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1 lecular weight peptides were cross-linked by transglutaminase.
2 sign better substrates or inhibitors of this transglutaminase.
3 which then trigger the enzymatic activity of transglutaminase.
4 e fibrin network and cross-linking by plasma transglutaminase.
5 results from the polyamination of tubulin by transglutaminase.
6 that neuronal tubulin can be polyaminated by transglutaminase.
7 odification method involving thermolysin and transglutaminase.
8  followed by repolymerization with microbial transglutaminase.
9 l differences making FXIII unique from other transglutaminases.
10 n of antibodies against epidermal and tissue transglutaminases.
11 b, -2f, and -2g proteins), the cross-linking transglutaminase 1 enzyme (Tg-1) and Muc5AC mRNA transcr
12 ucocerebrosidase, acid sphingomyelinase, and transglutaminase 1.
13 ldren tested positive for IgA against tissue transglutaminase (1.0%; 95% confidence interval, 0.4%-1.
14 into the pathophysiology of TGM1 ichthyoses, transglutaminase-1 enzymatic activity, and potential the
15 sive, temperature-sensitive mutations in the transglutaminase-1 gene (TGM1).
16 , and anti-IgA autoantibodies against tissue transglutaminase (12%).
17  celiac disease or antibodies against tissue transglutaminase 2 (controls), healthy family members of
18 to both human Factor XIII (FXIII) and tissue transglutaminase 2 (hTG2).
19 isease (CD), a condition in which antitissue transglutaminase 2 (TG2) Abs are suspected to contribute
20 evious work in our laboratory has shown that transglutaminase 2 (TG2) acting as a coreceptor for inte
21 s of total IgA antibodies, IgA antibodies to transglutaminase 2 (TG2) and endomysium, as well as IgA
22           The interaction between the enzyme transglutaminase 2 (TG2) and fibronectin (FN) is involve
23 um domain of the fibronectin binding site on transglutaminase 2 (TG2) and its importance in mediating
24                Antibodies to the autoantigen transglutaminase 2 (TG2) are a hallmark of celiac diseas
25       Autoantibodies specific for the enzyme transglutaminase 2 (TG2) are a hallmark of the gluten-se
26 A plasma cells (PCs) specific for the enzyme transglutaminase 2 (TG2) are abundant in the small intes
27                            Autoantibodies to transglutaminase 2 (TG2) are hallmarks of celiac disease
28     We and others have previously identified transglutaminase 2 (TG2) as a participant in adverse fib
29                                              Transglutaminase 2 (TG2) catalyzes transamidation or dea
30                                              Transglutaminase 2 (TG2) expression is required for epid
31                                       Tissue transglutaminase 2 (TG2) has been of particular interest
32                        Molecular deletion of transglutaminase 2 (TG2) has been shown to improve funct
33 the autoantibody response against the enzyme transglutaminase 2 (TG2) in celiac disease patients by g
34                   Genetic ablation of enzyme transglutaminase 2 (TG2) in Mgp(-/-) mice dramatically r
35               The mechanism of activation of transglutaminase 2 (TG2) in the extracellular matrix rem
36 e interferon-gamma (IFNgamma) and the enzyme transglutaminase 2 (TG2) in tissue destruction.
37  that it is a substrate for cross-linking by transglutaminase 2 (TG2) into higher-order species.
38                                              Transglutaminase 2 (TG2) is a Ca(2+)-dependent cross-lin
39                                       Tissue transglutaminase 2 (TG2) is a multifunctional protein pr
40                                              Transglutaminase 2 (TG2) is a ubiquitously expressed enz
41                                              Transglutaminase 2 (TG2) is a ubiquitously expressed enz
42                                              Transglutaminase 2 (TG2) is a ubiquitously expressed enz
43                                              Transglutaminase 2 (TG2) is secreted by a non-classical
44   The multifunctional, protein cross-linking transglutaminase 2 (TG2) is the main autoantigen in celi
45                   The multifunctional enzyme transglutaminase 2 (TG2) is the target of autoantibodies
46 l commercial assays that measure IgA against transglutaminase 2 (TG2) or IgG against DGP.
47 e is presented in this study that shows that transglutaminase 2 (TG2) plays a critical role in the ad
48     For example, Trx activates extracellular transglutaminase 2 (TG2) via reduction of an intramolecu
49 ssion by interacting with its ligand, tissue transglutaminase 2 (TG2), but the molecular basis for th
50 aspase-3 activation through stabilization of transglutaminase 2 (TG2), which cross-links and inactiva
51 ion plasma cells (PCs), we have analyzed the transglutaminase 2 (TG2)-specific VH:VL autoantibody rep
52 odies reactive to gluten or the self-antigen transglutaminase 2 (TG2).
53 on of autoantibodies specific for the enzyme transglutaminase 2 (TG2).
54 cific for gluten peptides or the autoantigen transglutaminase 2 (TG2).
55 isfolded keratins; 2) elevated levels of the transglutaminase 2 (TG2); 3) increased K8 phosphorylatio
56            Interactions among IgA, CD71, and transglutaminase 2 (Tgase2) were analyzed by flow cytome
57 es the AP-1 transcription factor Fos and the transglutaminase 2 (TGase2), a cross-linking enzyme with
58                                We identified transglutaminase 2 (TGM2) as a putative tumor suppressor
59 up 10 secretory phospholipase A2, Wnt5a, and transglutaminase 2 (Tgm2).
60    EMA-positive sera were further tested for transglutaminase 2 antibodies (TG2-IgA).
61 activation in different species and identify Transglutaminase 2 as a conserved M2 marker that is high
62                   We demonstrate that tissue transglutaminase 2 can serotonylate histone H3 tri-methy
63  ERalpha as a direct regulator of macrophage transglutaminase 2 expression, a multifunctional atherop
64                                              Transglutaminase 2 has an affinity to the MUC2 CysD2 dom
65 patients, who have antibodies against tissue transglutaminase 2 in the absence of villous atrophy.
66          Transforming growth factor-beta and transglutaminase 2 stimulate Snail-dependent invadosome
67 e deposits (sCD89, transferrin receptor, and transglutaminase 2) decreased markedly 1 week after trea
68 rypsin (modified or unmodified by the enzyme transglutaminase 2) or the peptic-tryptic digest of glia
69 uced mesangial surface expression of TGase2 (transglutaminase 2), which in turn up-regulated TfR1 exp
70 l ECM, including the multi-functional enzyme transglutaminase 2, which we show is crucial for neuroge
71 infiltrating T cells, HLA-DR expression, and transglutaminase 2-targeted IgA deposits.
72 002-1E03 from a digest of gliadin treated by transglutaminase 2.
73 linical presentation (86%) and positive anti-transglutaminase 2/endomysium antibodies (99%) was obser
74  transition gene signature characteristic of transglutaminase 2/transforming growth factor-beta activ
75 tein keratin 8 (K8) and its cross-linking by transglutaminase-2 (TG2) are essential for MDB formation
76                                              Transglutaminase-2 (TG2) is a new anti-fibrotic target f
77                                              Transglutaminase-2 (TGM-2) has been implicated in severa
78                           The stress protein transglutaminase-2 (TGM2) was also significantly higher
79 ell culture models, and this requires active transglutaminase-2 (TGM2).
80                         Pep1 did not inhibit transglutaminase-2 activity, and incorporation of biotin
81 lts (for levels of antibodies against tissue transglutaminase-2 and deamidated gliadin peptide), symp
82 nct from the two CD hallmark antigens tissue transglutaminase-2 and deamidated gliadin, exhibiting 71
83 uired for MDB formation, including increased transglutaminase-2 and keratin overexpression, with a gr
84                                Additionally, transglutaminase-2 expression was determined after YAP s
85 ysyl oxidase, lysyl oxidase-like 2-4, tissue transglutaminase-2), and ECM turnover genes/enzymes (mat
86 en fibrillar collagens, introduced by tissue transglutaminase-2, are necessary for the self-assembly
87 elastin and fibrillin can be cross-linked by transglutaminase-2, but the function of cross-linking on
88 tabilized by the matrix cross-linking enzyme transglutaminase-2.
89                                  Recently, a transglutaminase 3 knockout (TGM3/KO) mouse was generate
90  PADI3 (peptidylarginine deiminase 3), TGM3 (transglutaminase 3), and TCHH (trichohyalin) in a total
91 ibodies toward the newly identified neuronal transglutaminase 6 (TG6).
92  mutations in the TGM6 gene, which codes for transglutaminase 6 (TG6).
93  epidermal desmosomes, upregulated epidermal transglutaminase activity and heightened resistance to S
94 -out mice, led to a rapid rise in intestinal transglutaminase activity in a manner that could be inhi
95            Inhibiting polyamine synthesis or transglutaminase activity significantly decreases microt
96                                              Transglutaminase activity was increased in fibrosis, and
97                                              Transglutaminase addition exerted significant effect in
98 his study aimed to evaluate the influence of transglutaminase addition on the technological propertie
99 f a substrate, called Plugin, by the seminal transglutaminase AgTG3.
100 L.) (flour or seed) and cold gelling agents (transglutaminase, alginate or gelatin).
101  B (SS-A and SS-B, respectively), antitissue transglutaminase and antiendomysial antibodies, and para
102 LA-DQ8 genes) and autoantibodies (antitissue transglutaminase and antiendomysial).
103                These include elevated tissue transglutaminase and consequent inactivation through C-t
104 erologic tests for antibodies against tissue transglutaminase and deamidated gliadin peptide, greatly
105 f the profibrotic cleavage substrates tissue transglutaminase and endoglin accompanied MMP-14 suppres
106 esults of serum tests for IgA against tissue transglutaminase and endomysium or on both a health care
107 as well as for IgA antibodies against tissue transglutaminase and endomysium.
108 on-(gamma-glutamyl) lysine cross-linkages by transglutaminase and that enzymatic cross-linking increa
109 our antibody tests including IgA anti-tissue transglutaminase and/or IgA anti- endomysium permitted d
110 ults (IgA/IgG gliadin, endomysium, or tissue transglutaminase) and compared them with 213,208 age- an
111 bodies against gliadin, 1.22% against tissue transglutaminase, and 0.61% against endomysium (P>.05 vs
112 idase, gastric parietal cells (PCAs), tissue transglutaminase, and 21-hydroxylase was tested using a
113 g of VEGF165, catalysed by the enzyme tissue transglutaminase, and associates with MVs through its in
114        Concentrations of IgA, IgA antitissue transglutaminase, and endomysial antibodies were measure
115 anti-dpgli, IgG anti-dpgli, IgA anti- tissue transglutaminase, and IgA anti-endomysium) yielded posit
116 +SPI, WPI+CN and SPI+CN, were produced using transglutaminase, and their in vitro IgE reactivity and
117 high levels of autoantibodies against tissue transglutaminase (anti-TG2) are strongly indicative of a
118  years and analyzed for antibodies to tissue transglutaminase (anti-TG2A) using a radiobinding assay,
119  Serum samples were analyzed for anti-tissue transglutaminase (anti-tTG) concentrations at age 6 y.
120  between levels of antibodies against tissue transglutaminase (anti-tTG, a marker of celiac disease)
121 , antinuclear antibodies (ANAs), anti-tissue transglutaminase antibodies (AGTAs), and anti-thyroid pe
122 tive results from serologic tests for tissue transglutaminase antibodies (anti-TG2) but normal duoden
123 d optimized for determination of anti-tissue transglutaminase antibodies (anti-tTG) in human serum.
124 gneto immunosensor for the detection of anti-transglutaminase antibodies (ATG2) in celiac disease was
125 tive results from serologic tests for tissue transglutaminase antibodies (tTGA) without endoscopic de
126 ific screening serologic tests (anti- tissue transglutaminase antibodies IgA [anti-TTG] and ednomysea
127                    Positivity for IgA tissue transglutaminase antibodies was detected in 97%.
128 itivities of Simtomax, endomysial and tissue-transglutaminase antibodies were compared in 133 prospec
129                  Simultaneously, anti-tissue transglutaminase antibodies, questionnaire results, clin
130                It captures the target tissue transglutaminase antibody (anti-tTG), and finally allows
131 ning for celiac disease using the IgA tissue transglutaminase antibody (IgA tTG) and, if positive, te
132 ounger (P = .03), had lower titers of tissue transglutaminase antibody (P = .001), and less frequentl
133     The majority (98%) of FPs requested anti-transglutaminase antibody (tTG-Ab) titres for CD diagnos
134 ions revealed a slightly elevated IgA tissue transglutaminase antibody level in the setting of serum
135                                              Transglutaminases are also involved in fibrin(ogen) rete
136 n with lactoferrin and sodium alginate using transglutaminase as a cross-linking agent.
137 independent of coagulation factor 13 (FXIII) transglutaminase, as ANIT challenge in FXIII-deficient m
138 for up to 20 years for development of tissue transglutaminase autoantibodies (tTGA).
139 from birth were screened annually for tissue transglutaminase autoantibodies (tTGAs).
140 efined as being positive for islet or tissue transglutaminase autoantibodies at 2 consecutive clinic
141 ase autoimmunity, defined as positive tissue transglutaminase autoantibodies found in 2 consecutive s
142     Screening for celiac disease with tissue transglutaminase autoantibodies was performed annually i
143 Y study who were tested for islet and tissue transglutaminase autoantibodies, respectively.
144 bodies (HR, 0.99; 95% CI, 0.95-1.03), or the transglutaminase autoantibody (HR, 1.00; 95% CI, 0.98-1.
145 ntestinal biopsy or persistently high tissue transglutaminase autoantibody levels.
146 sults suggest that a risk of islet or tissue transglutaminase autoimmunity need not influence the rec
147                            Through microbial transglutaminase-catalysed transamidation of gluten prot
148                                              Transglutaminases catalyze the covalent linkage of prote
149 cation of a disulfide bond and pH-controlled transglutaminase-catalyzed modification of lysine, respe
150                             Endogenous brain transglutaminase-catalyzed polyaminated tubulins have th
151        Together, these findings suggest that transglutaminase-catalyzed polyamination of tubulins sta
152                                              Transglutaminase-catalyzed posttranslational incorporati
153               Moreover, we observed that the transglutaminase coagulation factor XIIIA (FXIIIA) was o
154 uggest that the proposed evolution of animal transglutaminase cross-linking activity from ancestral b
155             Dynamic rheometry indicated that transglutaminase cross-linking and niosomes charging of
156                                              Transglutaminase cross-linking prior to emulsification s
157                            Here we show that transglutaminase cross-linking supports formation of a 1
158 techniques for monitoring the intramolecular transglutaminase cross-links of pea proteins, based on p
159 f 5.7 mum were fabricated and charged into a transglutaminase-cross-linked whey protein solution that
160                                              Transglutaminase crosslinked faba bean protein extensive
161 lf-assembled zein networks through microbial transglutaminase crosslinking were investigated.
162 elets stabilized FXIII-depleted thrombi in a transglutaminase-dependent manner.
163 of celiac disease is believed to involve the transglutaminase-dependent response of CD4(+) T cells to
164                                              Transglutaminase diseases: from biochemistry to the beds
165                            Cyk3p possesses a transglutaminase domain that is essential for function,
166     Cross-linking the dialyzed emulsion with transglutaminase eliminated the detection of free lactas
167  beads (MBs) as a solid support in which the transglutaminase enzyme (TG2) is covalently immobilized
168 inocytes, produced TGM1 protein, and rescued transglutaminase enzyme function.
169 sed on the covalent immobilization of tissue transglutaminase enzyme in its open conformation (open-t
170 cornified envelope precursors and the tissue transglutaminase enzyme that cross-links them.
171 gelatin and gum arabic with or without using transglutaminase enzymes and to develop a functional kef
172 ion tomography/magnetic resonance imaging of transglutaminase factor XIII (FXIII) and myeloperoxidase
173                                  Coagulation transglutaminase factor XIII (FXIII) exists in circulati
174 nt study, we determined that activity of the transglutaminase factor XIII (FXIII) is critical for rbc
175    Here, we demonstrate that the coagulation transglutaminase, factor XIII (fXIII), drives arthritis
176 ogical importance of inactive members of the transglutaminase family, which are found throughout euka
177                            Crosslinking with transglutaminase followed by freeze drying resulted in a
178 factor of 10 below the recommended amount of transglutaminase for raw as well as heated restructured
179                                              Transglutaminase from Streptomyces mobaraensis (MTG) is
180 faba bean protein isolates were treated with transglutaminase from Streptomyces mobaraensis and tyros
181                    Using a gelatin microbial transglutaminase (gelatin-mTG) cell culture platform tun
182 hypothesised that the dosing of xylanase and transglutaminase has a positive impact on rye dough and
183 l cycle regulated and we unearth a bacterial transglutaminase homolog, HvyA, as restriction factor th
184  potency, and their selectivity toward other transglutaminase homologues is largely unknown.
185                                 Human tissue transglutaminase (hTG2) is a multifunctional enzyme.
186                     Her positive anti-tissue transglutaminase IgA antibodies and ensuing duodenal bio
187 ts with concentrations of IgA against tissue transglutaminase (IgA-TTG) >10-fold the upper limit of n
188 tients should also be tested for anti-tissue transglutaminase, IgA against deamidated gliadin peptide
189       However, using the crosslinking enzyme Transglutaminase II to alter microstructure independentl
190 cient between the CDAT score and anti-tissue transglutaminase immunoglobulin A (anti-t-TG-IgA) titer
191 ion and diagnosis of CD, particularly tissue transglutaminase-immunoglobulin A (TG2-IgA), IgA testing
192 vidence for an increased risk of anti-tissue transglutaminase in patients with IBD vs controls (RR 1.
193 ibuted to glutamine deamidation by microbial transglutaminase in the absence of sufficient lysine thr
194 s have circulating antibodies against tissue transglutaminase; in children, European guidelines allow
195  assay using an assay for IgA against tissue transglutaminase; in subjects with positive test results
196                                              Transglutaminase increased the absolute zeta-potential v
197                                          All transglutaminases increased post-SNx peaking at loss of
198 vitro antioxidative hydrolysate, followed by transglutaminase-induced cross-linking and microemulsifi
199 ts into the mechanism and inhibition of this transglutaminase-induced ubiquitination by MavC, but als
200  Furthermore, treatment of mice with the pan-transglutaminase inhibitor cystamine resulted in signifi
201 I deficiency had normal retention, and a pan-transglutaminase inhibitor T101 had only a modest inhibi
202  encoding factor XIII subunit A (FXIII-A), a transglutaminase involved in hemostasis, wound healing,
203 here is no confirmation that TG2 is the only transglutaminase involved, neither there are strategies
204           Gluten deamidation by human tissue transglutaminase is critical to activate celiac disease
205                The dimeric FXIII-A(2), a pro-transglutaminase is the catalytic part of the heterotetr
206              In this study, we have profiled transglutaminase isozymes in the rat subtotal nephrectom
207 ins are previously undetected members of the transglutaminase-like cysteine protease superfamily, and
208 e diphosphate via its catalytically inactive transglutaminase-like domain.
209                         We have identified a transglutaminase-like protein (Cyk3p) that functions in
210                         Similar to canonical transglutaminases, MavC possess deamidase activity that
211 )-Lys92(Ube2N)) isopeptide crosslink using a transglutaminase mechanism.
212 observed ratio allowed the identification of transglutaminase-mediated gamma-glutamyl isomers as inte
213 rgenic protein products could be produced by transglutaminase-mediated heterologous polymerization of
214 terogeneous lysine conjugation and bacterial transglutaminase-mediated site-specific conjugation.
215                                          The transglutaminase-mediated, covalent cross-linking of pro
216                                    Microbial transglutaminase (mTG) catalyses the formation of protei
217       The substrate promiscuity of microbial transglutaminase (mTG) has been exploited in various app
218                         Impacts of microbial transglutaminase (MTGase) (0-0.6 units/g sample) on gel
219             Key modifying roles of microbial transglutaminase (MTGase) in the development of innovati
220                                    Microbial transglutaminase (MTGase) is an enzyme of the class of t
221 ia sp. C1112 for the production of microbial transglutaminase (MTGase, EC 2.3.2.13).
222                                    Microbial transglutaminases (MTGs) catalyze the formation of Gln-L
223  general role for the catalytically inactive transglutaminases of fungi and animals, some of which ha
224 ) during storage in emulsion gels containing transglutaminase or alginate.
225 er limit of detection and IgG against tissue transglutaminase or deamidated gliadin peptide, or endom
226                  Red rice cakes treated with transglutaminase presented a lower glucose release rate
227   Among tests for celiac disease, IgA tissue transglutaminase presented the best diagnostic test accu
228  has been indicated previously that during a transglutaminase reaction activated factor XIII (FXIIIa)
229 ng only the final deacylation portion of the transglutaminase reaction.
230      Analysis of heparin binding of the main transglutaminases revealed that although the interaction
231        The binding of SLPI with Cementoin to transglutaminase seems to be an effective strategy to tr
232 ic ATPase, anti-thyroid peroxidase, and anti-transglutaminase seropositivity, and these autoantibodie
233 use a proteomic approach in combination with transglutaminase-specific labeling to identify FXIIIa pl
234 lar methodologies to demonstrate that tissue transglutaminase (TG) activates downstream NF-kappaB sig
235                            The popularity of transglutaminase (TG) by the food industry and the varia
236                  In humans, 9 members of the transglutaminase (TG) family have been identified, of wh
237 -MS/MS-method for the detection of microbial transglutaminase (TG) from Streptomyces mobaraensis in d
238  for the simultaneous detection of microbial transglutaminase (TG) from Streptomyces mobaraensis, and
239 A2), cyclooxygenase 2 (COX-2), thrombin, and transglutaminase (TG).
240 ckpea protein (w/w) with/without addition of transglutaminase (TG).
241                            Expression of the transglutaminase TG2 has been linked to constitutive act
242   The ubiquitous cross-linking enzyme tissue transglutaminase (TG2) has been implicated in irreversib
243                                       Tissue transglutaminase (TG2) is a multifunctional Ca(2+)-activ
244                                       Tissue transglutaminase (TG2) is a multifunctional enzyme invol
245                                       Type 2 transglutaminase (TG2) is an important cancer stem cell
246 ssion of the pro-inflammatory protein tissue transglutaminase (TG2) reprograms the transcription regu
247 inhibits melanoma growth and binds to tissue transglutaminase (TG2), a major crosslinking enzyme in E
248                                       Tissue transglutaminase (TG2), an enzyme involved in cell proli
249 tural GPR56 ligands, collagen III and tissue transglutaminase (TG2), and one small-molecule agonist,
250 performance of antibody tests against tissue transglutaminase (TG2), deamidated gliadin peptide (DGP)
251 ated with the sulfhydryl-rich protein tissue transglutaminase (TG2), thereby endowing the membrane su
252 ignificant increases in the levels of type-2 transglutaminase (TG2, which is implicated in transamida
253 nd levels of immunoglobulin A against tissue-transglutaminase (TGA-IgA) 10-fold or more the upper lim
254 nomer sample that had been cross-linked with transglutaminase (TGase) and digested with pepsin.
255                                Extracellular transglutaminase (TGase) has been shown previously to pl
256                                              Transglutaminase (TGase) is a Ca(2+)-dependent cross-lin
257                                              Transglutaminase (TGase) is an enzyme for improving the
258  a milk base in the absence or presence of a transglutaminase (TGase) protein cross-linking step on t
259 atin hydrolysates and glucosamine (GlcN) via transglutaminase (TGase), as well as glycation between f
260 designed to detect the catalytic activity of transglutaminase (TGase), which creates a covalent bond
261 /50 degrees C) in the presence or absence of transglutaminase (TGase).
262 ll epitope through treatment with the enzyme transglutaminase (TGase).
263                                              Transglutaminases (TGases) are ubiquitous enzymes that t
264 evident 2 d after birth, followed by reduced transglutaminase (TGM) activity, transepidermal water lo
265                                              Transglutaminase (TGM)-2 has been shown to contribute to
266 in, angiopoietin-like factor (ANGPTL)-7, and transglutaminase (TGM)-2.
267                              In vitro tissue transglutaminase (Tgm2) cross-linking was monitored and
268         By gene expression screening, tissue transglutaminase (TGM2) was identified as one of the gen
269 t extents on stiffer substrates; TAZ, tissue transglutaminase (TGM2), and soluble frizzled-related pr
270 ins (Krts), keratin-associated proteins, and transglutaminases (Tgms) and their substrates were signi
271 ormed by extensive cross-linking activity of transglutaminases (TGs) during terminal epidermal differ
272                We demonstrate that MavC is a transglutaminase that catalyses covalent linkage of ubiq
273       Coagulation factor XIIIa (FXIIIa) is a transglutaminase that covalently cross-links fibrin and
274  Plasma coagulation factor XIII (FXIII) is a transglutaminase that promotes cross-linking of the extr
275                      Thus, unlike the active transglutaminases that activate RhoA, the multidomain pr
276 edicted after GC-1g, and the mean IgA-tissue transglutaminase titers declined, contrary to the predic
277 ne, TIG3 interacts with and activates type I transglutaminase to enhance keratinocyte terminal differ
278 lyacrylamide gel electrophoresis profiles of transglutaminase-treated low concentration (0.01% w/w) p
279 milar with known CD-specific antigens tissue transglutaminase (tTG) and deamidated gliadin, and the c
280  Immunoglobulin A (IgA) antibodies to tissue transglutaminase (tTG) are the serologic test of choice
281                                       Tissue transglutaminase (tTG) assay was available, but one-thir
282                                       Tissue transglutaminase (tTG) functions as a GTPase and an acyl
283  often relies on the presence of anti-tissue transglutaminase (tTG) IgA autoantibodies.
284                                       Tissue transglutaminase (tTG) is an acyltransferase/GTP-binding
285 ecognition scaffold is the following: tissue transglutaminase (tTG) is immobilized as a capturing age
286  glycated haemoglobin (HbA1c) and IgA tissue transglutaminase (tTg) were collected prior to, and foll
287 al antibodies (EMA) and antibodies to tissue transglutaminase (tTG) were developed to screen for celi
288  to different antigens, in particular tissue transglutaminase (tTG), gliadin (Glia), and endomysium.
289 ind the protein cross-linking enzyme, tissue transglutaminase (tTG), in cancer cell migration.
290                                       Tissue transglutaminase (tTG), involved in PTM of gluten antige
291 disease have serum antibodies against tissue transglutaminase (tTG).
292  such as increased antibody levels to tissue transglutaminase (tTG).
293                                              Transglutaminase type 2 (TG2) is an extracellular matrix
294      The positive predictive value of tissue transglutaminase type 2 (tTG) antibodies performed with
295             Rates of elevated levels of anti-transglutaminase type 2 and antigliadin antibodies were
296                                         Anti-transglutaminase type 2 and antigliadin antibodies were
297 re than 12000 participants) found IgA tissue transglutaminase was associated with high accuracy (sens
298     HLA DQ2/DQ8 and serum antibodies against transglutaminase were analysed.
299         Coagulation factor XIII (FXIII) is a transglutaminase with a well defined role in the final s
300  protein was crosslinked to some extent with transglutaminase with higher dosages (100 and 1000nkat/g

 
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