ライフサイエンスコーパス: transferrin saturation

1 ygous for TFR2 mutations, all with increased transferrin saturation.

2 pathogens was strongly associated with serum transferrin saturation.

3 -off value was used to define elevated serum transferrin saturation.

4 re correlated positively with maternal serum transferrin saturation.

5 M significantly increased serum ferritin and transferrin saturation.

6 um ferritin less than 30 ng/mL regardless of transferrin saturation.

7 disorder was associated with serum iron and transferrin saturation.

8 defined as low ferritin, high TIBC, and low transferrin saturation.

9 oncentrations for iron, ferritin and percent transferrin saturation.

10 ron levels, total iron-binding capacity, and transferrin saturation.

11 els, and serum iron correlated strongly with transferrin saturation.

12 higher ferritin/transferrin levels and lower transferrin saturation.

13 eived iron depletion therapy; all had normal transferrin saturation.

14 epcidin, accompanied by a major reduction in transferrin saturation.

15 rum ferritin and normal to slightly elevated transferrin saturation.

16 ations were proportional to the increment in transferrin saturation.

17 245X) mutant mice also demonstrated elevated transferrin saturations.

18 for serum iron (0.30, 95% CI 0.09, 0.51) and transferrin saturation (0.34, 95% CI 0.15, 0.54), but lo

19 1.83 (95% CI: 1.21, 2.76; P < 0.01); HR for transferrin saturation: 1.68 (95% CI: 1.18, 2.38; P < 0.

20 : 2.45 (95% CI:1.12, 5.34; P < 0.05); HR for transferrin saturation: 1.90 (95% CI:1.02, 3.56; P < 0.0

21 (CKD) stages 3-5, hemoglobin 8.5-10.0 g/dl, transferrin saturation 15% or more, and ferritin 50 ng/m

22 rline low ferritin level (40 ng/mL) with low transferrin saturation (2%); 86 participants (26.5%) inc

23 ntrol (ferritin=628+/-367 ng/ml [mean+/-SD]; transferrin saturation=30%+/-12%; P<0.001 for both).

24 eters (ferritin=899+/-488 ng/ml [mean+/-SD]; transferrin saturation=39%+/-17%) versus subjects on act

25 t alcohol use, viral hepatitis, or increased transferrin saturation, 4,568 participants with NAFLD we

26 l/L during the first 6-24 h after birth) and transferrin saturation (50.2 +/- 16.7% to 14.4 +/- 6.1%)

27 otes with normal levels of both ferritin and transferrin saturation (6.49, 2.09 to 20.18).

28 itin (304 ng/mL; 95% CI, 217-391; P<0.0001), transferrin saturation (6.8%; 95% CI, 2.7-10.8; P=0.002)

29 r iron (29.1% vs. 34.5 umol/L; P < 0.05) and transferrin saturation (60.9% vs. 79.1%; P < 0.01), but

30 pared with HFE+/+ littermates, with elevated transferrin saturations (68.4% vs. 49.8%) and elevated l

31 high-sensitivity C-reactive peptide), higher transferrin saturation (a marker of iron stores), and th

32 tivation of the Smad1/5/8 signaling pathway, transferrin saturation activates the downstream Smad1/5/

33 s and reducing serum iron concentrations and transferrin saturation (all P < 0.01).

34 ate with increased serum iron, ferritin, and transferrin saturation and decreased transferrin levels,

35 nostically categorized based on quartiles of transferrin saturation and ferritin as "Iron Replete" (2

36 cular volume increased after conversion, but transferrin saturation and ferritin did not change.

37 Screening for transferrin saturation and ferritin levels does not dete

38 ipants with HFE mutations, the average serum transferrin saturation and ferritin levels were slightly

39 A (average 7.7-fold), despite their elevated transferrin saturation and hepatic iron content.

40 normal ALT levels (P < 0.05) included higher transferrin saturation and iron and selenium concentrati

41 fe(-/-)) significantly decreased serum iron, transferrin saturation and liver iron accumulation.

42 We demonstrated that both transferrin saturation and liver iron content independen

43 erythropoiesis, and significantly decreased transferrin saturation and lung iron stores after 2 week

44 /fl);Cre(+) mice exhibited approximately 90% transferrin saturation and massive liver iron overload,

45 C326Y, which can be associated with greater transferrin saturation and more prominent iron depositio

46 us including iron, ferritin, transferrin and transferrin saturation and serum lipid profile on a rout

47 The balance between serum transferrin saturation and serum transferrin-receptor co

48 he latter category of FPN mutation have high transferrin saturation and tend to deposit iron througho

49 roots of the proportion with the lowest mean transferrin saturation and the proportion with the highe

50 findings were supported in the analyses for transferrin saturation and total iron binding capacity,

51 emochromatosis C282Y homozygotes with normal transferrin saturation and/or ferritin, not recommended

52 rom all three strains demonstrated increased transferrin saturations and liver iron concentrations co

53 Single measurements of serum transferrin saturations and serum ferritin levels.

54 associated with increases in iron transport (transferrin saturation) and stores (serum ferritin) amon

55 0.08 (0.09) for serum iron, 0.08 (0.07) for transferrin saturation, and - 0.09 (- 0.17) for ferritin

56 refore, we tested whether high and low iron, transferrin saturation, and ferritin are associated with

57 um iron, total iron binding capacity (TIBC), transferrin saturation, and ferritin in a Hispanic/Latin

58 When studied according to levels of iron, transferrin saturation, and ferritin in a single blood s

59 We measured serum iron, transferrin saturation, and ferritin in all heterozygote

60 Iron, transferrin saturation, and ferritin were measured in 13

61 Plasma TAS, serum ferritin, serum iron, transferrin saturation, and hemoglobin were measured at

62 study-wise type 1 error for serum ferritin, transferrin saturation, and intravenous iron and erythro

63 haracterized by high serum ferritin, reduced transferrin saturation, and macrophage iron loading.

64 er operator curve analysis of CHr, ferritin, transferrin saturation, and MCV demonstrates that CHr ha

65 es of iron status (including serum ferritin, transferrin saturation, and non-transferrin-bound iron)

66 rtal distribution and increased plasma iron, transferrin saturation, and non-transferrin-bound iron,

67 atory tests, including complete blood count, transferrin saturation, and other chemistries; serum fer

68 sed serum iron, total iron binding capacity, transferrin saturation, and serum albumin.

69 ical data, including hemoglobin, serum iron, transferrin saturation, and serum ferritin concentration

70 t group had significantly higher hemoglobin, transferrin saturation, and serum ferritin values and a

71 ded determination of hemoglobin, hematocrit, transferrin saturation, and serum ferritin values.

72 >/=2 of 3 tests (erythrocyte protoporphyrin, transferrin saturation, and serum ferritin) and a low he

73 complex significantly increased hemoglobin, transferrin saturation, and serum ferritin, and it signi

74 sed indicators, such as serum ferritin (SF), transferrin saturation, and soluble transferrin receptor

75 Ferritin and/or transferrin saturation are required for diagnosis and sc

76 uration subpopulation and support the use of transferrin saturation as an inexpensive screening test

77 testinal (59)Fe uptake, lower serum iron and transferrin saturation, but no change in liver non-heme

78 yed the decrease in hepcidin until after the transferrin saturation, but not the ferritin concentrati

79 can children when defined using ferritin and transferrin saturation, but not when defined by hepcidin

80 ion (by 48.4%; 95% CI: 15.4%, 91.0%), higher transferrin saturation (by 52.3%; 95% CI: 17.9%, 96.7%),

81 Increased transferrin saturation correlated positively with the se

82 low iron levels (cutoff 10.5 mumol/mL), low transferrin saturation (cutoff 55%), and high serum tran

83 Statistical mixture modeling was applied to transferrin saturation data for African Americans and Ca

84 In previous analyses of transferrin saturation data in African Americans and Cau

85 days 2, 3), serum iron (depressed days 2-4), transferrin saturation (depressed days 2-4), and retinol

86 airs, we measured ferritin, transferrin, and transferrin saturation during early pregnancy.

87 arrow iron depletion in patients with normal transferrin saturations, elevated serum ferritins, eleva

88 Diet did not affect hemoglobin, transferrin saturation, erythrocyte protoporphyrin, or s

89 no significant difference between groups in transferrin saturation, erythrocyte zinc protoporphyrin

90 ity (F ratio 12.8, P < 0.0001) and 25% lower transferrin saturation (F ratio 4.3, P < 0.0001) compare

91 At initial diagnosis, fasting transferrin saturation, ferritin level, routine chemistr

92 creases in serum iron levels, percentages of transferrin saturation, ferritin levels, elevations in h

93 Transferrin saturation, ferritin, and hepcidin were redu

94 agnosis of COVID-19 and measured serum iron, transferrin saturation, ferritin, hepcidin and serum cat

95 3 +/- 5.6 mumol/L (P < 0.0001) and increased transferrin saturation from 35.7 +/- 16.3% to 45.4 +/- 1

96 ID) diet for 24 hours, the rapid decrease of transferrin saturation from 71% to 24% (control vs ID di

97 ron levels, total iron-binding capacity, and transferrin saturation from baseline to week 12 ( P < 0.

98 ted with high LIC, as well as an increase of transferrin saturation > 45% (p < 0.001).

99 versus 37.5% of compound heterozygotes with transferrin saturation > 50%; p = 0.003), serum ferritin

100 on, serum ferritin levels of 300-2000 ng/mL, transferrin saturation >/= 45%, and no known history of

101 300 mug/L in men and >200 mug/L in women and transferrin saturation >45% in women and 50% in men.

102 L (OR 5.53, CI95 [1.43-21.42]) and increased transferrin saturation >= 45% (OR 2.63, CI95 [1.32-5.23]

103 in this pilot study, ferritin >= 600 ug/L, transferrin saturation >= 45% and a family history of hy

104 tely 11% to 22% of those with elevated serum transferrin saturation had concurrently elevated serum f

105 a whole, these subpopulations of increasing transferrin saturations had progressively increasing mea

106 Hepcidin, serum iron, transferrin, transferrin saturation, haptoglobin, c-reactive protein,

107 hromatosis screening program that uses serum transferrin saturation has been proposed, but few data e

108 hich has high levels of hemoglobin, iron and transferrin saturation, has hitherto been used as a prox

109 s increased in C282Y homozygotes with normal transferrin saturation (hazard ratio 2.00, 95% CI 1.04 t

110 inflammation, FeM-1269 increases serum iron, transferrin saturation, hemoglobin and hematocrit.

111 ther with serum ferritin, serum transferrin, transferrin saturation, hemoglobin, and alanine aminotra

112 n, soluble transferrin receptor, serum iron, transferrin saturation, hemoglobin, hematocrit) in males

113 symptomatic Australians to estimate the mean transferrin saturation in hemochromatosis heterozygotes

114 aims of this study were to estimate the mean transferrin saturation in hemochromatosis heterozygotes

115 The prevalence of elevated serum transferrin saturation in non-Hispanic black persons and

116 The prevalence of elevated serum transferrin saturation in persons 20 to 49 years of age

117 with Kupffer-cell iron deposition and normal transferrin saturation in vivo, whereas patients with th

118 that was seen at both high and low levels of transferrin saturation in white women should be confirme

119 To determine if transferrin saturations in African Americans may reflect

120 addition, only after the older transfusion, transferrin saturation increased progressively over 4 ho

121 groups initially had 30% declines in plasma transferrin saturation, increases in plasma ferritin con

122 iduals without inflammatory conditions or by transferrin saturation (iron/total iron binding capacity

123 icantly, but it had no discernible effect on transferrin saturation, iron-deficient erythropoiesis, h

124 To test the hypothesis that relatively high transferrin saturation is associated with increased stro

125 An elevated transferrin saturation is the earliest phenotypic abnorm

126 aditional population screening measuring the transferrin saturation is unlikely to be cost-effective

127 iency definitions were studied: (1) combined transferrin saturation less than 10% and serum ferritin

128 eft ventricular ejection fraction <=45%) and transferrin saturation less than 20% or serum ferritin l

129 in the overall cohort or in patients with a transferrin saturation less than 20%, or reduce the tota

130 ritin greater than or equal to 30 ng/mL with transferrin saturation less than 20%.

131 t failure hospitalization in patients with a transferrin saturation less than 20%.

132 nked to clinically relevant indices, such as transferrin saturation level.

133 iron concentrations (+31.3%, P = 0.027) and transferrin saturation levels (+28.4%, P = 0.009) increa

134 males showed differences across genotypes in transferrin saturation levels (100% of homozygotes versu

135 e marrow iron stores are absent, even though transferrin saturation levels are normal.

136 The increase of transferrin saturation levels persisted after 57 d of HD

137 ablished primary hematopoietic cultures with transferrin saturation levels that restricted erythropoi

138 Consistent with this, iron, ferritin, and transferrin saturation levels were reduced and red cell

139 Initial transferrin-saturation levels exceeding the threshold as

140 patients (serum ferritin < 100 mug/L and/or transferrin saturation < 20%) with chronic kidney diseas

141 n < 100 ng/mL or ferritin 100-299 ng/mL with transferrin saturation < 20%).

142 rations of < 100 ng/mL or 100-299 ng/mL with transferrin saturation < 20%.

143 erum ferritin at a level of < 150 ng/mL, and transferrin saturation < 21%.

144 nsferrin receptors > or = 8.4 microg/mL, and transferrin saturation < or = 13.2%).

145 contractile performance and explaining why a transferrin saturation < ~15%-16% predicts the ability o

146 erritin <100 mug/L (75.4% versus 68.1%), and transferrin saturation <20% (87.9% versus 81.4%).

147 defined as a ferritin level <100 ng/mL or a transferrin saturation <20% and a ferritin level 100 to

148 tricular ejection fraction <=45%, and either transferrin saturation <20% or serum ferritin <100 mug/L

149 t failure hospitalization in patients with a transferrin saturation <20%) occurred in 103 patients in

150 <100 ng/mL or ferritin 100-300 ng/mL with a transferrin saturation <20%), and documented HF hospital

151 s ferritin <100 mug/L, or 100-299 mug/L with transferrin saturation <20%), and had a left ventricular

152 tration between 100 and 300 mug/L along with transferrin saturation <20%), and IDA through laboratory

153 cy (ferritin<100 mug/L or 100-300 mug/L with transferrin saturation <20%).

154 o 30 ml/min, 46% of women and 19% of men had transferrin saturation <20%, and 47% of women and 44% of

155 <100 ng/ml or ferritin 100 to 300 ng/ml with transferrin saturation <20%.

156 Transferrin saturation <21% was also associated with inc

157 ia) and ND-CKD (serum ferritin < 200 ug/L or transferrin saturation <= 20% and serum ferritin 200-299

158 itin <or= 450 pmol/L or <or= 675 pmol/L with transferrin saturation <or= 19%) receiving subcutaneousl

159 rin receptor >or=8.4 or >10.0 microg/mL, and transferrin saturation <or=13.2% or <10.0%, respectively

160 Low transferrin saturation (<10%) was similarly associated w

161 me effect: +10.7 +/- 9.6 mug/L, P < 0.0001), transferrin saturation (main time effect: +5.1% +/- 18.7

162 ron measures, including serum ferritin (SF), transferrin saturation, mean cell volume, and hemoglobin

163 icant difference in baseline serum ferritin, transferrin saturation, mean cell volume, mean cell hemo

164 a for iron overload using serum ferritin and transferrin saturation measurements and HFE genotyping.

165 randomized in DAPA-HF, 3009 had ferritin and transferrin saturation measurements available at baselin

166 A profound reduction in transferrin saturation occurred within the first 12 h of

167 ciated positively with increasing deciles of transferrin saturation (odds ratio [OR] per decile, 1.10

168 In practice, a fasting transferrin saturation of > or = 45% identifies virtuall

169 Participants with a transferrin saturation of >55% or >45% and an elevated s

170 When erythropoietin resistance is present, transferrin saturation of < 27% or serum ferritin < 300

171 tin concentration of <200 mug per liter or a transferrin saturation of <20% being a trigger for iron

172 .2% of the truncated sample had a lower mean transferrin saturation of 24.1%, whereas 11.8% had an in

173 f 24.1%, whereas 11.8% had an increased mean transferrin saturation of 37.3%.

174 Patients with a serum transferrin saturation of 45% or more on initial testing

175 A transferrin saturation of 50% had a sensitivity of only

176 Those who had a fasting serum transferrin saturation of 55% or more and a serum ferrit

177 hemochromatosis; and 23 patients had a serum transferrin saturation of 55% or more with no identifiab

178 opulation with a mean +/- standard deviation transferrin saturation of 63.4% +/- 5.7% (postulated hom

179 ation, and 65 of the 66 patients (98%) had a transferrin saturation of at least 45%.

180 erum ferritin level of 101 to 299 ng/mL with transferrin saturation of less than 20%.

181 t erythropoiesis as judged by the NKF-K/DOQI transferrin saturation or serum ferritin targets.

182 Higher transferrin saturation or serum iron concentrations were

183 a high-iron diet revealed no differences in transferrin saturation or tissue iron stores between WT

184 =2 of 3 abnormal concentrations in ferritin, transferrin saturation, or erythrocyte protoporphyrin (t

185 ion in hematocrit value, reticulocyte count, transferrin saturation, or ferritin level in the experim

186 n C282Y homozygotes with normal plasma iron, transferrin saturation, or ferritin, and in C282Y homozy

187 Even C282Y homozygotes with normal iron, transferrin saturation, or ferritin, not currently recom

188 in individuals with low or high plasma iron, transferrin saturation, or ferritin.

189 h levels of free erythrocyte protoporphyrin, transferrin saturation, or hemoglobin (percent change =

190 est did not significantly change serum iron, transferrin saturation, or hepcidin levels.

191 but little effect on hemoglobin, hematocrit, transferrin saturation, or plasma iron.

192 iding with a 50% reduction in serum iron and transferrin saturation over the 24-hour period.

193 wth rates were very strongly correlated with transferrin saturation (p < 0.0001 in all cases).

194 increases in AUC for serum iron (P < 0.01), transferrin saturation (P < 0.001), and nontransferrin-b

195 oots of the proportions with the lowest mean transferrin saturation (P = .925) and the highest (q = 0

196 tate significantly reduced tHcy (P = 0.017), transferrin saturation (P = 0.027), and ferritin (P = 0.

197 Serum transferrin saturations (percent saturation of serum tra

198 or length gain or in hemoglobin, hematocrit, transferrin saturation, plasma zinc, or erythrocyte ribo

199 who ingested 65 mg of iron, the increase in transferrin saturation preceded by hours the increase in

200 , older age, higher urea reduction ratio and transferrin saturation, prescription of intravenous iron

201 NTBPI values in TM correlated with transferrin saturation (r = .6, P = .03) but not with se

202 The prevalence of initially elevated serum transferrin saturation ranged from 1% to 6%.

203 Cut-off values used to define elevated serum transferrin saturation ranged from greater than 45% to g

204 um ferritin was higher than 21,000 ng/mL and transferrin saturation reached 102%.

205 Determination of transferrin saturation remains the most useful noninvasi

206 s more severe in the restrictive group (peak transferrin saturation: restrictive group 83.9+/-13.0%,

207 tion studies for serum iron, serum ferritin, transferrin saturation (SAT) and total iron binding capa

208 To evaluate potential transferrin saturation screening levels, modeling result

209 normal individuals and to evaluate potential transferrin saturation screening levels.

210 Serum transferrin saturation screening tests were offered to a

211 Transferrin saturation (serum iron concentration divided

212 um ferritin, total iron-binding capacity and transferrin saturation), serum hepcidin and genome-wide

213 Measures included transferrin saturation, serum ferritin level, and self-r

214 y was defined as > or =2 abnormal values for transferrin saturation, serum ferritin, and erythrocyte

215 The transferrin saturation, serum ferritin, and liver iron b

216 nces in hematocrit, mean corpuscular volume, transferrin saturation, serum ferritin, and serum iron.

217 Measurement of serum ferritin, transferrin saturation, serum soluble transferrin recept

218 emochromatosis heterozygotes form a distinct transferrin saturation subpopulation and support the use

219 of this new study was to determine if these transferrin saturation subpopulations have different lev

220 consistent with the concept that population transferrin saturation subpopulations reflect different

221 ritin concentrations were determined for the transferrin saturation subpopulations that were identifi

222 1) to encourage laboratories to provide the transferrin saturation test as part of routine laborator

223 f 45% or more on initial testing had a serum transferrin saturation test done under fasting condition

224 eening for hemochromatosis done by using the transferrin saturation test has been advocated by expert

225 screening for hemochromatosis done by using transferrin saturation testing can be recommended, labor

226 parameters of iron metabolism, particularly transferrin saturation, that reflect serum iron availabi

227 e in hematocrit, hemoglobin, serum iron, and transferrin saturation, the appearance of microcytic ani

228 serum ferritin, total-iron-binding capacity, transferrin saturation, the ratio of enzymatic to immuno

229 A transferrin saturation threshold of 45% identified 98% o

230 d evaluation of iron status (ferritin level, transferrin saturation, transferrin receptor level, reti

231 hereditary hemochromatosis (HHC) by means of transferrin saturation (TS) levels has been advocated an

232 t possible to assess the association between transferrin saturation (TS) subpopulations and HFE mutat

233 globin, mean corpuscular volume (MCV), serum transferrin saturation (TS), serum ferritin, and white b

234 end maintaining ferritin > or =100 ng/ml and transferrin saturation (TSAT) > or =20% to ensure adequa

235 45%, and either serum ferritin < 100 ug/L or transferrin saturation (TSAT) < 20% were randomized to i

236 Thirteen patients with serum transferrin saturation (TSAT) < 25% received ITDI, and 1

237 rritin levels between 500 and 1200 ng/ml and transferrin saturation (TSAT) < or = 25%.

238 erritin <100 ng/mL or, when 100-299 ng/mL, a transferrin saturation (TSAT) <20%.

239 ferritin level <100 or 100 to 299 ng/mL with transferrin saturation (TSAT) <20%.

240 08 for all), but defining iron deficiency as transferrin saturation (TSAT) <21% did.

241 r regression analyses, ferritin (B = -0.43), transferrin saturation (TSAT) (B = -0.17), hepcidin (B =

242 so observed reduced ferritin (p = 0.003) and transferrin saturation (TSAT) (p = 0.008) levels from Ph

243 ractice Patterns Study, with first available transferrin saturation (TSAT) and ferritin levels as exp

244 One-half of them (10 of 22) proposed transferrin saturation (TSAT) as an alternative or compl

245 dose, intravenous (IV) iron dose, ferritin, transferrin saturation (TSAT) concentration, parathyroid

246 Subjects on FC had increased ferritin and transferrin saturation (TSAT) levels compared with subje

247 occurs with serum ferritin >500 ng/ml and/or transferrin saturation (TSAT) of 20 to 30%.

248 unsaturated iron-binding capacity (UIBC) and transferrin saturation (TSAT) with a single allele effec

249 ce (PAD) was developed for the assessment of transferrin saturation (TSAT), which is defined as the r

250 dialysis patients with high ferritin and low transferrin saturation (TSAT).

251 e remaining 369 patients, 256 (69%) had high transferrin saturation (TSAT; 45%) and 199 (53%) had con

252 patients with CKD stage G3-4 with percentage transferrin saturation (%TSAT) <= 30% and serum ferritin

253 ferritin level was <100 mug/L, regardless of transferrin saturation [TSAT], or 100 to 299 mug/L if TS

254 ents who are responsive to erythropoietin, a transferrin saturation value < 18% or serum ferritin lev

255 atosis be defined by repeated elevated serum transferrin saturation values(with or without DNA test r

256 The mean serum iron concentrations and transferrin-saturation values were higher in heterozygot

257 ptoms of hemochromatosis or who had elevated transferrin-saturation values.

258 concentration was >700 mug per liter or the transferrin saturation was >=40%), or low-dose iron sucr

259 ency (serum ferritin level <100 ng/mL; or if transferrin saturation was <20%, a serum ferritin level

260 n value was 62 +/- 4.8 percent, and the mean transferrin saturation was 20 +/- 9 percent.

261 The median baseline transferrin saturation was 8% (range, 2-58%).

262 Transferrin saturation was also associated with a greate

263 ion and the proportion with the highest mean transferrin saturation was approximately 1.

264 However, transferrin saturation was decreased more with chromium

265 ated with overexpression of hepcidin and low transferrin saturation was found to be associated with b

266 n of less than 100 mug/L or 100-300 mug/L if transferrin saturation was less than 20% or C-reactive p

267 Transferrin saturation was lower in male stressed neonat

268 The prevalence of elevated serum transferrin saturation was lower in women than in men wh

269 matocrit was measured weekly for four weeks, transferrin saturation was measured, and coexisting illn

270 a standard diet, by 10 weeks of age, fasting transferrin saturation was significantly elevated compar

271 Transferrin saturation was significantly lower in C57BL/

272 Low transferrin saturation was similarly associated with red

273 of septic ICU subjects, low iron levels and transferrin saturation went along with a nonlethal outco

274 ancer), higher serum iron concentrations and transferrin saturation were associated with increased ri

275 ncreasing weight, urea reduction ration, and transferrin saturation were associated with lower EPO do

276 ochromatosis homozygotes, two populations of transferrin saturation were identified in asymptomatic A

277 t for diurnal variation, 3 subpopulations of transferrin saturation were identified in each racial gr

278 In summary, low and high plasma iron and transferrin saturation were independently associated wit

279 Three subpopulations based on transferrin saturation were present (P < .0001) and the

280 m baseline in hemoglobin, serum ferritin and transferrin saturation were significantly greater with F

281 Findings for transferrin saturation were similar, whereas infection r

282 Samples pooled by transferrin saturation were used to conduct ex-vivo grow

283 in, serum iron, total-iron-binding-capacity, transferrin saturation) were significantly elevated duri

284 results indicated a close correlation of low transferrin saturation with decreased hepcidin mRNA.

285 served a significant U-shaped association of transferrin saturation with risk of incident stroke (> 4

286 total-iron-binding capacity and increase in transferrin saturation (%) with resistive training are l

287 For transferrin saturation, with 6194 CHD cases in 5 studies

288 demonstrated strain-dependent differences in transferrin saturation, with the highest values in AKR m

289 ansferrin receptor (sTfR) concentrations and transferrin saturation without affecting hemoglobin conc

290 of greater than 60% to define elevated serum transferrin saturation would identify an estimated 1.4 t