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1 n receptor-related protein 1, MIC2/CD99, and transglutaminase 2.
2 002-1E03 from a digest of gliadin treated by transglutaminase 2.
3 tabilized by the matrix cross-linking enzyme transglutaminase-2.
4 /K18) and ubiquitin that are cross-linked by transglutaminase-2.
5 pounds were not acting through inhibition of transglutaminase 2 activity.
6                         Pep1 did not inhibit transglutaminase-2 activity, and incorporation of biotin
7  indicate that suprabasin is a substrate for transglutaminase 2 and 3 activity.
8 sease requires a positive serology (IgA anti-transglutaminase 2 and anti-endomysial antibodies) and v
9                            Transfection with transglutaminase 2 and htt-N63-148Q-myc followed by trea
10         Calmodulin coimmunoprecipitates with transglutaminase 2 and huntingtin in cells transfected w
11 -links in the HD cortex that colocalize with transglutaminase 2 and huntingtin.
12 dulin colocalizes at the confocal level with transglutaminase 2 and with huntingtin in HD intranuclea
13 lts (for levels of antibodies against tissue transglutaminase-2 and deamidated gliadin peptide), symp
14 nct from the two CD hallmark antigens tissue transglutaminase-2 and deamidated gliadin, exhibiting 71
15 uired for MDB formation, including increased transglutaminase-2 and keratin overexpression, with a gr
16 her epidermally expressed transglutaminases, transglutaminase-2 and transglutaminase-3, on chromosome
17 re down-regulation of cyclin D1, COBRA1, and transglutaminase-2 and up-regulation of tumor necrosis f
18 ysyl oxidase, lysyl oxidase-like 2-4, tissue transglutaminase-2), and ECM turnover genes/enzymes (mat
19    EMA-positive sera were further tested for transglutaminase 2 antibodies (TG2-IgA).
20  type and detection of anti-gliadin and anti-transglutaminase 2 antibodies to identify a subgroup of
21                                              Transglutaminase 2 appears to be an important component
22 en fibrillar collagens, introduced by tissue transglutaminase-2, are necessary for the self-assembly
23 activation in different species and identify Transglutaminase 2 as a conserved M2 marker that is high
24 polyglutamine repeats (htt-N63-148Q-myc) and transglutaminase 2 but not in cells transfected with myc
25 elastin and fibrillin can be cross-linked by transglutaminase-2, but the function of cross-linking on
26                   We demonstrate that tissue transglutaminase 2 can serotonylate histone H3 tri-methy
27          In celiac disease, small intestinal transglutaminase 2 causes deamidation of glutamine resid
28  celiac disease or antibodies against tissue transglutaminase 2 (controls), healthy family members of
29              We demonstrated previously that transglutaminase 2 cross-links mutant huntingtin in cell
30                                          IgA-transglutaminase 2 decreased significantly within 4 week
31 e deposits (sCD89, transferrin receptor, and transglutaminase 2) decreased markedly 1 week after trea
32 loped recently, such as assessing intestinal transglutaminase 2 deposits, flow cytometry technique, m
33 linical presentation (86%) and positive anti-transglutaminase 2/endomysium antibodies (99%) was obser
34  autoimmunity was defined as a positive anti-transglutaminase 2 enzyme-linked immunosorbent assay tes
35  ERalpha as a direct regulator of macrophage transglutaminase 2 expression, a multifunctional atherop
36                                Additionally, transglutaminase-2 expression was determined after YAP s
37 Short interfering RNA was used to knock down transglutaminase-2 expression.
38 re Mesothelin, Muc4, Muc5A/C, Kallikrein 10, Transglutaminase 2, Fascin, TMPRSS3 and stratifin.
39                                              Transglutaminase 2 has an affinity to the MUC2 CysD2 dom
40 to both human Factor XIII (FXIII) and tissue transglutaminase 2 (hTG2).
41              Whereas the development of anti-transglutaminase 2 IgA is linked with gastrointestinal d
42 patients, who have antibodies against tissue transglutaminase 2 in the absence of villous atrophy.
43 eek treatment with ZED1227, a selective oral transglutaminase 2 inhibitor, at three dose levels as co
44                                Inhibition of transglutaminase 2 is a potential treatment for celiac d
45                                  Analysis of transglutaminase 2 knock-out mice confirmed that these c
46 R2+ breast cancer cell line as a function of transglutaminase 2 levels, even in the presence of fibro
47 rypsin (modified or unmodified by the enzyme transglutaminase 2) or the peptic-tryptic digest of glia
48 biochemically by blotting with antibodies to transglutaminase-2/p62 proteins and to K8/K18/ubiquitin
49  was cross-linked to germ tubes catalyzed by transglutaminase 2 prior to cell fractionation, immunopr
50  transfection with both htt-N63-148Q-myc and transglutaminase 2 resulted in cross-linking of mutant h
51          Transforming growth factor-beta and transglutaminase 2 stimulate Snail-dependent invadosome
52 infiltrating T cells, HLA-DR expression, and transglutaminase 2-targeted IgA deposits.
53 isease (CD), a condition in which antitissue transglutaminase 2 (TG2) Abs are suspected to contribute
54 evious work in our laboratory has shown that transglutaminase 2 (TG2) acting as a coreceptor for inte
55 s of total IgA antibodies, IgA antibodies to transglutaminase 2 (TG2) and endomysium, as well as IgA
56           The interaction between the enzyme transglutaminase 2 (TG2) and fibronectin (FN) is involve
57 um domain of the fibronectin binding site on transglutaminase 2 (TG2) and its importance in mediating
58                Antibodies to the autoantigen transglutaminase 2 (TG2) are a hallmark of celiac diseas
59       Autoantibodies specific for the enzyme transglutaminase 2 (TG2) are a hallmark of the gluten-se
60 A plasma cells (PCs) specific for the enzyme transglutaminase 2 (TG2) are abundant in the small intes
61                            Autoantibodies to transglutaminase 2 (TG2) are hallmarks of celiac disease
62     We and others have previously identified transglutaminase 2 (TG2) as a participant in adverse fib
63  lymphoid and myeloid subsets alongside anti-transglutaminase 2 (TG2) autoantibody production.
64                                              Transglutaminase 2 (TG2) catalyzes transamidation or dea
65                                              Transglutaminase 2 (TG2) expression is required for epid
66 to hypertrophy, associated with up-regulated transglutaminase 2 (TG2) expression, mediates not only p
67                                       Tissue transglutaminase 2 (TG2) has been of particular interest
68                        Molecular deletion of transglutaminase 2 (TG2) has been shown to improve funct
69 duction of autoantibodies against the enzyme transglutaminase 2 (TG2) in celiac disease likely result
70 the autoantibody response against the enzyme transglutaminase 2 (TG2) in celiac disease patients by g
71                   Genetic ablation of enzyme transglutaminase 2 (TG2) in Mgp(-/-) mice dramatically r
72               The mechanism of activation of transglutaminase 2 (TG2) in the extracellular matrix rem
73 e interferon-gamma (IFNgamma) and the enzyme transglutaminase 2 (TG2) in tissue destruction.
74  that it is a substrate for cross-linking by transglutaminase 2 (TG2) into higher-order species.
75                                              Transglutaminase 2 (TG2) is a Ca(2+)-dependent cross-lin
76                                              Transglutaminase 2 (TG2) is a distinctive member of the
77                                              Transglutaminase 2 (TG2) is a highly expressed mammalian
78                                              Transglutaminase 2 (TG2) is a multifunctional enzyme tha
79                                       Tissue transglutaminase 2 (TG2) is a multifunctional protein pr
80                                              Transglutaminase 2 (TG2) is a multifunctional protein th
81                                              Transglutaminase 2 (TG2) is a ubiquitously expressed enz
82                                              Transglutaminase 2 (TG2) is a ubiquitously expressed enz
83                                              Transglutaminase 2 (TG2) is a ubiquitously expressed enz
84                                        Human transglutaminase 2 (TG2) is believed to play an importan
85                                       Tissue transglutaminase 2 (TG2) is overexpressed in epithelial
86                                              Transglutaminase 2 (TG2) is secreted by a non-classical
87   The multifunctional, protein cross-linking transglutaminase 2 (TG2) is the main autoantigen in celi
88                   The multifunctional enzyme transglutaminase 2 (TG2) is the target of autoantibodies
89 l commercial assays that measure IgA against transglutaminase 2 (TG2) or IgG against DGP.
90 e is presented in this study that shows that transglutaminase 2 (TG2) plays a critical role in the ad
91                                              Transglutaminase 2 (TG2) plays a pivotal role in the pat
92 oimmune disorder whose pathogenesis requires transglutaminase 2 (TG2) to deamidate select glutamine r
93     For example, Trx activates extracellular transglutaminase 2 (TG2) via reduction of an intramolecu
94 y cells release microvesicles rich in tissue transglutaminase 2 (Tg2) which activate murine fibroblas
95 n 1 (MTA1), a master chromatin modifier, and transglutaminase 2 (TG2), a multifunctional enzyme, are
96                     Macrophage expression of transglutaminase 2 (TG2), a multifunctional protein with
97  and H3Q5dop, respectively) are catalysed by transglutaminase 2 (TG2), and alter both local and globa
98 ssion by interacting with its ligand, tissue transglutaminase 2 (TG2), but the molecular basis for th
99  cross-linked by transamidation catalyzed by transglutaminase 2 (TG2), itself an inflammation-regulat
100 aspase-3 activation through stabilization of transglutaminase 2 (TG2), which cross-links and inactiva
101 ion plasma cells (PCs), we have analyzed the transglutaminase 2 (TG2)-specific VH:VL autoantibody rep
102 cific for gluten peptides or the autoantigen transglutaminase 2 (TG2).
103 odies reactive to gluten or the self-antigen transglutaminase 2 (TG2).
104 on of autoantibodies specific for the enzyme transglutaminase 2 (TG2).
105 isfolded keratins; 2) elevated levels of the transglutaminase 2 (TG2); 3) increased K8 phosphorylatio
106                                              Transglutaminase 2 (TG2, a.k.a. tissue transglutaminase)
107 tein keratin 8 (K8) and its cross-linking by transglutaminase-2 (TG2) are essential for MDB formation
108                                              Transglutaminase-2 (TG2) is a new anti-fibrotic target f
109                                              Transglutaminase 2 (TGase 2) expression is increased in
110                 The transamidase activity of transglutaminase 2 (TGase 2) is considered to be importa
111            Interactions among IgA, CD71, and transglutaminase 2 (Tgase2) were analyzed by flow cytome
112 es the AP-1 transcription factor Fos and the transglutaminase 2 (TGase2), a cross-linking enzyme with
113                                              Transglutaminase-2 (TGM-2) has been implicated in severa
114 y enriched in tumor tissues that overexpress transglutaminase 2 (TGM2) and regulate the three-dimensi
115                                We identified transglutaminase 2 (TGM2) as a putative tumor suppressor
116          This study investigates the role of transglutaminase 2 (TGM2) in enhancing mucosal pellicle
117 iver of this phenotype and astrocyte-derived transglutaminase 2 (TGM2) was identified as a promoter o
118  binding 1(Id1), fos-like antigen 1 (FOSL1), transglutaminase 2 (TGM2), asparagine synthetase (ASNS),
119 e H3Q5ser is known to be installed by tissue transglutaminase 2 (TGM2), the substrate characteristics
120 reveals that serpinB2 upregulation activates transglutaminase 2 (TGM2), which selectively deamidates
121 up 10 secretory phospholipase A2, Wnt5a, and transglutaminase 2 (Tgm2).
122                           The stress protein transglutaminase-2 (TGM2) was also significantly higher
123 al cancer, and breast cancer are coated with transglutaminase-2 (TGM2)-dependent covalent CXCL12-kera
124 ell culture models, and this requires active transglutaminase-2 (TGM2).
125  transition gene signature characteristic of transglutaminase 2/transforming growth factor-beta activ
126 uced mesangial surface expression of TGase2 (transglutaminase 2), which in turn up-regulated TfR1 exp
127 l ECM, including the multi-functional enzyme transglutaminase 2, which we show is crucial for neuroge

 
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