologic exam, complete blood count, transcranial Doppler ultrasound (TCD), measurement of in
137 od flow velocities (CBFV) were measured with transcranial Doppler ultrasound along with noninvasive b
138 to determine sensitivity and specificity of transcranial Doppler ultrasound and cerebral angiography
139 he middle cerebral artery was measured using transcranial Doppler ultrasound and postural control (i.
142 bolic signals can be detected in patients by transcranial Doppler ultrasound despite aspirin and hepa
154 ars) receiving chronic hemodialysis, we used transcranial Doppler ultrasound to measure cerebral arte
160 internal carotid and vertebral arteries, and transcranial Doppler ultrasound was used to assess middl
162 vessels, the sensitivity and specificity of transcranial Doppler ultrasound were middle cerebral art
165 ings of MAP (Finometer), CBF velocity (CBFV; transcranial Doppler ultrasound), end-tidal CO(2) (capno
166 omatic circulating emboli can be detected by transcranial Doppler ultrasound, are frequent in patient
167 al artery velocity (MCAV) was measured using transcranial Doppler ultrasound, as an index of CBF, in
170 rate of all ancillary studies was 4% (5% of transcranial Doppler ultrasounds, 4% of nuclear studies,
174 sis, results of seven of the 26 preoperative transcranial Doppler US studies were abnormal, and all o
176 en were examined with nonimaging and imaging transcranial Doppler US techniques on the same day, for
179 g, 47 (24%) of 196 participants had elevated transcranial Doppler velocities (43 [22%] conditional, f
180 maximum tolerated dose significantly lowers transcranial Doppler velocities and reduces primary stro
181 years with sickle cell anaemia with abnormal transcranial Doppler velocities conducted at three teach
183 ildren with sickle cell anaemia and abnormal transcranial Doppler velocities results in a 92% relativ
186 splant indications included stroke (n = 12), transcranial Doppler velocity >200 cm/s (n = 2), >/=3 va
189 el and reduced risk of albuminuria, abnormal transcranial Doppler velocity, and stroke (absolute valu
190 intracranial pressure, mean cerebral artery transcranial Doppler velocity, PaCO2, cerebral perfusion
191 g optic nerve sheath diameter (ONSD), venous transcranial Doppler (vTCD) of straight sinus systolic f
192 e, and asymptomatic) indicated that abnormal transcranial Doppler was more common in the seizure (4/6
194 cranial Dopplers before stroke, and positive transcranial Dopplers were not associated with delayed s
195 learning difficulty, headaches, or abnormal transcranial Doppler), who also underwent bolus-tracking
196 ur shaft fracture with RLS evaluation, daily transcranial Doppler with embolus detection studies, and