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1 ion mutations in skin barrier genes increase transepidermal water loss.
2 Skin barrier function was measured by transepidermal water loss.
3 lated with the SC water content but not with transepidermal water loss.
4 n barrier function as evidenced by increased transepidermal water loss.
5 robial activity, paracellular diffusion, and transepidermal water loss.
6 ells in blood, correlating with severity and transepidermal water loss.
7 primary cell population responsible for high transepidermal water loss.
8 oglobulin levels, skin symptom scores, or on transepidermal water loss.
9 id envelope and die shortly after birth from transepidermal water loss.
10 icle-treated animals), assessed as increased transepidermal water loss.
11 tures improve barrier repair, as assessed by transepidermal water loss.
16 unction, as measured by corneometry, pH, and transepidermal water loss also normalized with treatment
19 STS proteins was highly correlative to skin transepidermal water loss and allergic sensitization.
20 rier development, evidenced by a decrease in transepidermal water loss and an enhanced outside-in bar
26 interactions with the TGM1 genotype included transepidermal water loss and emollient and retinoid use
27 of the epidermis associated with a decreased transepidermal water loss and increased proinflammatory
28 oms, accompanied by significant decreases in transepidermal water loss and increases in water content
31 to barrier disruption, indicated by elevated transepidermal water loss and reduced lipid synthesis en
32 S1pr2(-/-) mouse showed significantly higher transepidermal water loss and required another 24 hours
33 was to investigate the relationship between transepidermal water loss and skin permeability to triti
34 certained using confocal Raman spectroscopy; transepidermal water loss and skin surface pH were measu
35 more prematurely have elevated rates of both transepidermal water loss and transcutaneous heat loss,
36 on of the barrier function of skin increases transepidermal water loss and up-regulates inflammatory
38 KLK5 activity in TgKLK5 mouse skin, reduced transepidermal water loss, and decreased proinflammatory
39 l epidermis, corneocyte fragility, increased transepidermal water loss, and local inflammation in the
40 measurements of percentage of skin redness, transepidermal water loss, and participant-assessed pain
41 permeability indicated by higher pH, greater transepidermal water loss, and reduced lipid synthesis e
44 phils, cutaneous expression of Il4 and Il13, transepidermal water loss, antigen-specific IgE antibody
45 44, P = .0006) and skin barrier dysfunction (transepidermal water loss area under the curve r = 0.31,
46 children had higher lesional and nonlesional transepidermal water loss (both P < .001) as well as dec
49 ximately 0.5 U); (ii) enhanced SC integrity (transepidermal water loss change with sequential tape st
50 ing IL-13Ralpha2 had significantly increased transepidermal water loss, cutaneous inflammation, perip
51 mal permeability barrier defects with severe transepidermal water loss, decreased intercellular lipid
53 d in Ric(EKO) mice, as revealed by increased transepidermal water loss, enhanced corneocyte fragility
55 lents in SCID/NOD mice demonstrated enhanced transepidermal water loss following s.c. administration
56 fetal rats displayed no measurable barrier (transepidermal water loss > 10 mg per cm2 per h), a meas
58 on, and cytokine production were measured by transepidermal water loss, histopathology, molecular bio
60 m, with SerpinB2(-/-) mice showing increased transepidermal water loss, increased overt loss of strat
61 hat cutaneous barrier formation, measured as transepidermal water loss, is delayed in male fetal rats
62 active plaque phenotypes displayed elevated transepidermal water loss levels, increased numbers of e
64 ut allergy (PA) is associated with increased transepidermal water loss; low urocanic acid (UCA) and p
65 ut allergy (PA) is associated with increased transepidermal water loss; low urocanic acid (UCA) and p
66 se curve, with maximal sweating (measured as transepidermal water loss) (mean 70 g m(-2) hour(-1)) af
69 sebopsoriasis displayed a lesser increase in transepidermal water loss, normal numbers of lamellar bo
70 lg(-/-) neonates showed little alteration in transepidermal water loss or lipid- or corneocyte-relate
73 present in these dogs, as well as increased transepidermal water loss, particularly in sites charact
74 tic administration of all activators tested (transepidermal water loss range 4.0-8.5 mg per cm2 per h
76 Similarly, there was no correlation between transepidermal water loss rates and the 3H2O permeabilit
81 exercise, on several dermatologic measures: transepidermal water loss, recovery of skin barrier func
82 re), Scoring for Atopic Dermatitis (SCORAD), transepidermal water loss, skin filaggrin (FLG) expressi
83 e markers also significantly correlated with transepidermal water loss, suggesting a link between the
84 r associations with skin filaggrin (FLG) and transepidermal water loss (TEWL) (assesses skin barrier
87 ipid barrier as measured by skin resistance, transepidermal water loss (TEWL) and Fourier transform i
90 determine whether eczema, dry skin, and high transepidermal water loss (TEWL) at 3 months were associ
91 ering techniques of laser Doppler imaging, a transepidermal water loss (TEWL) device and a skin therm
92 We have evaluated the content of lipids and transepidermal water loss (TEWL) in lesional and non-les
93 his methylation is associated with increased transepidermal water loss (TEWL) in risk allele carriers
95 ough parameters like skin hydration (SH) and transepidermal water loss (TEWL) is vital for diagnosing
96 tional Endpoints (BASELINE) birth cohort had transepidermal water loss (TEWL) measured in the early n
98 by electric impedance spectroscopy (EIS) and transepidermal water loss (TEWL) measurements after the
99 ing test (LAST):'stinger' and 'non-stinger'; transepidermal water loss (TEWL) measurements; and sensi
101 healing endpoint and recurrence by measuring transepidermal water loss (TEWL) post-closure at the sit
102 rthermore, to determine whether increases in transepidermal water loss (TEWL) predate the development
104 were applied to the upper arm; impedance and transepidermal water loss (TEWL) were measured at baseli
107 of the skin and measurements of the rate of transepidermal water loss (TEWL) were recorded sequentia
109 function, impaired skin barrier function by transepidermal water loss (TEWL), eczema, and filaggrin
111 rneum was assessed by measuring capacitance, transepidermal water loss (TEWL), rates of absorption-de
112 sure in wild-type and Serpinb3a-null mice on transepidermal water loss (TEWL), sensitization, and inf
117 es (body surface area/BSA, pruritus ADQ, and transepidermal water loss/TEWL) with immune and barrier
118 by reduced transglutaminase (TGM) activity, transepidermal water loss, up-regulation of the proinfla
119 orneocytes and its contribution to increased transepidermal water loss was confirmed by tape strippin
121 od samples were assessed for hormone levels, transepidermal water loss was measured to assess skin ba
125 nce analysis to measure hydration status and transepidermal water loss, we show that the epidermal ba
126 ith severe dehydration, suggesting excessive transepidermal water loss, which was confirmed by in vit
127 y normal, and exhibited an increase in basal transepidermal water loss without alteration in basal mi